Search results for "interleukin-2"

showing 10 items of 269 documents

Differential Regulatory Capacity of CD25+ T Regulatory Cells and Preactivated CD25+ T Regulatory Cells on Development, Functional Activation, and Pro…

2004

Abstract CD25+ T regulatory (Treg) cells play a central role regarding the maintenance of peripheral tolerance via suppression of autoaggressive CD4+ T cells, CD8+ T cells, and Th1 cells. In this study we demonstrate that CD25+ Treg cells can also suppress the differentiation of murine conventional CD4+ T cells toward Th2 cells in a contact-dependent manner. However, the cytokine production and proliferation of established Th2 cells could not be inhibited by freshly isolated CD25+ Treg cells, whereas a strong inhibition of differentiated Th2 cells by in vitro preactivated CD25+ Treg cells could be observed. Inhibition of both conventional CD4+ T cells and Th2 cells is accompanied by a stron…

CD4-Positive T-LymphocytesImmunologySuccinimideschemical and pharmacologic phenomenaLymphocyte ActivationMiceInterleukin 21Th2 CellsT-Lymphocyte SubsetsAnimalsImmunology and AllergyCytotoxic T cellIL-2 receptorAntigen-presenting cellInterleukin 3Mice Inbred BALB CCD40biologyPeripheral toleranceForkhead Transcription FactorsReceptors Interleukin-2hemic and immune systemsFluoresceinsCell biologyDNA-Binding ProteinsMice Inbred C57BLbiology.proteinInterleukin 12CytokinesThe Journal of Immunology
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Nitric oxide enhances Th9 cell differentiation and airway inflammation

2014

International audience; Th9 cells protect hosts against helminthic infection but also mediate allergic disease. Here we show that nitric oxide (NO) promotes Th9 cell polarization of murine and human CD4(+) T cells. NO de-represses the tumour suppressor gene p53 via nitrosylation of Mdm2. NO also increases p53-mediated IL-2 production, STAT5 phosphorylation and IRF4 expression, all essential for Th9 polarization. NO also increases the expression of TGFβR and IL-4R, pivotal to Th9 polarization. OVA-sensitized mice treated with an NO donor developed more severe airway inflammation. Transferred Th9 cells induced airway inflammation, which was exacerbated by NO and blocked by anti-IL-9 antibody.…

CD4-Positive T-LymphocytesInterleukin 2[SDV]Life Sciences [q-bio]Cellular differentiationNitric Oxide Synthase Type IIGeneral Physics and AstronomyMice TransgenicInflammationCell SeparationNitric OxideArticleGeneral Biochemistry Genetics and Molecular BiologyNitric oxideMicechemistry.chemical_compoundEosinophiliaSTAT5 Transcription FactormedicineAnimalsHumansInterleukin 9Cells CulturedInflammationMice Inbred BALB CMultidisciplinarybiologyNitrosylationInterleukin-9Cell DifferentiationGeneral Chemistryrespiratory systemFlow Cytometry3. Good healthCell biologyMice Inbred C57BLchemistryInterferon Regulatory FactorsImmunologyLeukocytes Mononuclearbiology.proteinInterleukin-2Mdm2Tumor Suppressor Protein p53medicine.symptomAntibodymedicine.drugNature Communications
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Murine liver antigen presenting cells control suppressor activity of CD4+CD25+ regulatory T cells.

2005

CD4(+)CD25(+) regulatory T cells (Treg) are important mediators of peripheral immune tolerance; however, whether Treg participate also in hepatic immune tolerance is not clear. Therefore, we tested the potential of Treg to suppress stimulation of CD4(+) T cells by liver sinusoidal endothelial cells (LSEC), Kupffer cells (KC), or hepatocytes. In the absence of Treg, all 3 types of liver cells could stimulate CD4(+) T cell proliferation; in the presence of Treg, however, CD4(+) T cell proliferation was suppressed. Interaction with KC even stimulated the expansion of the Treg population; LSEC or hepatocytes, in contrast, could not induce proliferation of Treg. Because liver inflammation can be…

CD4-Positive T-LymphocytesLiver cytologyKupffer CellsT cellT-LymphocytesAntigen-Presenting Cellschemical and pharmacologic phenomenaBiologyImmune toleranceMiceAntigenmedicineImmune ToleranceAnimalsIL-2 receptorAntigen-presenting cellCell ProliferationInflammationHepatologyLiver cellKupffer cellEndothelial Cellshemic and immune systemsReceptors Interleukin-2medicine.anatomical_structureLiverImmunologyCancer researchHepatocytesHepatology (Baltimore, Md.)
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Epicutaneous and Oral Low-Zone Tolerance Protects from Colitis in Mice

2016

Tolerance to environmental antigens that encounter the organism at interfaces like skin or gut prevents deleterious systemic immune responses. The aim of this study was to analyze whether and how low doses of haptens, by entry through the skin or gastrointestinal tract, affect the outcome of the predominantly Th1/Th17-mediated 2,4,6-trinitro-benzenesulfonic acid-induced colitis, which mimics an autoimmune bowl disease in man. Epicutaneous and oral applications of low doses of the allergen resulted in the induction of low-zone tolerance (LZT) and protected from colitis development, demonstrated by a significantly reduced inflammatory response of the gut in vivo. In line with this observation…

CD4-Positive T-LymphocytesMale0301 basic medicineAdoptive cell transferT cellAdministration Oralchemical and pharmacologic phenomenaDermatologyAdministration CutaneousDermatitis ContactT-Lymphocytes RegulatoryBiochemistryImmune toleranceMice03 medical and health sciences0302 clinical medicineImmune systemAntigenImmune TolerancemedicineAnimalsHumansIL-2 receptorColitisMolecular Biologybusiness.industryInterleukin-2 Receptor alpha SubunitFOXP3Cell BiologyAllergensColitismedicine.diseaseAdoptive TransferInterleukin-10Disease Models Animal030104 developmental biologymedicine.anatomical_structure030220 oncology & carcinogenesisImmunologyFemalebusinessJournal of Investigative Dermatology
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The Programmed Death (PD)‐1/PD‐Ligand 1 Pathway Regulates Graft‐Versus‐Host‐Reactive CD8 T Cells After Liver Transplantation

2008

Acute graft-versus-host disease (aGVHD) is a life-threatening complication after solid-organ transplantation, which is mediated by host-reactive donor T cells emigrating from the allograft. We report on two liver transplant recipients who developed an almost complete donor chimerism in peripheral blood and bone marrow-infiltrating T cells during aGVHD. By analyzing these T cells directly ex vivo, we found that they died by apoptosis over time without evidence of rejection by host T cells. The host-versus-donor reactivity was selectively impaired, as anti-third-party and antiviral T cells were still detectable in the host repertoire. These findings support the acquired donor-specific allotol…

CD4-Positive T-LymphocytesMaleCell TransplantationProgrammed Cell Death 1 ReceptorGraft vs Host DiseaseCD8-Positive T-LymphocytesTCIRG1MiceInterleukin 21Immune systemAntigenAntigens CDAnimalsHumansImmunology and AllergyCytotoxic T cellMedicinePharmacology (medical)IL-2 receptorMice KnockoutTransplantationbusiness.industryInterleukin-2 Receptor alpha SubunitForkhead Transcription FactorsMiddle AgedLiver TransplantationTransplantationsurgical procedures operativeGene Expression RegulationAntigens SurfaceImmunologyInterleukin 12Apoptosis Regulatory ProteinsbusinessImmunosuppressive AgentsAmerican Journal of Transplantation
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Activated glycoprotein A repetitions predominant (GARP)-expressing regulatory T cells inhibit allergen-induced intestinal inflammation in humanized m…

2015

Background Recently, we developed a humanized mouse model of allergen-induced IgE-dependent gut inflammation in PBMC-engrafted immunodeficient mice. Objective In the present study, we wanted to investigate the role of regulatory T (Treg) cells and their activation status in this model. Methods Nonobese diabetic-severe combined immunodeficiency-γc −/− mice were injected intraperitoneally with human PBMCs from allergic donors together with the respective allergen or NaCl as control in the presence or absence of different concentrations of CD4 + CD25 + Treg cells of the same donor. After an additional allergen boost 1 week later, mice were challenged with the allergen rectally on day 21 and gu…

CD4-Positive T-LymphocytesMalemedicine.medical_treatmentImmunologyInflammationNodMice SCIDBiologyImmunoglobulin ET-Lymphocytes RegulatoryMicemedicineHypersensitivityImmunology and AllergyAnimalsHumansIL-2 receptorAntibodies BlockingCells CulturedCell ProliferationImmunosuppression TherapyInflammationSevere combined immunodeficiencyInterleukin-2 Receptor alpha SubunitMembrane Proteinshemic and immune systemsForkhead Transcription FactorsDendritic cellAllergensImmunoglobulin Emedicine.diseaseIntestinesDisease Models AnimalCytokineImmunologyHumanized mouseAntibody FormationCD4 Antigensbiology.proteinLeukocytes MononuclearFemalemedicine.symptomThe Journal of allergy and clinical immunology
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Induction of CD4+/CD25+ regulatory T cells by targeting of antigens to immature dendritic cells

2003

AbstractCoupling of ovalbumin (OVA) to anti–DEC-205 monoclonal antibody (mAb) (αDEC) induced the proliferation of OVA-specific T cells in vivo. Expansion was short-lived, caused by dendritic cells (DCs), and rendered T cells anergic thereafter. Phenotypic analysis revealed the induction of CD25+/CTLA-4+ T cells suppressing proliferation and interleukin-2 (IL-2) production of effector CD4+ T cells. The findings were supported by 2 disease models: (1) CD4+ T-cell–mediated hypersensitivity reactions were suppressed by the injection of αDEC-OVA and (2) the application of hapten-coupled αDEC-205 reduced CD8+ T-cell–mediated allergic reactions. Thus, targeting of antigens to immature DCs through …

CD4-Positive T-LymphocytesOvalbuminT-LymphocytesImmunologyCD8-Positive T-LymphocytesDermatitis ContactLymphocyte ActivationBiochemistryMiceInterleukin 21Antigens CDHypersensitivityAnimalsCytotoxic T cellCTLA-4 AntigenHypersensitivity DelayedLymphocyte CountIL-2 receptorAntigensAntigen-presenting cellAntigen PresentationMice Inbred BALB CCD40biologyAntibodies MonoclonalReceptors Interleukin-2Dendritic CellsCell BiologyHematologyDendritic cellNatural killer T cellAntigens DifferentiationCell biologyImmunologyInterleukin 12biology.proteinInterleukin-2HaptensBlood
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miR-155 inhibition sensitizes CD4+ Th cells for TREG mediated suppression.

2009

BackgroundIn humans and mice naturally occurring CD4(+)CD25(+) regulatory T cells (nTregs) are a thymus-derived subset of T cells, crucial for the maintenance of peripheral tolerance by controlling not only potentially autoreactive T cells but virtually all cells of the adaptive and innate immune system. Recent work using Dicer-deficient mice irrevocably demonstrated the importance of miRNAs for nTreg cell-mediated tolerance.Principal findingsDNA-Microarray analyses of human as well as murine conventional CD4(+) Th cells and nTregs revealed a strong up-regulation of mature miR-155 (microRNA-155) upon activation in both populations. Studying miR-155 expression in FoxP3-deficient scurfy mice …

CD4-Positive T-LymphocytesScienceImmunology/ImmunomodulationBiologyModels BiologicalT-Lymphocytes RegulatoryImmune tolerancemiR-155MiceDownregulation and upregulationImmune ToleranceAnimalsHumansIL-2 receptorOligonucleotide Array Sequence AnalysisMultidisciplinaryInnate immune systemGenetics and Genomics/Functional GenomicsQInterleukin-2 Receptor alpha SubunitRPeripheral toleranceFOXP3Forkhead Transcription FactorsTransfectionImmunity InnateCell biologyUp-RegulationKineticsMicroRNAsImmunologyImmunology/Immune ResponseMedicineGenetics and Genomics/Genetics of the Immune SystemResearch ArticlePLoS ONE
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Modulation of proliferation and lymphokine secretion of murine CD4+ T cells and cloned Th1 cells by proteins of the extracellular matrix.

1997

In this study we investigated the co-stimulatory signaling capacity of diverse proteins of the extracellular matrix (ECM) for murine resting CD4+ T cells and Th1 clone cells, activated by immobilized anti-CD3 mAb. ECM proteins used in various concentrations had no effect on IL-2 production or proliferation of highly purified CD4+ T cell populations. When the preparation of CD4+ T cells contained contaminating accessory cells, IL-2 secretion and proliferation was enhanced in the presence of co-immobilized collagens or fibronectin. However, the level of proliferation attainable by added irradiated splenocytes was not reached. Using Th1 cell clone M4, enhanced production of IL-2 in the presenc…

CD4-Positive T-LymphocytesT cellImmunologyLymphocyte ActivationExtracellular matrixInterleukin 21MicemedicineImmunology and AllergyCytotoxic T cellAnimalsSecretionAntigen-presenting cellExtracellular Matrix ProteinsLymphokinesMice Inbred BALB CMice Inbred C3HbiologyChemistryIntegrin beta1LymphokineReceptors Interleukin-2General MedicineTh1 CellsMolecular biologyCell biologyClone CellsFibronectinMice Inbred C57BLmedicine.anatomical_structurebiology.proteinInternational immunology
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NFATc2 and NFATc3 transcription factors play a crucial role in suppression of CD4+ T lymphocytes by CD4+ CD25+ regulatory T cells

2005

The phenotype of NFATc2(-/-) c3(-/-) (double knockout [DKO]) mice implies a disturbed regulation of T cell responses, evidenced by massive lymphadenopathy, splenomegaly, and autoaggressive phenomena. The population of CD4(+) CD25(+) T cells from DKO mice lacks regulatory capacity, except a small subpopulation that highly expresses glucocorticoid-induced tumor necrosis factor receptor family-related gene (GITR) and CD25. However, neither wild-type nor DKO CD4(+) CD25(+) regulatory T cells (T reg cells) are able to suppress proliferation of DKO CD4(+) CD25(-) T helper cells. Therefore, combined NFATc2/c3 deficiency is compatible with the development of CD4(+) CD25(+) T reg cells but renders c…

CD4-Positive T-LymphocytesT cellImmunologyPopulationchemical and pharmacologic phenomenaReceptors Nerve Growth FactorBiologyLymphocyte ActivationReceptors Tumor Necrosis FactorInterleukin 21MiceT-Lymphocyte SubsetsGlucocorticoid-Induced TNFR-Related ProteinmedicineImmunology and AllergyCytotoxic T cellAnimalsIL-2 receptorReceptoreducationTranscription factorMice Knockouteducation.field_of_studyNFATC Transcription FactorsZAP70Brief Definitive ReportNuclear Proteinshemic and immune systemsReceptors Interleukin-2Molecular biologyCoculture TechniquesDNA-Binding Proteinsmedicine.anatomical_structureTranscription FactorsThe Journal of Experimental Medicine
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