Search results for "inbred c57bl"

showing 10 items of 1287 documents

Obesogen effect of bisphenol S alters mRNA expression and DNA methylation profiling in male mouse liver

2020

International audience; Environmental pollution is increasingly considered an important factor involved in the obesity incidence. Endocrine disruptors (EDs) are important actors in the concept of DOHaD (Developmental Origins of Health and Disease), where epigenetic mechanisms play crucial roles. Bisphenol A (BPA), a monomer used in the manufacture of plastics and resins is one of the most studied obesogenic endocrine disruptor. Bisphenol S (BPS), a BPA substitute, has the same obesogenic properties, acting at low doses with a sex-specific effect following perinatal exposure. Since the liver is a major organ in regulating body lipid homeostasis, we investigated gene expression and DNA methyl…

Malemedicine.medical_specialtyEnvironmental EngineeringHealth Toxicology and Mutagenesis[SDV]Life Sciences [q-bio]0208 environmental biotechnologyEnvironmental pollution02 engineering and technologyEndocrine Disruptors010501 environmental sciencesBiology01 natural sciencesEpigenesis GeneticPhenolsPregnancyInternal medicineToxicity TestsGene expressionmedicineAnimalsHumansEnvironmental ChemistryObesityRNA MessengerSulfonesEpigeneticsGene0105 earth and related environmental sciencesDose-Response Relationship DrugPublic Health Environmental and Occupational HealthGeneral MedicineGeneral ChemistryDNA MethylationLipid MetabolismPollution3. Good health020801 environmental engineeringMice Inbred C57BLEndocrinologyGene Expression RegulationLiverEndocrine disruptorPrenatal Exposure Delayed EffectsDNA methylationFemaleObesogenhormones hormone substitutes and hormone antagonistsDNA hypomethylation
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Inhibitory effects of indicaxanthin on mouse ileal contractility: analysis of the mechanism of action.

2011

Recently, we have showed that indicaxanthin, the yellow betalain pigment abundant in the fruit of Opuntia ficus indica, has remarkable spasmolytic effects on the intestinal contractility in vitro. Thus, the purpose of the present study was to investigate the mechanism of action underlying the observed response. We used organ bath technique to record the mechanical activity of the mouse ileum longitudinal muscle and ELISA to measure the levels of cAMP. Indicaxanthin induced inhibitory effects on spontaneous mechanical activity, which were unaffected by indomethacin, a non-selective inhibitor of cycloxygenase; 1H-[1,2,4]oxadiazolo[4,3-a]quinoxalin-1-one, a selective inhibitor of nitric oxide-…

Malemedicine.medical_specialtyIBMXPyridinesIndicaxanthinBiologyIn Vitro TechniquesContractilitySettore BIO/09 - FisiologiaAdenylyl cyclaseContractilitychemistry.chemical_compoundMiceSmooth muscleCactus pear fruitIleumSettore BIO/10 - BiochimicaInternal medicinemedicineCyclic AMPAnimalsEnzyme InhibitorsPharmacologyForskolinPhosphodiesteraseMuscle SmoothBetaxanthinsBiomechanical PhenomenaMice Inbred C57BLEndocrinologychemistryPhosphodiesterasesCarbacholZaprinastSoluble guanylyl cyclaseIndicaxanthinMuscle ContractionSignal TransductionEuropean journal of pharmacology
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Increased calcium influx is responsible for the sustained mechanical tone in colon from dystrophic (mdx) mice

2001

Abstract Background & Aims: Proximal colon from dystrophic mice develops spontaneous tone increment, but the mechanisms involved in its development have not been investigated. This study examined whether alterations in the properties of cell membrane calcium channels and/or sarcoplasmic reticular (SR) Ca 2+ -adenosine triphosphatase (ATPase) contribute to tone development. Methods: Effects of calcium-free solution, nifedipine, pinaverium (calcium channel blockers), and cyclopiazonic acid (CPA; SR Ca 2+ -ATPase inhibitor) on the contractile activity of colon from mdx and control mice were determined. Results Calcium-free solution abolished spontaneous contractions in both preparations, but d…

Malemedicine.medical_specialtyIndolesNifedipineColonSarcoplasmchemistry.chemical_elementCalciumMuscular DystrophiesCalcium in biologyMicechemistry.chemical_compoundNifedipineInternal medicinemedicineAnimalsHepatologyVoltage-dependent calcium channelCalcium channelGastroenterologyPinaveriumMice Inbred C57BLEndocrinologychemistryMice Inbred mdxCalciumCyclopiazonic acidMuscle Contractionmedicine.drugGastroenterology
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Paradoxically, iron overload does not potentiate doxorubicin-induced cardiotoxicity in vitro in cardiomyocytes and in vivo in mice

2015

Doxorubicin (DOX) is known to induce serious cardiotoxicity, which is believed to be mediated by oxidative stress and complex interactions with iron. However, the relationship between iron and DOX-induced cardiotoxicity remains controversial and the role of iron chelation therapy to prevent cardiotoxicity is called into question. Firstly, we evaluated in vitro the effects of DOX in combination with dextran-iron on cell viability in cultured H9c2 cardiomyocytes and EMT-6 cancer cells. Secondly, we used an in vivo murine model of iron overloading (IO) in which male C57BL/6 mice received a daily intra-peritoneal injection of dextran-iron (15mg/kg) for 3weeks (D0-D20) and then (D21) a single su…

Malemedicine.medical_specialtyIron OverloadCell SurvivalHeart VentriclesIronCardiomegaly030204 cardiovascular system & hematologyToxicologymedicine.disease_causeCell LineVentricular MyosinsMice03 medical and health sciences0302 clinical medicine[SDV.MHEP.CSC]Life Sciences [q-bio]/Human health and pathology/Cardiology and cardiovascular systemAtrial natriuretic peptideIn vivoCell Line TumorInternal medicineNatriuretic Peptide Brainpolycyclic compoundsmedicineAnimalsMyocytes CardiacDoxorubicinViability assay030304 developmental biologyPharmacology0303 health sciencesCardiotoxicityCell growthChemistryDextransBrain natriuretic peptideCardiotoxicity[SDV.MHEP.CSC] Life Sciences [q-bio]/Human health and pathology/Cardiology and cardiovascular systemUp-Regulation3. Good healthMice Inbred C57BLOxidative Stresscell proliferationEndocrinologyDoxorubicincardiovascular systemOxidative stressmedicine.drugToxicology and Applied Pharmacology
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Decreased kynurenine pathway potentiate resilience to social defeat effect on cocaine rewa

2021

The kynurenine (KYN) pathway of tryptophan (TRP) degradation is activated by stress and inflammatory factors. It is now well established that social stress induces the activation of the immune system, with central inflammation and KYN metabolism being two of the main factors linking stress with depression. The aim of the present study was to evaluate the long-lasting changes in the KYN pathway induced by social defeat (SD) associated with the resilience or susceptibility to an increase in the conditioned rewarding effects of cocaine. Mice were exposed to repeated SD and 3 weeks later, a conditioned place preference (CPP) induced by a subthreshold dose of cocaine (1.5 mg/kg) was developed. K…

Malemedicine.medical_specialtyKynurenine pathwayIndomethacinStriatumEnvironmentOxytocinSocial defeatSocial DefeatCellular and Molecular Neurosciencechemistry.chemical_compoundMiceCocaineRewardInternal medicineCerebellumMedicineAnimalsKynureninePharmacologySocial stressEnvironmental enrichmentbusiness.industryTryptophanResilience PsychologicalConditioned place preferenceMice Inbred C57BLEndocrinologyPsicobiologiaOxytocinchemistryConditioning OperantbusinessKynureninemedicine.drugSignal Transduction
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Hepatic steatosis is not due to impaired fatty acid oxidation capacities in C57BL/6J mice fed the conjugated trans-10,cis-12-isomer of linoleic acid.

2004

Decreased body fat mass and liver steatosis have been reported in mice fed diets containing the conjugated linoleic acid trans-10,cis-12-C18:2 (CLA2), but not in those fed diets containing cis-9,trans-11-C18:2 (CLA1). Because the decrease in fatty acid (FA) oxidation may cause fat accumulation, we questioned whether the effects of both CLAs on enzyme activities and mRNA expression were related to liver FA oxidation. To address this question, 7-wk-old male C57BL/6J mice were fed for 4 wk a diet supplemented with 1% CLA1, CLA2, or cis-9-C18:1 (control) esterified as triacylglycerols. In CLA2-fed mice, the proportions of CLA2 in the total FA of liver lipids were substantially lower than those …

Malemedicine.medical_specialtyLinoleic acidConjugated linoleic acidMedicine (miscellaneous)Mitochondria LiverBiologychemistry.chemical_compoundMiceDietary Fats UnsaturatedInternal medicinemedicineAnimalsLinoleic Acids ConjugatedCarnitineRNA MessengerEnzyme InhibitorsUnsaturated fatty acidTriglycerideschemistry.chemical_classificationNutrition and DieteticsCarnitine O-PalmitoyltransferaseEsterificationReverse Transcriptase Polymerase Chain ReactionFatty liverFatty AcidsFatty acidmedicine.diseaseFatty LiverMalonyl Coenzyme AMice Inbred C57BLEndocrinologychemistryBiochemistryLiverCarnitine palmitoyltransferase IOxidation-ReductionPolyunsaturated fatty acidmedicine.drug
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Low Protein Intake Is Associated with a Major Reduction in IGF-1, Cancer, and Overall Mortality in the 65 and Younger but Not Older Population

2014

SummaryMice and humans with growth hormone receptor/IGF-1 deficiencies display major reductions in age-related diseases. Because protein restriction reduces GHR-IGF-1 activity, we examined links between protein intake and mortality. Respondents aged 50–65 reporting high protein intake had a 75% increase in overall mortality and a 4-fold increase in cancer death risk during the following 18 years. These associations were either abolished or attenuated if the proteins were plant derived. Conversely, high protein intake was associated with reduced cancer and overall mortality in respondents over 65, but a 5-fold increase in diabetes mortality across all ages. Mouse studies confirmed the effect…

Malemedicine.medical_specialtyLow proteinnutrition protein intake caloric restriction nutrientsPhysiologymedicine.medical_treatmentLongevityCalorie restrictionBreast NeoplasmsGrowth hormone receptorBiologyArticleMiceLow-protein dietNeoplasmsDiabetes mellitusInternal medicineDiabetes MellitusDiet Protein-RestrictedmedicineAnimalsHumansInsulin-Like Growth Factor IMelanomaMolecular BiologyAgedProportional Hazards ModelsMice KnockoutMice Inbred BALB CIncidence (epidemiology)CancerCell BiologyMiddle Agedmedicine.diseaseMiddle ageMice Inbred C57BLCross-Sectional StudiesEndocrinologyFemaleCarrier ProteinsFollow-Up StudiesSignal TransductionCell Metabolism
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Imeglimin Normalizes Glucose Tolerance and Insulin Sensitivity and Improves Mitochondrial Function in Liver of a High-Fat, High-Sucrose Diet Mice Mod…

2015

International audience; Imeglimin is the first in a new class of oral glucose-lowering agents currently in phase 2b development. Although imeglimin improves insulin sensitivity in humans, the molecular mechanisms are unknown. This study used a model of 16-week high-fat, high-sucrose diet (HFHSD) mice to characterize its antidiabetic effects. Six-week imeglimin treatment significantly decreased glycemia, restored normal glucose tolerance, and improved insulin sensitivity without modifying organs, body weights, and food intake. This was associated with an increase in insulin-stimulated protein kinase B phosphorylation in the liver and muscle. In liver mitochondria, imeglimin redirects substra…

Malemedicine.medical_specialtyMale Animals Mice Inbred C57BL Insulin Resistance/*physiology Diet High-Fat/adverse effects Hypoglycemic Agents/*therapeutic use Liver/*drug effects/*metabolism Mitochondria/*drug effects/*metabolism Triazines/*therapeutic useImegliminMitochondria/*drug effects/*metabolismEndocrinology Diabetes and Metabolism[SDV]Life Sciences [q-bio]High-Fat/adverse effectsBiologyMitochondrionDiet High-Fatmedicine.disease_causeInbred C57BLchemistry.chemical_compoundMiceLipid oxidationInternal medicineInternal MedicinemedicineHypoglycemic Agents/*therapeutic useHypoglycemic AgentsAnimalsProtein kinase BBeta oxidationComputingMilieux_MISCELLANEOUS2. Zero hungerchemistry.chemical_classificationReactive oxygen speciesTriazines/*therapeutic useTriazinesMitochondria3. Good healthDietMice Inbred C57BL[SDV] Life Sciences [q-bio]EndocrinologyLiver/*drug effects/*metabolismLiverchemistryInsulin Resistance/*physiologyCoenzyme Q – cytochrome c reductaseInsulin ResistanceOxidative stress
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Sex differences in nucleus accumbens transcriptome profiles associated with susceptibility versus resilience to subchronic variable stress

2015

Depression and anxiety disorders are more prevalent in females, but the majority of research in animal models, the first step in finding new treatments, has focused predominantly on males. Here we report that exposure to subchronic variable stress (SCVS) induces depression-associated behaviors in female mice, whereas males are resilient as they do not develop these behavioral abnormalities. In concert with these different behavioral responses, transcriptional analysis of nucleus accumbens (NAc), a major brain reward region, by use of RNA sequencing (RNA-seq) revealed markedly different patterns of stress regulation of gene expression between the sexes. Among the genes displaying sex differe…

Malemedicine.medical_specialtyMethyltransferaseStreRepression PsychologyNucleus accumbensBiologyAnxietyMotor ActivityGene Expression Regulation EnzymologicNucleus AccumbensDNA Methyltransferase 3ATranscriptomeMiceInternal medicineGene expressionmedicineTranscriptional regulationAnimalsNucleus accumbenEpigeneticsDNA (Cytosine-5-)-MethyltransferasesGene Knock-In TechniquesSwimmingGeneticsMice KnockoutSex CharacteristicsBehaviorNeuroscience (all)DepressionGeneral NeuroscienceEpigeneticFeeding BehaviorArticlesResilience PsychologicalSex differenceMice Inbred C57BLEndocrinologyChronic DiseaseBrain stimulation rewardFemaleTranscriptomeStress PsychologicalSex characteristics
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Mitochondrial glutathione oxidation correlates with age-associated oxidative damage to mitochondrial DNA

1996

Mitochondria may be primary targets of free radical damage associated with aging. We have found that mitochondrial glutathione is markedly oxidized with aging in rats and mice. The oxidized to reduced glutathione ratio rises with aging in the liver, kidney, and brain. The magnitude of these changes is much higher than that previously found in whole cells of any species previously studied. In the liver, this ratio (expressing GSSG as a percent of GSH) changed from 0.77 +/- 0.19% (n=5) in young rats to 2.47 +/- 1.25% (n=5) in old ones, i.e., 320% of the controls. In the brain and kidney, values for old rats were, respectively, 600 and 540% higher than those of young rats. A marked oxidation o…

Malemedicine.medical_specialtyMitochondrial DNAAgingAntioxidantmedicine.medical_treatmentMitochondrionmedicine.disease_causeBiochemistryDNA MitochondrialAntioxidantschemistry.chemical_compoundMiceInternal medicineGeneticsmedicineDeoxyguanosineAnimalsRats WistarMolecular BiologyFree-radical theory of agingKidneyGlutathione DisulfideChemistryDeoxyguanosineGlutathioneGlutathioneRatsMice Inbred C57BLOxidative StressEndocrinologymedicine.anatomical_structure8-Hydroxy-2'-DeoxyguanosineRabbitsOxidation-ReductionOxidative stressBiotechnologyDNA Damage
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