Search results for "interleukin-1"

showing 10 items of 660 documents

IL-10 and TGF-β1 gene polymorphisms in Greek patients with recurrent aphthous stomatitis.

2022

Recurrent aphthous stomatitis (RAS) is one of the most frequent inflammatory disorders of the oral mucosa. Cytokines, which play an important role in RAS pathogenesis, participate directly or indirectly in normal, immunological and inflammatory processes and are secreted from cells belonging to innate and adaptive immunity as a consequence of microbial and antigenic stimuli. Gene polymorphisms in specific cytokines may predispose to RAS development. The aim of this study was the investigation and association of IL-10 and TGF-β1 gene polymorphisms with RAS.Study's cohort consisted of 60 Greek patients diagnosed with RAS, including 40 patients with minor, 10 patients with major and 10 with he…

GenotypeGreecePolymorphism Single NucleotideInterleukin-10Transforming Growth Factor beta1OtorhinolaryngologyCase-Control StudiesHumansSurgeryGenetic Predisposition to DiseaseStomatitis Aphthousrecurrent aphthous stomatitis aphthae IL-10 TGF-β gene polymorphisms oral mucosaCodonGeneral DentistryUNESCO:CIENCIAS MÉDICASMedicina oral, patologia oral y cirugia bucal
researchProduct

GM-CSF restores innate, but not adaptive, immune responses in glucocorticoid-immunosuppressed human blood in vitro.

2003

Abstract Infection remains the major complication of immunosuppressive therapy in organ transplantation. Therefore, reconstitution of the innate immunity against infections, without activation of the adaptive immune responses, to prevent graft rejection is a clinically desirable status in transplant recipients. We found that GM-CSF restored TNF mRNA and protein expression without inducing IL-2 production and T cell proliferation in glucocorticoid-immunosuppressed blood from either healthy donors or liver transplant patients. Gene array experiments indicated that GM-CSF selectively restored a variety of dexamethasone-suppressed, LPS-inducible genes relevant for innate immunity. A possible ex…

Graft RejectionLipopolysaccharidesT-LymphocytesCell Cycle ProteinsCell SeparationOrgan transplantationDexamethasoneMiceCDC2-CDC28 KinasesConcanavalin ATumor Cells CulturedImmunology and AllergySkin TransplantationMiddle AgedCyclin-Dependent KinasesUp-RegulationSurvival Ratemedicine.anatomical_structureImmunity ActiveTumor necrosis factor alphaGlucocorticoidCell DivisionCyclin-Dependent Kinase Inhibitor p27Immunosuppressive Agentsmedicine.drugAdultmedicine.medical_specialtyT cellImmunologyDown-RegulationBiologyProtein Serine-Threonine KinasesImmune systemAdjuvants ImmunologicIn vivomedicineAnimalsHumansDexamethasoneAgedSalmonella Infections AnimalInnate immune systemTumor Suppressor ProteinsCyclin-Dependent Kinase 2Granulocyte-Macrophage Colony-Stimulating FactorImmunity InnateGene Expression RegulationImmunologyLeukocytes MononuclearMice Inbred CBAInterleukin-2Interleukin-1Journal of immunology (Baltimore, Md. : 1950)
researchProduct

Uterine rejection after allogeneic uterus transplantation in the rat is effectively suppressed by tacrolimus

2013

Objective To evaluate the effects of the immunosuppressant tacrolimus on rejection of a transplanted uterus and on uterine expression of markers of inflammation and implantation. Design Experimental study. Setting University laboratory. Animal(s) Female rats. Intervention(s) Uteri from brown Norway rats were transplanted to Lewis rats, receiving either tacrolimus or no treatment. Sham groups underwent either hemihysterectomy or tacrolimus treatment. Main Outcome Measure(s) Gross morphology, histology, density of T-lymphocytes by immunohistochemistry, and mRNA levels of interleukin (IL)-1α, leukemia inhibitory factor (LIF), galectin-1, CD200, IL-15, interferon-inducible protein-10 (IP-10), a…

Graft Rejectionmedicine.medical_specialtyNecrosisGalectin 1UterusHysterectomyTacrolimusAndrologyNecrosisInterleukin-1alphaRats Inbred BNInternal medicineUterus transplantationmedicineAnimalsTransplantation HomologousInflammationbusiness.industryUterusObstetrics and GynecologyInterleukinHistologyOrgan TransplantationTacrolimusRatsChemokine CXCL10Transplantationsurgical procedures operativeEndocrinologymedicine.anatomical_structureReproductive MedicineRats Inbred LewFemalemedicine.symptombusinessLeukemia inhibitory factorBiomarkersImmunosuppressive AgentsFertility and Sterility
researchProduct

The many roads to inflammatory bowel diseases.

2006

Two independent studies by Rakoff-Nahoum et al. (2006) and Uhlig et al. (2006) in this issue of Immunity have illuminated a unique pathogenic role of innate immunity via Toll-like receptor and interleukin-23 signaling, respectively, in intestinal inflammation. These data define new roads to gut inflammation and future avenues for therapy.

Gut inflammationInnate immune systemInterleukinsImmunologyToll-Like ReceptorsInflammatory Bowel DiseasesBiologyInflammatory Bowel DiseasesInterleukin-12Interleukin-23Immunity InnateInfectious DiseasesImmunityIntestinal inflammationImmunologyImmunology and AllergyAnimalsSignal TransductionImmunity
researchProduct

HBV-specific immune defect in chronic hepatitis B (CHB) is correlated with a dysregulation of pro- and anti-inflammatory cytokines.

1999

SUMMARY The aim of this study was to examine the immunomodulating effects of rhIL-12 on the immune response induced by hepatitis B virus (HBV) antigens in clinical subgroups of patients with HBV infection. Peripheral blood mononuclear cells (PBMC) of 80 patients were stimulated with HBsAg, HBcAg, pre-S1Ag and tetanus toxoid in the absence or presence of IL-12 (0.01, 0.1 and 1 ng/ml). Stimulation by anti-CD3 + anti-CD28 and lipopolysaccharide (LPS) were used as controls. Proliferation and cytokine production were determined by 3H-thymidine uptake and ELISA after 72 h. After stimulation with HBV antigens only, production of tumour necrosis factor-alpha (TNF-α) or IL-10 was observed in all pat…

HBsAgHepatitis B virusImmunologyAntigen-Presenting CellsIn Vitro Techniquesmedicine.disease_causeLymphocyte ActivationHepatitis B AntigensInterferon-gammaHepatitis B ChronicOrthohepadnavirusmedicineImmunology and AllergyHumansHepatitis B AntibodiesHepatitisHepatitis B virusbiologybusiness.industryTumor Necrosis Factor-alphavirus diseasesOriginal ArticlesHepatitis Bmedicine.diseasebiology.organism_classificationVirologyInterleukin-12digestive system diseasesRecombinant ProteinsInterleukin-10HBcAgHBeAgHepadnaviridaeImmunologyDNA ViralLeukocytes MononuclearCytokinesInflammation MediatorsbusinessClinical and experimental immunology
researchProduct

Human cytomegalovirus (HCMV)-specific CD4+ T lymphocyte response in AIDS patients with no past or current HCMV disease following HAART.

2003

Abstract Background: The incidence of Human Cytomegalovirus (HCMV) end-organ disease has dramatically decreased since the implementation of highly active antiretroviral therapies (HAARTs), but the precise immune mechanism whereby HCMV is controlled remains to be elucidated. Objectives: To investigate the effect of (HAART) on CD4 + T-cell immunity to HCMV in AIDS patients with no past or current HCMV disease. Study design: Seventeen patients were prospectively examined for CD4 + (CD45RO + and CD45 RA + ) T-cell counts (flow cytometry), HIV RNA load (Amplicor HIV test), HCMV leukoDNAemia and HCMV DNA in urine (nested PCR), lymphoproliferative response (LPR) to HCMV, phytohemagglutinin (PHA) a…

Human cytomegalovirusAdultCD4-Positive T-LymphocytesMalevirusesCytomegalovirusmedicine.disease_causeLymphocyte ActivationHerpesviridaeVirusInterferon-gammaBetaherpesvirinaeT-Lymphocyte SubsetsVirologyImmunopathologyAntiretroviral Therapy Highly ActivemedicineHumansViremiaAcquired Immunodeficiency SyndromebiologyAIDS-Related Opportunistic Infectionsvirus diseasesHIVbiochemical phenomena metabolism and nutritionMiddle AgedViral Loadbiology.organism_classificationmedicine.diseaseVirologyCD4 Lymphocyte CountInterleukin-10Infectious DiseasesImmunologyCytomegalovirus InfectionsDNA ViralCytokinesRNA ViralCytokine secretionFemaleViral diseaseInterleukin-4Lymphoproliferative responseJournal of clinical virology : the official publication of the Pan American Society for Clinical Virology
researchProduct

The Role of Interleukin 10 in the Regulation of Allergic Immune Responses

2001

Several clinical studies and animal models have shown that Th2 lymphocytes play a key role in the pathophysiology of IgE-mediated allergic immune responses like allergic rhinitis and asthma or venom anaphylaxis. Classical specific immunotherapy (SIT) that has been proven to be clinically effective can serve as a role model for immunological changes that are associated with amelioration of allergic diseases. During SIT, the Th2-dominated immune response is modified towards a Th1 response leading to a decline in allergen-specific IgE and an increase in allergen-specific IgG production. Most importantly, however, production of the immunosuppressive/-regulatory cytokine interleukin 10 (IL-10) i…

Hypersensitivity ImmediateAllergyT-Lymphocytesmedicine.medical_treatmentT cellImmunologyImmunoglobulin EImmune toleranceAtopyImmune systemHumansImmunology and AllergyMedicineClonal Anergybiologybusiness.industryDendritic CellsGeneral MedicineImmunotherapymedicine.diseaseInterleukin-10Interleukin 10medicine.anatomical_structureDesensitization ImmunologicImmunologybiology.proteinbusinessInternational Archives of Allergy and Immunology
researchProduct

Lymphokine profile and activation pattern of two unrelated antigen- or idiotype-specific T suppressor cell clones.

1992

Two T suppressor (Ts) clones of different specificity have been analyzed for their lymphokine spectrum. BVI/5 is an I-Ek-restricted bovine serum albumin (BSA)-specific Ts cell clone from a CBA/J mouse tolerized by low doses of BSA. It affects directly or indirectly the function of BSA-specific T helper (Th) cells. The Ts cell clone 178-4 from a BALB/c mouse is I-Ed restricted and recognizes the public J558 Id on B cells. It prevents alpha(1----3)dextran B 1355S (Dex)-specific IgG antibody production and drives Dex-specific J558 idiotype-bearing B cells into an anergic B IgG memory cell state. Both Ts cell clones thus cause specific suppression, yet in different experimental systems using di…

IdiotypeMalemedicine.medical_treatmentImmunologyLymphocyte ActivationT-Lymphocytes RegulatoryInterferon-gammaMiceAntigenInterferonAntibody SpecificityCell ClonemedicineImmunology and AllergyAnimalsAntigensLymphokinesCD40biologyLymphokineHistocompatibility Antigens Class IISerum Albumin BovineT-Lymphocytes Helper-InducerMolecular biologyClone CellsCytokineImmunologybiology.proteinMice Inbred CBAClone (B-cell biology)medicine.drugInterleukin-1European journal of immunology
researchProduct

Interferon-α Abrogates Tolerance Induction by Human Tolerogenic Dendritic Cells

2011

Background Administration of interferon-α (IFN-α) represents an approved adjuvant therapy as reported for malignancies like melanoma and several viral infections. In malignant diseases, tolerance processes are critically involved in tumor progression. In this study, the effect of IFN-α on tolerance induction by human tolerogenic dendritic cells (DC) was analyzed. We focussed on tolerogenic IL-10-modulated DC (IL-10 DC) that are known to induce anergic regulatory T cells (iTregs). Methodology/Principal Findings IFN-α promoted an enhanced maturation of IL-10 DC as demonstrated by upregulation of the differentiation marker CD83 as well as costimulatory molecules. IFN-α treatment resulted in an…

Immune CellsT cellImmunologylcsh:MedicineAntigen-Presenting CellsPriming (immunology)Adaptive ImmunityBiologyLymphocyte ActivationImmune SuppressionT-Lymphocytes RegulatoryImmunophenotypingImmune toleranceImmune ActivationImmunomodulationImmune TolerancemedicineHumansCytotoxic T celllcsh:ScienceAntigen-presenting cellBiologyImmune ResponseClonal AnergyMultidisciplinaryClonal anergyT Cellslcsh:RImmunityImmunoregulationInterferon-alphaCell DifferentiationDendritic CellsInterleukin-10Tolerance inductionmedicine.anatomical_structureImmune SystemImmunologyCancer researchCytokineslcsh:QImmunizationCD8Research ArticlePLoS ONE
researchProduct

Suppressor activity of anergic T cells induced by IL-10-treated human dendritic cells: association with IL-2- and CTLA-4-dependent G1 arrest of the c…

2003

We have previously shown that human IL-10-treated dendritic cells (DC) induce an antigen-specific anergy in CD4+ T lymphocytes. These anergic T cells are characterized by an inhibited proliferation, a reduced production of IL-2, and additionally display antigen-specific suppressor activity. In this study we investigated the mechanisms underlying the anergic state and regulatory function of these T cells. We did not observe enhanced rates of programmed cell death of anergic CD4+ suppressor T cells compared to T cells stimulated with mature DC. Cell cycle analysis by DNA staining and Western blot experiments revealed an arrest of anergic CD4+ T suppressor cells in the G1 phase. High levels of…

ImmunoconjugatesRegulatory T cellT-LymphocytesImmunologyApoptosisCell Cycle ProteinsAbataceptCyclin-dependent kinaseAntigens CDmedicineImmunology and AllergyHumansCTLA-4 AntigenIL-2 receptorClonal AnergybiologyTumor Suppressor ProteinsRetinoblastoma proteinDendritic cellDendritic CellsCell cycleAntigens DifferentiationCell biologyInterleukin-10Interleukin 10medicine.anatomical_structurebiology.proteinInterleukin-2CDK inhibitorCell DivisionCyclin-Dependent Kinase Inhibitor p27European journal of immunology
researchProduct