Search results for "immune disease"

showing 10 items of 359 documents

Disease-Modifying Therapies and Coronavirus Disease 2019 Severity in Multiple Sclerosis

2021

Objective: This study was undertaken to assess the impact of immunosuppressive and immunomodulatory therapies on the severity of coronavirus disease 2019 (COVID-19) in people with multiple sclerosis (PwMS). Methods: We retrospectively collected data of PwMS with suspected or confirmed COVID-19. All the patients had complete follow-up to death or recovery. Severe COVID-19 was defined by a 3-level variable: mild disease not requiring hospitalization versus pneumonia or hospitalization versus intensive care unit (ICU) admission or death. We evaluated baseline characteristics and MS therapies associated with severe COVID-19 by multivariate and propensity score (PS)-weighted ordinal logistic mod…

Male0301 basic medicineDimethyl FumarateNeurodegenerativemultiple sclerosis; coronavirus; pneumoniaSeverity of Illness Indexlaw.inventionImmunosuppressive AgentImmunologic Factor0302 clinical medicineNatalizumablawMonoclonalMultiple Sclerosi80 and overLungHumanizedResearch ArticlesAged 80 and overNatalizumabMiddle AgedIntensive care unitHospitalizationSettore MED/26 - NEUROLOGIAIntensive Care UnitsNeurologyMethylprednisoloneNeurologicalPneumonia & InfluenzaInterferonFemaleImmunosuppressive AgentsResearch ArticleHumanmedicine.drugAdultmedicine.medical_specialtyMusc-19 Study GroupMultiple SclerosisAdolescentClinical SciencesIntensive Care UnitClinical NeurologySettore MED/26Antibodies Monoclonal HumanizedAutoimmune DiseaseAntibodiesYoung Adult03 medical and health sciencesClinical ResearchInternal medicineSeverity of illnessmedicineHumansImmunologic FactorsMortalityAdolescent; Adult; Aged; Aged 80 and over; Antibodies Monoclonal Humanized; COVID-19; Dimethyl Fumarate; Female; Fingolimod Hydrochloride; Hospitalization; Humans; Immunologic Factors; Immunosuppressive Agents; Intensive Care Units; Interferons; Male; Middle Aged; Mortality; Multiple Sclerosis; Natalizumab; SARS-CoV-2; Severity of Illness Index; Young AdultAgedNeurology & NeurosurgeryExpanded Disability Status ScaleFingolimod HydrochlorideSARS-CoV-2business.industryMultiple sclerosisNeurosciencesCOVID-19PneumoniaOdds ratiomedicine.diseaseBrain DisordersGood Health and Well Being030104 developmental biologyOcrelizumabInterferonsNeurology (clinical)business030217 neurology & neurosurgery
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Regulatory T cell deficient scurfy mice exhibit a Th2/M2-like inflammatory response in the skin

2017

Abstract Background Scurfy mice have a functional defect in regulatory T cells (Treg), which leads to lethal multi-organ inflammation. The missing Treg function results in uncontrolled autoimmune cellular and humoral inflammatory responses. We and others have previously shown that during the course of disease scurfy mice develop severe skin inflammation and autoantibodies including anti-nuclear autoantibodies (ANA). Objective Autoimmune skin inflammation and ANA are hallmarks for the diagnosis of autoimmune connective tissue diseases; therefore we analyzed scurfy mice for typical signs of these diseases. Methods Indirect immunofluorescence was used to specify the ANA pattern in scurfy mice.…

Male0301 basic medicinePathologymedicine.medical_specialtyRegulatory T cellCD3Fluorescent Antibody TechniqueConnective tissueDermatitisInflammationDermatologyT-Lymphocytes RegulatoryBiochemistrySclerodermaAutoimmune DiseasesMice03 medical and health sciencesMixed connective tissue diseaseFibrosismedicineAnimalsMolecular BiologySkinCell NucleusScleroderma SystemicTissue Inhibitor of Metalloproteinase-1biologybusiness.industryMacrophagesAutoantibodyForkhead Transcription FactorsMacrophage ActivationFlow Cytometrymedicine.diseaseFibrosisUp-Regulation030104 developmental biologymedicine.anatomical_structureAntibodies AntinuclearImmunologybiology.proteinCytokinesFemaleCollagenmedicine.symptombusinessJournal of Dermatological Science
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ATP4A autoimmunity and Helicobacter pylori infection in children with type 1 diabetes

2014

Summary Persistent presence of ATP4A autoantibodies (ATP4AA) directed towards parietal cells is typical for atrophic body gastritis (ABG), an autoimmune disease associated with type 1 diabetes. We assessed whether Helicobacter pylori (Hp) infection might be associated with positivity for ATP4AA in children with type 1 diabetes. Sera were collected from 70 (38♀) type 1 diabetes children [aged 13·2 ± 4·5 years, age at diagnosis 8·8 ± 4·3 years, diabetes duration 4·5 ± 3·8 years, mean HbA1c 7·8 ± 1·6% (62 ± 17·5 mmol/mol)] seen at the regional diabetes clinic in Katowice, Poland. Patients were tested concurrently for Hp infection by means of a 13C urea breath test. ATP4AA were measured using a…

MaleAdolescentAutoimmune Gastritisautoantibodiestype 1 diabetesImmunologyAutoimmunityautoimmune gastritismedicine.disease_causeparietal cellAutoimmunityAutoimmune DiseasesHelicobacter InfectionsH(+)-K(+)-Exchanging ATPaseYoung AdultSex FactorsmedicineImmunology and AllergyHumansChildAutoimmune diseaseType 1 diabetesbiologyHelicobacter pyloribusiness.industryAutoantibodyOriginal ArticlesHelicobacter pyloribiology.organism_classificationmedicine.diseaseConfidence intervalCross-Sectional StudiesDiabetes Mellitus Type 1Child PreschoolImmunologyFemaleGastritismedicine.symptombusinessClinical and Experimental Immunology
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Immunological Effects of Allopurinol in the Treatment of Experimental Autoimmune Uveitis (EAU) after Onset of the Disease

2003

Purpose Allopurinol reduces oxidative tissue damage and exerts immunomodulating effects in the treatment of experimental autoimmune uveitis (EAU). However, the mechanism of the immunologic pathway remains unclear. In previous studies, treatment was started at the time of immunization. Therefore, whether allopurinol prevents the onset of the disease (i.e., acts in a protective manner) is not known. Methods Sixteen male Lewis rats were used: 6 EAU without therapy [control]; 4 EAU with allopurinol treatment starting 7 days after immunization [AL7]; and 6 EAU with allopurinol treatment starting 11 days after immunization [AL11]. Their sera were tested against Western blots of sodium dodecyl sul…

MaleAllopurinolmedicine.medical_treatmentBlotting WesternAllopurinolmedicine.disease_causeAutoantigensRetinaAutoimmune DiseasesAutoimmunity03 medical and health sciences0302 clinical medicineAdjuvants ImmunologicImmunopathologymedicineAnimalsAutoantibodiesAutoimmune diseaseChemotherapyArrestinbusiness.industryAutoantibodyUveitis PosteriorGeneral Medicinemedicine.diseaseRatsDisease Models AnimalOphthalmologyImmunizationRats Inbred LewImmunology030221 ophthalmology & optometryElectrophoresis Polyacrylamide GelImmunizationbusiness030217 neurology & neurosurgeryUveitismedicine.drugEuropean Journal of Ophthalmology
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Treatment of chronic type B hepatitis with recombinant alpha-interferon induces autoantibodies not specific for autoimmune chronic hepatitis.

1989

Recombinant human alpha-interferon is now under intensive investigation as therapy for chronic Type B hepatitis. Recent reports have suggested that prolonged alpha-interferon therapy may induce autoimmune reactions. We have evaluated the problem of autoimmunity related to alpha-interferon therapy by testing for 15 different antibodies in the sera of 31 patients treated with alpha-interferon. No patient had autoantibodies before treatment; 27 (87%) of 31 patients developed at least one autoantibody. Eleven patients had antinuclear antibodies and 21 had smooth muscle antibodies, both of which usually developed during alpha-interferon therapy. In contrast, antibodies to endocrine organs such a…

MaleAnti-nuclear antibodymedicine.medical_treatmentmedicine.disease_causeAutoimmunityAutoimmune DiseasesHepatitisPrimary biliary cirrhosisAntibody SpecificityEndocrine GlandsMedicineHumansAutoantibodiesHepatitisHepatologybiologybusiness.industryAutoantibodymedicine.diseaseHepatitis BAnti-thyroid autoantibodiesRecombinant ProteinsImmunologyChronic DiseaseInterferon Type Ibiology.proteinThyroglobulinFemaleAntibodybusinessHepatology (Baltimore, Md.)
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Autoimmune hepatitis following hepatitis A virus infection.

1995

A 7-year-old patient is reported who suffered from fatigue and jaundice due to chronic hepatitis. He had acquired hepatitis A virus infection in his community and communicated the disease to his German family 4 weeks later. While the other family members recovered from acute viral hepatitis A, the patient presented 10 weeks after the onset of hyperbilirubinemia (12 mg/dl) with the histology of chronic hepatitis, absence of markers for viral persistence, presence of autoantibodies against smooth muscle (1:320) and the asialoglycoprotein receptor (1:600), and marked hypergammaglobulinemia (3700 mg/dl), leading to the diagnosis of autoimmune hepatitis. The patient received immunosuppressive th…

MaleAsialoglycoproteinsReceptors Cell SurfaceAutoimmune hepatitisAsialoglycoprotein ReceptorAutoimmune DiseasesHepatitisLiver diseaseHypergammaglobulinemiamedicineHumansChildAutoantibodiesHepatitisHepatologybusiness.industryAutoantibodyHypergammaglobulinemiaMuscle SmoothJaundiceHepatitis Amedicine.diseaseImmunologyChronic DiseaseViral diseaseDisease Susceptibilitymedicine.symptomViral hepatitisbusinessJournal of hepatology
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Increased helper cell activity of NZB mice against H-2-identical allogeneic cells.

1988

The T cells of NZB mice become hyperreactive after stimulation with minor histocompatibility (MIH) antigens. This hyperreactivity has previously been demonstrated only for cytotoxic T cells of NZB, although there was some evidence for an increase of their T-helper cell activity facilitating the response. Here we report a quantitative analysis of T-cell help and help of T-cell subpopulations against autologous, MIH, and H-2 antigens in a limiting dilution assay. After stimulation of NZB T cells with autologous and H-2 antigens, the T-helper cell frequencies did not differ from that of normal mice. After stimulation with MIH antigens however, Lyt 1<sup>+</sup>2<sup>+</sup…

MaleCellular immunityImmunologyAntigen-Presenting Cellschemical and pharmacologic phenomenaStimulationMice Inbred StrainsBiologyAutoimmune DiseasesMiceAntigenmedicineImmunology and AllergyCytotoxic T cellAnimalsAutoantibodiesAutoimmune diseaseMice Inbred BALB CMice Inbred NZBH-2 AntigensGeneral MedicineT lymphocyteT-Lymphocytes Helper-Inducermedicine.diseaseHistocompatibilityDisease Models AnimalHumoral immunityImmunologyFemaleInternational archives of allergy and applied immunology
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Complete congenital heart block is associated with increased autoantibody titers against calreticulin.

1996

Complete congenital heart block (CCHB) is associated with anti-Ro/SS-A and anti-La/SS-B antibodies. Calreticulin, a calcium-binding, multi-functional protein of the endoplasmic reticulum with C-terminal KDEL-sequence, is not part of the Ro/SS-A ribonucleoprotein complex. In this study anti-calreticulin autoantibody responses in serum samples from 18 infants with CCHB, their mothers and in a control group of 11 anti-Ro/SS-A or anti-La/SS-B positive infants without heart block and their mothers were analysed. Specific enzyme-linked immunosorbent assays were performed. Nine out of 18 sera with CCHB contained IgG anti-calreticulin antibodies. Four sera of those with IgG antibodies also had IgM …

MaleClinical BiochemistryBlotting WesternEnzyme-Linked Immunosorbent AssayBiochemistryAutoantigensImmunopathologymedicineHumansNeonatal lupus erythematosusAutoantibodiesAutoimmune diseaseLupus erythematosusbiologybusiness.industryCalcium-Binding ProteinsAutoantibodyInfant NewbornGeneral Medicinemedicine.diseaseConnective tissue diseaseHeart BlockImmunoglobulin MRibonucleoproteinsImmunoglobulin GImmunologybiology.proteinFemaleAntibodybusinessCalreticulinCalreticulinEuropean journal of clinical investigation
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Allopurinol has Immunomodulating Activity following Topical and Systemic Application in Experimental Autoimmune Uveitis

2001

PurposeAllopurinol has beneficial effects in the systemic treatment of lens-induced uveitis and experimental autoimmune uveitis (EAU). This is believed to be due to a reduction of oxidative tissue damage through its dose-dependent free radical scavenging ability, and to an immunomodulating effect. The purpose of this study was to demonstrate the immunological effects on the IgG-antibody repertoire in EAU after topical and systemic allopurinol and steroids.MethodsWe assigned 43 male Lewis rats to 6 different groups: healthy rats (BASE, n=3), EAU without therapy (CTRL, n=9); systemic treatment with allopurinol (ALSYS, n=9, 50 mg/kg body wt.i.v., given every three days for two weeks), topical …

MaleDrugmedicine.medical_specialtyAdministration TopicalAllopurinolmedicine.medical_treatmentmedia_common.quotation_subjectBlotting WesternAllopurinol hypersensitivity syndromeAllopurinol030204 cardiovascular system & hematologyPharmacologyAutoantigens030226 pharmacology & pharmacyAutoimmune DiseasesUveitis03 medical and health sciences0302 clinical medicineAdjuvants ImmunologicInternal medicinemedicineAnimalsGlucocorticoidsAutoantibodiesmedia_commonAutoimmune diseaseChemotherapybusiness.industryAutoantibodyFree Radical ScavengersGeneral Medicinemedicine.diseaseRatsDisease Models AnimalOphthalmologyEndocrinologyMethylprednisoloneRats Inbred LewImmunoglobulin GInjections IntravenousElectrophoresis Polyacrylamide GelbusinessUveitismedicine.drugEuropean Journal of Ophthalmology
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Treatment of experimental autoimmune encephalomyelitis with adenylate deaminase from Penicillium lanoso-viride.

2000

The effect of intramuscularly administered immunomodulator, adenylate deaminase (E.C. 3.5.4.6), from Penicillium lanoso-viride on the clinical score of acute experimental autoimmune encephalomyelitis (EAE), a T cell-mediated autoimmune disease, was examined by inoculation of guinea pigs with rabbit brain and spinal cord homogenate (encephalitogen) and complete Freund's adjuvant. Adenylate deaminase (ADA) was effective in delaying the onset of clinical disease. ADA inhibited the severity of EAE. There was a significant decrease in clinical signs. A decrease in the number of morbid and dead animals was observed. Of ADA treated animals, 50-80% developed no clinical manifestations of EAE. The o…

MaleEncephalomyelitis Autoimmune ExperimentalEncephalomyelitisImmunologyGuinea PigsCross Reactionsmedicine.disease_causeInjections IntramuscularAutoimmunityAMP DeaminaseMiceBlood serumAdjuvants Immunologicimmune system diseasesImmunology and AllergyMedicineAnimalsHypersensitivity DelayedComplement ActivationSkin TestsAutoimmune diseaseMice Inbred BALB Cbiologybusiness.industryMultiple sclerosisExperimental autoimmune encephalomyelitisPenicilliumBrainAMP deaminasemedicine.diseaseSpinal CordImmunologybiology.proteinFemaleImmunizationRabbitsAntibodybusiness2'3'-Cyclic-Nucleotide PhosphodiesterasesJournal of autoimmunity
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