Search results for "interleukin"

showing 10 items of 1856 documents

A mutation in the second intracellular loop of the pituitary adenylate cyclase activating polypeptide type I receptor confers constitutive receptor a…

2000

AbstractThe pituitary adenylate cyclase activating polypeptide (PACAP) type I receptor belongs to the glucagon/secretin/vasoactive intestinal polypeptide (VIP) receptor family. We mutated and deleted an amino acid residue (E261) which is located within the second intracellular loop of the rat PACAP type I receptor and which is highly conserved among the receptor family. The wild-type receptor and the mutant receptors were efficiently expressed at the surface of COS-7 cells at nearly the same level and revealed the same high affinity for the agonist PACAP-27. The cAMP contents of COS cells transfected with the E261A, E261Q, and the deletion mutant receptor were 4.6-, 5.7-, and 6.7-fold highe…

endocrine systemGrowth-hormone-releasing hormone receptorMolecular Sequence DataReceptors Pituitary Adenylate Cyclase-Activating PolypeptideBiophysicsGlutamic AcidSignal transductionTransfectionBiochemistryBeta-1 adrenergic receptorConstitutive activityStructural BiologycAMPCyclic AMPGeneticsEnzyme-linked receptorAnimals5-HT5A receptorAmino Acid SequenceReceptors Pituitary HormoneMolecular BiologySequence DeletionPeptide hormone receptorSite-directed mutagenesisPituitary adenylate cyclase activating polypeptideChemistryLiver receptor homolog-1Cell BiologyMolecular biologyRatsInterleukin-21 receptorCOS CellsMutagenesis Site-DirectedEstrogen-related receptor gammaSequence AlignmentGlucagon receptor familyhormones hormone substitutes and hormone antagonistsAdenylyl CyclasesReceptors Pituitary Adenylate Cyclase-Activating Polypeptide Type IFEBS Letters
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Human interleukin-6 facilitates hepatitis B virus infection in vitro and in vivo.

2000

Abstract Background and aim. Research on hepatitis B virus (HBV) infection in vivo has been limited due to the absence of a suitable animal model. We have developed a human–mouse radiation chimera in which normal mice, preconditioned by lethal total body irradiation and radioprotected with SCID mouse bone marrow cells, are permissive for engraftment of human hematopoietic cells and solid tissues. This resulting human–mouse model, which comprises three genetically disparate sources of tissue, is therefore termed Trimera. This study was aimed at assessing the effect of human IL-6 on HBV infection in vivo in Trimera mice. Methods. Trimera mice were transplanted with human liver tissue fragment…

endocrine systemHepatitis B virusMice SCIDmedicine.disease_causeVirus ReplicationMiceIn vivoVirologymedicineAnimalsHumansHepatitis B virusbiologychimeric miceInterleukin-6Hepatitis BVirologyMolecular biologydigestive system diseasesIn vitroTransplantationDisease Models Animalmedicine.anatomical_structureCell cultureRadiation Chimerabiology.proteinviral infectionBone marrowAntibodyviral receptorEx vivoVirology
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IL-17 and TNF-α Are Key Mediators of Moraxella catarrhalis Triggered Exacerbation of Allergic Airway Inflammation

2017

Alterations of the airway microbiome is often associated with pulmonary diseases. For example, detection of the bacterial pathogen Moraxella catarrhalis in the upper airways is linked with an increased risk to develop or exacerbate asthma. However, the mechanisms by which M. cattarhalis augments allergic airway inflammation (AAI) remains unclear. We here characterized the cellular and soluble mediators of M. catarrhalis triggered excacerbation of AAI in wt and IL-17 deficient as well as in animals treated with TNF-alpha and IL-6 neutralizing antibodies. We compared the type of inflammatory response in M. catarrhalis infected, HDM-allergic and animals infected with M. catarrhalis at differen…

exacerbation of allergic reactionslcsh:Immunologic diseases. Allergy0301 basic medicineExacerbationT cellImmunologyexacerbation of pulmonary inflammationpulmonary inflammationMoraxella catarrhalisAllergic sensitization03 medical and health sciences0302 clinical medicineMoraxellaceae infectionsinfection and allergyImmunology and AllergyMedicineSensitizationOriginal Researchbiologybusiness.industrymicrobial exacerbation of pulmonary inflammationbiology.organism_classificationIL-17030104 developmental biologymedicine.anatomical_structureTNF-αImmunologyTumor necrosis factor alphaInterleukin 17lcsh:RC581-607businessAirway030215 immunologyFrontiers in Immunology
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Regulatory cytokine gene polymorphisms and risk of colorectal carcinoma.

2006

It is well established that cancer arises in chronically inflamed tissue, and this is particularly notable in the gastrointestinal tract. Classic examples include Helicobacter pylori-associated gastric cancer, hepatocellular carcinoma, and inflammatory bowel disease-associated colorectal cancer. Growing evidence suggests that these associations might be not casual findings. Focusing on individual cytokines has generated evidence that anti-inflammatory cytokine interleukin (IL)-10 and transforming growth factor-beta1 (TGF-beta1) may have a complex role in gastrointestinal carcinogenesis. As an example, IL-10-deficient mice develop severe atrophic gastritis and a chronic enterocolitis, develo…

gene polymorphismsMaleRiskProlineColorectal cancerAtrophic gastritisil-10colorectal cancerMouse model of colorectal and intestinal cancerBiologymedicine.disease_causePolymorphism Single NucleotideGeneral Biochemistry Genetics and Molecular BiologyMetastasisTransforming Growth Factor beta1colorectal cancercytokine genepolymorphismsHistory and Philosophy of ScienceGene FrequencyLeucineGenotypemedicineHumansGenetic Predisposition to DiseaseAllelesGeneral Neurosciencetgf-β1CarcinomaCancermedicine.diseaseInterleukin-10Amino Acid SubstitutionItalyTumor progressionCase-Control StudiesImmunologycolorectal cancer; gene polymorphisms; il-10; tgf-β1FemaleCarcinogenesisColorectal NeoplasmsAnnals of the New York Academy of Sciences
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Metallic nanoparticles exhibit paradoxical effects on oxidative stress and pro-inflammatory response in endothelial cells in vitro

2007

Particulate matter is associated with different human diseases affecting organs such as the respiratory and cardiovascular systems. Very small particles (nanoparticles) have been shown to be rapidly internalized into the body. Since the sites of internalization and the location of the detected particles are often far apart, a distribution via the blood stream must have occurred. Thus, endothelial cells, which line the inner surface of blood vessels, must have had direct contact with the particles. In this study we tested the effects of metallic nanoparticles (Co and Ni) on oxidative stress and proinflammatory response in human endothelial cells in vitro. Exposure to both nanoparticle types…

human diseasesmedia_common.quotation_subjectImmunologyNanoparticleCell CountInflammationBiologymedicine.disease_cause03 medical and health sciences0302 clinical medicineMicroscopy Electron TransmissionNickelmedicineHumansImmunology and AllergyParticle SizeCytotoxicityInternalizationCells CulturedChemokine CCL2media_commonInflammationPharmacologyInterleukin-8Endothelial CellsCobaltIntercellular Adhesion Molecule-1GlutathioneIn vitroCell biologyOxidative StressMetalsNanotoxicology030220 oncology & carcinogenesisNanoparticlesGentian Violetmedicine.symptomSignal transductionReactive Oxygen SpeciesOxidation-ReductionOxidative stress030215 immunology
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Effects of intrinsic aerobic capacity, aging and physical activity on interleukin-15 protein level in serum and skeletal muscle

2015

Myokines are cytokines and other peptides produced in muscle that can transmit autocrine, paracrine or endocrine signals to other tissues. This is a potential way for exercise to carry out its beneficial effects on different parts of the body. Interleukin-15 (IL-15) is a myokine that has been shown to exert beneficial effects on multiple tissues: skeletal muscle, adipose tissue, bone and skin. Muscle-derived IL-15 signaling remains to be fully elucidated. It is known that the expression and signaling is affected by aging, exercise, and intrinsic aerobic capacity. The purpose of this study was to assess the effects of intrinsic aerobic capacity, aging and physical activity separately and in …

ikääntymineninterleukiinitagingintrinsic running capacityphysical activityinterleukin-15human activitiesmyokinefyysinen aktiivisuusjuoksu
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Ultraviolet B light attenuates the systemic immune response in central nervous system autoimmunity

2014

Objective: Environmental conditions (eg, latitude) play a critical role in the susceptibility and severity of many autoimmune disorders, including multiple sclerosis (MS). Here, we investigated the mechanisms underlying the beneficial effects of immune regulatory processes induced in the skin by moderate ultraviolet B (UVB) radiation on central nervous system (CNS) autoimmunity. Methods: Effects of UVB light were analyzed in a murine model of CNS autoimmunity (experimental autoimmune encephalomyelitis). Additionally, patients with relapsing–remitting MS were treated with narrowband UVB phototherapy. Immunomodulatory effects were examined in skin biopsies, serum samples, and immune cells of …

integumentary systemmedicine.medical_treatmentExperimental autoimmune encephalomyelitischemical and pharmacologic phenomenaInflammationBiologymedicine.disease_causemedicine.diseaseAutoimmunityImmune toleranceInterleukin 21Immune systemCytokineNeurologyImmunityImmunologymedicineNeurology (clinical)medicine.symptomAnnals of Neurology
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IL-13 and IL-33 Serum Levels Are Increased in Systemic Sclerosis Patients With Interstitial Lung Disease

2022

ObjectiveSystemic sclerosis (SSc) mortality is extremely variable in its internal organ involvement. Pulmonary fibrosis occurs in up to 30% of the cases. Animal models provide evidence that IL-33 is able to induce both cutaneous and pulmonary fibrosis via increased IL-13 and in SSc patients the levels of IL-33 correlate with skin fibrosis. Our aim was to test whether both IL-33 and IL-13 are higher in patients with diffuse SSc and interstitial lung disease (SSc-ILD) compared to SSc patients without ILD and healthy controls.MethodsSerum levels of IL-13 and IL-33 were measured in 30 SSc patients with diffuse disease and 30 healthy controls by enzyme-linked immunosorbent assay. The extent of p…

interstitial lung diseaseMedicine (General)R5-920interleukinsintegumentary systemsystemic sclerosisIL-13IL-13; IL-33; interleukins; interstitial lung disease; systemic sclerosisIL-33General Medicinerespiratory systemskin and connective tissue diseases
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Human Memory Th17 Cell Populations Change Into Anti-inflammatory Cells With Regulatory Capacity Upon Exposure to Active Vitamin D

2019

Autoimmune diseases are characterized by an aberrantly activated immune system, resulting in tissue damage and functional disability in patients. An important therapeutic goal is to restore the deregulated immunological balance between pro- A nd anti-inflammatory T cells. This imbalance is illustrated by elevated levels and activity of memory Th17 cell populations, such as Th17, Th1/Th17, and Th17.1 cells, in various autoimmune diseases. These cells are characterized by the chemokine receptor CCR6, RORC expression and production of IL-17A, IFNγ, and TNFα. Using rheumatoid arthritis (RA) as a model of autoimmune disease, we here demonstrate that pro-inflammatory memory CCR6+ Th cells can swi…

lcsh:Immunologic diseases. Allergy0301 basic medicineAdultMaleReceptors CCR6rheumatoid arthritisCD3 ComplexCD3CellImmunologyAnti-Inflammatory Agentschemical and pharmacologic phenomenavitamin DC-C chemokine receptor type 6Autoimmune DiseasesArthritis Rheumatoid03 medical and health sciencesChemokine receptor0302 clinical medicineImmune systemsynovial fluidRAR-related orphan receptor gammamedicineImmunology and AllergyHumansCells CulturedOriginal ResearchAutoimmune diseasebiologyChemistryTumor Necrosis Factor-alphaInterleukinsMiddle Agedmedicine.diseaseTreg030104 developmental biologymedicine.anatomical_structureImmunologybiology.proteinTh17 CellsTumor necrosis factor alphaFemaleTh17lcsh:RC581-607Immunologic Memory030215 immunologyFrontiers in Immunology
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Reciprocal influence of B cells and tumor macro and microenvironments in the ApcMin/+ model of colorectal cancer

2017

One of the most fascinating aspects of the immune system is its dynamism, meant as the ability to change and readapt according to the organism needs. Following an insult, we assist to the spontaneous organization of different immune cells which cooperate, locally and at distance, to build up an appropriate response. Throughout tumor progression, adaptations within the systemic tumor environment, or macroenvironment, result in the promotion of tumor growth, tumor invasion and metastasis to distal organs, but also to dramatic changes in the activity and composition of the immune system. In this work, we show the changes of the B-cell arm of the immune system following tumor progression in the…

lcsh:Immunologic diseases. Allergy0301 basic medicineColorectal cancerImmunologySpleenintestinal cancerBiologylcsh:RC254-282Metastasis03 medical and health sciencesImmune systemPeritoneummedicineImmunology and Allergyapcmin/+ miceApcMin/+mice; B lymphocytes; IgA; IL-10; intestinal cancer; Immunology and Allergy; Immunology; OncologyB lymphocyteApcMin/+micelcsh:Neoplasms. Tumors. Oncology. Including cancer and carcinogensmedicine.diseasePhenotypeInterleukin 10030104 developmental biologymedicine.anatomical_structureOncologyTumor progressionIL-10Immunologyb lymphocyteslcsh:RC581-607IgAOncoImmunology
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