Search results for "harm"

showing 10 items of 13866 documents

Antagonism by SR 48692 of mechanical responses to neurotensin in rat intestine.

1996

Abstract 1. The effects of SR 48692 on neurotensin (NT)-induced mechanical responses were investigated in rat duodenum and proximal colon by use of isometric, isovolumic preparations. 2. SR 48692 inhibited the relaxant responses to NT in duodenal circular and longitudinal muscle. It also antagonized the NT-induced contractile effects in duodenal circular muscle and in proximal colon (both muscular layers). 3. From Schild analysis and pA2 value for SR 48692 was 8.2 in tissues where NT induced relaxant effects and 7.5 in tissues where NT induced contractile effects and the slope of the regression line was not significantly different from unity, indicating competitive antagonism. 4. SR 48692 d…

medicine.medical_specialtyCarbacholColonDuodenumMuscle RelaxationNeuropeptideSubstance PBiologyPeptide hormoneIn Vitro Techniqueschemistry.chemical_compoundNorepinephrineInternal medicineIsometric ContractionmedicineAnimalsReceptors NeurotensinVasoconstrictor AgentsRats WistarReceptorNeurotensinPharmacologyMuscle SmoothRatsMuscle relaxationmedicine.anatomical_structureEndocrinologychemistryDuodenumQuinolinesPyrazolesmedicine.drugNeurotensinResearch ArticleBritish journal of pharmacology
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Pre- and postsynaptic effects of muscarinic agonists in the guinea-pig ileum

1980

The effects of several muscarinic agonists on smooth muscle (postsynaptic effect) and on acetylcholine release (presynaptic effect) were compared in the longitudinal muscle-myenteric plexus preparation of the guinea-pig ileum. 1. For release experiments the acetylcholine stores of the preparation were labelled with 3H-choline. Electrical field stimulation in the absence of a cholinesterase inhibitor caused an outflow of tritium that reflected release of 3H-acetylcholine. The agonists oxotremorine, arecaidinepropargylester, methylfurmethide, muscarine, carbachol, arecoline and pilocarpine inhibited the stimulation-induced outflow in a concentration-dependent manner. At the highest concentrat…

medicine.medical_specialtyCarbacholGuinea PigsNeuromuscular JunctionIn Vitro TechniquesReceptors NicotinicTritiumInhibitory postsynaptic potentialchemistry.chemical_compoundIleumPostsynaptic potentialInternal medicineMuscarinic acetylcholine receptormedicineMuscarinic acetylcholine receptor M4OxotremorineAnimalsReceptors CholinergicPharmacologyMuscarineOxotremorineGeneral MedicineReceptors MuscarinicAcetylcholineEndocrinologyParasympathomimeticsSolubilitychemistryAcetylcholineMuscle Contractionmedicine.drugNaunyn-Schmiedeberg's Archives of Pharmacology
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The release of choline from phospholipids mediated by beta-adrenoceptor activation in isolated hearts.

1986

The resting efflux of choline into the perfusate (Tyrode's solution) of isolated hearts was equal to the rate, at which choline was liberated from phospholipid degradation (Lindmar et al. 1986). Infusion of isoprenaline (2 X 10(-7) mol/l), forskolin (1-3 X 10(-6) mol/l) or 3-isobutyl-1-methylxanthine (IBMX; 3 X 10(-4) mol/l) for 40 min markedly enhanced the efflux of choline. The increase was linear during the experimental period and, in the case of isoprenaline, was blocked by 3 X 10(-7) mol/l atenolol. In the guinea-pig heart, IBMX at a threshold concentration of 10(-4) mol/l shifted the concentration-response curve for the effect of forskolin on the efflux of choline to the left by one l…

medicine.medical_specialtyCarbacholIBMXGuinea PigsPhospholipidIn Vitro TechniquesCholinechemistry.chemical_compoundInternal medicineIsoprenaline1-Methyl-3-isobutylxanthineReceptors Adrenergic betamedicineCyclic AMPCholineAnimalsPhospholipidsCholinesterasePharmacologyForskolinbiologyMyocardiumColforsinGeneral MedicineMyocardial ContractionReceptors MuscarinicEndocrinologychemistryQuinacrinebiology.proteinCalciumChickensAcetylcholinemedicine.drugNaunyn-Schmiedeberg's archives of pharmacology
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Zinc acexamate inhibits gastric acid and pepsinogen secretion in the rat.

1990

Abstract Pretreatment with zinc acexamate (25–100 mg kg−1 i.p.) inhibited acid and pepsinogen secretion in the pylorus-ligated rat. Zinc acexamate (5–50 mg kg−1 p.o.) also inhibited the increases in acid secretion induced by carbachol (10 μg kg−1) and 2-deoxy-D-glucose (200 mg kg−1) in the perfused stomach of the anaesthetized rat. A delayed antisecretory effect was observed with this drug on histamine induced responses. High concentrations of zinc acexamate (10−5-10−2 M) did not modify the in-vitro activity of pepsin. Administration of zinc acexamate resulted in an increase in the presence of pepsinogen at the mucosal level. A morphological examination of the gastric mucosa confirmed an ac…

medicine.medical_specialtyCarbacholPharmaceutical Sciencechemistry.chemical_elementZincGastric Acidchemistry.chemical_compoundPepsinInternal medicinemedicineGastric mucosaAnimalsAnesthesiaPylorusPharmacologyAminocaproatesbiologyPepsinogensChemistryStomachRatsGastric chief cellPerfusionmedicine.anatomical_structureEndocrinologyGastric MucosaAminocaproic Acidbiology.proteinGastric acidHistaminemedicine.drugThe Journal of pharmacy and pharmacology
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Screening and surveillance for hepatocellular carcinoma: perspective of a new era?

2016

Hepatocellular carcinoma (HCC) is the fifth most common cancer and the third most common cause of cancer-related death with an increasing prevalence worldwide. Early diagnosis of HCC is important since observational studies have reported that, in patients undergoing surveillance, cancer is diagnosed at an earlier stage with increased chances of curative therapies. Anyway, despite the extensive use of screening for HCC, its effectiveness is still a controversial topic since supporting evidence is not unequivocal and some issues need to be explored. Areas covered: The aim of this paper is to review main literature data supporting performance and effectiveness of screening for early detection …

medicine.medical_specialtyCarcinoma HepatocellularEarly detectionGastroenterology03 medical and health sciences0302 clinical medicineInternal medicinemedicineHumansMass ScreeningPharmacology (medical)In patientStage (cooking)Intensive care medicineCancer specific mortalityEarly Detection of CancerNeoplasm StagingRandomized Controlled Trials as Topicbusiness.industryLiver NeoplasmsCancermedicine.diseasedigestive system diseasesOncology030220 oncology & carcinogenesisHepatocellular carcinomaPopulation Surveillance030211 gastroenterology & hepatologyObservational studybusinessExpert review of anticancer therapy
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A mathematical model of cardiovascular dynamics for the diagnosis and prognosis of hemorrhagic shock

2020

Abstract A variety of mathematical models of the cardiovascular system have been suggested over several years in order to describe the time-course of a series of physiological variables (i.e. heart rate, cardiac output, arterial pressure) relevant for the compensation mechanisms to perturbations, such as severe haemorrhage. The current study provides a simple but realistic mathematical description of cardiovascular dynamics that may be useful in the assessment and prognosis of hemorrhagic shock. The present work proposes a first version of a differential-algebraic equations model, the model dynamical ODE model for haemorrhage (dODEg). The model consists of 10 differential and 14 algebraic e…

medicine.medical_specialtyCardiac outputMean arterial pressureShock HemorrhagicSettore ING-INF/01 - ElettronicaCardiovascular SystemGeneral Biochemistry Genetics and Molecular Biologycardiovascular dynamicshemorrhagic shockHeart RateInternal medicineHeart ratemedicineQuantitative assessmentAnimalsmathematical modellingCardiac OutputGeneral Environmental SciencePharmacologyGeneral Immunology and MicrobiologyMathematical modelbusiness.industryApplied MathematicsGeneral NeuroscienceSettore ING-IND/34 - Bioingegneria IndustrialeExperimental dataGeneral MedicineModels Theoreticalhemorrhagic shock;Blood pressureModeling and SimulationSettore ING-INF/06 - Bioingegneria Elettronica E InformaticaHemorrhagic shockCardiologybusinessMathematical Medicine and Biology: A Journal of the IMA
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Expert opinion on managing chronic HCV in patients with cardiovascular disease

2018

International audience; Extrahepatic manifestations of chronic HCV infection include cardiovascular diseases and an increase in cardiovascular mortality. The pathogenic mechanisms by which HCV contributes to cardiovascular disease are not well defined, however, it is likely that systemic inflammation, and the promotion of other metabolic diseases are involved. In this Review, the evidence for HCV infection as a non-traditional risk factor for cardiovascular disease is evaluated. Furthermore, practical advice to evaluate cardiovascular disease risk and disease in chronic hepatitis C patients are included for help in daily clinical practice. Despite the advances in therapies for the treatment…

medicine.medical_specialtyCardiotonic AgentsHepacivirusDisease030204 cardiovascular system & hematologySystemic inflammationAntiviral Agents03 medical and health sciences0302 clinical medicine[SDV.MHEP.CSC]Life Sciences [q-bio]/Human health and pathology/Cardiology and cardiovascular systemRisk Factors[SDV.MHEP.MI]Life Sciences [q-bio]/Human health and pathology/Infectious diseasesOdds RatioHumansMedicineDrug InteractionsPharmacology (medical)In patient030212 general & internal medicineRisk factorIntensive care medicineExpert TestimonyCardiovascular mortalityInflammationPharmacologybusiness.industryDisease progressionDisease ManagementHepatitis C Chronic3. Good healthInfectious DiseasesCardiovascular DiseasesExpert opinionPractice Guidelines as TopicDisease riskmedicine.symptombusiness
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A Review of the Cardiovascular and Anti-Atherogenic Effects of Ghrelin

2013

Ghrelin is a peptide hormone produced mainly in the stomach that has widespread tissue distribution and diverse hormonal, metabolic and cardiovascular activities. The circulating ghrelin concentration increases during fasting and decreases after food intake. Ghrelin secretion may thus be initiated by food intake and is possibly controlled by nutritional factors. Lean subjects have increased levels of circulating ghrelin compared with obese subjects. Recent reports show that low plasma ghrelin is associated with elevated fasting insulin levels, insulin resistance and type 2 diabetes mellitus. Factors involved in the regulation of ghrelin secretion have not yet been defined; however, it is as…

medicine.medical_specialtyCardiotonic AgentsHyperlipidemias030209 endocrinology & metabolismPeptide hormoneBiologyCardiovascular System03 medical and health sciences0302 clinical medicineInsulin resistanceInternal medicineDiabetes mellitusInsulin SecretionDrug DiscoverymedicineAnimalsHumansHypoglycemic AgentsInsulinAntiatherogenic agentHypolipidemic Agents030304 developmental biology2. Zero hungerPharmacology0303 health sciencesEvidence-Based Medicinedigestive oral and skin physiologyType 2 Diabetes MellitusLipid Metabolismmedicine.diseaseGhrelin3. Good healthEndocrinologyDiabetes Mellitus Type 2Cardiovascular DiseasesGastric MucosaHyperglycemiaGhrelinhormones hormone substitutes and hormone antagonistsGhrelin secretionHormoneCurrent Pharmaceutical Design
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Special Considerations for Antihypertensive Agents in Dialysis Patients

2010

Hypertension is present in most patients with end-stage renal disease and likely contributes to the premature cardiovascular disease in dialysis patients. Previous practice guidelines have recommended that, in patients on chronic dialysis, blood pressure (BP) should be reduced below 130/80 mm Hg. This is based on opinions but not strong evidence, since no concrete information exists about which BP values should be the parameter to follow and which should be the target BP values. The majority of the antihypertensive agents can be used in this population, but the pharmacokinetics altered by the impaired kidney function and dialyzability influence the appropriate dosage as well as the time and…

medicine.medical_specialtyCardiotonic AgentsHypertension RenalCombination therapyMetabolic Clearance Ratemedicine.drug_classVasodilator Agentsmedicine.medical_treatmentAdrenergic beta-AntagonistsPopulationAngiotensin-Converting Enzyme InhibitorsCardiotonic AgentsRenal DialysisInternal medicinemedicineHumansDrug InteractionsDiureticseducationAntihypertensive drugAntihypertensive AgentsDialysisRandomized Controlled Trials as Topiceducation.field_of_studybusiness.industryHematologyGeneral MedicineCalcium Channel Blockersmedicine.diseaseEndocrinologyBlood pressureCardiovascular DiseasesNephrologyPractice Guidelines as TopicPolypharmacyKidney Failure ChronicDrug Therapy CombinationHemodialysisbusinessAngiotensin II Type 1 Receptor BlockersKidney diseaseBlood Purification
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Assessment of the cardiostimulant action of propionyl-L-carnitine on chronically volume-overloaded rat hearts.

1993

Chronic volume overload was induced in young rats of Wistar strain by surgical opening of the aorto-caval fistula. Three months later, during in vitro perfusion with exogenous palmitate, left ventricular function and energy turnover (QO2) of hypertrophied hearts were severely depressed. This seemed to be related to impaired long-chain fatty acid utilization, as reflected by decreased 14CO2 production from U-14C-palmitate and decreased tissue levels of L-carnitine. Another group of rats exposed to chronic volume overload was pretreated for 2 weeks before sacrifice with propionyl-L-carnitine (250 mg/kg/day), and the hearts were perfused with 1.2 mM palmitate and 10 mM propionyl-L-carnitine. I…

medicine.medical_specialtyCardiotonic AgentsVolume overloadPalmitatesCardiomegalyMitochondria HeartVentricular Function LeftMuscle hypertrophyContractilityOxygen ConsumptionInternal medicineCarnitinemedicineAnimalsPharmacology (medical)Rats WistarPharmacologychemistry.chemical_classificationVentricular functionbusiness.industryMyocardiumFatty acidGeneral MedicinePropionyl l carnitineCarbon DioxideMyocardial ContractionRatsEndocrinologychemistryVolume (thermodynamics)Cardiac hypertrophyCardiology and Cardiovascular MedicinebusinessCardiovascular drugs and therapy
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