Search results for "hereditary"

showing 10 items of 650 documents

Hereditary motor sensory neuropathy type II with neurofilament accumulation: new finding or new disorder?

1985

Peroneal muscular atrophy is now known to be heterogeneous and to be due to various underlying genetic mechanisms. Exploring this heterogeneity further, we report on a German kinship with the clinical, genetic, and nerve conduction features of hereditary motor and sensory neuropathy type II (HMSN type II) but whose sural nerves on biopsy were found to show infrequent axonal swellings with neurofilament accumulations not previously described. The dominant inheritance and absence of kinky hair set this disorder apart from giant axonal neuropathy. There was no history of toxic exposure to industrial chemicals. We conclude that the disorder either is a new type of HMSN or is HMSN type II with p…

AdultMaleNeurofilamentAdolescentBiopsyCardiomyopathyNeural ConductionCardiovascular System03 medical and health sciences0302 clinical medicineSural NerveEvoked Potentials SomatosensoryBiopsymedicineHumansAxonHereditary Sensory and Autonomic NeuropathiesChildCytoskeleton030304 developmental biologyGiant axonal neuropathyAged0303 health sciencesmedicine.diagnostic_testbusiness.industryElectromyographyPeroneal muscular atrophyMiddle Agedmedicine.diseaseAxonsPedigreeMicroscopy ElectronMuscular Atrophymedicine.anatomical_structureNeurologyEvoked Potentials VisualFemaleNeurology (clinical)AbnormalityHereditary motor and sensory neuropathybusinessNeuroscience030217 neurology & neurosurgeryAnnals of neurology
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High prevalence of BRCA1 deletions in BRCAPRO-positive patients with high carrier probability.

2007

Mutation screening of the BRCA1 and BRCA2 genes in probands with familial breast/ovarian cancer has been greatly improved by the multiplex ligation-dependent probe amplification (MLPA) assay able to evidence gene rearrangements not detectable by standard screening methods. However, no criteria for selection of cases to be submitted to the MLPA test have been reported yet. We used the BRCAPro software for the selection of familial breast/ovarian cancer probands investigated with the MLPA approach after negative BRCA1/2 conventional mutation screening. One hundred and seventy-seven probands were investigated for germline BRCA1/2 mutations after assessment of genetic risk using BRCAPro. Proban…

AdultMaleOncologyProbandcongenital hereditary and neonatal diseases and abnormalitiesmedicine.medical_specialtyendocrine system diseasesBreast NeoplasmsGermlineBreast Neoplasms MaleGermline mutationBreast cancerRisk FactorsInternal medicinePrevalenceHumansMedicineGenetic Predisposition to DiseaseMultiplexMultiplex ligation-dependent probe amplificationskin and connective tissue diseasesAgedSequence DeletionOvarian NeoplasmsGeneticsBRCA1 Proteinbusiness.industryGenetic Carrier ScreeningProstatic NeoplasmsHematologyMiddle Agedmedicine.diseaseBRCA1 BRCA2 BRCAPro breast cancer MLPA ovarian cancerPedigreeOncologyMutation (genetic algorithm)FemalebusinessOvarian cancerSoftware
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Somatic loss of an EXT2 gene mutation during malignant progression in a patient with hereditary multiple osteochondromas

2015

Multiple osteochondromas (MO) is an autosomal-dominant skeletal disorder caused by mutations in the exostosin-1 ( EXT1 ) or exostosin-2 ( EXT2 ) genes. In this study, we report the analysis of the mutational status of the EXT2 gene in tumor samples derived from a patient affected by hereditary MO, documenting the somatic loss of the germline mutation in a giant chondrosarcoma and in a rapidly growing osteochondroma. The sequencing of all exons and exon–intron junctions of the EXT1 and EXT2 genes from blood DNA of the proband did not reveal any mutation in the EXT1 gene but did demonstrate the presence of the transition point mutation c.67C > T in the EXT2 gene, determining the introduction …

AdultMaleOsteochondromaCancer ResearchMultiple osteochondromaSettore MED/06 - Oncologia MedicaChondrosarcomaLoss of HeterozygositySettore BIO/11 - Biologia MolecolareBone NeoplasmsGene mutationBiologyN-Acetylglucosaminyltransferasesmedicine.disease_causeGermlineLoss of heterozygosityGermline mutationGeneticChondrosarcoma; Hereditary cancer; Hereditary multiple osteochondromas; Tumor suppressor gene; Molecular Biology; Genetics; Cancer ResearchSkeletal disorderGeneticsmedicineHumansTumor suppressor geneHereditary multiple osteochondromaMolecular BiologyGeneticsMutationChromosomes Human Pair 11DNA Neoplasmmedicine.diseaseHereditary cancerSettore MED/18 - Chirurgia GeneraleSettore MED/03 - Genetica MedicaMutationDisease ProgressionCancer Genetics
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Long-term outcomes with subcutaneous C1-inhibitor replacement therapy for prevention of hereditary angioedema attacks

2019

Background For the prevention of attacks of hereditary angioedema (HAE), the efficacy and safety of subcutaneous human C1-esterase inhibitor (C1-INH[SC]; HAEGARDA, CSL Behring) was established in the 16-week Clinical Study for Optimal Management of Preventing Angioedema with Low-Volume Subcutaneous C1-Inhibitor Replacement Therapy (COMPACT). Objective To assess the long-term safety, occurrence of angioedema attacks, and use of rescue medication with C1-INH(SC). Methods Open-label, randomized, parallel-arm extension of COMPACT across 11 countries. Patients with frequent angioedema attacks, either study treatment-naive or who had completed COMPACT, were randomly assigned (1:1) to 40 IU/kg or …

AdultMalePediatricsmedicine.medical_specialtyAdolescentInjections SubcutaneousAttack rateC1-inhibitor03 medical and health sciencesYoung Adult0302 clinical medicinemedicineLong term outcomesImmunology and AllergyHumans030212 general & internal medicineddc:610Adverse effectChildAgedbiologyAngioedemabusiness.industryIncidence (epidemiology)Angioedemas HereditaryMiddle Agedmedicine.diseaseOptimal managementTreatment Outcome030228 respiratory systemHereditary angioedemabiology.proteinFemalemedicine.symptombusinessComplement C1 Inhibitor Protein
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A nationwide survey of hereditary angioedema due to C1 inhibitor deficiency in Italy

2014

Introduction Hereditary angioedema due to C1-inhibitor deficiency (C1-INH-HAE type I) or dysfunction (C1-INH-HAE type II) is a rare disease characterized by recurrent episodes of edema with an estimated frequency of 1:50,000 in the global population without racial or gender differences. In this study we present the results of a nationwide survey of C1-INH-HAE patients referring to 17 Italian centers, the Italian network for C1-INH-HAE, ITACA. Methods Italian patients diagnosed with C1-INH-HAE from 1973 to 2013 were included in the study. Diagnosis of C1-INH-HAE was based on family and/or personal history of recurrent angioedema without urticaria and on antigenic and/or functional C1-INH def…

AdultMalePediatricsmedicine.medical_specialtyAdolescentPopulationNationwide surveyC1-inhibitorYoung AdultmedicineHumansGenetics(clinical)C1 inhibitor; C4; Hereditary angioedema; Adolescent; Adult; Angioedemas Hereditary; Female; Humans; Italy; Male; Middle Aged; Young Adult; Medicine (all); Genetics (clinical); Pharmacology (medical)Pharmacology (medical)Young adulteducationGenetics (clinical)C4Medicine(all)Hereditary angioedemaeducation.field_of_studyC1 inhibitorAngioedemabiologybusiness.industryResearchMedicine (all)Angioedemas HereditaryAngioedemasGeneral MedicineMiddle Agedbacterial infections and mycosesmedicine.diseaseSettore MED/16 - ReumatologiaHereditaryItalyHereditary angioedemabiology.proteinFemalemedicine.symptomDifferential diagnosisbusinessRare diseaseOrphanet Journal of Rare Diseases
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Asphyxiation by laryngeal edema in patients with hereditary angioedema.

2000

Objective To describe the occurrence of fatal laryngeal edema in patients with hereditary angioedema due to C1 esterase inhibitor deficiency. Patients and Methods We describe 6 patients from various regions of Germany who died from laryngeal edema within the last 10 years. Furthermore, we conducted a retrospective survey of 58 patients with hereditary angioedema, originating from 46 affected families. The data were obtained from the attending physicians and from the patients' relatives. Results Among the 6 reported patients, aged 9 to 78 years, hereditary angioedema had been diagnosed in 3 and was undiagnosed in 3. None of them had an emergency cricothyrotomy or received C1 inhibitor concen…

AdultMalePediatricsmedicine.medical_specialtyAdolescentmedicine.medical_treatmentComplement C1 Inactivator ProteinsLaryngeal EdemaC1-inhibitorLaryngeal DiseasesEcallantideAsphyxiaEdemamedicineHumansCricothyrotomyAngioedemaChildAgedRetrospective StudiesFirst episodeAsphyxiabiologybusiness.industryGeneral MedicineMiddle Agedmedicine.diseaseAnesthesiaHereditary angioedemabiology.proteinFemalemedicine.symptombusinessmedicine.drugMayo Clinic proceedings
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Helicobacter pylori Infection as a Triggering Factor of Attacks in Patients with Hereditary Angioedema

2007

  Udgivelsesdato: 2007-Jun BACKGROUND: Helicobacter pylori infection is considered among the causative factors of urticaria and angioedema. Having conducted a study on 65 patients, Hungarian authors reported in 2001 that successful eradication of H. pylori is followed by a significant reduction in the number of attacks in patients with hereditary angioedema (HAE). The present study aimed to reinvestigate the relationship between H. pylori infection and the attack rate in the framework of an international collaborative study. MATERIALS AND METHODS: Within the framework of the PREHAEAT project launched by the European Union, further 152 patients were studied in seven collaborating centers, an…

AdultMalePeptic UlcerHelicobacter pylori infectionmedicine.medical_specialtyAdolescentImmunologyAttack ratemacromolecular substancesHelicobacter InfectionsDisease severityInternal medicinemedicineImmunology and AllergyHumansmedia_common.cataloged_instanceIn patientAttack frequencyAngioedemaEuropean unionChildAgedmedia_commonClinical Trials as TopicHelicobacter pyloribiologyAngioedemabusiness.industryGastroenterologyGeneral MedicineMiddle AgedHelicobacter pylorimedicine.diseasebiology.organism_classificationInfectious DiseasesChild PreschoolGastritisHereditary angioedemaImmunologyFemalemedicine.symptombusinessHelicobacter
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Gastric outlet obstruction in a neonate because of Peutz-Jeghers syndrome

2012

Neonatal detection of Peutz-Jeghers syndrome is unusual with only 2 cases previously reported in the literature. We describe a neonate presenting with gastric outlet obstruction owing to 2 large Peutz-Jeghers polyps. The child's father and grandmother were known to have Peutz-Jeghers syndrome. On the ninth day of life, the infant underwent colonoscopy, abdominal exploration, and complete surgical resection of 3 polyps. The postoperative course was uneventful, and the patient was discharged home at the age of 3 weeks on full oral feeds. This is the first case report of inherited Peutz-Jeghers syndrome causing gastric outlet obstruction in a neonate.

AdultMalePolyhydramniosSurgical resectioncongenital hereditary and neonatal diseases and abnormalitiesmedicine.medical_specialtyDay of lifePeutz-Jeghers SyndromeColonoscopyPeutz–Jeghers syndromePolypsPregnancyStomach NeoplasmsAbdominal explorationmedicineHumansskin and connective tissue diseasesmedicine.diagnostic_testGastric Outlet Obstructionbusiness.industryInfant NewbornGastric outlet obstructionGeneral Medicinemedicine.diseasedigestive system diseasesSurgeryPediatrics Perinatology and Child HealthFemaleSurgerybusinessJournal of Pediatric Surgery
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A new germline mutation in BRCA1 gene in a sicilian family with ovarian cancer

2005

A group of 103 sicilian patients with hereditary and familiar breast and/or ovarian cancer were screened for Breast Cancer 1 gene (BRCA1) mutations by direct sequencing PCR products spanning the coding region and partial intronic regions of the BRCA1 gene. In this study, we report a new germline mutation in BRCA1 gene, not previously reported in the BIC database, in a woman with ovarian cancer at 46 years old. Mother's proband has been diagnosed the same histotype of ovarian cancer at 42 age. The mutational analyses that shown a 4843delC frameshift mutation in exon 16 of BRCA1 gene was extended to other family members including the proband's brother and her two sons. Direct automatic sequen…

AdultMaleProbandcongenital hereditary and neonatal diseases and abnormalitiesCancer Researchendocrine system diseasesGenetic counselingCystadenocarcinomaGenes BRCA1BiologyFrameshift mutationExonGermline mutationBreast cancermedicineHumansGenetic Predisposition to DiseaseFrameshift MutationSicilyGerm-Line MutationOvarian NeoplasmsBRCA1 Direct automatic sequencing Germline mutation Ovarian cancerGeneticsMiddle Agedmedicine.diseasePedigreeOncologyMutation (genetic algorithm)Cancer researchFemaleOvarian cancerBreast Cancer Research and Treatment
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Benefits and risks of danazol in hereditary angioedema: a long-term survey of 118 patients

2008

Background Hereditary angioedema (HAE) due to C1 inhibitor deficiency is clinically characterized by relapsing skin swellings, abdominal pain attacks, and life-threatening upper airway obstruction. Treatment with androgens prevents attacks for those with this condition. Objective To examine the benefits and risks of long-term treatment with danazol. Methods Data were generated retrospectively from 118 German and Danish patients who had HAE due to C1 inhibitor deficiency and were treated with danazol from 2 months to 30 years. The frequency and severity of acute attacks were registered before and during danazol treatment, and adverse effects to the treatment were noted. Data were collected b…

AdultMalePulmonary and Respiratory Medicinemedicine.medical_specialtyAbdominal painPediatricsImmunologyDose-Response Relationship ImmunologicRisk AssessmentSeverity of Illness IndexEcallantideSeverity of illnessmedicineHumansImmunology and AllergyAdverse effectDepression (differential diagnoses)Retrospective StudiesDanazolbusiness.industryDanazolAngioedemas HereditaryEstrogen AntagonistsMiddle Agedmedicine.diseaseSurgeryDiscontinuationHereditary angioedemaFemalemedicine.symptombusinessFollow-Up Studiesmedicine.drugAnnals of Allergy, Asthma & Immunology
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