Search results for "fibroblast"

showing 10 items of 667 documents

Gingival fibroblasts ?in vitro? and Down's Syndrome

2012

Gingival fibroblast cultures from four patients with Down's Syndrome (DS) and periodontal disease were compared with four in vitro age-matched fibroblast cultures of handicapped subjects (ND) also affected by periodontitis. The extra copy of cromosome 21 could alter growth regulation and biochemical mechanisms, so we examined quantitatively some DS phenotypical aspects to detect possible differences from those of controls. The growth properties of gingival fibroblast cultures from DS patients were more elevated than their ND age-matched controls. There were no differences in plasma membrane polarization and in neutral endopeptidase activity. The succinate-cytochrome C reductase activity dec…

Periodontitismedicine.medical_specialtyS syndromeClinical BiochemistryBiomedical EngineeringBioengineeringCell BiologyBiologymedicine.diseasePhenotypeIn vitroEndocrinologyPeriodontal diseaseInternal medicinemedicineReductase activityGingival fibroblastNeprilysinBiotechnologyCytotechnology
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Regulation of Anti-Apoptotic SOD2 and BIRC3 in Periodontal Cells and Tissues.

2021

Made available in DSpace on 2021-06-25T10:49:07Z (GMT). No. of bitstreams: 0 Previous issue date: 2021-01-02 Fundação de Amparo à Pesquisa do Estado de São Paulo (FAPESP) Deutscher Akademischer Austauschdienst Coordenação de Aperfeiçoamento de Pessoal de Nível Superior (CAPES) Deutsche Forschungsgemeinschaft The aim of the study was to clarify whether orthodontic forces and periodontitis interact with respect to the anti-apoptotic molecules superoxide dismutase 2 (SOD2) and baculoviral IAP repeat-containing protein 3 (BIRC3). SOD2, BIRC3, and the apoptotic markers caspases 3 (CASP3) and 9 (CASP9) were analyzed in gingiva from periodontally healthy and periodontitis subjects by real-time PCR…

Periodontium0301 basic medicineGingivaApoptosislcsh:ChemistryGingivitis0302 clinical medicineskin and connective tissue diseases610 Medicine &amp; healthperiodontitislcsh:QH301-705.5Cells CulturedSpectroscopybiologyCaspase 3General MedicineGingivitisBaculoviral IAP Repeat-Containing 3 ProteinCaspase 93. Good healthComputer Science Applicationsorthodontic tooth movementHost-Pathogen Interactionscardiovascular systemmedicine.symptomgingivitismedicine.medical_specialtySOD2610 Medicine & healthArticleCatalysisInorganic ChemistrySuperoxide dismutase03 medical and health sciencesstomatognathic systemOrthodontic tooth movementInternal medicinemedicineAnimalsHumansPeriodontal fiberddc:610Physical and Theoretical ChemistryPeriodontitisMolecular BiologyPeriodontitisFusobacterium nucleatumSuperoxide Dismutasebusiness.industry<i>Fusobacterium nucleatum</i>periodontal ligamentOrganic Chemistry030206 dentistryPeriodontiumFibroblastsmedicine.diseasebiology.organism_classificationRatsstomatognathic diseases030104 developmental biologyEndocrinologyGene Expression Regulationlcsh:Biology (General)lcsh:QD1-999Apoptosisbiology.proteinFusobacterium nucleatumbusinessperiodontiumPeriodontal ligament
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Influence of Agents Used for Topical Wound Treatment on Phagocyte Stimulation and Fibroblast Growth

1995

Wound healing has always been a fascinating phenomenon for physicians and surgeons. Wounds usually close by forming new granulation tissue, contracting, and re-epithelializing. Before granulation begins, polymor-phonuclear leukocytes (PMNs) and monocytes/macrophages (MCs) are chemotactically attracted to sites of injury. Upon contact with various stimuli in the wound including bioactive lipids, complement components, certain cytokines, e.g., tumor necrosis factor-α (TNF-α) [1], granulocyte/ monocyte-colony stimulating factor (GM-CSF), interferon-γ (IFN-γ), and interleukin-8, and particles such as microorganisms, phagocytes respond by a respiratory burst consisting of a markedly enhanced upt…

PhagocyteSuperoxideGranulation tissueGranulocyteMicrobiologyRespiratory burstchemistry.chemical_compoundmedicine.anatomical_structurechemistryImmunologymedicineTumor necrosis factor alphaFibroblastWound healing
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Multiple Receptors Mediate apoJ-Dependent Clearance of Cellular Debris into Nonprofessional Phagocytes

2001

Phagocytosis of apoptotic, senescent, and dying cells by macrophages is a well characterized process. More recently it has been shown that in addition to macrophages vital neighboring cells in the affected tissue participate in the cellular clearance. While scavenger receptors have been shown to mediate uptake into macrophages, it is poorly understood how cellular debris is internalized by nonprofessional phagocytes. We here analyze the endocytic activity of vital fibroblasts and epithelial cells exposed to cellular debris and membrane remnants. We show a mutual stimulation in the endocytosis of debris and apolipoproteinJ (clusterin) in these cells. Experiments using RAP (receptor-associate…

Phagocytosismedia_common.quotation_subjectEndocytic cycleAntineoplastic AgentsApoptosisTretinoinBiologyEndocytosisCulture Media Serum-FreeCell LineTumor Cells CulturedAnimalsScavenger receptorReceptorInternalizationGlycoproteinsReceptors LipoproteinYolk Sacmedia_commonPhagocytesClusterinEpithelial CellsCell BiologyFibroblastsEndocytosisCell biologyLow Density Lipoprotein Receptor-Related Protein-2ClusterinBucladesineCell culturebiology.proteinLow Density Lipoprotein Receptor-Related Protein-1Molecular ChaperonesExperimental Cell Research
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Gene amplification in fibroblasts from ataxia telangiectasia (AT) patients and in X-ray hypersensitive AT-like Chinese hamster mutants.

2001

In search of functions involved in the regulation of gene amplification, and given the relevance of chromosome breakage in initiating the process, we analyzed the gene amplification ability of cells hypersensitive to inducers of DNA double-strand breaks and defective in cell cycle control: two human fibroblast strains derived from patients affected by ataxia telangiectasia (AT) and two hamster mutant cell lines belonging to complementation group XRCC8 of the rodent X-ray-sensitive mutants. These mutants are considered hamster models of AT cells. To measure gene amplification, the frequency and the rate of occurrence of N-(phosphonacetyl)-L-aspartate resistant cells were determined. In both …

Phosphonoacetic AcidCancer ResearchAntimetabolites AntineoplasticMutantHamstermedicine.disease_causeRadiation ToleranceChinese hamsterCell LineAtaxia TelangiectasiaCricetulusMultienzyme ComplexesCricetinaeGene duplicationmedicineAspartate CarbamoyltransferaseAnimalsHumansDihydroorotaseMutationAspartic AcidbiologyX-RaysGenetic Complementation TestGene AmplificationGeneral MedicineCell cycleFibroblastsmedicine.diseasebiology.organism_classificationMolecular biologyDrug Resistance NeoplasmAtaxia-telangiectasiaMutationCarbamoyl-Phosphate Synthase (Glutamine-Hydrolyzing)Chromosome breakageCarcinogenesis
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Differential gene expression in p53-mediated G(1) arrest of human fibroblasts after gamma-irradiation or N-phosphoacetyl-L-aspartate treatment.

2000

In human fibroblasts, N:-phosphoacetyl-L-aspartate (PALA) and gamma-radiation induce reversible and irreversible p53-mediated G(1) cell cycle arrest, respectively. By coupling the premature chromosome condensation technique to fluorescence in situ hybridization, we found no evidence of DNA damage after PALA treatment. We used representational difference analysis (cDNA-RDA) to study changes in gene expression after PALA treatment and gamma-radiation in normal human fibroblasts. The mammary-derived growth inhibitor (MDGI) gene was expressed in PALA-treated cells. Ectopic MDGI expression arrested PALA-treated but not irradiated RKO cells. Expression of an antisense RNA against MDGI resulted in…

Phosphonoacetic AcidCancer ResearchTumor suppressor geneIn situ hybridizationBiologyFatty Acid-Binding ProteinsCell LineGene expressionHumansGeneIn Situ Hybridization FluorescenceMetaphaseSkinExpressed Sequence TagsExpressed sequence tagAspartic AcidCell CycleG1 PhaseChromosome MappingG0 phaseGeneral MedicineCell cycleFibroblastsMolecular biologyGrowth InhibitorsGene Expression RegulationGamma RaysKaryotypingRepresentational difference analysisTumor Suppressor Protein p53Carrier ProteinsCell Adhesion MoleculesFatty Acid Binding Protein 3Chromosomes Human Pair 7Carcinogenesis
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The depletion of nuclear glutathione impairs cell proliferation in 3t3 fibroblasts.

2009

BACKGROUND:Glutathione is considered essential for survival in mammalian cells and yeast but not in prokaryotic cells. The presence of a nuclear pool of glutathione has been demonstrated but its role in cellular proliferation and differentiation is still a matter of debate. PRINCIPAL FINDINGS:We have studied proliferation of 3T3 fibroblasts for a period of 5 days. Cells were treated with two well known depleting agents, diethyl maleate (DEM) and buthionine sulfoximine (BSO), and the cellular and nuclear glutathione levels were assessed by analytical and confocal microscopic techniques, respectively. Both agents decreased total cellular glutathione although depletion by BSO was more sustaine…

PhysiologyGlutathione reductaseCell Biology/Cell Growth and Divisionlcsh:MedicineBiology3T3 cellschemistry.chemical_compoundMicemedicineBiochemistry/Cell Signaling and Trafficking StructuresAnimalsButhionine sulfoximinelcsh:ScienceTranscription factorButhionine SulfoximineCell ProliferationGlutathione TransferaseCell NucleusMultidisciplinaryMicroscopy ConfocalCell growthlcsh:RMaleatesGlutathione3T3 CellsFibroblastsMolecular biologyGlutathioneCell biologyCell nucleusmedicine.anatomical_structureGlutathione ReductasechemistryCytoplasmlcsh:QResearch ArticlePloS one
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Imbalance of expression of bFGF and PK1 is associated with defective maturation and antenatal placental insufficiency.

2013

Abstract Objective Defective placental maturation is associated with restricted functional capacity and adverse perinatal fetal outcomes. The aim of the study was a comparative analysis of the role of mRNA expression of various angiogenic factors in placental maturation defects. Study design We examined the mRNA expression patterns of prokineticin 1 (PK1), its receptors (PKRs), basic-fibroblast growth factor (bFGF), vascular endothelial growth factor (VEGF) and placental growth factor (PlGF) in tissue from third-trimester placentae that exhibited delayed or accelerated villous maturation. Results The expression of PK1 and PKR2 was elevated in placental tissue exhibiting accelerated maturati…

Placental growth factorVascular Endothelial Growth Factor Amedicine.medical_specialtyPlacenta DiseasesReceptors Peptidemedicine.medical_treatmentPlacentaPregnancy Trimester ThirdPlacental insufficiencyBiologyPregnancy ProteinsReceptors G-Protein-CoupledGastrointestinal Hormoneschemistry.chemical_compoundPregnancyInternal medicinemedicineHumansReceptorPlacenta Growth FactorFetusGrowth factorObstetrics and Gynecologymedicine.diseaseProkineticinVascular endothelial growth factorEndocrinologyReproductive MedicinechemistryPIGFFemaleFibroblast Growth Factor 2Vascular Endothelial Growth Factor Endocrine-Gland-DerivedEuropean journal of obstetrics, gynecology, and reproductive biology
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Mouse embryonic stem cells are hypersensitive to apoptosis triggered by the DNA damage O(6)-methylguanine due to high E2F1 regulated mismatch repair.

2007

Exposure of stem cells to genotoxins may lead to embryonic lethality or teratogenic effects. This can be prevented by efficient DNA repair or by eliminating genetically damaged cells. Using undifferentiated mouse embryonic stem (ES) cells as a pluripotent model system, we compared ES cells with differentiated cells, with regard to apoptosis induction by alkylating agents forming the highly mutagenic and killing DNA adduct O(6)-methylguanine. Upon treatment with N-methyl-N'-nitro-N-nitrosoguanidine (MNNG), ES cells undergo apoptosis at much higher frequency than differentiated cells, although they express a high level of the repair protein O(6)-methylguanine-DNA methyltransferase (MGMT). Apo…

Pluripotent Stem CellsMethylnitronitrosoguanidineDNA ComplementaryGuanineDNA damageDNA repairCellular differentiationApoptosisBiologyDNA Mismatch RepairModels BiologicalDNA AdductsMiceO(6)-Methylguanine-DNA MethyltransferaseDNA adductAnimalsMolecular BiologyEmbryonic Stem CellsSwiss 3T3 CellsBase SequenceCell DifferentiationCell BiologyDNA MethylationFibroblastsEmbryonic stem cellMolecular biologyDNA-Binding ProteinsMutS Homolog 2 ProteinDNA methylationDNA mismatch repairStem cellE2F1 Transcription FactorDNA DamageCell death and differentiation
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In Vitro Generation of Pancreatic Endocrine Cells from Human Adult Fibroblast-Like Limbal Stem Cells

2012

Stem cells might provide unlimited supply of transplantable cells for β-cell replacement therapy in diabetes. The human limbus is a highly specialized region hosting a well-recognized population of epithelial stem cells, which sustain the continuous renewal of the cornea, and the recently identified stromal fibroblast-like stem cells (f-LSCs), with apparent broader plasticity. However, the lack of specific molecular markers for the identification of the multipotent limbal subpopulation has so far limited the investigation of their differentiation potential. In this study we show that the human limbus contains uncommitted cells that could be potentially harnessed for the treatment of diabete…

Pluripotent Stem CellsStromal cellCellular differentiationPopulationBiomedical Engineeringlcsh:MedicineEnteroendocrine cellLimbus CorneaeBiologyLimbus; β-CellsSettore MED/13 - EndocrinologiaLimbuInsulin-Secreting CellsInsulin SecretionDiabetes MellitusHumansInsulineducationInduced pluripotent stem cellCells CulturedProinsulinTransplantationeducation.field_of_studyDiabeteslcsh:RCell DifferentiationCell Biologyβ-CellsCell biologyAdult Stem CellsStem cellFibroblast-like stem cellsBiomarkersAdult stem cellCell Transplantation
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