Search results for "hate"

showing 10 items of 2099 documents

Lack of effect of oxytocin on the numbers of ?synaptic? ribbons, cyclic guanosine monophosphate and serotonin N-acetyltransferase activity in organ-c…

1993

In addition to the stimulating influence of the sympathetic system on the function of the mammalian pineal gland, neuropeptides such as neuropeptide Y, vasoactive intestinal polypeptide and arginine-vasopressin (AVP) are thought to function as modulators. Since AVP has been shown to influence pineal melatonin synthesis, the aim of the present study was to investigate the possible effects of the second hypothalamic nonapeptide oxytocin (OT), which likewise has been detected in the pineal gland. We therefore studied "synaptic" ribbon (SR) numbers, N-acetyltransferase (NAT) activity and the intracellular concentration of cyclic guanosine monophosphate (cGMP) following in vitro incubation of ra…

Maleendocrine systemmedicine.medical_specialtyHistologyArylamine N-AcetyltransferaseVasoactive intestinal peptideNeuropeptideCell CommunicationBiologyOxytocinPineal GlandPathology and Forensic MedicineRats Sprague-DawleyNorepinephrinechemistry.chemical_compoundPineal glandOrgan Culture TechniquesInternal medicinemedicineAnimalsCyclic GMPCyclic guanosine monophosphateOrganellesRats BrattleboroRats Inbred StrainsCell BiologyNeuropeptide Y receptorCircadian RhythmRatsArginine Vasopressinmedicine.anatomical_structureEndocrinologynervous systemOxytocinchemistrySerotoninhormones hormone substitutes and hormone antagonistsmedicine.drugEndocrine glandCell & Tissue Research
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Blood Glutathione as an Index of Radiation-Induced Oxidative Stress in Mice and Humans

1997

Abstract The effect of x-rays on GSH and GSSG levels in blood was studied in mice and humans. An HPLC method that we recently developed was applied to accurately determine GSSG levels in blood. The glutathione redox status (GSH/GSSG) decreases after irradiation. This effect is mainly due to an increase in GSSG levels. Mice received single fraction radiotherapy, at total doses of 1.0 to 7.0 Gy. Changes in GSSG in mouse blood can be detected 10 min after irradiation and last for 6 h within a range of 2.0–7.0 Gy. The highest levels of GSSG (20.1 ± 2.9 μ M), a 4.7-fold increase as compared with controls) in mouse blood are found 2 h after radiation exposure (5 Gy). Breast and lung cancer patien…

Maleinorganic chemicalsmedicine.medical_specialtyLung NeoplasmsRadicalBreast NeoplasmsRadiation inducedOxidative phosphorylationGlucosephosphate Dehydrogenasemedicine.disease_causeBiochemistryMicechemistry.chemical_compoundfluids and secretionsPhysiology (medical)Internal medicinemedicineAnimalsHumansIrradiationRadiation InjuriesChromatography High Pressure LiquidGlutathione TransferaseGlutathione PeroxidaseGlutathione DisulfideChemistryDose-Response Relationship RadiationGlutathioneGlutathioneRedox statusSingle fractionOxidative StressGlutathione ReductaseEndocrinologyBiochemistryFemaleOxidation-ReductionOxidative stressFree Radical Biology and Medicine
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TORC1 Inhibition by Rapamycin Promotes Antioxidant Defences in a Drosophila Model of Friedreich’s Ataxia

2015

Friedreich's ataxia (FRDA), the most common inherited ataxia in the Caucasian population, is a multisystemic disease caused by a significant decrease in the frataxin level. To identify genes capable of modifying the severity of the symptoms of frataxin depletion, we performed a candidate genetic screen in a Drosophila RNAi-based model of FRDA. We found that genetic reduction in TOR Complex 1 (TORC1) signalling improves the impaired motor performance phenotype of FRDA model flies. Pharmacologic inhibition of TORC1 signalling by rapamycin also restored this phenotype and increased the lifespan and ATP levels. Furthermore, rapamycin reduced the altered levels of malondialdehyde + 4-hydroxyalke…

Malelcsh:MedicineGene Expressionmedicine.disease_causeAntioxidantsAnimals Genetically ModifiedAdenosine Triphosphate0302 clinical medicineRNA interferenceIron-Binding ProteinsMalondialdehydeDrosophila Proteinslcsh:ScienceAconitate HydrataseGenetics0303 health sciencesMultidisciplinaryReverse Transcriptase Polymerase Chain ReactionGlutathione3. Good healthCell biologyDrosophila melanogasterRNA Interferencemedicine.symptomImmunosuppressive AgentsDrosophila ProteinResearch ArticleAtaxiaLongevityMotor ActivityBiologyAconitase03 medical and health sciencesmedicineAnimalsHumans030304 developmental biologySirolimusAldehydesSuperoxide Dismutaselcsh:RAutophagyRepressor ProteinsDisease Models AnimalOxidative StressFriedreich AtaxiaFrataxinbiology.proteinlcsh:Q030217 neurology & neurosurgeryOxidative stressTranscription FactorsGenetic screenPLOS ONE
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Acute toxicity of dichlorvos to Aphanius iberus (Cuvier & Valenciennes, 1846) and its anti-cholinesterase effects on this species

2008

This study evaluates the toxic effects of the organophosphate pesticide (OP) dichlorvos to the endangered Iberian toothcarp (Aphanius iberus). To this end, the lethal toxicity of dichlorvos based on 96 h-LC50 bioassays was determined in saline water (50 g/L), and in vivo effects of dichlorvos on cholinesterase (ChE) activity were investigated in adult female and male specimens. The 96 h-LC50 value determined by probit analysis was 3.17 mg/L (95% confidence limits: 1.34-3.97). The characterisation of the ChE using different substrates and specific inhibitors was also carried out in head and muscle tissues. Acetylthiocholine was the substrate preferred by both head and muscle ChE in males and…

Malemedicine.medical_specialtyAchéHealth Toxicology and MutagenesisAquatic ScienceToxicologychemistry.chemical_compoundInternal medicineDichlorvosmedicineAnimalsCholinesterasesCholinesterase (ChE)Cholinesterasebiologyintegumentary systemToxicityKillifishesMusclesOrganophosphateAcetylcholinesteraseSurvival AnalysisAcute toxicitylanguage.human_languageEnzyme assayEndocrinologyFishchemistryAphaniusToxicityDichlorvosbiology.proteinlanguageFemaleCholinesterase InhibitorsWater Pollutants ChemicalBiomarkers
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Hypothermic Oscillating Liver Perfusion Stimulates ATP Synthesis prior to Transplantation

1998

Abstract Background. ATP and glycogen depletion often have been demonstrated during cold storage of the liver prior to transplantation. Suppression of events that lead to metabolic depression and to lipid peroxidation could contribute to improvement of liver preservation. A new method of liver preservation for transplantation is therefore suggested, an oscillating oxygenated hypothermic liver perfusion. Methods. Biochemical analysis of liver tissue samples and perfusate after 10 h of perfusion by the presented oscillating perfusion model were compared with results after continuous liver perfusion for 10 h as well as with data derived from cold-stored livers over a period of 10 h. Particular…

Malemedicine.medical_specialtyAdenosineTime FactorsAllopurinolmedicine.medical_treatmentOrgan Preservation SolutionsCold storageBiologyLiver transplantationchemistry.chemical_compoundAdenosine TriphosphateRaffinoseRats Inbred BNInternal medicinemedicineAnimalsInsulinEnergy chargeLiver preservationMachine perfusionGlycogenOrgan PreservationGlutathioneLiver GlycogenLiver TransplantationRatsCold TemperatureOxygenPerfusionTransplantationEndocrinologyLiverBiochemistrychemistryEvaluation Studies as TopicSurgeryEnergy MetabolismPerfusionJournal of Surgical Research
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Reduced basal and stimulated (isoprenaline, Gpp(NH)p, forskolin) adenylate cyclase activity in Alzheimer's disease correlated with histopathological …

1991

Cyclic adenosine monophosphate (cAMP) is an adenylate cyclase borne second messenger involved in basic metabolic events. The beta-adrenoceptor sensitive adenylate cyclase was studied in post-mortem hippocampi of controls and Alzheimer patients. Virtually identical subsets of each hippocampus homogenate were stimulated by 100 mumol isoprenaline, Gpp(NH)p and forskolin, respectively, in presence of an ATP-regenerating system. The determination of cAMP formed was carried out by means of a radioassay. The observed significant 50% reduction in basal as well as in stimulated adenylate cyclase activity in Alzheimer's disease is negatively correlated with semiquantitative evaluations of amyloid pla…

Malemedicine.medical_specialtyAdenylate kinaseCyclasechemistry.chemical_compoundAlzheimer DiseaseReference ValuesInternal medicineIsoprenalinemedicineHumansCyclic adenosine monophosphateSenile plaquesMolecular BiologyAgedAged 80 and overGuanylyl ImidodiphosphateForskolinChemistryGeneral NeuroscienceColforsinIsoproterenolBrainKineticsEndocrinologyPostmortem ChangesSecond messenger systemFemaleNeurology (clinical)Cyclase activityAdenylyl CyclasesDevelopmental Biologymedicine.drugBrain Research
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Gene Transcription Alterations Associated with Decrease of Ethanol Intake Induced by Naltrexone in the Brain of Wistar Rats

2006

Preclinical and clinical studies suggest that the administration of the opioid antagonist naltrexone decreases the intake of ethanol. However, the neuroplastic adaptations in the brain associated to reduction of ethanol consumption remains to be elucidated. The aim of the study was to identify gene transcription alterations underlying the attenuation of voluntary ethanol intake by administration of naltrexone in rats. Increasing doses of naltrexone (0.7 mg/kg, 4 days and 1.4 mg/kg/day, 4 days) to rats with acquired high preferring ethanol consumption (>3.5 g of ethanol/kg/day) decreased voluntary ethanol intake (50%). Voluntary ethanol consumption altered mu-opioid receptor function in the …

Malemedicine.medical_specialtyAlcohol DrinkingTranscription Geneticmedicine.drug_classNarcotic AntagonistsNucleus accumbensPharmacologyNaltrexoneInternal medicineImage Processing Computer-AssistedmedicineAnimalsRats WistarOpioid peptideIn Situ HybridizationBrain ChemistryPharmacologyEthanolTyrosine hydroxylaseChemistryOlfactory tubercleCentral Nervous System DepressantsEnkephalin Ala(2)-MePhe(4)-Gly(5)-NaltrexoneRatsAnalgesics OpioidVentral tegmental areaPsychiatry and Mental healthmedicine.anatomical_structureEndocrinologynervous systemGuanosine 5'-O-(3-Thiotriphosphate)HypothalamusAutoradiographyOpioid antagonistmedicine.drugNeuropsychopharmacology
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Mildronate, the inhibitor of l-carnitine transport, induces brain mitochondrial uncoupling and protects against anoxia-reoxygenation

2013

Abstract The preservation of mitochondrial function is essential for normal brain function after ischaemia-reperfusion injury. l -carnitine is a cofactor involved in the regulation of cellular energy metabolism. Recently, it has been shown that mildronate, an inhibitor of l -carnitine transport, improves neurological outcome after ischaemic damage of brain tissues. The aim of the present study was to elucidate the mitochondria targeted neuroprotective action of mildronate in the model of anoxia-reoxygenation-induced injury. Wistar rats were treated daily with mildronate ( per os ; 100 mg/kg) for 14 days. The acyl-carnitine profile was determined in the brain tissues. Mitochondrial respirati…

Malemedicine.medical_specialtyBioenergeticsCell RespirationMitochondrionBiologyNeuroprotectionCarnitine transportAdenosine TriphosphateCarnitineInternal medicineRespirationmedicineAnimalsCarnitineRats WistarHypoxiaPharmacologyBrainMetabolismMitochondriaRatsOxygenCitric acid cycleNeuroprotective AgentsEndocrinologyCarnitine AcyltransferasesAcyl Coenzyme AMethylhydrazinesmedicine.drugEuropean Journal of Pharmacology
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Antagonistic effects of hypertrehalosemic neuropeptide on the activities of 6-phosphofructo-1-kinase and fructose-1,6-bisphosphatase in cockroach fat…

2001

Hypertrehalosemic neuropeptides from the corpora cardiaca such as the decapeptide Bld HrTH bring about a profound switch in the metabolic activity of cockroach fat body during which production of the blood sugar trehalose is stimulated while the catabolism of carbohydrate (glycolysis) is inhibited. The mechanisms of the metabolic switch are not fully understood. Incubation of isolated fat body from the cockroach Blaptica dubia with 10(-8) M Bld HrTH, for 10-60 min, stimulated glycogen breakdown and increased the content of the substrates of both the glycolytic enzyme 6-phosphofructo-1-kinase (PFK, EC 2.7.1.11) and the gluconeogenic enzyme fructose-1,6-bisphosphatase (FBPase, EC 3.1.3.11) in…

Malemedicine.medical_specialtyBlaptica dubiaPhosphofructokinase-1Fat BodyFructose 16-bisphosphataseCockroachesIn Vitro TechniquesBiologyBiochemistryGene Expression Regulation Enzymologicchemistry.chemical_compoundInternal medicineFructosediphosphatesmedicineAnimalsGlycolysisPhosphofructokinase 1Molecular BiologyCatabolismNeuropeptidesTrehaloseFructosebiology.organism_classificationAdenosine MonophosphateFructose-BisphosphataseKineticsEndocrinologyFructose 26-bisphosphatechemistryBiochemistryInsect HormonesInsect Sciencebiology.proteinGlycogenPhosphofructokinaseInsect Biochemistry and Molecular Biology
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The serum level of free testosterone is reduced in amyotrophic lateral sclerosis

2002

Sporadic amyotrophic lateral sclerosis (ALS) is a progressive neurodegenerative disorder affecting upper and lower motoneurons. There is an approximately 2:1 higher incidence of ALS in men compared to women, and this has raised the hypothesis of an involvement of sex hormones in the etiopathogenesis of the disorder. In this work, the serum levels of dehydroepiandrosterone sulphate (DHEAS), 17-betaestradiol, free and total testosterone were measured in 35 patients with defined or probable ALS, according to the El-Escorial/WFN revisited criteria, and compared to those obtained from 57 disease controls, matched for age and gender to the ALS group. We found no differences between ALS cases and …

Malemedicine.medical_specialtyCentral nervous system diseaseDehydroepiandrosterone sulphateDegenerative diseaseSex hormone-binding globulinInternal medicinemedicineHumans17-βestradiol; Amyotrophic lateral sclerosis; Dehydroepiandrosterone sulphate; Motoneurons; Sex hormone binding globulin; TestosteroneTestosteroneAmyotrophic lateral sclerosisAged17-βestradiolAged 80 and overSex Characteristicsbiologybusiness.industryTestosterone (patch)Middle Agedmedicine.diseaseAmyotrophic lateral sclerosisPathophysiologySex hormone binding globulinMotoneuronsEndocrinologyNeurologybiology.proteinFemaleNeurology (clinical)businessSex characteristicsHormone
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