Search results for "interleukin-1"

showing 10 items of 660 documents

Differential effects of anti-TNF-α and anti-IL-12/23 agents on human leukocyte–endothelial cell interactions

2015

AbstractEnhanced leukocyte recruitment is an inflammatory process that occurs during early phases of the vascular dysfunction that characterises atherosclerosis. We evaluated the impact of anti-TNF-α (adalimumab, infliximab and etanercept) and anti-IL-12/23 (ustekinumab) on interactions between human leukocytes and endothelial cells in a flow chamber that reproduced in vivo conditions. Clinical concentrations of anti-TNF-α were evaluated on the leukocyte recruitment induced by a variety of endothelial (TNF-α, interleukin-1β, lymphotoxin-α and angiotensin-II) and leukocyte (PAF, IL-12 and IL-23) stimuli related to inflammation and atherosclerosis. Treatment with anti-TNF-α, even before or af…

EndotheliumInflammationAnti-IL-12/23 agentsCardiovascular side effectsBiologicsInterleukin-23Rheumatic diseasesIn vivoPsoriasisHuman Umbilical Vein Endothelial CellsInterleukin 23HumansMedicineAnti-TNF-α agentsPharmacologyTumor Necrosis Factor-alphabusiness.industryCell adhesion moleculeAdalimumabEndothelial Cellsmedicine.diseaseInterleukin-12Leukocyte–endothelial cell interactionsEndothelial stem cellmedicine.anatomical_structureImmunologyLeukocytes MononuclearTumor necrosis factor alphamedicine.symptombusinessEuropean Journal of Pharmacology
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The Binomial “Inflammation-Epigenetics” in Breast Cancer Progression and Bone Metastasis: IL-1β Actions Are Influenced by TET Inhibitor in MCF-7 Cell…

2022

The existence of a tight relationship between inflammation and epigenetics that in primary breast tumor cells can lead to tumor progression and the formation of bone metastases was investigated. It was highlighted how the induction of tumor progression and bone metastasis by Interleukin-1 beta, in a non-metastatic breast cancer cell line, MCF-7, was dependent on the de-methylating actions of ten-eleven translocation proteins (TETs). In fact, the inhibition of their activity by the Bobcat339 molecule, an inhibitor of TET enzymes, determined on the one hand, the modulation of the epithelial-mesenchymal transition process, and on the other hand, the reduction in the expression of markers of bo…

Epithelial-Mesenchymal TransitionDNA methylation; bone metastasis; inflammation; Interleukin-1β; ten-eleven translocation proteins; MCF-7 cell lineInterleukin-1betaBreast NeoplasmsBone NeoplasmsMCF-7 cell lineCatalysisEpigenesis GeneticInorganic ChemistrySettore BIO/13 - Biologia ApplicataCell Line TumorHumansPhysical and Theoretical ChemistryMolecular BiologySpectroscopybone metastasisDNA methylationten-eleven translocation proteinsOrganic ChemistryGeneral MedicineInterleukin-1βComputer Science ApplicationsSettore BIO/18 - GeneticainflammationMCF-7 CellsFemaleInflammatory Breast NeoplasmsInternational Journal of Molecular Sciences
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TGF-beta regulates airway responses via T cells.

2003

Abstract Allergic asthma is characterized by airway hyperreactivity, inflammation, and a Th2-type cytokine profile favoring IgE production. Beneficial effects of TGF-β and conflicting results regarding the role of Th1 cytokines have been reported from murine asthma models. In this study, we examined the T cell as a target cell of TGF-β-mediated immune regulation in a mouse model of asthma. We demonstrate that impairment of TGF-β signaling in T cells of transgenic mice expressing a dominant-negative TGF-β type II receptor leads to a decrease in airway reactivity in a non-Ag-dependent model. Increased serum levels of IFN-γ can be detected in these animals. In contrast, after injection of OVA …

Epitopes T-LymphocyteNitric Oxide Synthase Type IIImmunoglobulin EMiceAntibody SpecificityCell MovementT-Lymphocyte SubsetsTransforming Growth Factor betaImmunology and AllergyInterferon gammaLungInterleukin-13biologymedicine.diagnostic_testrespiratory systemImmunohistochemistrymedicine.anatomical_structureInterleukin 13Alum Compoundsmedicine.symptomBronchial HyperreactivityBronchoalveolar Lavage Fluidmedicine.drugGenetically modified mousemedicine.medical_specialtyOvalbuminT cellImmunologyCD2 AntigensInflammationMice Inbred StrainsMice TransgenicProtein Serine-Threonine KinasesInterferon-gammaInternal medicineAdministration InhalationmedicineAnimalsHumansAerosolsInflammationbusiness.industryReceptor Transforming Growth Factor-beta Type IITransforming growth factor betaImmunoglobulin ETh1 Cellsrespiratory tract diseasesEndocrinologyBronchoalveolar lavageImmunologybiology.proteinNitric Oxide SynthasebusinessReceptors Transforming Growth Factor betaJournal of immunology (Baltimore, Md. : 1950)
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Immunological insights into the pathogenesis of active CMV infection in non-immunosuppressed critically ill patients.

2011

Dissociation of cytomegalovirus (CMV) DNA loads between the lower respiratory tract and blood, with high levels in the former compartment and low or undetectable levels in the latter, commonly occurs during active CMV infection in critically ill patients despite the presence of high frequencies of CMV-specific IFN-γ-producing CD8+ and CD4+ T cells in blood. Data presented in this case report suggest that inter-compartmental differences in interleukin-10 (IL-10) levels may, in part, explain the pathobiology of this phenomenon. In the absence of ganciclovir treatment, a significant correlation was observed between IL-10 levels and CMV DNA loads in lower respiratory tract specimens (P = 0.016)…

GanciclovirMalemedicine.medical_treatmentCritical IllnessRespiratory SystemCytomegalovirusPathogenesisVirologymedicineHumansInterleukin 6AgedAged 80 and overLungbiologyvirus diseasesImmunosuppressionMiddle AgedViral LoadVirologyInterleukin-10Interleukin 10Infectious Diseasesmedicine.anatomical_structureBloodImmunologyCytomegalovirus Infectionsbiology.proteinFemaleBronchoalveolar Lavage FluidCD8Respiratory tractmedicine.drugJournal of medical virology
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Orthogonal electrophoretic fractionation of DNA in agarose gels.

2008

We developed an electrophoretic procedure, using Voltage Gradient Gel Electrophoresis (VGGE), which allows to obtain both an improvement of the resolution power of the system in orthogonal fractionation of DNA and, mainly, an about fourfold enhancement of hybridization signals in Southern blotting applications.

Gel electrophoresisElectrophoresis Agar GelChromatographyGel electrophoresis of nucleic acidsCell BiologyFractionationDNABiologyMolecular biologyInterleukin-10chemistry.chemical_compoundElectrophoresischemistryAgaroseRNA MessengerMolecular BiologyDNATemperature gradient gel electrophoresisSouthern blotMolecular and cellular probes
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Cytomegalovirus Interleukin-10 Expression in Infected Cells Does Not Impair MHC Class I Restricted Peptide Presentation on Bystanding Antigen-Present…

2006

Human cytomegalovirus (HCMV) has evolved strategies to counteract its surveillance by the immune system. Mitigation of antiviral immune responses is considered critical for establishment of viral latency and for spread. Recently, a gene encoding an interleukin-10 homologue (cmvIL-10) has been discovered in the HCMV genome. Using recombinant cmvIL-10, several mostly immunosuppressive functions of the molecule have been described. However, the role of cmvIL-10 in the context of viral infection was not addressed. To be able to analyze this issue, we generated cmvIL- 10-negative viral mutants. Using these mutants, we tested whether the expression of cmvIL-10 by infected cells would render bysta…

Gene Expression Regulation ViralHuman cytomegalovirusvirusesImmunologyCongenital cytomegalovirus infectionAntigen-Presenting CellsCytomegalovirusContext (language use)Viral ProteinsImmune systemVirologyMHC class ImedicineHumansAntigen-presenting cellCells CulturedAntigen PresentationbiologyHistocompatibility Antigens Class IBystander EffectFibroblastsmedicine.diseaseVirologyInterleukin-10CTL*Interleukin 10MutationImmunologybiology.proteinMolecular MedicineGene DeletionViral Immunology
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TGFbeta regulates the CD4+CD25+ T-cell pool and the expression of Foxp3 in vivo.

2004

Factors influencing the development of CD4+CD25+ T-cells in vivo are poorly understood. In order to investigate the contribution of TGFbeta1 to the development and function of CD4+CD25+ T-cells, we generated a gain of function mutation resulting in the overexpression of an active form of TGFbeta1 in T-cells under control of the human CD2 promoter. In peripheral lymphoid organs and in the thymus, the frequency of CD4+CD25+ T-cells was increased in transgenic mice. This appeared to be due to an autocrine effect of TGFbeta on T-cells, since concomitant impairment of TGFbeta-signaling in double transgenic mice resulted in a phenotype similar to wild type. In contrast, in single transgenic mice …

Genetically modified mouseTransgeneT cellImmunologyCD2 AntigensMice TransgenicBiologyMiceIn vivoT-Lymphocyte SubsetsTransforming Growth Factor betamedicineImmunology and AllergyAnimalsAutocrine signallingTranscription factorWild typeFOXP3Forkhead Transcription FactorsReceptors Interleukin-2General MedicineMolecular biologyCell biologyInterleukin-10DNA-Binding Proteinsmedicine.anatomical_structureCD4 AntigensInternational immunology
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Severe hepatic injury in interleukin 18 (IL-18) transgenic mice: a key role for IL-18 in regulating hepatocyte apoptosis in vivo.

2004

Background: Interleukin 18 (IL-18) is a cytokine with pleiotropic activity that augments T helper 1 responses and cytotoxic activity of natural killer cells. Methods: To assess the function of IL-18 in vivo, we generated IL-18 transgenic (IL-18 Tg) mice under the control of a CD2 promoter/enhancer construct. Results: Macroscopically, IL-18 Tg mice showed reduced relative liver weight compared with wild-type littermates. TUNEL assays demonstrated increased hepatocyte apoptosis, and primary hepatocytes isolated from IL-18 Tg mice exhibited an increased spontaneous apoptosis rate. Furthermore, cross linking of Fas increased significantly the apoptosis rate in hepatocytes isolated from wild- ty…

Genetically modified mousemedicine.medical_specialtyTransgeneT-LymphocytesApoptosisMice TransgenicMice SCIDBiologyTransfectionTranslocation GeneticInterferon-gammaMiceIn vivoInternal medicinemedicineCytotoxic T cellAnimalsfas ReceptorL-SelectinCells CulturedLiver injuryTumor Necrosis Factor-alphaGastroenterologyInterleukin-18NF-kappa BOrgan Sizemedicine.diseaseAdoptive TransferEndocrinologymedicine.anatomical_structureLiverApoptosisHepatocyteLymphocyte TransfusionCancer researchHepatocytesInterleukin 18Gut
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Disrupting immune regulation incurs transient costs in male reproductive function.

2014

9 pages; International audience; BACKGROUND: Immune protection against pathogenic organisms has been shown to incur costs. Previous studies investigating the cost of immunity have mostly focused on the metabolic requirements of immune maintenance and activation. In addition to these metabolic costs, the immune system can induce damage to the host if the immune response is mis-targeted or over-expressed. Given its non-specific nature, an over-expressed inflammatory response is often associated with substantial damage for the host. Here, we investigated the cost of an over-expressed inflammatory response in the reproductive function of male mice. METHODOLOGY/PRINCIPAL FINDINGS: We experimenta…

Genetics and Molecular Biology (all)0106 biological sciencesMalemedicine.medical_treatmentlcsh:MedicineBiochemistry01 natural sciencesMiceMonoclonalReceptorsTestis[ SDV.EE.IEO ] Life Sciences [q-bio]/Ecology environment/Symbiosis[ SDV.IMM ] Life Sciences [q-bio]/ImmunologyReceptors Interleukin-10Animals; Antibodies Monoclonal; Body Weight; Inflammation; Male; Mice; Organ Size; Receptors Interleukin-10; Reproduction; Spermatozoa; Testis; Immunomodulation; Medicine (all); Biochemistry Genetics and Molecular Biology (all); Agricultural and Biological Sciences (all)lcsh:ScienceReceptorImmune Response0303 health sciencesMultidisciplinaryReproductive functionMedicine (all)ReproductionAging and ImmunityAntibodies MonoclonalOrgan SizeSpermatozoaInterleukin-103. Good healthCytokineCytokines[SDV.IMM]Life Sciences [q-bio]/Immunologymedicine.symptomResearch ArticleInfertilityEvolutionary ImmunologyImmunologyInflammationBiology010603 evolutionary biologyImmune SuppressionAntibodiesImmunomodulation03 medical and health sciencesImmune systemImmunitymedicineAnimalsBiology030304 developmental biologyInflammationEvolutionary Biology[ SDE.BE ] Environmental Sciences/Biodiversity and Ecologylcsh:RBody WeightImmunityImmunoregulationmedicine.diseaseBlockadeAgricultural and Biological Sciences (all)Immune SystemImmunologyHumoral Immunitylcsh:Q[SDE.BE]Environmental Sciences/Biodiversity and Ecology[SDV.EE.IEO]Life Sciences [q-bio]/Ecology environment/Symbiosis
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Interleukin-10 promoter polymorphism in sporadic Alzheimer's disease.

2003

Proinflammatory cytokines and acute-phase proteins play an important role in Alzheimer's disease (AD) neurodegeneration, and common polymorphisms of genes controlling their high production have been shown to be associated with AD. Thus, AD patients display a proinflammatory genotype and the control of inflammation might play a protective role in AD development. By sequence-specific probes, we have evaluated the role of anti-inflammatory cytokine interleukin(IL)-10 in AD, by analysing in 132 AD patients and 213 healthy controls the prevalence of three different haplotypes, involving three single-nucleotide polymorphisms (SNPs) at -1082 (G--A), -819 (C--T) and -592 (C--A) nucleotides of IL-10…

GeneticsInflammationImmunologyHaplotypeInterleukinSingle-nucleotide polymorphismBiologyProinflammatory cytokineInterleukin-10Interleukin 10Alzheimer DiseaseImmunologyGenotypeGeneticsSNPHumansAllelePromoter Regions GeneticGenetics (clinical)Genes and immunity
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