Search results for "genotype"

showing 10 items of 1725 documents

Design of the Coronary ARtery DIsease Genome-Wide Replication And Meta-Analysis (CARDIoGRAM) Study

2010

Background— Recent genome-wide association studies (GWAS) of myocardial infarction (MI) and other forms of coronary artery disease (CAD) have led to the discovery of at least 13 genetic loci. In addition to the effect size, power to detect associations is largely driven by sample size. Therefore, to maximize the chance of finding novel susceptibility loci for CAD and MI, the Coronary ARtery DIsease Genome-wide Replication And Meta-analysis (CARDIoGRAM) consortium was formed. Methods and Results— CARDIoGRAM combines data from all published and several unpublished GWAS in individuals with European ancestry; includes >22 000 cases with CAD, MI, or both and >60 000 controls; and unifies …

AdultMaleGenotypeMultifunction cardiogramMyocardial InfarctionSingle-nucleotide polymorphismGenome-wide association studyCoronary Artery Disease030204 cardiovascular system & hematologyBioinformaticsPolymorphism Single NucleotideArticleCoronary artery disease03 medical and health sciences0302 clinical medicineGeneticsHumansMedicineGenetic Predisposition to DiseaseMyocardial infarctionGenetics (clinical)Aged030304 developmental biologyGenetic association0303 health sciencesbusiness.industryMiddle Agedmedicine.disease3. Good healthGenetic epidemiologyResearch DesignFemaleCardiology and Cardiovascular MedicinebusinessAlgorithmsImputation (genetics)Genome-Wide Association StudyCirculation: Cardiovascular Genetics
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The val158met polymorphism of human catechol-O-methyltransferase (COMT) affects anterior cingulate cortex activation in response to painful laser sti…

2010

Background: Pain is a complex experience with sensory, emotional and cognitive aspects. Genetic and environmental factors contribute to pain-related phenotypes such as chronic pain states. Genetic variations in the gene coding for catechol-O-methyltransferase ( COMT) have been suggested to affect clinical and experimental pain-related phenotypes including regional μ-opioid system responses to painful stimulation as measured by ligand-PET (positron emission tomography). The functional val158met single nucleotide polymorphism has been most widely studied. However, apart from its impact on pain-induced opioid release the effect of this genetic variation on cerebral pain processing has not been…

AdultMaleGenotypePainSingle-nucleotide polymorphismStimulationCatechol O-MethyltransferaseGyrus CinguliCellular and Molecular NeuroscienceYoung Adultmedicinelcsh:PathologyHumansddc:610AlleleAnterior cingulate cortexCerebral CortexCatechol-O-methyl transferasePolymorphism Geneticmedicine.diagnostic_testResearchLasersChronic painMiddle Agedmedicine.diseaseMagnetic Resonance ImagingAnesthesiology and Pain Medicinemedicine.anatomical_structureCerebral cortexPositron-Emission TomographyMolecular MedicineFemaleFunctional magnetic resonance imagingPsychologyNeurosciencelcsh:RB1-214Molecular Pain
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Prevalence of fimA genotypes of Porphyromonas gingivalis and other periodontal bacteria in a Spanish population with chronic periodontitis

2012

Objectives: The aim of this study was to determine the prevalence of the different fimA genotypes of Porphyromonas gingivalis in adult Spanish patients with chronic periodontitis, patients with gingivitis and periodontally healthy subjects, and the relationship between these genotypes and other periodontopathogenic bacteria. Study design: Samples of subgingival plaque were taken from 86 patients (33 with chronic periodontitis, 16 with gingivitis, and 37 periodontally healthy) in the course of a full periodontal examination. PCR was employed to determine the presence of the 6 fimA genotypes of Porphyromonas gingivalis (I-V and Ib) and of Aggregatibacter actinomycetemcomitans, Tannerella fors…

AdultMaleGenotypePeriodontal examinationDentistryOdontologíaMicrobiologyGingivitismedicineTannerella forsythiaHumansPeriodontologyGeneral DentistryPorphyromonas gingivalisPeriodontitisbiologybusiness.industryAggregatibacter actinomycetemcomitansTreponema denticolabiochemical phenomena metabolism and nutritionMiddle Aged:CIENCIAS MÉDICAS [UNESCO]medicine.diseasebiology.organism_classificationCiencias de la saludChronic periodontitisstomatognathic diseasesOtorhinolaryngologySpainUNESCO::CIENCIAS MÉDICASChronic PeriodontitisbacteriaSurgeryResearch-ArticleFemaleFimbriae Proteinsmedicine.symptombusinessPorphyromonas gingivalisMedicina Oral, Patología Oral y Cirugía Bucal
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Pathological implications of Th1/Th2 cytokine genetic variants in Behçet's disease: Data from a pilot study in a Sicilian population

2013

Cytokines act as pleiotropic polypeptides able to regulate inflammatory/immune responses and to provide important signals in physiological and pathological processes. Several cytokines (Th1, Th2, and Th17) seem to be involved in the pathophysiology of Behçet's disease, a chronic immune-mediated disease characterized by oral and genital lesions and ocular inflammation. Its individual susceptibility seems to be modulated by genetic variants in genes codifying these cytokines. Th1 and Th17 seem to be involved in the disease's active phases, and Th2 seems to affect the development or severity of the disease; however, contrasting data are reported. In this study, some genetic variants of the Th1…

AdultMaleGenotypePopulationPilot ProjectsBehcet's diseaseDiseaseBiologyPolymorphism Single NucleotideBiochemistryYoung AdultImmune systemTh1 and Th2 cytokines Immune imbalance Behc¸et’s disease Polymorphisms SusceptibilityGene FrequencyGenotypeGeneticsmedicineHumansSettore MED/05 - Patologia ClinicaeducationSicilyMolecular BiologyPathologicalEcology Evolution Behavior and SystematicsSettore MED/04 - Patologia Generaleeducation.field_of_studyBehcet SyndromeInterleukinsGenetic VariationGeneral MedicineMiddle Agedmedicine.diseaseHuman geneticsPathophysiologySettore MED/16 - ReumatologiaImmunologyFemale
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Variability in human hepatic MRP4 expression: influence of cholestasis and genotype

2007

The multidrug resistance protein 4 (MRP4) is an efflux transporter involved in the transport of endogenous substrates and xenobiotics. We measured MRP4 mRNA and protein expression in human livers and found a 38- and 45-fold variability, respectively. We sequenced 2 kb of the 5'-flanking region, all exons and intron/exon boundaries of the MRP4 gene in 95 patients and identified 74 genetic variants including 10 non-synonymous variations, seven of them being located in highly conserved regions. None of the detected polymorphisms was significantly associated with changes in the MRP4 mRNA or protein expression. Immunofluorescence microscopy indicated that none of the non-synonymous variations af…

AdultMaleGenotypeProtein ConformationBiologyPolymorphism Single NucleotideExonCholestasisTerminology as TopicGenotypeGenetic variationGeneticsmedicineHumansRNA MessengerGeneCellular localizationPharmacologyMessenger RNACholestasisPolymorphism GeneticReverse Transcriptase Polymerase Chain ReactionIntronGenetic VariationDNAmedicine.diseaseImmunohistochemistryMolecular biologyIntronsGene Expression RegulationHaplotypesLiverMicroscopy FluorescenceMolecular MedicineFemaleMultidrug Resistance-Associated ProteinsThe Pharmacogenomics Journal
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Further evidence that D90A-SOD1 mutation is recessively inherited in ALS patients in Italy.

2008

Mutations in the Cu/Zn superoxide dismutase 1 (SOD1) gene have been reported to cause adult-onset autosomal dominant amyotrophic lateral sclerosis (FALS). In sporadic cases (SALS), de novo mutations in the SOD1 gene have occasionally been observed. All the SOD1 mutations are autosomal dominantly inherited with the exception of D90A. To date, in Italy, only two sporadic ALS cases carrying the D90A mutation have been reported in a homozygous state. We investigated for the presence of this mutation in 169 unrelated ALS patients from southern Italy. The genetic analysis revealed three ALS patients (1.8%) with mild phenotype carrying the homozygous D90A mutation.

AdultMaleGenotypeSOD1DNA Mutational AnalysisGenes RecessiveBiologyGenetic analysisSuperoxide dismutaseSuperoxide Dismutase-1GenotypemedicineHumansGenetic Predisposition to DiseaseAmyotrophic lateral sclerosisGeneDe novo mutationsAgedGeneticsSuperoxide DismutaseAmyotrophic Lateral SclerosisSOD1; SLA;General MedicineSOD1Middle Agedmedicine.diseaseMolecular biologyNeurologyItalyMutation (genetic algorithm)Mutationbiology.proteinFemaleNeurology (clinical)SLA
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Increased frequency of the CTLA-4 49 A/G polymorphism in patients with acquired haemophilia A compared to healthy controls

2007

Acquired haemophilia (AH) is an autoimmune disorder characterized by autoantibodies against endogenous factor VIII (FVIII). Half of the patients present with an underlying disease known to cause the FVIII autoantibodies whereas in the other half the disease is of idiopathic nature. Recently, it has been shown that variants of the polymorphic cytotoxic T lymphocyte antigen-4 (CTLA-4) gene are associated with autoimmune diseases and also represent a risk factor for inhibitor formation in inherited haemophilia A. In the present study, we investigated whether CTLA-4 variants also play a role in the pathogenesis of AH. Therefore, we analyzed three single nucleotide polymorphisms (SNPs) of the CT…

AdultMaleGenotypeSingle-nucleotide polymorphismHemophilia AHaemophiliaPolymorphism Single NucleotideGene FrequencyAntigens CDGenotypemedicineHumansCTLA-4 AntigenGenetic Predisposition to DiseaseAlleleAllele frequencyGenetics (clinical)AgedAutoantibodiesAged 80 and overAutoimmune diseaseFactor VIIIbusiness.industryAutoantibodyHematologyGeneral MedicineMiddle Agedmedicine.diseaseAntigens DifferentiationCase-Control StudiesImmunologyFemaleGene polymorphismbusinessHaemophilia
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Precision Medicine in Graves’ Disease: CD40 Gene Variants Predict Clinical Response to an Anti-CD40 Monoclonal Antibody

2021

BackgroundCD40, a key co-stimulatory molecule expressed on antigen-presenting cells, is genetically associated with a number of autoimmune diseases including Graves’ disease (GD). Therefore, recent therapies targeting CD40 have been developed, including the anti-CD40 monoclonal antibody Iscalimab. In a recent pilot study, Iscalimab was shown to induce clinical remission in ~ 50% of GD patients, but the reason why only 50% of GD patients responded is not known. The aim of our study was to test the hypothesis that specific CD40 single nucleotide polymorphism (SNP) genotypes and haplotypes are associated with clinical response of GD patients to Iscalimab.MethodsWe extracted genomic DNA from th…

AdultMaleGenotypemedicine.drug_classprecision medicineEndocrinology Diabetes and MetabolismGraves' diseaseSingle-nucleotide polymorphismmedicine.disease_causeMonoclonal antibodyPolymorphism Single NucleotideDiseases of the endocrine glands. Clinical endocrinologyAutoimmunityYoung AdultEndocrinologyGenotypeCD40HumansMedicineSNPRNA MessengerCD40 AntigensgeneGeneAgedOriginal Researchbusiness.industryHaplotypeAntibodies MonoclonalMiddle AgedRC648-665medicine.diseaseGraves DiseasevariantImmunologyFemaleGraves’ diseasebusinessFrontiers in Endocrinology
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The -308G/A polymorphism of TNF-alpha influences immunological parameters in old subjects affected by infectious diseases.

2005

Abnormal increments of pro-inflammatory cytokines (IL-6 and TNF-alpha) characterize the outbreak of infectious diseases, which are the major cause of death in the elderly. A counterbalance to the inflammation is exerted by IL-10 with an inhibitory role on TNF-alpha production. As is well known, some cytokine gene polymorphisms influence the cytokine production, playing a role as susceptibility or resistance factors against immune-mediated and infectious disease. Genetic variations in the -308A/G locus for TNF-alpha seems to affect the clinical outcome of some infectious diseases. In fact, the -308A allele is associated with severe septic shock and death. On this basis, we have screened heal…

AdultMaleGenotypemedicine.medical_treatmentImmunologyCommunicable DiseasesGene FrequencyGenotypeGeneticsmedicineBronchopneumoniaHumansAlleleInterleukin 6Molecular BiologyAllele frequencyGenetics (clinical)AgedPolymorphism GeneticbiologyInterleukin-6Tumor Necrosis Factor-alphaOutbreakGeneral MedicineMiddle AgedBronchitis ChronicKiller Cells NaturalZincCytokineInfectious disease (medical specialty)Immunologybiology.proteinTumor necrosis factor alphaFemaleMetallothioneinInternational journal of immunogenetics
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Can the serological status of anti-HBc alone be considered a sentinel marker for detection of occult HBV infection?

2008

Some individuals have “occult” infection with hepatitis B virus (HBV), defined as presence of HBV genome in the serum or liver tissue without HBV surface antigen (HBsAg) in the serum. The aim of this study was to investigate whether serum antibodies against HBV core antigen in isolation (“anti-HBc alone”) are a useful marker of “occult” HBV in patients with or without hepatitis C virus (HCV) infection. “Anti-HBc alone” was detected in the sera of 119/6,544 (1.8%) asymptomatic outpatients referred to the diagnostic laboratory for routine testing for viral hepatitis, 62/607 (10.2%) drug users, and 42/195 (21.5%) patients with hepatocellular carcinoma. Using three in-house nested-PCR amplifica…

AdultMaleHBV serologic markerHBsAgHepatitis B virusGenotypeHepatitis C virusHBV genotypemedicine.disease_causeSettore MED/42 - Igiene Generale E ApplicataPolymerase Chain ReactionSensitivity and Specificity‘‘occult’’ HBVSerologyOrthohepadnavirusVirologymedicinePrevalenceHumansSerologic TestsHCCAgedHepatitis B virusAged 80 and overbiologybusiness.industryvirus diseasesSequence Analysis DNAHepatitis C AntibodiesMiddle Agedbiology.organism_classificationmedicine.diseaseHepatitis BVirologyHepatitis B Core AntigensHepatitis Cdigestive system diseasesInfectious DiseasesHepadnaviridaeImmunologyDNA Viralanti-HBcFemaleViral diseasebusinessViral hepatitisSentinel Surveillance
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