Search results for "immunology and allergy"

showing 10 items of 2868 documents

CIMT 2016: Mechanisms of efficacy in cancer immunotherapy — Report on the 14th Annual Meeting of the Association for Cancer Immunotherapy May 10–12 2…

2016

0301 basic medicineOncologymedicine.medical_specialtyCombination therapymedicine.medical_treatmentImmunologyMeeting Reportcombination therapyCell therapy03 medical and health sciencesCancer immunotherapyInternal medicineantibodiestumor microenvironmentImmunology and AllergyMedicinetumor vaccinationPersonalized therapypersonalized therapyPharmacologyTumor microenvironmentcancer immunotherapybusiness.industryCIMTcellular therapy030104 developmental biologyImmunologycheckpoint blockadebusinessHuman Vaccines & Immunotherapeutics
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CIMT 2015: The right patient for the right therapy - Report on the 13th annual meeting of the Association for Cancer Immunotherapy

2015

The 13th Annual Meeting of the Association for Cancer Immunotherapy (CIMT) brought together more than 800 scientists in Mainz, Germany, from May 11–13, 2015, to present and discuss current research...

0301 basic medicineOncologymedicine.medical_specialtyCombination therapymedicine.medical_treatmentImmunologyMeeting Reportcombination therapyCell therapy03 medical and health sciencesCancer immunotherapyInternal medicinemedicineImmunology and Allergytumor microenvironmentPersonalized therapytumor vaccinationPharmacologypersonalized therapyTumor microenvironmentcancer immunotherapybusiness.industryCIMTcellular therapy030104 developmental biologyImmunologybusinessHuman Vaccines & Immunotherapeutics
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CIMT 2017: Anniversary symposium - Report on the 15th CIMT Annual Meeting of the Association for Cancer Immunotherapy

2017

The 15th Annual Meeting of the Association for Cancer Immunotherapy (CIMT) took place May 10–11, 2017, Mainz, Germany during which scientists and CIMT members from all over the world not only celeb...

0301 basic medicineOncologymedicine.medical_specialtyCombination therapymedicine.medical_treatmentImmunologyPhysiologyMeeting Reportcombination therapyCell therapy03 medical and health sciences0302 clinical medicineCancer immunotherapyInternal medicinemedicineImmunology and Allergyantibodiestumor microenvironmentPersonalized therapytumor vaccinationPharmacologypersonalized therapycancer immunotherapybusiness.industryCIMTcellular therapy030104 developmental biology030220 oncology & carcinogenesischeckpoint blockadebusinessHuman Vaccines & Immunotherapeutics
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A catch-22: Interleukin-22 and cancer.

2017

Barrier surfaces of multicellular organisms are in constant contact with the environment and infractions to the integrity of epithelial surfaces is likely a frequent event. Interestingly, components of the immune system, that can be activated by environmental compounds such as the microbiota or nutrients, are interspersed among epithelial cells or directly underlie the epithelium. It is now appreciated that immune cells continuously receive and integrate signals from the environment. Curiously, such continuous reception of stimulation does not normally trigger an inflammatory response but mediators produced by immune cells in response to such signals seem to rather promote barrier integrity…

0301 basic medicineOncologymedicine.medical_specialtymedicine.medical_treatmentImmunologyBiologyPolymorphism Single NucleotideEpitheliumMalignant transformationTight JunctionsInterleukin 2203 medical and health sciences0302 clinical medicineImmune systemInternal medicineNeoplasmsmedicineImmunology and AllergyAnimalsHumansLymphocytesIntestinal MucosaReceptorWound HealingInterleukinsMicrobiotaInnate lymphoid cellEpithelial CellsEpitheliumImmunity InnateCell biology030104 developmental biologyCytokinemedicine.anatomical_structureCell Transformation NeoplasticWound healing030215 immunologyEuropean journal of immunology
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Can Immunogenic Chemotherapies Relieve Cancer Cell Resistance to Immune Checkpoint Inhibitors?

2019

The unprecedented clinical activity of checkpoint blockade in several types of cancers has formally demonstrated that anti-tumor immune responses are crucial in cancer therapy. Durable responses seen in patients treated with immune checkpoint inhibitors (ICI) show that they can trigger the establishment of long-lasting immunologic memory. This beneficial outcome is however achieved for a limited number of patients. In addition, late relapses are emerging suggesting the development of acquired resistances that compromise the anticancer efficacy of ICI. How can this be prevented through combination therapies? We here review the functions of immune checkpoints, the successes of ICI in treating…

0301 basic medicineOrganoplatinum CompoundsImmune checkpoint inhibitorsmedicine.medical_treatmentProgrammed Cell Death 1 ReceptorLeucovorinReviewLymphocyte ActivationchemotherapyimmunomodulationB7-H1 AntigenMice0302 clinical medicineAntineoplastic Agents ImmunologicalcheckpointT-Lymphocyte SubsetsNeoplasmsAntineoplastic Combined Chemotherapy ProtocolsTumor MicroenvironmentImmunology and AllergyCTLA-4 AntigenMolecular Targeted TherapyClinical Trials as TopicLymphokinesDrug Synergism3. Good healthNeoplasm ProteinsFluorouracillcsh:Immunologic diseases. AllergyImmunologyCancer therapyT cells03 medical and health sciencesImmune systemmedicineAnimalsHumanscancerIn patientChemotherapybusiness.industryCancermedicine.diseaseIpilimumabBlockade030104 developmental biologyDrug Resistance NeoplasmCancer cellCancer researchlcsh:RC581-607business030215 immunologyFrontiers in immunology
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IL-33 promotes food anaphylaxis in epicutaneously sensitized mice by targeting mast cells

2016

Background Cutaneous exposure to food allergens predisposes to food allergy, which is commonly associated with atopic dermatitis (AD). Levels of the epithelial cytokine IL-33 are increased in skin lesions and serum of patients with AD. Mast cells (MCs) play a critical role in food-induced anaphylaxis and express the IL-33 receptor ST2. The role of IL-33 in patients with MC-dependent food anaphylaxis is unknown. Objective We sought to determine the role and mechanism of action of IL-33 in patients with food-induced anaphylaxis in a model of IgE-dependent food anaphylaxis elicited by oral challenge of epicutaneously sensitized mice. Methods Wild-type, ST2-deficient, and MC-deficient Kit W-sh/…

0301 basic medicineOvalbuminImmunologyMice TransgenicAdministration CutaneousImmunoglobulin Emedicine.disease_causeArticleDermatitis Atopic03 medical and health sciences0302 clinical medicineAllergenFood allergymedicineAnimalsHumansImmunology and AllergyMast CellsRNA MessengerAnaphylaxisSkinMice Inbred BALB Cbiologybusiness.industryDegranulationAllergensImmunoglobulin EInterleukin-33medicine.diseaseMast cellInterleukin 33Ovalbumin030104 developmental biologymedicine.anatomical_structureImmunologybiology.proteinFemalebusinessFood HypersensitivityAnaphylaxis030215 immunologyJournal of Allergy and Clinical Immunology
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Promises and Pitfalls in the Use of PD-1/PD-L1 Inhibitors in Multiple Myeloma

2018

In the biology of multiple myeloma (MM), immune dysregulation has emerged as a critical component for novel therapeutic strategies. This dysfunction is due to a reduced antigen presentation, a reduced effector cell ability and a loss of reactive T cells against myeloma, together with a bone marrow microenvironment that favors immune escape. The Programmed Death-1 (PD-1) pathway is associated with the regulation of T cell activation and with the apoptotic pathways of effector memory T cells. Specifically, the binding with PD-1 ligand (PD-L1) on the surface of tumor plasma cells down-regulates T cell-proliferation, thus contributing to the immune escape of tumor cells. In relapsed and/or refr…

0301 basic medicinePD-L1lcsh:Immunologic diseases. AllergyDurvalumabMini ReviewT-LymphocytesT cellProgrammed Cell Death 1 ReceptorImmunologyAntigen presentationT cellsPembrolizumabmedicine.disease_causeB7-H1 Antigen03 medical and health sciences0302 clinical medicineBone Marrowimmune dysregulationPD-L1PD-1Tumor MicroenvironmentmedicineAnimalsHumansImmunology and AllergyImmune dysregulation; Multiple myeloma; PD-1; PD-L1; T cells; Animals; B7-H1 Antigen; Bone Marrow; Humans; Multiple Myeloma; Programmed Cell Death 1 Receptor; T-Lymphocytes; Tumor MicroenvironmentMultiple myelomabiologybusiness.industryImmune dysregulationmedicine.diseasemultiple myeloma030104 developmental biologymedicine.anatomical_structure030220 oncology & carcinogenesisbiology.proteinCancer researchNivolumabbusinesslcsh:RC581-607Frontiers in Immunology
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Modulating allergic response by engineering the major Parietaria allergens.

2017

0301 basic medicineParietariaImmunologySettore BIO/11 - Biologia MolecolareBiologyAllergensAntigens Plantmedicine.disease_causebiology.organism_classificationPlants Genetically Modified03 medical and health sciencesDisease Models AnimalMice030104 developmental biology0302 clinical medicineParietaria030228 respiratory systemAllergic responseImmunologymedicineHypersensitivityImmunology and AllergyAnimalsHumansPlant ProteinsThe Journal of allergy and clinical immunology
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Intracellular fluoride influences TASK mediated currents in human T cells.

2019

The expression of Kv1.3 and KCa channels in human T cells is essential for maintaining cell activation, proliferation and migration during an inflammatory response. Recently, an additional residual current, sensitive to anandamide and A293, compounds specifically inhibiting currents mediated by TASK channels, was observed after complete pharmacological blockade of Kv1.3 and KCa channels. This finding was not consistently observed throughout different studies and, an in-depth review of the different recording conditions used for the electrophysiological analysis of K+ currents in T cells revealed fluoride as major anionic component of the pipette intracellular solutions in the initial studie…

0301 basic medicinePatch-Clamp TechniquesTime FactorsPotassium CompoundsT-LymphocytesImmunologyMagnesium ChlorideMembrane Potentials03 medical and health scienceschemistry.chemical_compoundFluorides0302 clinical medicinePotassium Channels Tandem Pore DomainPotassium Channel BlockersImmunology and AllergyHumansCells CulturedKv1.3 Potassium ChannelActivator (genetics)ChemistryPipetteAnandamideElectrophysiology030104 developmental biologyMembraneBiophysicsCell activationFluorideIntracellular030215 immunologyJournal of immunological methods
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Parvovirus B19V Nonstructural Protein NS1 Induces Double-Stranded Deoxyribonucleic Acid Autoantibodies and End-Organ Damage in Nonautoimmune Mice

2018

Abstract Background Viral infection is implicated in development of autoimmunity. Parvovirus B19 (B19V) nonstructural protein, NS1, a helicase, covalently modifies self double-stranded deoxyribonucleic acid (dsDNA) and induces apoptosis. This study tested whether resulting apoptotic bodies (ApoBods) containing virally modified dsDNA could induce autoimmunity in an animal model. Methods BALB/c mice were inoculated with (1) pristane-induced, (2) B19V NS1-induced, or (3) staurosporine-induced ApoBods. Serum was tested for dsDNA autoantibodies by Crithidia luciliae staining and enzyme-linked immunosorbent assay. Brain, heart, liver, and kidney pathology was examined. Deposition of self-antigens…

0301 basic medicinePathogenesis and Host ResponseviruksetvirusesB19VKidney GlomerulusSLEApoptosisAutoimmunityanti-dsDNA antibodyViral Nonstructural Proteinsmedicine.disease_causeAutoimmunityautoimmuniteettiMice0302 clinical medicineGlomerulonephritisParvovirus B19 HumanImmunology and Allergy030212 general & internal medicineEnzyme InhibitorstolerancebiologyChemistryapoptosisBrainInfectious DiseasesLivervirustauditAntibodies AntinuclearmaksatulehdusFemaleAntibodyImmunosuppressive Agentsta3111infektiot03 medical and health sciencesohjelmoitunut solukuolemaMajor Articles and Brief ReportsExtracellular VesiclesAntigenmedicineCrithidia luciliaeAnimalsapoptotic bodiesparvoviruksetParvovirusTerpenesAnti-dsDNA antibodiesMyocardiumta1183parvovirusAutoantibodyta1182DNAbiology.organism_classificationStaurosporineMolecular biology030104 developmental biologyApoptosisbiology.proteinautovasta-aineetglomerulonephritisThe Journal of Infectious Diseases
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