Search results for "lcsh:Neurology. Diseases of the nervous system"

showing 10 items of 162 documents

Standardised Neuropsychological Assessment for the Selection of Patients Undergoing DBS for Parkinson’s Disease

2017

DBS is an increasingly offered advanced treatment for Parkinson’s disease (PD). Neuropsychological assessment is considered to be an important part of the screening for selection of candidates for this treatment. However, no standardised screening procedure currently exists. In this study, we examined the use of our standardised neuropsychological assessment for the evaluation of surgical candidates and to identify risk factors for subsequent decline in cognition and mood. A total of 40 patients were assessed before and after DBS. Evaluation of mood and case notes review was also undertaken. Before DBS, patients with PD demonstrated frequent impairments in intellectual functioning, memory, …

Article Subjectbehavioral disciplines and activitieslcsh:Neurology. Diseases of the nervous systemlcsh:RC346-429Research ArticleParkinson's Disease
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Altered neural responses to social fairness in bipolar disorder

2020

Highlights • Bipolar disorder is characterized by impaired processing of social fairness. • BD patients exhibit increased rejection of moderate unfairness in Ultimatum Game. • BD patients display decreased response to moderate unfairness in anterior insula. • BD patients deactivate posterior and middle insula in response to unfairness. • Trait impulsivity positively correlated with deactivations in posterior insula.

Audiologylcsh:RC346-4290302 clinical medicineSocial decision makingRATING-SCALEBRAINSocial informationhealth care economics and organizationsBrain Mappingmedicine.diagnostic_test05 social sciencesRegular ArticleMagnetic Resonance ImaginghumanitiesNeurologyFMRIECONOMIC DECISION-MAKINGlcsh:R858-859.7Fairness ; Bipolar disorder ; Ambiguity ; Ultimatum game ; Social decision-makingmedicine.symptomPsychologyPsychosocialpsychological phenomena and processesUltimatum gamemedicine.medical_specialtyAmbiguityFairnessSocial decision-makingBipolar disorderCognitive NeuroscienceDecision MakingImpulsivitylcsh:Computer applications to medicine. Medical informaticsbehavioral disciplines and activities050105 experimental psychologyMECHANISMS03 medical and health sciencesmental disordersmedicineContextual informationHumans0501 psychology and cognitive sciencesRadiology Nuclear Medicine and imagingBipolar disorderSocial BehaviorINSULAMETAANALYSISlcsh:Neurology. Diseases of the nervous systemPERFORMANCEmedicine.diseaseGames ExperimentalIMPULSIVENESSNeurology (clinical)Functional magnetic resonance imagingInsula030217 neurology & neurosurgeryNeuroImage: Clinical
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Transcriptomic metaanalyses of autistic brains reveals shared gene expression and biological pathway abnormalities with cancer

2019

Este es el artículo que se ha publicado de forma definitiva en: https://molecularautism.biomedcentral.com/articles/10.1186/s13229-019-0262-8 En este artículo también participa Joan Climent, Vera Pancaldi, Lourdes Fañanás, Celso Arango, Mara Parellada, Anaïs Baudot, Daniel Vogt, John L. Rubenstein, Alfonso Valencia y Rafael Tabarés-Seisdedos. Background: Epidemiological and clinical evidence points to cancer as a comorbidity in people with autism spectrum disorders (ASD). A significant overlap of genes and biological processes between both diseases has also been reported. Methods: Here, for the first time, we compared the gene expression profiles of ASD frontal cortex tissues and 22 cancer t…

Autism.AutismComorbidityBioinformaticsAutismo.lcsh:RC346-429Expresión génica.TranscriptomeAfectividad - Trastornos.0302 clinical medicineNeoplasmsGene expression2.1 Biological and endogenous factorsAetiologyCàncerCáncer - Aspectos genéticos.ComputingMilieux_MISCELLANEOUSCancer0303 health sciencesProstate CancerBrainAffective disorders.3. Good healthPsychiatry and Mental healthMental HealthSignal transductionSignal TransductionBiotechnologyUrologic DiseasesIntellectual and Developmental Disabilities (IDD)1.1 Normal biological development and functioningClinical SciencesBiologyASDBiological pathway03 medical and health sciencesDevelopmental NeuroscienceUnderpinning researchmental disordersGeneticsmedicineHumansAutistic DisorderIntellectual and Developmental DisabilitiesGeneMolecular Biologylcsh:Neurology. Diseases of the nervous systemPI3K/AKT/mTOR pathwayCancer - Genetic aspects.030304 developmental biologyResearchNeurosciencesMultimorbidityCancermedicine.diseaseExpressió gènicaHuman geneticsBrain DisordersMeta-analysisGene expression.AutismGene expressionAutisme[INFO.INFO-BI]Computer Science [cs]/Bioinformatics [q-bio.QM]TranscriptomeKidney cancer030217 neurology & neurosurgeryDevelopmental BiologyMolecular Autism
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Impaired circadian heart rate variability in Parkinson’s disease: a time-domain analysis in ambulatory setting

2020

Abstract Background Heart rate variability (HRV) decreases in Parkinson’s disease (PD) and it can be considered a marker for cardiovascular dysautonomia. The purpose of this pilot study is to evaluate long-term time-domain analysis of HRV of PD patients and compare the results with those of matched healthy individuals. Methods Idiopathic PD patients without comorbidity impairing HRV, and age-matched healthy individuals were recruited in a pilot study. A long-term time domain analysis of HRV using 24-h ambulatory ECG was performed. Results Overall, 18 PD patients fulfilling inclusion criteria completed the evaluation (mean age was 55.6 ± 8.8, disease duration: 5.0 ± 4.7). Mean SCOPA-AUT scor…

Autonomic disordersmedicine.medical_specialtyParkinson's diseaseNeurologyAutonomic disorderPopulationPilot ProjectsNon-motor symptomsPrimary DysautonomiasAutonomic disorderlcsh:RC346-429Antiparkinson AgentsLevodopaHeart RateInternal medicinemedicineHumansHeart rate variabilityeducationlcsh:Neurology. Diseases of the nervous systemAgededucation.field_of_studybusiness.industryDysautonomiaParkinson DiseaseGeneral MedicineMiddle Agedmedicine.diseaseComorbidityCircadian RhythmHeart rate variability SCOPA-AUTCardiovascular DiseasesAmbulatoryCardiologyNeurology (clinical)medicine.symptombusinessResearch ArticleBMC Neurology
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Lipofuscin Hypothesis of Alzheimer’s Disease

2011

The primary culprit responsible for Alzheimer’s disease (AD) remains unknown. Aβ protein has been identified as the main component of amyloid of senile plaques, the hallmark lesion of AD, but it is not definitively established whether the formation of extracellular Aβ deposits is the absolute harbinger of the series of pathological events that hit the brain in the course of sporadic AD. The aim of this paper is to draw attention to a relatively overlooked age-related product, lipofuscin, and advance the hypothesis that its release into the extracellular space following the death of neurons may substantially contribute to the formation of senile plaques. The presence of intraneuronal Aβ, sim…

Aβ proteinNeurofibrillary tanglesAmyloidAmyloidCognitive Neurosciencelcsh:Geriatricslcsh:RC346-429LipofuscinLipofuscinLesionExtracellularMedicineOriginal Research ArticleSenile plaquesPathologicallcsh:Neurology. Diseases of the nervous systembusiness.industryMacular degenerationAlzheimer's diseaseMacular degenerationmedicine.diseaseBiochemistry of Alzheimer's diseaselcsh:RC952-954.6Psychiatry and Mental healthmedicine.symptombusinessAlzheimer’s diseaseNeuroscienceDementia and Geriatric Cognitive Disorders Extra
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Clinico-Immunological Status and Neurocognitive Function of Perinatally Acquired HIV-Positive Children on cART: A Cross-Sectional Correlational Study…

2020

Despite the undisputed benefits of combination antiretroviral therapy (cART), perinatally acquired human immunodeficiency virus (PHIV) children on treatment often present with a spectrum of neurological deficits known as HIV-associated neurocognitive impairment. Even higher CD4 cell count does not seem to prevent the development of neurocognitive impairment in children with PHIV. While CD4 cell count has shown to have the greatest prognostic value, its association with neurocognitive abilities remains to be clarified. This study aimed at determining the correlation between plasma CD4+ lymphocyte and neurocognitive function in children with PHIV on cART. In total, 152 purposively recruited h…

CartPediatricsmedicine.medical_specialtyWechsler Preschool and Primary Scale of Intelligencebusiness.industryWorking memorycombination antiretroviral therapy (cART)Psychological interventionCognitionlcsh:RC346-429neurocognitive deficitsperinatally acquired HIV (PHIV)Neurologyimmunological statusneurocognitive assessmentMedicineNeurology (clinical)Early childhoodbusinessNeurocognitivePsychosocialneurological deficitslcsh:Neurology. Diseases of the nervous systemplasma CD4+ lymphocyte countOriginal ResearchFrontiers in neurology
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ALS-linked FUS mutations confer loss and gain of function in the nucleus by promoting excessive formation of dysfunctional paraspeckles

2019

Mutations in the FUS gene cause amyotrophic lateral sclerosis (ALS-FUS). Mutant FUS is known to confer cytoplasmic gain of function but its effects in the nucleus are less understood. FUS is an essential component of paraspeckles, subnuclear bodies assembled on a lncRNA NEAT1. Paraspeckles may play a protective role specifically in degenerating spinal motor neurons. However it is still unknown how endogenous levels of mutant FUS would affect NEAT1/paraspeckles. Using novel cell lines with the FUS gene modified by CRISPR/Cas9 and human patient fibroblasts, we found that endogenous levels of mutant FUS cause accumulation of NEAT1 isoforms and paraspeckles. However, despite only mild cytoplasm…

Cell NucleusResearchAmyotrophic Lateral SclerosisIntranuclear Inclusion BodiesNEAT1lcsh:RC346-429Cell LineLoss of Function MutationCell Line TumorFused in sarcoma (FUS)ParaspeckleHumansProtein IsoformsRNA-Binding Protein FUSRNA Long NoncodingAmyotrophic lateral sclerosis (ALS)CRISPR-Cas Systemslcsh:Neurology. Diseases of the nervous systemActa Neuropathologica Communications
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Single cell cultures of Drosophila neuroectodermal and mesectodermal central nervous system progenitors reveal different degrees of developmental aut…

2009

Abstract Background The Drosophila embryonic central nervous system (CNS) develops from two sets of progenitor cells, neuroblasts and ventral midline progenitors, which behave differently in many respects. Neuroblasts derive from the neurogenic region of the ectoderm and form the lateral parts of the CNS. Ventral midline precursors are formed by two rows of mesectodermal cells and build the CNS midline. There is plenty of evidence that individual identities are conferred to precursor cells by positional information in the ectoderm. It is unclear, however, how far the precursors can maintain their identities and developmental properties in the absence of normal external signals. Results To s…

Central Nervous Systemanimal structuresEmbryo NonmammalianCentral nervous systemEctodermApoptosisBiologylcsh:RC346-429MesodermNeuroblastDevelopmental NeurosciencePrecursor cellmedicineAnimalsDrosophila ProteinsCell LineageProgenitor celllcsh:Neurology. Diseases of the nervous systemCells CulturedEmbryonic Stem CellsBody PatterningNeural PlatefungiCell DifferentiationEmbryonic stem cellmedicine.anatomical_structureCell cultureembryonic structuresDrosophilaNeuroscienceDevelopmental biologyCell DivisionResearch ArticleNeural development
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Neuropsicología y anorexia nerviosa. Hallazgos cognitivos y radiológicos

2012

Resumen: Introducción: El estudio de las alteraciones neuropsicológicas halladas en pacientes diagnosticadas de anorexia nerviosa (AN) ha experimentado, a través de las últimas décadas, un espectacular avance proporcionando un amplio campo de conocimientos acerca de los cambios observados en la morfología cerebral y las capacidades cognitivas, así como de la reversibilidad o estabilidad de estos, conformando un perfil de alteración neuropsicológico característico de este trastorno de la conducta alimentaria. Desarrollo: Se presenta una revisión actualizada hasta diciembre de 2010 de los resultados obtenidos en la literatura acerca de las alteraciones, tanto de la morfología cerebral como de…

Clinical NeurologyNeurology (clinical)lcsh:Neurology. Diseases of the nervous systemlcsh:RC346-429Neurología
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Tratamiento del dolor orofacial en pacientes con síndrome del ligamento estilomandibular (síndrome de Ernest)

2013

Resumen: Introducción: El síndrome de Ernest se define como una alteración del ligamento estilomandibular, caracterizado por la presencia de dolor en la región preauricular y en el ángulo mandibular, irradiado al cuello, el hombro y el ojo del mismo lado, asociado a dolor durante la palpación del ligamento estilomandibular. El objetivo es presentar las características clínicas, el tratamiento y la evolución de una serie de pacientes con el síndrome de Ernest. Métodos: Se realizó un estudio clínico, observacional, retrospectivo, entre los años 1998 y 2008. Se recogieron todos los datos con respecto a la edad, el sexo, el tiempo de evolución y las características del dolor. A todos los pacien…

Clinical NeurologyNeurology (clinical)lcsh:Neurology. Diseases of the nervous systemlcsh:RC346-429Neurología
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