Search results for "giant cell"

showing 10 items of 125 documents

The ultrastructure of multinucleate giant cells

2002

Abstract A survey of the available ultrastructural data on physiologically and pathologically occurring and virally-induced multinucleate giant cells (MNGCs) is presented. Emphasis is initially placed upon the bone osteoclast, the skeletal muscle myotube and the placental syncytiotrophoblast. The widespread occurence of MNGCs in a range of pathological situations is discussed, with emphasis upon the broad involvement of the macrophage in inflammatory responses. Many viruses produce cell fusion in vivo and in vitro when cell cultures are infected. Several examples are given. A clear distinction is drawn between viral fusion from “without” and viral fusion from “within” the cell. The cytopath…

PopulationsyncytiotrophoblastGeneral Physics and AstronomyEndogenous retrovirusBiologyArticleSyncytiotrophoblastMultinucleateStructural Biologyendogenous retrovirusmedicineGeneral Materials ScienceeducationsyncytiaCytopathic effectSyncytiumeducation.field_of_studyCell fusioncell fusionCell BiologyCell biologymedicine.anatomical_structureGiant cellMultinucleate giant cellImmunologyHIV-1Micron (Oxford, England : 1993)
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Second-generation Langerhans cells originating from epidermal precursors are essential for CD8+ T cell priming.

2014

Abstract In vivo studies questioned the ability of Langerhans cells (LCs) to mediate CD8+ T cell priming. To address this issue, we used intradermal immunization with plasmid DNA, a system in which activation of CD8+ T cells depends on delayed kinetics of Ag presentation. We found that dendritic cells (DCs) located in the skin at the time of immunization have limited ability to activate CD8+ T cells. This activity was mediated by a second generation of DCs that differentiated in the skin several days after immunization, as well as by lymph node–resident DCs. Intriguingly, CD8+ T cell responses were not affected following treatment with clodronate liposomes, immunization of CCR2−/− mice, or …

Receptors CCR2T cellImmunologyPriming (immunology)CD11cchemical and pharmacologic phenomenaBiologyCD8-Positive T-LymphocytesLymphocyte ActivationMiceImmune systemGiant Cells LanghansmedicineImmunology and AllergyCytotoxic T cellAnimalsSkinMice KnockoutChemokine CCL20integumentary systemhemic and immune systemsCell DifferentiationDendritic CellsMolecular biologyCD11c AntigenCCL20Mice Inbred C57BLmedicine.anatomical_structureImmunologyIntercellular Signaling Peptides and ProteinsClodronic AcidCD8Ex vivoHeparin-binding EGF-like Growth FactorPlasmidsJournal of immunology (Baltimore, Md. : 1950)
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Establishment of an HIV cell-cell fusion assay by using two genetically modified HeLa cell lines and reporter gene.

2003

Infection of human cells with the human immunodeficiency virus type I (HIV-1) can be mimicked by a fusion process between cells expressing the HIV envelope protein (Env) and cells expressing both human CD4 together with the appropriate human chemokine receptors. In this study, a T-tropic HIV cell-cell fusion assay was established that utilized CD4, human CXCR4 and HIV NL4-3 gp160 as fusion components and a T7 polymerase-activated luciferase as a reporter system. The HeLa T4 cells used, expressed CD4 and CXCR4, and the applied HeLa KS386 cells expressed HIV NL4-3 gp160. By combining HeLa T4 cells with HeLa KS386 cells, an approximately about 100- to 300-fold increase in luciferase activity c…

Reporter geneReceptors CXCR4Cell fusionbiologyvirusesvirus diseasesHIV envelope proteinTransfectionGp41biology.organism_classificationTransfectionMolecular biologyGiant CellsHIV Envelope Protein gp160HeLaCell FusionCell cultureGenes ReporterVirologyCD4 AntigensHIV-1HumansLuciferaseBiological AssayHeLa CellsJournal of virological methods
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Non-cross-linked porcine-based collagen I-III membranes do not require high vascularization rates for their integration within the implantation bed: …

2012

There are conflicting reports concerning the tissue reaction of small animals to porcine-based, non-cross-linked collagen I-III membranes/matrices for use in guided tissue/bone regeneration. The fast degradation of these membranes/matrices combined with transmembrane vascularization within 4 weeks has been observed in rats compared with the slow vascularization and continuous integration observed in mice. The aim of the present study was to analyze the tissue reaction to a porcine-based non-cross-linked collagen I-III membrane in mice. Using a subcutaneous implantation model, the membrane was implanted subcutaneously in mice for up to 60 days. The extent of scaffold vascularization, tissue …

ScaffoldMaterials scienceBarrier membraneSus scrofaBiomedical EngineeringFibroinNeovascularization PhysiologicBiochemistryCollagen Type IBiomaterialsProsthesis ImplantationMicemedicineAnimalsBone regenerationMolecular BiologyPolytetrafluoroethyleneMembranesTissue ScaffoldsGranulation tissueMembranes ArtificialGeneral MedicineImmunohistochemistryTransmembrane proteinRatsmedicine.anatomical_structureMembraneCollagen Type IIICross-Linking ReagentsGiant cellBiophysicsMicroscopy Electron ScanningFemaleFibroinsBiotechnologyBiomedical engineeringActa biomaterialia
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The chemical composition of synthetic bone substitutes influences tissue reactions in vivo : histological and histomorphometrical analysis of the cel…

2012

Bone substitute material properties such as granule size, macroporosity, microporosity and shape have been shown to influence the cellular inflammatory response to a bone substitute material. Keeping these parameters constant, the present study analyzed the in vivo tissue reaction to three bone substitute materials (granules) with different chemical compositions (hydroxyapatite (HA), beta-tricalcium phosphate (TCP) and a mixture of both with a HA/TCP ratio of 60/40 wt%). Using a subcutaneous implantation model in Wistar rats for up to 30 days, tissue reactions, including the induction of multinucleated giant cells and the extent of implantation bed vascularization, were assessed using histo…

ScaffoldMaterials scienceGranule (cell biology)Biomedical EngineeringBioengineeringInflammationAnatomyPhosphateBone tissueBiomaterialschemistry.chemical_compoundmedicine.anatomical_structurechemistryIn vivoGiant cellmedicineBiophysicsHydroxyapatitesmedicine.symptomBiomedical Materials
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AF/R2 adhesin and cytopathic effect as virulence traits of diarrhea-inducing Escherichia coli O103 in European rabbit

1995

Escherichia coli strains belonging to 0103:K-:H2 serovar and rhamnose negative biovars are responsible for frequent life-threatening diarrheas in weaned rabbits from national breeding units in western Europe (Blanco et al., 1994; Camguilhem and A. Milon, 1989). According to their mechanisms of pathogenesis, these strains may be considered as enteropathogenic E. coli (EPEC)-like. They adhere to intestinal brush border and to HeLa cells by mean of an adhesin called AF/R2 (Adhesive Factor/Rabbit 2) (Milon et al., 1990). They do not produce known toxins (i.e. ST, LT, SLT, CNF, CLDT) (Blanco et al., 1994; Mariani-Kurkdjian et al., 1993) and bear sequences homologous to EPEC eaeA (Leroy et al., 1…

Serotype0303 health sciencesBrush border030306 microbiologyRhamnose[SDV]Life Sciences [q-bio]VirulenceBiologymedicine.disease_cause3. Good healthMicrobiologyBacterial adhesin[SDV] Life Sciences [q-bio]03 medical and health scienceschemistry.chemical_compoundchemistryGiant cellmedicineEscherichia coliComputingMilieux_MISCELLANEOUS030304 developmental biologyCytopathic effect
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Thymidine-, uridine- and choline-kinase in rabbit kidney cells infected with herpesvirus hominis, type I and II.

1972

Data are presented about the activity of the thymidine-, uridine- and choline-kinase after infection with 21 strains ofherpesvirus hominis of serotype I or II in rabbit kidney cells. Type I strains increase the activity of the thymidine-kinase 15–20 fold over the controls, whereas the type II strains demonstrate a moderate activity, the level of the enzyme is increased 2–5 fold. One giant cell forming strain exhibits unusual properties, the TK activity decreases in correlation to the controls. The uridine- and choline-kinases induce the respective enzymes in different manner. The strains tested are divided into 5 groups depending upon the activity of the enzymatic activity. The implications…

SerotypeCholine kinaseBiologyKidneyThymidine KinaseCholinechemistry.chemical_compoundSpecies SpecificityVirologyCulture TechniquesRabbit kidneyAnimalsSimplexvirusSerotypingUridineCells Culturedchemistry.chemical_classificationStrain (chemistry)PhosphotransferasesGeneral MedicineVirologyUridineEnzymechemistryGiant cellRabbitsThymidineArchiv fur die gesamte Virusforschung
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Die Hemmung der Riesenzellbildung durch das sogenannte Compound 48/80 nach der Infektion mit dem Herpesvirus hominis

1969

Es wurde uber die Wirkung des Histamin-Liberators Compound 48/80 auf die Phasen der Synthese des Herpesvirus hominis und auf normale Zellstoffwechselprozessein vitro berichtet.

Simplexvirusfood.ingredientP-Methoxy-N-methylphenethylamineGeneral MedicineCompound 48/80BiologyVirologyMolecular biologychemistry.chemical_compoundfoodHerpesvirus hominischemistryViral replicationGiant cellVirologyArchiv f�r die gesamte Virusforschung
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Evidence for a multistep mechanism for cell-cell fusion by herpes simplex virus with mutations in the syn 3 locus using heparin derivatives during fu…

1994

Addition of heparin-Na+ as well as related substances of high and intermediate MW (Arteparon and polyanion SP54) 3 h after infection inhibit fusion from within (FFWI) induced by HSV strains with mutations in the syn 3 locus only. The concentration of heparin-Na+ required to inhibit FFWI is 10-fold higher (1 mg/ml) than that needed to inhibit adsorption. Instead of fusion, cell rounding is observed. The effect is readily reversible. A low MW heparin disaccharide is ineffective. Neomycin, at a concentration of 8 mM, inhibits FFWI induced by all HSV-1 but not HSV-2 strains, whereas adsorption is inhibited at 3 mM. We conclude from our observations that cell-cell fusion (FFWI) induced by syn 3 …

SyncytiumCell fusionHeparinCellMutantGeneral MedicineBiologyGiant CellsVirologyCell membranemedicine.anatomical_structureMutagenesisCell cultureCell surface receptorVirologyChlorocebus aethiopsmedicineVero cellAnimalsSimplexvirusVero CellsCells CulturedArchives of Virology
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Cell Immunity in Inflammatory Vasculitis

2015

The vasculitides are a highly heterogeneous group of disorders characterized by the presence of inflammatory leukocytes in the vessel walls and reactive inflammation. Giant cell arteritis (GCA) and Takayasu’s arteritis (TA) are the two primary large-vessel vasculitides. Two distinct cellular pathways have been identified in GCA: Th17 polarization and IL-17 secretion and generation of Th1 cells which secrete IFN-γ. These two pathways may play different roles in the pathogenesis of vasculitides. The antineutrophil cytoplasmic antibody (ANCA)-associated vasculitides (AAVs) are small vessel vasculitis associated with antibodies directly to myeloperoxidase (MPO-ANCA) such as eosinophilic granulo…

TakayasuVasculitisSettore MED/16 - ReumatologiaANCAHortonImmunologyVasculitideImmunology and AllergyBehçet syndromeGiant cell vasculiti
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