Search results for "glutamine"

showing 10 items of 122 documents

Induction of Transglutaminase 2 by a Liver X Receptor/Retinoic Acid Receptor α Pathway Increases the Clearance of Apoptotic Cells by Human Macrophages

2009

Rationale: Liver X receptors (LXRs) are oxysterol-activated nuclear receptors that are involved in the control of cholesterol homeostasis and inflammatory response. Human monocytes and macrophages express high levels of these receptors and are appropriate cells to study the response to LXR agonists. Objective: The purpose of this study was to identify new LXR targets in human primary monocytes and macrophages and the consequences of their activation. Methods and Results: We show that LXR agonists significantly increase the mRNA and protein levels of the retinoic acid receptor (RAR)α in primary monocytes and macrophages. LXR agonists promote RARα gene transcription through binding to a spec…

Agonistmedicine.medical_specialtyReceptors Retinoic AcidPhysiologymedicine.drug_classResponse elementReceptors Cytoplasmic and NuclearApoptosisBiologyCell LinePhagocytosisGTP-Binding ProteinsInternal medicinemedicineHumansMacrophageProtein Glutamine gamma Glutamyltransferase 2ReceptorLiver X receptorLiver X ReceptorsTransglutaminasesMacrophagesRetinoic Acid Receptor alphaMacrophage ActivationAtherosclerosisOrphan Nuclear ReceptorsCell biologyDNA-Binding ProteinsRetinoic acid receptorEndocrinologyNuclear receptorRetinoic acid receptor alphaEnzyme InductionCardiology and Cardiovascular MedicineCirculation Research
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Human sweat odour conjugates in human milk, colostrum and amniotic fluid

2012

International audience; Using ultra performance liquid chromatography-mass spectrometry we identified for the first time glutamine-N-alpha-conjugates of the fatty acids (E)/(Z)-3-methylhex-2-enoic acid and (R)/(S)-3-hydroxy-3-methylhexanoic acid as well as cysteinylglycine-S-conjugates of (R)/(S)-3-methyl-3-sulphanylhexan-1-ol and (R)/(S)-3-sulphanylhexan-1-ol as constituents of human milk and colostrum. The glutamine-N-alpha-conjugates were detected also in human amniotic fluids. The mean values of glutamine-N-alpha-conjugate of (R)/(S)-3-hydroxy-3-methylhexanoic acid were highest in colostrums with a range of <0.1-382 mu g/kg, followed by the mature human milk with values from <0.1 to 39.…

Amniotic fluid[ SDV.AEN ] Life Sciences [q-bio]/Food and Nutritionglutamine-n-alpha-conjugateAnalytical ChemistrySWEATCow milk03 medical and health sciences0302 clinical medicinefluids and secretions030225 pediatricsLactationmedicineodour precursorMature milkComputingMilieux_MISCELLANEOUS030304 developmental biology0303 health sciencesChromatographycysteinylglycine-s-conjugateChemistryfood and beverageshuman milkamniotic fluidGeneral MedicineGlutaminemedicine.anatomical_structureColostrum[SDV.AEN]Life Sciences [q-bio]/Food and NutritionFood ScienceConjugate
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Mitochondrial glutathione depletion by glutamine in growing tumor cells.

2000

The effect of L-glutamine (Gln) on mitochondrial glutathione (mtGSH) levels in tumor cells was studied in vivo in Ehrlich ascites tumor (EAT)-bearing mice. Tumor growth was similar in mice fed a Gln-enriched diet (GED; where 30% of the total dietary nitrogen was from Gln) or a nutritionally complete elemental diet (SD). As compared with non-tumor-bearing mice, tumor growth caused a decrease of blood Gln levels in mice fed an SD but not in those fed a GED. Tumor cells in mice fed a GED showed higher glutaminase and lower Gln synthetase activities than did cells isolated from mice fed an SD. Cytosolic glutamate concentration was 2-fold higher in tumor cells from mice fed a GED ( approximately…

AnionsMalemedicine.medical_specialtyFree RadicalsGlutamineOxidative phosphorylationBiologyMitochondrionMitochondrial Sizemedicine.disease_causeBiochemistryGlutaminase activitychemistry.chemical_compoundMiceAdenosine TriphosphatePhysiology (medical)Internal medicinemedicineAnimalsHumansAmino AcidsCarcinoma Ehrlich TumorGlutaminaseTumor Necrosis Factor-alphaGlutathioneHydrogen-Ion ConcentrationGlutathioneRecombinant ProteinsMitochondriaGlutamineOxidative StressEndocrinologyBiochemistrychemistryOxidative stressFree radical biologymedicine
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Glutamine synthetases of green and etiolated leaves ofSinapis alba : Evidence of the identity of the respective enzyme proteins.

1989

Studies on the glutamine synthetases (GS, EC 6.3.1.2) of green (GS2) and etiolated leaves (GSet) ofSinapis alba L. (cv. Steinacher) revealed striking similarities between the respective enzyme proteins. The enzymes showed corresponding chromatographic properties, both on dimethylaminoethyl-Sephacel and on hydroxylapatite columns. The purified GS proteins were also identical with regard to the molecular weight of their subunits. Isoelectrofocusing of pure GSet yielded two distinct polypeptide bands in the pH 5.6 region of the gels. This pattern corresponded to the two strong bands of GS2. Two charge variants of GS polypeptides could be detected by Western-blot analysis of the soluble protein…

Antiserumchemistry.chemical_classificationGel electrophoresisImmunoprecipitationPlant ScienceBiologyGlutamineEnzymeBiochemistrychemistryHoloenzymesGlutamine synthetaseEtiolationGeneticsPlanta
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High Plasma Glutamate and a Low Glutamine-to-Glutamate Ratio Are Associated with Increased Risk of Heart Failure but Not Atrial Fibrillation in the P…

2020

[Background] Although the association between glutamate and glutamine in relation to cardiometabolic disorders has been evaluated, the role of these metabolites in the development of atrial fibrillation (AF) and heart failure (HF) remains unknown.

Blood GlucoseMalePREDIMEDmedicine.medical_specialtyGlutaminePopulationGlutamic AcidMedicine (miscellaneous)Heart failureDiet MediterraneanBody Mass IndexRisk FactorsOriginal Research ArticlesInternal medicinemedicineHumanseducationAgededucation.field_of_studyNutrition and Dieteticsbusiness.industryIncidence (epidemiology)Glutamate receptorAtrial fibrillationMiddle Agedmedicine.diseaseLipidsPredimedAtrial fibrillationGlutamineEndocrinologyQuartileCase-Control StudiesHeart failureFemaleGlutamatebusiness
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Delivering all in one: Antigen-nanocapsule loaded with dual adjuvant yields superadditive effects by DC-directed T cell stimulation

2018

Therapeutic vaccination is and remains a major challenge, particularly in cancer treatment. In this process, the effective activation of dendritic cells by a combination of distinctly acting adjuvants and an antigen is crucial for success. While most common vaccine formulations lack the efficiency to trigger sufficient T cell responses in a therapeutic tumor treatment, nanovaccines offer unique properties to tackle that challenge. Here, we report the stepwise development of a nanocapsule for vaccination approaches, comprising a shell consisting of antigen and loaded with a superadditive adjuvant combination. In a first initial step, we identified the combination of resiquimod (R848) and mur…

CD4-Positive T-Lymphocytes0301 basic medicineCell SurvivalOvalbuminT-Lymphocytesmedicine.medical_treatmentT cellPharmaceutical ScienceMice Transgenic02 engineering and technologyCD8-Positive T-LymphocytesCancer VaccinesCell Line03 medical and health scienceschemistry.chemical_compoundNanocapsulesAntigenmedicineAnimalsHumansAntigensCytotoxicityAdjuvants PharmaceuticCell ProliferationChemistryImidazolesDextransDendritic CellsDendritic cell021001 nanoscience & nanotechnologyCell biologyMice Inbred C57BL030104 developmental biologymedicine.anatomical_structureCytokinesSpermineResiquimod0210 nano-technologyAcetylmuramyl-Alanyl-IsoglutamineAdjuvantMuramyl dipeptideCD8Journal of Controlled Release
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Glioblastoma cells induce differential glutamatergic gene expressions in human tumor-associated microglia/macrophages and monocyte-derived macrophages

2015

Glioblastoma cells produce and release high amounts of glutamate into the extracellular milieu and subsequently can trigger seizure in patients. Tumor-associated microglia/macrophages (TAMs), consisting of both parenchymal microglia and monocytes-derived macrophages (MDMs) recruited from the blood, are known to populate up to 1/3 of the glioblastoma tumor environment and exhibit an alternative, tumor-promoting and supporting phenotype. However, it is unknown how TAMs respond to the excess extracellular glutamate in the glioblastoma microenvironment. We investigated the expressions of genes related to glutamate transport and metabolism in human TAMs freshly isolated from glioblastoma resecti…

Cancer ResearchAntigens Differentiation MyelomonocyticGlutamic AcidglutamateAMPA receptorSLC7A11Antigens CDTumor Cells CulturedExtracellularmedicineHumansReceptors AMPAGRIA2PharmacologyCD11b AntigenbiologyMicrogliaBrain NeoplasmsMacrophagesmonocyte-derived macrophagesCalcium-Binding ProteinsMicrofilament Proteinsglioblastomatumor-associated microglia/macrophagesGlutamate receptorSLC1A2Coculture TechniquesDNA-Binding ProteinsGlutaminemedicine.anatomical_structureGene Expression RegulationOncologyAstrocytesImmunologybiology.proteinCancer researchLeukocyte Common AntigensMolecular MedicineMicrogliaResearch PaperCancer Biology &amp; Therapy
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TMIC-49. POTASSIUM CHANNEL KIR4.1 AND GLUTAMINE SYNTHETASE ARE DYSREGULATED IN GLIOMA

2017

The potassium channel KIR4.1 (KCNJ10) and the glutamate catalyzing enzyme glutamine synthetase (GS) are highly expressed in glial cells of the central nervous system. Both glial proteins play important roles in the maintenance of neuronal activity and neurotransmission. Dysfunction of both proteins can result in altered neuronal excitability and may lead to excitotoxicity. We analyzed 35 snap frozen tissue blocks (glioblastoma [GBM], n=22; low grade astrocytoma (LGA), n=8; oligodendroglioma (OG), n=3; oligoastrocytoma, n=2). All glioma samples had a matching tissue specimen from both the tumor core and the adjacent normal-appearing infiltration zone. Molecular subtyping (MGMT, IDH1/2, 1p/19…

Cancer ResearchChemistryGlutamate receptorExcitotoxicitymedicine.diseasemedicine.disease_causePotassium channelAbstractsmedicine.anatomical_structureOncologyGlutamine synthetaseGliomaGene expressionCancer researchmedicineNeurogliaNeurology (clinical)Oligodendroglioma
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Lactate-induced inhibition of tumor cell proliferation.

1988

Abstract Culture medium that was recovered from tumor cell or fibroblast cultures during the plateau phase, and that was replenished by addition of glucose, glutamine, and serum and readjustment of pH had a distinct growth-inhibiting effect on monolayer cell cultures. The effect, which was not specific for a given cell strain, may be partially responsible for the "density inhibition" commonly observed in malignant cells grown in monolayer cultures. By modifying fresh growth media, it was shown that the growth inhibition observed can be partly attributed to the accumulation of lactate in the culture medium of plateau phase cells. This substance reduced the plating efficiency and the number o…

Cancer ResearchPlating efficiencyPlateau (mathematics)law.inventionchemistry.chemical_compoundMiceIn vivolawMonolayerTumor Cells CulturedMedicineAnimalsHumansRadiology Nuclear Medicine and imagingLactic AcidAmino AcidsRadiationbusiness.industryPetri dishCell biologyCulture MediaGlutamineOncologychemistryCell cultureImmunologyLactatesGrowth inhibitionbusinessCell DivisionInternational journal of radiation oncology, biology, physics
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Bcl-2 and glutathione depletion sensitizes B16 melanoma to combination therapy and eliminates metastatic disease.

2007

Abstract Purpose: Advanced melanoma resists all current therapies, and metastases in the liver are particularly problematic. Prevalent resistance factors include elevated glutathione (GSH) and increased expression of bcl-2 in melanoma cells. GSH has pleiotropic effects promoting cell growth and broad resistance to therapy, whereas Bcl-2 inhibits the activation of apoptosis and contributes to elevation of GSH. This study determined the in vivo efficacy of combination therapies administered while GSH and Bcl-2 were individually and simultaneously decreased in metastatic melanoma lesions. Experimental Design: Highly metastatic murine B16 melanoma (B16M-F10) cells have elevated levels of both G…

Cancer Researchmedicine.medical_specialtySkin NeoplasmsCombination therapyPaclitaxelGlutamineMelanoma ExperimentalBiologyMetastasischemistry.chemical_compoundMiceIn vivoInternal medicinemedicineAnimalsNeoplasm MetastasisAcivicinHematologyTumor Necrosis Factor-alphaMelanomaX-RaysGlutathioneThionucleotidesmedicine.diseaseAntineoplastic Agents PhytogenicCombined Modality TherapyGlutathioneTreatment OutcomeOncologychemistryProto-Oncogene Proteins c-bcl-2ToxicityCancer researchClinical cancer research : an official journal of the American Association for Cancer Research
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