Search results for "lcsh:Neoplasms. Tumors. Oncology. Including cancer and carcinogens"

showing 10 items of 382 documents

Assessing molecular subtypes of gastric cancer: microsatellite unstable and Epstein-Barr virus subtypes. Methods for detection and clinical and patho…

2018

Background The molecular classification of gastric cancer recognises two subtypes prone to immune checkpoint blockade: the microsatellite unstable and the Epstein-Barr virus (EBV)-related tumours. We aim to assess the concordance between immunohistochemistry and PCR for microsatellite status evaluation, and explore the value of microsatellite instability (MSI) and EBV as predictive survival factors. Material and methods We collected 246 consecutively diagnosed gastric cancer cases in all stages and evaluated the microsatellite status using immunohistochemistry for mismatched repair (MMR) proteins and PCR. EBV expression was studied through in situ hybridisation. Results Forty-five (18%) cas…

OncologyCancer Researchmedicine.medical_specialtyConcordancemedicine.disease_causelcsh:RC254-282Internal medicinemedicine1506PathologicalOriginal Researchepstein-barr virusbusiness.industrygastric cancerCancerMicrosatellite instabilitylcsh:Neoplasms. Tumors. Oncology. Including cancer and carcinogensmedicine.diseaseEpstein–Barr virusImmune checkpointdigestive system diseasesOncologyImmunohistochemistryMicrosatellitemicrosatellite instabilitybusinessESMO open
researchProduct

In the literature: October 2019

2019

Gastrointestinal cancers are a subset of molecularly heterogeneous diseases. In the era of personalised medicine, major efforts are being made towards stratifying patients according to molecular profiling. However, although most treatments are currently based on targeted therapy in relation to specific genomic alterations, acquired resistance emerges during anticancer therapies and subsequently treatment failure occurs. Intratumour heterogeneity plays a significant role in the acquisition of resistance by clonal evolution of tumour cell populations under therapeutic pressure. Despite a single tumour biopsy represents the standard for cancer research and drives our therapeutic decisions, lim…

OncologyCancer Researchmedicine.medical_specialtyMetastatic lesionsmedicine.diagnostic_testbusiness.industrymedicine.medical_treatmentDisease progressionliteratureNewslcsh:Neoplasms. Tumors. Oncology. Including cancer and carcinogensmedicine.diseaselcsh:RC254-282Somatic evolution in cancerMetastasisTargeted therapyTumour tissueOncologyBiopsy SiteInternal medicineBiopsymedicine1506businessESMO Open
researchProduct

In the literature: April 2019

2019

Glioblastoma (GBM) remains an unmet need in Medical Oncology considering its poor prognosis and the lack of advances in therapeutics in more than one decade.1 Despite the initial enthusiasm, the development of immunotherapy in GBM has proved to be challenging, with a disappointing negative phase III clinical trial.2 Some of the phenotypic hallmarks of GBM make immunotherapy difficult. Its relatively low mutational load, its immunologically ‘cold’ microenvironment with scarce infiltrating immune effector cells, a dominant myeloid compartment composed by microglia and myeloid-derived suppressor cells and a strong immunosuppression, both local, mediated by immunosuppressive regulatory T cells …

OncologyCancer Researchmedicine.medical_specialtyMyeloidMicrogliabusiness.industrymedicine.medical_treatmentliteratureImmunosuppressionPembrolizumabImmunotherapyNewslcsh:Neoplasms. Tumors. Oncology. Including cancer and carcinogenslcsh:RC254-282medicine.anatomical_structureImmune systemOncologyInternal medicinemedicine1506NivolumabbusinessDexamethasonemedicine.drugESMO Open
researchProduct

Treatment of Lenalidomide Exposed or Refractory Multiple Myeloma: Network Meta-Analysis of Lenalidomide-Sparing Regimens.

2021

Over the past 10 years, the treatment of multiple myeloma (MM) dramatically changed due to the introduction of a number of new agents and combination regimens both in the frontline and in the relapsed/refractory setting. Currently, at least 11 classes of therapeutic agents, including steroids, alkylators (melphalan and cyclophosphamide), proteasome inhibitors (PI: bortezomib, carfilzomib, ixazomib), immunomodulatory agents (thalidomide, lenalidomide, pomalidomide), monoclonal antibodies (mAbs: elotuzumab, daratumumab), HDAC-inhibitors (panobinostat), BCL2 inhibitors (venetoclax), selective inhibitors of nuclear export (selinexor), drug-conjugated mAbs (belantamab mafodotin), bispecific agen…

OncologyCancer Researchmedicine.medical_specialtyOpinionlenalidomidelcsh:RC254-282chemistry.chemical_compoundInternal medicinemedicineLenalidomideIsatuximabcarfilzomibBortezomibbusiness.industrynetwork meta analysisbortezomibDaratumumabRefractory Multiple Myelomalcsh:Neoplasms. Tumors. Oncology. Including cancer and carcinogensdaratumumabCarfilzomibmyelomachemistryOncologyMeta-analysisbusinessmedicine.drugisatuximabFrontiers in oncology
researchProduct

In the literature: October 2020.

2020

Immune checkpoint inhibitors are widely used as treatment for an increasing number of solid tumours. Nevertheless, the lack of predictive biomarker represents a limitation across several cancer types. During the last years, the possibility to dynamically study tumour evolution through circulating tumour DNA (ctDNA) in plasma has opened novel possibility in evaluating disease status and therapeutic response, especially in localised disease to predict the possibility of relapse. However, the specific opportunities for application in the context of immunotherapy remain to be clarified.1 In an article recently published in Cancer Discovery by Zhang et al ,2 a comprehensive analysis of ctDNA dat…

OncologyCancer Researchmedicine.medical_specialtyPrognostic variableDurvalumabbusiness.industryCancerMicrosatellite instabilityContext (language use)DiseaseNewsmedicine.diseaselcsh:Neoplasms. Tumors. Oncology. Including cancer and carcinogenslcsh:RC254-282not applicableOncologyInternal medicinemedicine1506Lung cancerbusinessTremelimumabmedicine.drugESMO open
researchProduct

Diffusion-weighted imaging in oncology: An update

2020

To date, diffusion weighted imaging (DWI) is included in routine magnetic resonance imaging (MRI) protocols for several cancers. The real additive role of DWI lies in the “functional” information obtained by probing the free diffusivity of water molecules into intra and inter-cellular spaces that in tumors mainly depend on cellularity. Although DWI has not gained much space in some oncologic scenarios, this non-invasive tool is routinely used in clinical practice and still remains a hot research topic: it has been tested in almost all cancers to differentiate malignant from benign lesions, to distinguish different malignant histotypes or tumor grades, to predict and/or assess treatment resp…

OncologyCancer Researchmedicine.medical_specialtyReviewCancer imaginglcsh:RC254-282030218 nuclear medicine & medical imaging03 medical and health sciences0302 clinical medicineMagnetic resonance imagingInternal medicinemedicineEffective diffusion coefficientcardiovascular diseasesapparent diffusion coefficient; cancer imaging; diffusion weighted imaging; magnetic resonance imaging; oncologic imagingmedicine.diagnostic_testbusiness.industryMagnetic resonance imagingDiffusion weighted imaginglcsh:Neoplasms. Tumors. Oncology. Including cancer and carcinogensOncologic imagingClinical PracticeOncologyApparent diffusion coefficient030220 oncology & carcinogenesisCancer imagingbusinessDiffusion MRI
researchProduct

In the literature: June 2019

2019

Biliary tract cancer (BTC) includes cholangiocarcinoma and gallbladder cancer. BTCs are known to have a poor prognosis, with a 5-year overall survival below 20%.1 Unfortunately, majority of patients are diagnosed with advanced stage, being palliative chemotherapy with cisplatin and gemcitabine the current standard of care.2 Poor prognosis is due to the fact that only 20% of patients are diagnosed in early stages3 and the high risk of relapse following curative surgery. Unfortunately, the lack of randomised studies has made the role of adjuvant treatment in BTC following surgery an unresolved matter for many years.4 5 Adjuvant therapy (either in the form of chemotherapy or chemoradiotherapy)…

OncologyCancer Researchmedicine.medical_specialtybusiness.industryliteratureGemOxNewsmedicine.diseaselcsh:Neoplasms. Tumors. Oncology. Including cancer and carcinogenslcsh:RC254-282GemcitabineBile duct cancerOxaliplatinCapecitabineOncologyInternal medicinemedicineAdjuvant therapy1506Gallbladder cancerbusinessChemoradiotherapymedicine.drugESMO Open
researchProduct

Correction to: Assessment of intratumor immune-microenvironment in colorectal cancers with extranodal extension of nodal metastases

2019

No data is available on the molecular background of the extra-nodal extension (ENE) of lymph node metastasis (LN) in colorectal cancer (CRC).A series of 22 ENE-positive CRCs was considered and three samples per case were selected (the primary CRC, an ENE-negative and an ENE-positive metastatic LN). Samples (n = 66) were analysed by immunohistochemistry for PD-L1, CD4, CD8, CD68 and CD80. Fifteen out of twenty-two cases were further profiled through a hotspot multigene mutational custom panel, including 164 hotspot regions ofA significantly higher percentage of CD4-, CD8- and CD68-positive cells was observed at the invasive front of both CRCs and in ENE in contrast with what observed at the …

OncologyCancer Researchmedicine.medical_specialtylcsh:Cytologybusiness.industryImmune microenvironmentExtranodal ExtensionCorrectionlcsh:Neoplasms. Tumors. Oncology. Including cancer and carcinogenslcsh:RC254-282not known03 medical and health sciences0302 clinical medicineOncology030220 oncology & carcinogenesisInternal medicineCorretionGeneticsmedicinelcsh:QH573-671medicine.symptomNODALbusinessConfusion
researchProduct

A polymorphism at the 3'-UTR region of the aromatase gene defines a subgroup of postmenopausal breast cancer patients with poor response to neoadjuva…

2009

Abstract Background Aromatase (CYP19A1) regulates estrogen biosynthesis. Polymorphisms in CYP19A1 have been related to the pathogenesis of breast cancer (BC). Inhibition of aromatase with letrozole constitutes the best option for treating estrogen-dependent BC in postmenopausal women. We evaluate a series of polymorphisms of CYP19A1 and their effect on response to neoadjuvant letrozole in early BC. Methods We analyzed 95 consecutive postmenopausal women with stage II-III ER/PgR [+] BC treated with neoadjuvant letrozole. Response to treatment was measured by radiology at 4th month by World Health Organization (WHO) criteria. Three polymorphisms of CYP19A1, one in exon 7 (rs700519) and two in…

OncologyCancer Researchmedicine.medical_specialtymedicine.drug_classmedicine.medical_treatmentAnastrozoleAntineoplastic AgentsBreast Neoplasmslcsh:RC254-282AromataseBreast cancerSurgical oncologyInternal medicineNitrilesGeneticsmedicineHumansAromatase3' Untranslated RegionsNeoadjuvant therapyAgedAged 80 and overPolymorphism GeneticAromatase inhibitorbiologybusiness.industryLetrozoleMiddle AgedTriazoleslcsh:Neoplasms. Tumors. Oncology. Including cancer and carcinogensmedicine.diseaseImmunohistochemistryNeoadjuvant TherapyPostmenopauseTreatment OutcomeEndocrinologyOncologyLetrozoleDisease Progressionbiology.proteinFemalebusinessTamoxifenResearch Articlemedicine.drugBMC Cancer
researchProduct

Moving the target on the optimal adjuvant strategy for resected pancreatic cancers: A systematic review with meta-analysis

2020

Combination regimens have shown superiority over single agents in the adjuvant treatment of resected pancreatic cancer (PC), but there are no data supporting definition of the best regimen. This work aimed to compare the efficacy and safety of mFOLFIRINOX, gemcitabine+capecitabine, and gemcitabine+nab/paclitaxel in PC patients. A meta-analysis was performed for direct comparison between trials comparing combination regimens and gemcitabine monotherapy. Subsequently, an indirect comparison was made between trials investigating the efficacy and safety of mFOLFIRINOX, gemcitabine+capecitabine, and gemcitabine+nab/paclitaxel because of the same control arm (gemcitabine). A total of three studie…

OncologyCancer Researchmedicine.medical_specialtymedicine.medical_treatmentReviewNeutropenialcsh:RC254-282Capecitabine03 medical and health scienceschemistry.chemical_compound0302 clinical medicinePancreatic cancerInternal medicinemedicineChemotherapyMeta-analysi030212 general & internal medicineAdjuvantChemotherapybusiness.industryMFOLFIRINOXPancreatic cancerlcsh:Neoplasms. Tumors. Oncology. Including cancer and carcinogensmedicine.diseaseGemcitabineRegimenMeta-analysisOncologyPaclitaxelchemistryAdjuvant Chemotherapy Meta-analysis MFOLFIRINOX Pancreatic cancer Systematic review030220 oncology & carcinogenesisSystematic reviewbusinessAdjuvantmedicine.drug
researchProduct