Search results for "lcsh:QP351-495"

showing 10 items of 18 documents

Targeting Voltage-Dependent Calcium Channels with Pregabalin Exerts a Direct Neuroprotective Effect in an Animal Model of Multiple Sclerosis

2018

Background/aims Multiple sclerosis (MS) is a prototypical autoimmune central nervous system (CNS) disease. Particularly progressive forms of MS (PMS) show significant neuroaxonal damage as consequence of demyelination and neuronal hyperexcitation. Immuno-modulatory treatment strategies are beneficial in relapsing MS (RMS), but mostly fail in PMS. Pregabalin (Lyrica®) is prescribed to MS patients to treat neuropathic pain. Mechanistically, it targets voltage-dependent Ca2+ channels and reduces harmful neuronal hyperexcitation in mouse epilepsy models. Studies suggest that GABA analogues like pregabalin exert neuroprotective effects in animal models of ischemia and trauma. Methods We tested t…

0301 basic medicineCentral nervous systemPregabalinPregabalinPharmacologyNeuroprotectionlcsh:RC346-429Multiple sclerosis03 medical and health sciencesCellular and Molecular NeuroscienceDevelopmental Neurosciencemedicinelcsh:Neurology. Diseases of the nervous systemExperimental autoimmune encephalomyelitisMicrogliaVoltage-dependent calcium channelbusiness.industryMultiple sclerosislcsh:QP351-495Experimental autoimmune encephalomyelitismedicine.diseaseNeuroprotectionlcsh:Neurophysiology and neuropsychology030104 developmental biologymedicine.anatomical_structureNeurologyNeuropathic painbusinessmedicine.drugNeurosignals
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26th Annual Computational Neuroscience Meeting (CNS*2017): Part 2

2017

International audience; No abstract available

0301 basic medicineCerebellumComputer science[SDV]Life Sciences [q-bio]General Neurosciencelcsh:QP351-495Meeting Abstractslcsh:RC321-57103 medical and health sciencesCellular and Molecular Neurosciencelcsh:Neurophysiology and neuropsychology030104 developmental biologymedicine.anatomical_structuremedicineNeuronlcsh:Neurosciences. Biological psychiatry. NeuropsychiatryNeuroscienceComputingMilieux_MISCELLANEOUScomputational neuroscienceBMC Neuroscience
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Linoleic acid: Is this the key that unlocks the quantum brain? Insights linking broken symmetries in molecular biology, mood disorders and personalis…

2017

Abstract In this paper we present a mechanistic model that integrates subneuronal structures, namely ion channels, membrane fatty acids, lipid rafts, G proteins and the cytoskeleton in a dynamic system that is finely tuned in a healthy brain. We also argue that subtle changes in the composition of the membrane’s fatty acids may lead to down-stream effects causing dysregulation of the membrane, cytoskeleton and their interface. Such exquisite sensitivity to minor changes is known to occur in physical systems undergoing phase transitions, the simplest and most studied of them is the so-called Ising model, which exhibits a phase transition at a finite temperature between an ordered and disorde…

0301 basic medicinePhase transitionLinoleic acidMood DisorderModels NeurologicalPhysical systemAntidepressantContext (language use)MicrotubuleReviewlcsh:RC321-57103 medical and health sciencesCellular and Molecular Neuroscience0302 clinical medicineAntidepressants; Cytoskeleton; Depression; Ion channels; Ising model; Linoleic acid; Lipid raft; Microtubule; Mood disorders; Quantum states; Linoleic Acid; Mood Disorders; Brain; Models Neurological; Neuroscience (all); Cellular and Molecular NeuroscienceIsing modelCytoskeletonlcsh:Neurosciences. Biological psychiatry. NeuropsychiatryLipid raftQuantumIon channelCytoskeletonNeuroscience (all)ChemistryDepressionGeneral Neurosciencelcsh:QP351-495BrainQuantum statesMood disorders Linoleic acid Ion channels Cytoskeleton Microtubule Lipid raft Depression Antidepressants Ising model Quantum statesAntidepressantsQuantum stateLipid raftlcsh:Neurophysiology and neuropsychology030104 developmental biologyIon channelsMood disordersIsing modelIon channelNeuroscience030217 neurology & neurosurgery
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Alterations of perineuronal nets in the dorsolateral prefrontal cortex of neuropsychiatric patients

2019

Abstract Background Alterations in the structure and physiology of interneurons in the prefrontal cortex (PFC) are important factors in the etiopathology of different psychiatric disorders. Among the interneuronal subpopulations, parvalbumin (PV) expressing cells appear to be specially affected. Interestingly, during development and adulthood the connectivity of these interneurons is regulated by the presence of perineuronal nets (PNNs), specialized regions of the extracellular matrix, which are frequently surrounding PV expressing neurons. Previous reports have found anomalies in the density of PNNs in the PFC of schizophrenic patients. However, although some studies have described alterat…

0301 basic medicinePsychosisBipolar disorderPerineuronal netsPrefrontal cortexlcsh:RC321-57103 medical and health sciences0302 clinical medicinemental disordersNeuroplasticitymedicineMajor depressionPsiquiatriaBipolar disorderPrefrontal cortexlcsh:Neurosciences. Biological psychiatry. NeuropsychiatrySalut mentalBiological PsychiatryParvalbuminbiologyResearchPerineuronal netlcsh:QP351-495medicine.diseaseDorsolateral prefrontal cortexPsychiatry and Mental healthlcsh:Neurophysiology and neuropsychology030104 developmental biologymedicine.anatomical_structurenervous systemSchizophreniaSchizophreniabiology.proteinEsquizofrèniaNeuroscience030217 neurology & neurosurgeryParvalbuminInternational Journal of Bipolar Disorders
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Synaptic scaling generically stabilizes circuit connectivity

2011

Neural systems regulate synaptic plasticity avoiding overly strong growth or shrinkage of the connections, thereby keeping the circuit architecture operational. Accordingly, several experimental studies have shown that synaptic weights increase only in direct relation to their current value, resulting in reduced growth for stronger synapses [1]. It is, however, difficult to extract from these studies unequivocal evidence about the underlying biophysical mechanisms that control weight growth. The theoretical neurosciences have addressed this problem by exploring mechanisms for synaptic weight change that contain limiting factors to regulate growth [2]. The effectiveness of these mechanisms i…

573.8Computer science612.8612Plasticity573530lcsh:RC321-57103 medical and health sciencesCellular and Molecular NeuroscienceSynaptic weight0302 clinical medicineHomeostatic plasticityBiological neural networklcsh:Neurosciences. Biological psychiatry. Neuropsychiatry030304 developmental biology0303 health sciencesSynaptic scalingGeneral NeuroscienceWeight changelcsh:QP351-495Hebbian theorylcsh:Neurophysiology and neuropsychologyPoster PresentationSynaptic plasticityNeuroscience030217 neurology & neurosurgeryBMC Neuroscience
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Chronic fluoxetine treatment in middle-aged rats induces changes in the expression of plasticity-related molecules and in neurogenesis

2012

Abstract Background Antidepressants promote neuronal structural plasticity in young-adult rodents, but little is known of their effects on older animals. The polysialylated form of the neural cell adhesion molecule (PSA-NCAM) may mediate these structural changes through its anti-adhesive properties. PSA-NCAM is expressed in immature neurons and in a subpopulation of mature interneurons and its expression is modulated by antidepressants in the telencephalon of young-adult rodents. Results We have analyzed the effects of 14 days of fluoxetine treatment on the density of puncta expressing PSA-NCAM and different presynaptic markers in the medial prefrontal cortex, hippocampus and amygdala of mi…

Doublecortin Domain ProteinsMaleTelencephalonmedicine.medical_specialtyDoublecortin ProteinVesicular glutamate transporter 1NeurogenesisGlutamate decarboxylaseSynaptophysinHippocampusSubventricular zoneCell CountNeural Cell Adhesion Molecule L1Hippocampal formationSubgranular zonelcsh:RC321-571Cellular and Molecular NeuroscienceInternal medicineFluoxetineLateral VentriclesmedicineAnimalsRats Wistarlcsh:Neurosciences. Biological psychiatry. NeuropsychiatryCell ProliferationbiologyGlutamate DecarboxylaseGeneral NeuroscienceNeurogenesisBody WeightNeuropeptideslcsh:QP351-495DoublecortinRatsEndocrinologymedicine.anatomical_structureKi-67 Antigenlcsh:Neurophysiology and neuropsychologyGene Expression Regulationnervous systemVesicular Glutamate Transport Protein 1biology.proteinSialic AcidsAntidepressive Agents Second-GenerationNeuroscienceMicrotubule-Associated ProteinsResearch ArticleBMC Neuroscience
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Atypical perceptual narrowing in prematurely born infants is associated with compromised language acquisition at 2 years of age

2010

Abstract Background Early auditory experiences are a prerequisite for speech and language acquisition. In healthy children, phoneme discrimination abilities improve for native and degrade for unfamiliar, socially irrelevant phoneme contrasts between 6 and 12 months of age as the brain tunes itself to, and specializes in the native spoken language. This process is known as perceptual narrowing, and has been found to predict normal native language acquisition. Prematurely born infants are known to be at an elevated risk for later language problems, but it remains unclear whether these problems relate to early perceptual narrowing. To address this question, we investigated early neurophysiolog…

First languageBrain mappingDevelopmental psychology0302 clinical medicineDiscrimination PsychologicalSurveys and QuestionnairesBRAIN10. No inequalityCerebral CortexBrain MappingLanguage TestsNEWBORNSGeneral Neurosciencelcsh:QP351-495ElectroencephalographySignal Processing Computer-AssistedLanguage acquisitionPARADIGMLanguage developmentChild PreschoolAuditory PerceptionPsychologyInfant PrematureResearch ArticleBIRTH515 PsychologyeducationPOTENTIALSPRETERM CHILDRENLanguage Developmentlcsh:RC321-57103 medical and health sciencesCellular and Molecular NeuroscienceLanguage assessment030225 pediatricsPerceptual narrowingHumansSpeechNOVELTYlcsh:Neurosciences. Biological psychiatry. NeuropsychiatryAnalysis of VarianceMEMORYInfant NewbornInfantlcsh:Neurophysiology and neuropsychologyAcoustic StimulationWORDSOn Language030217 neurology & neurosurgerySpoken languageFollow-Up StudiesBMC Neuroscience
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Infant Event-Related Potentials to Speech are Associated with Prelinguistic Development

2020

Highlights • Speech processing and prelinguistic skills studied in a large longitudinal sample. • Auditory ERPs predicted prelinguistic development in infancy in LCS models. • P1 amplitude at 6 months predicted prelinguistic development between 6 and 12 months. • MMR to a frequency change was associated with prelinguistic skills at 6 months. • Infants’ neural speech processing can help to predict early language development.

Male6162 Cognitive scienceMismatch negativityCHILDRENCOMMUNICATIONAudiologyevent-related potentials0302 clinical medicinekielellinen kehitysprelinguistic skillsBRAIN10. No inequalityEvoked PotentialsOriginal ResearchChange scoreBASIC RESEARCHRISKinfantslcsh:QP351-49505 social sciencesLanguage developmentFemalePsychologyInfantsEvent-related potentialsDYSLEXIAmedicine.medical_specialtyPrelinguistic skills515 PsychologyCognitive Neuroscienceeducationlapset (ikäryhmät)Latent change score modelLanguage Developmentbehavioral disciplines and activities050105 experimental psychologylatent change score model03 medical and health sciencesEvent-related potentialmedicineHumans0501 psychology and cognitive sciencesMISMATCH NEGATIVITYAssociation (psychology)DyslexiaInfantLinguisticsmedicine.diseaseSpeech processingPseudowordlcsh:Neurophysiology and neuropsychologyDISCRIMINATIONLANGUAGE IMPAIRMENT030217 neurology & neurosurgeryRESPONSES
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Modulatory effects of nitric oxide-active drugs on the anticonvulsant activity of lamotrigine in an experimental model of partial complex epilepsy in…

2007

Abstract Background The effects induced by administering the anticonvulsant lamotrigine, the preferential inhibitor of neuronal nitric oxide synthase 7-nitroindazole and the precursor of NO synthesis L-arginine, alone or in combination, on an experimental model of partial complex seizures (maximal dentate gyrus activation) were studied in urethane anaesthetized rats. The epileptic activity of the dentate gyrus was obtained through the repetitive stimulation of the angular bundle and maximal dentate gyrus activation latency, duration and post-stimulus afterdischarge duration were evaluated. Results Either Lamotrigine (10 mg kg-1) or 7-nitroindazole (75 mg kg-1) i.p. administration had an ant…

MalePARTIAL COMPLEX EPILEPSYIndazolesArgininemedicine.medical_treatmentLamotriginePharmacologyArginineLamotrigineNitric OxideSettore BIO/09 - Fisiologialcsh:RC321-571Nitric oxideCellular and Molecular Neurosciencechemistry.chemical_compoundEpilepsy Complex PartialmedicineAnimalsDrug InteractionsEnzyme InhibitorsRats Wistarlcsh:Neurosciences. Biological psychiatry. NeuropsychiatryNitric oxide Lamotrigine epilepsy controlbiologyTriazinesExperimental modelGeneral NeuroscienceDentate gyruslcsh:QP351-495BrainElectric StimulationRatsNitric oxide synthaseDisease Models Animallcsh:Neurophysiology and neuropsychologyAnticonvulsantnervous systemchemistryDentate Gyrusbiology.proteinAnticonvulsantsNitric Oxide SynthaseResearch Articlemedicine.drugBMC Neuroscience
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Increase of Substance P Concentration in Saliva after Pharyngeal Electrical Stimulation in Severely Dysphagic Stroke Patients – an Indicator of Decan…

2017

Background/Aims: Substance P (SP) is a neuropeptide, likely acting as a neurotransmitter in the pharyngeal mucosa enhancing the swallow and cough reflex. Pharyngeal Electrical Stimulation (PES) induces a temporary increase of salivary SP levels in healthy adults. Previous evidence suggests that post-stroke dysphagia is related to reduced SP levels. Here, we investigated the effects of PES on SP levels in severely dysphagic stroke patients and a possible link between increase of SP and treatment success. Methods: 23 tracheotomized stroke patients who could not be decannulated due to severe and persisting dysphagia according to endoscopic evaluation received PES for 10 minutes a day over thre…

MaleSalivaStroke patientCough reflexStimulationSubstance PSubstance Plcsh:RC346-42903 medical and health sciencesCellular and Molecular Neurosciencechemistry.chemical_compoundTracheostomy0302 clinical medicinePharyngeal electrical stimulationDevelopmental NeurosciencemedicineHumansProspective Studies030212 general & internal medicineSalivaStrokelcsh:Neurology. Diseases of the nervous systemAgedAged 80 and overbusiness.industrylcsh:QP351-495Middle Agedmedicine.diseaseDysphagiaElectric StimulationStrokePESlcsh:Neurophysiology and neuropsychologyNeurologychemistryAnesthesiaTracheal decannulationPharynxBiomarker (medicine)Femalemedicine.symptomDeglutition Disordersbusiness030217 neurology & neurosurgeryNeurosignals
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