Search results for "inheritance"

showing 10 items of 160 documents

Second study on the recurrence risk of isolated esophageal atresia with or without trachea-esophageal fistula among first-degree relatives: no eviden…

2013

BACKGROUND Esophageal atresia with/without trachea-esophageal fistula (EA/TEF) denotes a spectrum of severe congenital malformations. The aim of this systematic study was to determine both the recurrence risk for EA/TEF, and the risk for malformations of the VATER/VACTERL association spectrum, in first-degree relatives of patients with isolated EA/TEF. METHODS A total of 108 unrelated patients with isolated EA/TEF were included. These individuals had 410 first-degree relatives including 194 siblings. The presence of EA/TEF and malformations of the VATER/VACTERL association spectrum in relatives was systematically assessed. Data from the EUROCAT network were used for comparison. RESULTS None…

AdultHeart Defects CongenitalMaleRiskEmbryologymedicine.medical_specialtyAdolescentFistulaInheritance PatternsLimb Deformities CongenitalAnal CanalKidneyGastroenterologyRecurrence riskAnus ImperforateEsophagusInternal medicinemedicineHumansEsophageal FistulaFirst-degree relativesChildEsophageal Atresiabusiness.industrySiblingsVATER/VACTERL ASSOCIATIONGeneral Medicinemedicine.diseaseVACTERL associationSpinePedigreeTracheaRadiusAtresiaCase-Control Studiesembryonic structuresPediatrics Perinatology and Child HealthCohortFemalebusinessDevelopmental BiologyTracheoesophageal FistulaBirth defects research. Part A, Clinical and molecular teratology
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Examining the independent and joint effects of genomic and exposomic liabilities for schizophrenia across the psychosis spectrum

2020

The EUGEI project was supported by the European Community’s Seventh Framework Program under grant agreement no. HEALTH-F2-2009-241909 (Project EU-GEI). Dr O’Donovan is supported by MRC programme grant (G08005009) and an MRC Centre grant (MR/ L010305/1). Dr Rutten was funded by a VIDI award number 91718336 from the Netherlands Scientific Organisation. Drs Guloksuz and van Os are supported by the Ophelia research project, ZonMw grant number: 636340001. Dr Arango was supported by the Spanish Ministry of Science and Innovation; Instituto de Salud Carlos III (SAM16PE07CP1, PI16/02012, PI19/024); CIBERSAM; Madrid Regional Government (B2017/BMD-3740, AGES-CM-2); Fundación Familia Alonso and Fundac…

AdultMaleExposomePsychosisMultifactorial InheritanceEpidemiologyBIRTHSchizotypystructured interviewGENE-ENVIRONMENTPopulationschizotypypopulationEnvironment03 medical and health sciences0302 clinical medicineMedicineHumansSpectrum disorderGenetic Predisposition to DiseasegeneticspsychosiseducationMETAANALYSISvalidationRISKeducation.field_of_studychildhood traumareliabilitybusiness.industryPublic Health Environmental and Occupational HealthAbsolute risk reductionassociationRegression analysisGenomicsmedicine.disease3. Good health030227 psychiatryPsychiatry and Mental healthPsychotic DisordersSchizophreniaSchizophreniaFemaleGene-Environment InteractionSchizophrenic PsychologyOriginal Articlebusiness030217 neurology & neurosurgeryClinical psychologyEpidemiology and Psychiatric Sciences
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New syndrome: Autosomal dominant microcephaly and radio-ulnar synostosis

1994

To date, the combination of microcephaly and radio-ulnar synostosis has not been recognized as a distinct clinical and genetic entity. We report on 4 familial cases with this previously undescribed combination of defects, showing autosomal dominant inheritance (Fig. 1). © 1994 Wiley-Liss, Inc.

AdultMaleMicrocephalyAdolescentUlnaSupinationMedicineHumansAbnormalities MultiplemicrocephalyChildGenetics (clinical)Genes Dominantradioulnar synostosisbusiness.industryfungiInfantAnatomySyndromeSynostosisMiddle Agedmedicine.diseasePedigreeautosomal dominant inheritanceRadiusSynostosisRadioulnar synostosisFemaleCongenital diseasebusinessHand Deformities Congenital
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Replication of previous genome-wide association studies of psychiatric diseases in a large schizophrenia case-control sample from Spain.

2014

Genome wide association studies (GWAS) has allowed the discovery of some interesting risk variants for schizophrenia (SCZ). However, this high-throughput approach presents some limitations, being the most important the necessity of highly restrictive statistical corrections as well as the loss of statistical power inherent to the use of a Single Nucleotide Polymorphism (SNP) analysis approach. These problems can be partially solved through the use of a polygenic approach. We performed a genotyping study in SCZ using 86 previously associated SNPs identified by GWAS of SCZ, bipolar disorder (BPD) and autistic spectrum disorder (ASD) patients. The sample consisted of 3063 independent cases wit…

AdultMaleMultifactorial InheritanceAdolescentBipolar disorderSingle-nucleotide polymorphismGenome-wide association studyBiologyPolymorphism Single NucleotideODZ4White PeopleYoung AdultPolygenic scoremedicineGWASSNPHumansGenetic Predisposition to DiseaseBipolar disorderAlleleGenotypingBiological PsychiatryAgedGeneticsAged 80 and overMembrane GlycoproteinsModels GeneticCase-control studyMiddle Agedmedicine.diseasePsychiatry and Mental healthROC CurveSchizophreniaSpainArea Under CurveCase-Control StudiesReplication studySchizophreniaFemaleGenome-Wide Association StudySchizophrenia research
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IBD risk loci are enriched in multigenic regulatory modules encompassing putative causative genes

2018

GWAS have identified >200 risk loci for Inflammatory Bowel Disease (IBD). The majority of disease associations are known to be driven by regulatory variants. To identify the putative causative genes that are perturbed by these variants, we generate a large transcriptome data set (nine disease-relevant cell types) and identify 23,650 cis-eQTL. We show that these are determined by ∼9720 regulatory modules, of which ∼3000 operate in multiple tissues and ∼970 on multiple genes. We identify regulatory modules that drive the disease association for 63 of the 200 risk loci, and show that these are enriched in multigenic modules. Based on these analyses, we resequence 45 of the corresponding 100 ca…

AdultMaleMultifactorial InheritanceQUANTITATIVE TRAIT LOCUSGenotypeSEQUENCING DATAQuantitative Trait LociSUSCEPTIBILITYPolymorphism Single NucleotideArticleCohort StudiesCODING VARIANTSCrohn Disease80 and overJournal ArticleMedicine and Health SciencesLOCUSHumansGenetic Predisposition to DiseasePolymorphismGENOME-WIDE ASSOCIATIONGenetic Association StudiesAgedAged 80 and overScience & TechnologyAdult; Aged; Aged 80 and over; Cohort Studies; Crohn Disease; Female; Gene Expression Profiling; Genetic Association Studies; Genetic Predisposition to Disease; Genotype; Humans; Inflammatory Bowel Diseases; Male; Middle Aged; Polymorphism Single Nucleotide; Quantitative Trait Loci; Sequence Analysis DNA; Multifactorial InheritanceGene Expression ProfilingCOMPLEX TRAITSBiology and Life SciencesSequence Analysis DNASingle NucleotideDNAMiddle AgedInflammatory Bowel DiseasesCROHNS-DISEASEMultidisciplinary SciencesQUANTITATIVE TRAITRARE VARIANTSScience & Technology - Other TopicsFemaleLOW-FREQUENCYSequence AnalysisINFLAMMATORY-BOWEL-DISEASE
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A polygenic approach to the association between smoking and schizophrenia.

2021

Smoking prevalence in schizophrenia is considerably larger than in general population, playing an important role in early mortality. We compared the polygenic contribution to smoking in schizophrenic patients and controls to assess if genetic factors may explain the different prevalence. Polygenic risk scores (PRSs) for smoking initiation and four genetically correlated traits were calculated in 1108 schizophrenic patients (64.4% smokers) and 1584 controls (31.1% smokers). PRSs for smoking initiation, educational attainment, body mass index and age at first birth were associated with smoking in patients and controls, explaining a similar percentage of variance in both groups. Attention-defi…

AdultPsychosisMultifactorial InheritanceSociodemographic FactorsPopulationMedicine (miscellaneous)Nerve Tissue ProteinsReceptors NicotinicGenetic correlationBody Mass IndexNicotineRisk Factorsmental disordersmedicineGenetic predispositionTobacco SmokingHumansGenetic Predisposition to DiseaseRisk factoreducationPharmacologyeducation.field_of_studybusiness.industryMiddle Agedmedicine.diseasePsychiatry and Mental healthPhenotypeSchizophreniaAttention Deficit Disorder with HyperactivitySchizophreniabusinessBody mass indexDemographymedicine.drugGenome-Wide Association StudyAddiction biologyREFERENCES
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Genome-wide association studies identify 137 genetic loci for DNA methylation biomarkers of aging

2021

Abstract Background Biological aging estimators derived from DNA methylation data are heritable and correlate with morbidity and mortality. Consequently, identification of genetic and environmental contributors to the variation in these measures in populations has become a major goal in the field. Results Leveraging DNA methylation and SNP data from more than 40,000 individuals, we identify 137 genome-wide significant loci, of which 113 are novel, from genome-wide association study (GWAS) meta-analyses of four epigenetic clocks and epigenetic surrogate markers for granulocyte proportions and plasminogen activator inhibitor 1 levels, respectively. We find evidence for shared genetic loci ass…

AgingMultifactorial InheritanceBLOODEpigenetic clock05 Environmental SciencesbiomarkkeritGenome-wide association studyQH426-470Epigenesis Genetic/dk/atira/pure/core/keywords/icep0302 clinical medicineBiomarkers of agingGWASBiology (General)AdiposityGenetics11832 Microbiology and virology0303 health sciences318 Medical biotechnologyDNA methylation1184 Genetics developmental biology physiologygenomiikkaDna Methylation ; Epigenetic Clock ; Gwasddc:DNA-metylaatioINSIGHTSC-Reactive ProteinepigenetiikkaDNA methylationMENDELIAN RANDOMIZATION/dk/atira/pure/sustainabledevelopmentgoals/good_health_and_well_beingEducational StatusICEPGenetic MarkersPROVIDESSUSCEPTIBILITY LOCIBioinformaticsQH301-705.5GenomicsBiology03 medical and health sciencesNHLBI Trans-Omics for Precision Medicine (TOPMed) ConsortiumAGESDG 3 - Good Health and Well-beingPlasminogen Activator Inhibitor 1REGRESSIONGeneticsHumansEpigeneticsGeneMETAANALYSIS030304 developmental biologyGenome HumanResearchGenetics of DNA Methylation Consortium06 Biological SciencesLipid MetabolismHuman geneticsGenetic architectureImmunity InnateikääntyminenGenetic LociCpG Islands08 Information and Computing Sciences3111 BiomedicineENRICHMENTepigenetic clock030217 neurology & neurosurgeryBiomarkersGenome-Wide Association StudyGranulocytes
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Aproximaciones al apócrifo en la órbita de Max Aub : del modelo francés a las últimas manifestaciones peninsulares

2009

En este trabajo se realiza una lectura del mapa de manifestaciones apócrifas alrededor de la figura de Max Aub, escritor que construye un universo de superchería literaria desde sus obras iniciales, abarcando y fusionando diferentes géneros. A partir del contraste con algunos de los textos franceses que leyó, y que supusieron una herencia cultural de gran valor, se esbozará un panorama que incluya tanto a los maestros y predecesores en la práctica apócrifa aubiana, André Gide y Valéry Larbaud fundamentalmente, como a sus sucesores más representativos: los narradores leoneses creadores de Sabino Ordás. Entre unos y otros, se rastrearán los casos peninsulares de mayor trascendencia, con el fi…

ApócrifoLiterature and Literary Theorymax aubFrench literature - Italian literature - Spanish literature - Portuguese literatureApocryphea.o. barnaboothapócrifoMax AubSupercheríaFakeCultural inheritanceAndré WaltersupercheríaSabino Ordásfalso literarioA.O. BarnaboothSubversionOrder (virtue)LiteratureFalso literariolcsh:French literature - Italian literature - Spanish literature - Portuguese literatureA. O. Barnaboothbusiness.industryPhilosophyBiographysabino ordás:CIENCIAS DE LAS ARTES Y LAS LETRAS::Teoría análisis y crítica literarias::Análisis literario [UNESCO]Apocryphe ; Max Aub ; Fake ; Sabino Ordás ; André Walter ; A. O. Barnaboothlcsh:PQ1-3999UNESCO::CIENCIAS DE LAS ARTES Y LAS LETRAS::Teoría análisis y crítica literarias::Análisis literarioPQ1-3999businessHumanitiesApócrifo ; Max Aub ; Superchería ; Sabino Ordás ; André Walter ; A.O. Barnabooth ; Falso literarioandré walter
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Distal Arthrogryposis type 5 in an Italian family due to an autosomal dominant gain-of-function mutation of the PIEZO2 gene

2022

Abstract Background Arthrogryposis multiplex congenita (AMC) is a group of clinically and etiologically heterogeneous conditions, characterized by prenatal onset contractures affecting two or more joints. Its incidence is about 1 in 3000 live births. AMC may be distinguished into amyoplasia, distal and syndromic arthrogryposis. Distal arthrogryposis (DA) predominantly affects hands and feet. It is currently divided into more than ten subtypes (DA1, DA2A/B, DA3–10), based on clinical manifestations, gene mutations and inheritance pattern. Among them, only a few patients with DA5 have been reported. It is associated to a gain-of-function pathogenic variant of the PIEZO2 gene, encoding for an …

ArthrogryposisContractureOphthalmoplegiaArthrogryposis multiplex congenita Case report DA5 Gain-of-function mutation NGS Ophthalmoplegia PIEZO2 gene Gain of Function Mutation Humans Infant Newborn Inheritance Patterns Ion Channels Mutation Pedigree Retinal Diseases Arthrogryposis Contracture OphthalmoplegiaRetinal DiseasesGain of Function MutationMutationInfant NewbornInheritance PatternsHumansGeneral MedicineIon ChannelsPedigreeItalian Journal of Pediatrics
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ABG1 , a Novel and Essential Candida albicans Gene Encoding a Vacuolar Protein Involved in Cytokinesis and Hyphal Branching

2005

ABSTRACT Immunoscreening of a Candida albicans expression library resulted in the isolation of a novel gene encoding a 32.9-kDa polypeptide (288 amino acids), with 27.7% homology to the product of Saccharomyces cerevisiae YGR106c, a putative vacuolar protein. Heterozygous mutants in this gene displayed an a ltered b udding g rowth pattern, characterized by the formation of chains of buds, decreasingly in size towards the apex, without separation of the daughter buds. Consequently, this gene was designated ABG1 . A conditional mutant for ABG1 with the remaining allele under the control of the MET3 promoter did not grow in the presence of methionine and cysteine, demonstrating that ABG1 was e…

Blotting WesternGreen Fluorescent ProteinsSaccharomyces cerevisiaeMutantHyphaeVacuoleVacuole inheritanceMicrobiologyFungal ProteinsGene Expression Regulation FungalCandida albicansCloning MolecularCandida albicansMolecular BiologyGeneCytokinesisFungal proteinGenes EssentialBase SequencebiologyArticlesGeneral Medicinebiology.organism_classificationBiochemistryVacuolesElectrophoresis Polyacrylamide GelGenome FungalCytokinesisSubcellular FractionsEukaryotic Cell
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