Search results for "herpes"

showing 10 items of 307 documents

Suppression of humoral immune response against herpes simplex virus induced by defective strains, ts- and TK- mutants.

1988

Suppression of humoral antibody formation against HSV is not only induced by replicating Herpes simplex virus type 2 (HSV-2) but also by the defective strain ANG and the deletion mutant 1301 of Herpes simplex virus type 1 (HSV-1). Moreover, ts-mutants A, H, K, S, 1201 and 1208 of HSV-1 as well as some ts-mutants of HSV-2 and “defective-interfering” particles of HSV-1 after high multiplicity of infection-passages induced suppression. Treatment of infected mice with ACG reduced antibody-formation but did not result in suppression. UV-irradiation of the antibody producing strain Len of HSV-1 strongly reduces antibody formation and induces suppression. Experiments using a series of intertypic r…

Deletion mutantGenes ViralvirusesMutantBiologymedicine.disease_causeAntibodies ViralVirus ReplicationGenomeThymidine KinaseMiceImmune systemVirologyViral InterferencemedicineImmune ToleranceAnimalsSimplexvirusRecombination GeneticDefective VirusesGeneral MedicineVirologyHerpes simplex virusHumoral immunityMutationbiology.proteinViral diseaseAntibodyArchives of virology
researchProduct

The broad-spectrum antiinfective drug artesunate interferes with the canonical nuclear factor kappa B (NF-κB) pathway by targeting RelA/p65.

2015

Infection with human cytomegalovirus (HCMV) is a serious medical problem, particularly in immunocompromised individuals and neonates. The success of standard antiviral therapy is hampered by low drug compatibility and induction of viral resistance. A novel strategy is based on the exploitation of cell-directed signaling inhibitors. The broad antiinfective drug artesunate (ART) offers additional therapeutic options such as oral bioavailability and low levels of toxic side-effects. Here, novel ART-derived compounds including dimers and trimers were synthesized showing further improvements over the parental drug. Antiviral activity and mechanistic aspects were determined leading to the followi…

DrugHuman cytomegalovirusTranscriptional Activationmedia_common.quotation_subjectTranscription Factor RelAArtesunateCytomegalovirusPharmacologyCREBAntiviral Agentschemistry.chemical_compoundVirologyDrug Resistance ViralmedicineHumansCyclic AMP Response Element-Binding ProteinHerpesviridaemedia_commonPharmacologybiologyHEK 293 cellsNF-kappa BTranscription Factor RelANF-κBmedicine.diseaseIn vitroArtemisininsUp-RegulationHEK293 CellschemistryMutationbiology.proteinSignal transductionSignal TransductionAntiviral research
researchProduct

Analysis of the TK enzyme complex induced by HSV types 1 and 2 by means of isoelectric focusing and polyacrylamide gel electrophoresis.

1982

Recently we have described that the Herpes simplex virus (HSV)-induced thymidine kinase (TK) induces AMP- and ADP-dThd-5'-phosphotransferase activities. We now demonstrate the heterogeneity of the described activities in isoelectric focusing experiments and polyacrylamide gel electrophoresis. A TK--mutant of HSV type 1 fails to induce these activities. The activities of the type 1 enzyme complex was neutralized by an anti-HSV-serum. The TK-enzyme complex expressed in LTK--cells transformed to a TK+-phenotype by sheared HSV-1 DNA was compared with the wild type TK complex in isoelectric focusing experiments. Additionally we demonstrate that the HSV type 1 enzyme complex has thymidylate kinas…

Enzyme complexvirusesThymidylate kinase activityBiologymedicine.disease_causeThymidine KinaseFeedbackchemistry.chemical_compoundViral ProteinsVirologymedicineSimplexvirusThymine NucleotidesPolyacrylamide gel electrophoresisIsoelectric focusingWild typeGeneral MedicineVirologyMolecular biologyHerpes simplex virusBiochemistrychemistryThymidine kinaseElectrophoresis Polyacrylamide GelIsoelectric FocusingDNAArchives of virology
researchProduct

Frequency of CD8+ T Lymphocytes Specific for Lytic and Latent Antigens of Epstein–Barr Virus in Healthy Virus Carriers

1999

Abstract We investigated CD8 + T cell frequencies of five different Epstein–Barr virus-specific cytotoxic T lymphocyte epitopes located within proteins of the replicative cycle and the latent state in healthy long-term virus carriers with IFN-γ enzyme-linked immunospot assay. Frequencies of the HLA-A3-restricted epitope RVRAYTYSK (RVR) whose minimal length was mapped in this study to amino acid position 148–156 of the immediate-early protein BRLF1 were compared with those of a further known HLA-A3-restricted epitope within EBNA3A, RLRAEAQVK (RLR). Determination of frequencies of CD8 + T lymphocytes directed against lytic antigen epitope RVR revealed that only one of eight donors recognized …

Epstein-Barr Virus InfectionsHerpesvirus 4 HumanvirusesT cellEpitopes T-LymphocyteCD8-Positive T-LymphocytesBiologymedicine.disease_causeVirusEpitopeCell LineImmediate-Early ProteinsViral ProteinsAntigenVirologymedicineHumansCytotoxic T cellHematopoietic Stem CellsEpstein–Barr virusVirologyMolecular biologyBZLF1medicine.anatomical_structureEpstein-Barr Virus Nuclear AntigensCarrier StateTrans-ActivatorsCD8T-Lymphocytes CytotoxicVirology
researchProduct

Suppression of humoral antibody formation against sheep red blood cells by infections with HSV-2 and the influence of mouse cytomegalovirus

1987

HSV-2 infections suppress the antibody response to HSV-1 but do not impair the mouse cytomegalo virus (MCMV) antibody generating system. In contrast, the sheep red blood cell (SRBC) IgM response is impaired by preinfections with HSV-2 but not with HSV-1. From the time kinetics of this suppression it can be concluded that only a certain submechanism "spills over" to the SRBC system. MCMV suppresses the antibody formation induced by HSV-1.

ErythrocytesTime FactorsvirusesCytomegalovirusAntibodies Viralmedicine.disease_causeVirusHerpesviridaeImmune toleranceBlood cellMiceVirologyAlphaherpesvirinaeImmune TolerancemedicineAnimalsSimplexvirusbiologyHerpes SimplexGeneral Medicinebiology.organism_classificationVirologyRed blood cellmedicine.anatomical_structureImmunoglobulin MAntibody FormationImmunologyHumoral immunitybiology.proteinAntibodyArchives of Virology
researchProduct

Replication of HSV-1 in murine peritoneal macrophages: comparison of various virus strains with different properties.

1984

The in vitro replication of eleven different strains of herpes simplex virus type 1 was studied in resident or thioglycollate-stimulated mouse macrophages. The strains of herpes simplex virus differed in the type of cytopathic effect, induction capacity for herpes simplex virus coded thymidine kinase and pathogenicity in the mouse. Herpes simplex virus replicated better in thioglycollate-stimulated macrophages than in resident macrophages. In vitro ageing of macrophages increased their replicative potency. Herpes simplex virus replicated better in macrophages from homozygous bg/bg C57/BL6J mice than in macrophages from their heterozygous littermates. Separation of macrophages on discontinuo…

ErythrocytesvirusesClone (cell biology)Mice Inbred StrainsBiologymedicine.disease_causeVirus ReplicationThymidine KinaseHerpesviridaeVirusMicrobiologychemistry.chemical_compoundMiceCytopathogenic Effect ViralPhagocytosisVirologymedicineMacrophageAnimalsAscitic FluidSimplexvirusCells CulturedCytopathic effectMacrophagesGeneral MedicineMacrophage ActivationVirologyMice Inbred C57BLHerpes simplex viruschemistryThymidine kinaseEnzyme InductionThymidineArchives of virology
researchProduct

Correlation between HHV-6 reactivation and multiple sclerosis disease activity.

2002

This study examined the association between HHV-6 infection and multiple sclerosis (MS) and the relationship between HHV-6 reactivation and disease activity. The frequency of HHV-6 genomic sequences in peripheral blood mononuclear cells (PBMCs), the incidence of plasma viremia (nPCR), the transcription of viral mRNA in PBMCs (RT-PCR), the presence of antiviral IgM and IgG class antibodies in the plasma (IFA) of 16 relapsing/remitting and secondary progressive MS patients were studied in comparison with clinical manifestations of the disease, magnetic resonance imaging (MRI) of brain, and serum interleukin (IL)-12 concentrations (ELISA). The prevalence of HHV-6 infection was significantly hi…

ExacerbationvirusesHerpesvirus 6 HumanViremiaEnzyme-Linked Immunosorbent Assaymedicine.disease_causePeripheral blood mononuclear cellHerpesviridaeCentral nervous system diseaseMultiple Sclerosis Relapsing-RemittingVirologymedicineHumansCells Culturedbiologybusiness.industryReverse Transcriptase Polymerase Chain ReactionMultiple sclerosisvirus diseasesInterleukinHerpesviridae Infectionsmedicine.diseaseVirologyInfectious DiseasesImmunoglobulin MImmunoglobulin GImmunologybiology.proteinLeukocytes MononuclearVirus ActivationAntibodybusinessJournal of medical virology
researchProduct

Rapid development of Epstein-Barr virus-associated Hodgkin's disease after cessation of foscarnet therapy in an HIV-infected patient.

2000

Epidemiological features suggest a link between Hodgkin's disease (HD) and Epstein-Barr virus (EBV) infection1. Indeed, EBV genome and expression of latent antigens can be found in Reed-Sternberg cells. In the majority of cases HD in HIV patients seems to be EBV-associated. We report on a 51-year-old HIV-infected patient in whom EBV-positive HD of mixed cellularity rapidly developed within one month after cessation of treatment with intravenous foscarnet.

FoscarnetMaleEpstein-Barr Virus InfectionsHIV InfectionsDermatologymedicine.disease_causeAntiviral AgentsVirusHerpesviridaehemic and lymphatic diseasesMedicineHumansPharmacology (medical)Sidabiologybusiness.industryPublic Health Environmental and Occupational HealthMiddle Agedmedicine.diseasebiology.organism_classificationEpstein–Barr virusVirologyHodgkin DiseaseLymphomaInfectious DiseasesFoscarnet SodiumImmunologyViral diseasebusinessmedicine.drugFoscarnetInternational journal of STDAIDS
researchProduct

Comparison of the genotoxic and apoptosis-inducing properties of ganciclovir and penciclovir in Chinese hamster ovary cells transfected with the thym…

2000

We studied the genotoxic and apoptosis-inducing properties of ganciclovir (GCV) and penciclovir (PCV) using Chinese hamster ovary cells stably transfected with the thymidine kinase (tk) gene of herpes simplex virus-1 (HSV-1). Cells expressing HSVtk were 300 and 100 times more sensitive than their isogenic HSVtk- counterparts to the cytotoxic effects of GCV and PCV, respectively. Using radiolabeled drugs, GCV was found to be incorporated into the genomic DNA much more effectively than PCV. GCV was highly potent in inducing chromosomal aberrations compared with PCV, which provoked less sister chromatid exchanges and chromosomal changes using equimolar or equitoxic doses. For both agents, apop…

GanciclovirDNA ReplicationCancer ResearchGuaninevirusesAcyclovirApoptosisCHO CellsHerpesvirus 1 HumanBiologymedicine.disease_causeTransfectioncomplex mixturesThymidine KinaseNecrosisCricetinaemedicineAnimalsMolecular BiologyGanciclovirMutagenicity TestsChinese hamster ovary cellCell CycleDNAGenetic TherapySuicide geneCell cycleMolecular biologyCell killingThymidine kinasePenciclovirMolecular MedicineSister Chromatid ExchangeGenotoxicitymedicine.drugCancer gene therapy
researchProduct

Comparative analysis of DNA breakage, chromosomal aberrations and apoptosis induced by the anti-herpes purine nucleoside analogues aciclovir, gancicl…

2002

Nucleoside analogues have been used in antiviral therapy and suicide cancer gene therapy. Therefore, it is of importance to compare their potential cytotoxic and genotoxic action. Using metabolically competent CHO cells expressing the thymidine kinase gene of herpes simplex virus type 1 (CHO-HSVtk cells) as a model system, the induction of DNA breaks was compared with the induction of structural chromosomal aberrations and apoptosis/necrosis after exposure to the anti-herpes nucleoside analogues aciclovir (ACV), ganciclovir (GCV) and penciclovir (PCV). After continuous treatment of CHO-HSVtk cells with the drugs, LD(10) in a colony-forming assay was 50, 0.5 and 1 microM for ACV, GCV and PCV…

GanciclovirGuanineDNA damagevirusesHealth Toxicology and MutagenesisAcyclovirApoptosisCHO CellsBiologymedicine.disease_causeAntiviral AgentsThymidine KinaseChromosomesColony-Forming Units AssayNecrosisCricetulusCricetinaeGeneticsmedicineCytotoxic T cellAnimalsSimplexvirusAciclovirEnzyme InhibitorsMolecular BiologyGanciclovirChromosome AberrationsDNAMolecular biologyHerpes simplex virusApoptosisPenciclovirNucleosidemedicine.drugDNA DamageMutation research
researchProduct