Search results for "lipas"

showing 10 items of 396 documents

Unexplained Elevated Serum Pancreatic Enzymes: A Reason to Suspect Celiac Disease

2006

BACKGROUND & AIMS: The frequency of elevated serum pancreatic enzymes in patients with celiac disease (CD) is unknown. The aim of this study was to evaluate the serum levels of pancreatic enzymes in CD patients. METHODS: Serum pancreatic isoamylase and lipase levels were assayed in 90 adult and 112 pediatric consecutive CD patients at diagnosis and after 12 months of gluten-free diet (GFD). Serum elastase and trypsin levels were assayed in a subgroup of adult CD patients. Pancreatic ultrasonography was also performed. RESULTS: Twenty-six adult (29%) and 29 pediatric (26%) CD patients exhibited elevated values of serum pancreatic amylase and/or lipase; trypsin was elevated in 69% and elastas…

AdultMalemedicine.medical_specialtyAbdominal painSettore MED/09 - Medicina InternaPancreatic diseaseAdolescentGlutensAsymptomaticInternal medicineDiabetes mellitusGFD gluten-free dietmedicineHumansTrypsinAmylaseLipaseChildPancreatic ElastaseHepatologybiologybusiness.industryElastaseGastroenterologyCase-control studyLipasemedicine.diseasetTGs transglutaminasePancreatic celiacCeliac DiseaseEmA anti-endomysiumEndocrinologyCase-Control StudiesChild PreschoolCD celiac diseaseAmylasesbiology.proteinFemalemedicine.symptomSD standard deviationbusinessIsoamylaseFollow-Up StudiesClinical Gastroenterology and Hepatology
researchProduct

Gender specific associations of the Trp64Arg mutation in the beta3-adrenergic receptor gene with obesity-related phenotypes in a Mediterranean popula…

2001

Abstract. Corella D, Guillen M, Portoles O, Sorli JV, Alonso V, Folch J, Saiz C (School of Medicine, University of Valencia, Valencia, Spain). Gender specific associations of the Trp64Arg mutation in the β3-adrenergic receptor gene with obesity-related phenotypes in a Mediterranean population: interaction with a common lipoprotein lipase gene variation. J Intern Med 2001; 250: 348–360. Objective. To investigate the association between the Trp64Arg β3-adrenergic receptor (ADRB3) mutation and obesity-related phenotypes in a Mediterranean Spanish population considering the effect of other genetic and environmental factors. Design and subject. Cross-sectional study in 1063 (476 men and 587 wome…

AdultMalemedicine.medical_specialtyAdolescentDNA Mutational AnalysisPopulationBiologyArginineGenetic determinismBody Mass IndexSex FactorsPolymorphism (computer science)Internal medicineGenotypeInternal MedicinemedicineHumansGenetic Predisposition to DiseaseObesityAlleleeducationAllelesAgedLipoprotein lipaseeducation.field_of_studyGenetic Carrier ScreeningBody WeightTryptophanGenetic VariationMiddle Agedmedicine.diseaseObesityLipoprotein LipasePhenotypeEndocrinologySpainReceptors Adrenergic beta-3FemaleBody mass indexJournal of Internal Medicine
researchProduct

Glycerophosphocholine is elevated in cerebrospinal fluid of Alzheimer patients.

2003

Experimental and clinical studies give evidence for breakdown of membrane phospholipids during neurodegeneration. In the present study, we measured the levels of glycerophosphocholine (GPCh), phosphocholine (PCh), and choline, that is, water-soluble metabolites of phosphatidylcholine (PtdCho), in human cerebrospinal fluid (CSF). Among 30 cognitively normal patients the average CSF levels of GPCh, phosphocholine and choline were 3.64, 1.28, and 1.93 microM, respectively; metabolite levels did not change with increasing age. When compared with age-matched controls, patients with Alzheimer's disease had elevated levels of all choline metabolites: GPCh was significantly increased by 76% (P<0.01…

AdultMalemedicine.medical_specialtyAgingAdolescentMetabolitePhosphorylcholineCholinechemistry.chemical_compoundPhospholipase A2Cerebrospinal fluidAlzheimer DiseasePhosphatidylcholineInternal medicinemedicineCholineHumansVascular dementiaPhosphocholineAgedAged 80 and overbiologyPhosphorylcholineGeneral NeuroscienceDementia VascularAge FactorsMiddle Agedmedicine.diseaseGlycerylphosphorylcholineEndocrinologychemistryImmunologybiology.proteinFemaleNeurology (clinical)Geriatrics and GerontologyBiomarkersDevelopmental BiologyNeurobiology of aging
researchProduct

Small and dense LDL in Familial Combined Hyperlipidemia and N291S polymorphism of the lipoprotein lipase gene

2009

18 pagesm 2 figures. -- PMID: 19335919 [PubMed]

AdultMalemedicine.medical_specialtyApolipoprotein BAdolescentEndocrinology Diabetes and MetabolismPopulationClinical BiochemistryHyperlipidemia Familial CombinedMutation MissenseBlood lipidschemistry.chemical_compoundYoung AdultHigh-density lipoproteinEndocrinologyInternal medicinePrevalenceMedicineShort PaperHumanseducationlcsh:RC620-627AgedBiochemistry medicalLipoprotein lipaseeducation.field_of_studyPolymorphism Geneticmedicine.diagnostic_testbiologyCholesterolbusiness.industryBiochemistry (medical)Cholesterol LDLMiddle AgedAtherosclerosisLipidslcsh:Nutritional diseases. Deficiency diseasesLipoprotein LipaseEndocrinologychemistrySpainLow-density lipoproteinCase-Control Studiesbiology.proteinFemalelipids (amino acids peptides and proteins)businessLipid profile
researchProduct

A Phase 3 Trial of Sebelipase Alfa in Lysosomal Acid Lipase Deficiency

2015

BackgroundLysosomal acid lipase is an essential lipid-metabolizing enzyme that breaks down endocytosed lipid particles and regulates lipid metabolism. We conducted a phase 3 trial of enzyme-replacement therapy in children and adults with lysosomal acid lipase deficiency, an underappreciated cause of cirrhosis and severe dyslipidemia. MethodsIn this multicenter, randomized, double-blind, placebo-controlled study involving 66 patients, we evaluated the safety and effectiveness of enzyme-replacement therapy with sebelipase alfa (administered intravenously at a dose of 1 mg per kilogram of body weight every other week); the placebo-controlled phase of the study was 20 weeks long and was followe…

AdultMalemedicine.medical_specialtyCirrhosisAdolescentHDLBiopsy[SDV]Life Sciences [q-bio]Lysosomal acid lipase deficiencyGastroenterologyLDLlaw.inventionYoung Adultchemistry.chemical_compoundDouble-Blind MethodRandomized controlled triallawInternal medicinemedicineHumansAdolescent; Adult; Aged; Alanine Transaminase; Biopsy; Child; Child Preschool; Cholesterol HDL; Cholesterol LDL; Double-Blind Method; Dyslipidemias; Female; Humans; Liver; Male; Middle Aged; Sterol Esterase; Wolman Disease; Young AdultChildPreschoolComputingMilieux_MISCELLANEOUSAgedDyslipidemiasbiologyCholesterolbusiness.industryWolman DiseaseAlanine TransaminaseLipid metabolismGeneral MedicineMiddle AgedSterol Esterasemedicine.disease3. Good healthCholesterolEndocrinologyLiverSebelipase alfachemistryAlanine transaminasebiology.proteinFemalebusinessDyslipidemia
researchProduct

Retrobulbar adipocytes and humoral immunity in Graves' ophthalmopathy

1993

As CT and MR-imaging revealed an enlargement of retrobulbar fat tissue in patients with Graves' ophthalmopathy, the role of the retrobulbar adipocytes in the pathogenesis of this disorder remains to be elucidated. To evaluate the in vitro influence of humoral immunity on retrobulbar adipose tissue, the effects of IgG and sera from 56 euthyroid ophthalmopathy patients and 53 controls on both porcine and human (patients' and controls') retrobulbar adipocytes were measured by means of several assays: An enzyme-linked immunosorbent assay was employed to reveal specific binding of antibodies to the adipocytes. Metabolic activity was determined by means of a colorimetric dimethyl thiazolium-diphe…

AdultMalemedicine.medical_specialtyEye DiseasesSwineEndocrinology Diabetes and MetabolismGraves' diseaseClinical BiochemistryAdipose tissueEnzyme-Linked Immunosorbent AssayEyeBiochemistryImmunoglobulin GGraves' ophthalmopathychemistry.chemical_compoundEndocrinologyInternal medicineAdipocytemedicineAdipocytesAnimalsHumansCells CulturedAgedLipoprotein lipasebiologybusiness.industryBiochemistry (medical)General MedicineMiddle Agedmedicine.diseaseGraves DiseaseKineticsEndocrinologychemistryImmunoglobulin GHumoral immunityAntibody Formationbiology.proteinFemaleAntibodybusiness
researchProduct

Familial HDL deficiency due to ABCA1 gene mutations with or without other genetic lipoprotein disorders

2004

Mutations in ABCA1 have been shown to be the cause of Tangier disease (TD) and some forms of familial hypoalphalipoproteinemia (HA), two genetic disorders characterized by low plasma HDL levels. Here we report six subjects with low HDL, carrying seven ABCA1 mutations, six of which are previously unreported. Two mutations (R557X and H160FsX173) were predicted to generate short truncated proteins; two mutations (E284K and Y482C) were located in the first extracellular loop and two (R1901S and Q2196H) in the C-terminal cytoplasmic domain of ABCA1. Two subjects found to be compound heterozygotes for ABCA1 mutations did not have overt clinical manifestations of TD. Three subjects, all with prema…

AdultMalemedicine.medical_specialtyHeterozygoteSettore MED/09 - Medicina InternaApolipoprotein BAdolescentPremature coronary artery diseaseTangier diseaseCoronary DiseaseBiologyGene mutationmedicine.disease_causeCompound heterozygosityTangier diseaseInternal medicineGenotypeABCA1 genemedicineHumansChildHypoalphalipoproteinemiaSelection BiasAgedApolipoproteins BGeneticsMutationFamilial defective Apo B (FDB)Apolipoprotein A-ICholesterol HDLnutritional and metabolic diseasesMiddle Agedmedicine.diseaseLipoprotein lipaseTangier disease; Familial HDL deficiency; ABCA1 gene; Familial defective Apo B (FDB); Lipoprotein lipase; Premature coronary artery diseaseEndocrinologyChild PreschoolMutationbiology.proteinlipids (amino acids peptides and proteins)Allelic heterogeneityATP-Binding Cassette TransportersFemaleCardiology and Cardiovascular MedicineFamilial HDL deficiencyATP Binding Cassette Transporter 1
researchProduct

Pain-induced alterations in the individual non-esterified fatty acids in serum.

1979

Changes of individual non-esterified fatty acids in serum were studied in 7 metabolically healthy patients, suffering from idiopathic trigeminal neuralgia, who underwent electrocoagulation of gasserian ganglion under neurolepthypalgesia. The mean concentration of fatty acids increased by about 40% during the strong surgical pain stimulus. The increased lipolysis affected all individual fatty acids in proportion to the ratio of their presence in adipose tissue. The mechanism of pain-induced activation of lipase and possible consequences of alterations in individual non-esterified fatty acids are discussed.

AdultMalemedicine.medical_specialtyIncreased LipolysisAdipose tissueBiologyFatty Acids NonesterifiedTrigeminal ganglionTrigeminal neuralgiaInternal medicinemedicineIdiopathic trigeminal neuralgiaElectrocoagulationHumansLipaseAgedchemistry.chemical_classificationLipid MobilizationFatty acidMiddle AgedTrigeminal Neuralgiamedicine.diseaseGanglionAnesthesiology and Pain MedicineEndocrinologymedicine.anatomical_structureNeurologychemistryTrigeminal Ganglionbiology.proteinFemaleNeurology (clinical)Pain
researchProduct

Macroamylasaemia after treatment with hydroxyethyl starch

1977

After infusion of 500 ml of 6% hydroxyethyl starch into fifty-four patients an increase of serum amylase was observed which in fifty-one cases exceeded the upper limit of normal (190 U/l). In most cases serum amylase reached twice the basal value. Renal function influenced the duration of the increase in serum amylase, but not the maximum increase (201+/-15 U/l; mean+/-SEM). In patients with advanced renal failure (glomerular filtration rate (GFR) = 2-10 ml/min) serum amylase was still markedly elevated after 72 h (298+/-24 U/l; mean+/-SEM). In patients with normal renal function (GFR greater than 90 ml/min) serum amylase decreased to 183+/-40 U/l (mean+/-SEM) within 72 h without reaching b…

AdultMalemedicine.medical_specialtyPancreatic diseaseClinical BiochemistryRenal functionUrineIn Vitro TechniquesHydroxyethyl starchBiochemistryHydroxyethyl Starch DerivativesExcretionBasal (phylogenetics)Renal DialysisInternal medicinemedicineHumansInfusions ParenteralAmylaseLipasePancreasbiologyChemistryStarchGeneral MedicineMiddle Agedmedicine.diseaseEndocrinologyAmylasesHypertensionChromatography Gelbiology.proteinKidney Failure ChronicFemaleGlomerular Filtration Ratemedicine.drugEuropean Journal of Clinical Investigation
researchProduct

Pancreatic enzymes in chronic renal failure and transplant patients.

1992

The aim of the present study was to determine the frequency and degree of elevated serum levels of Total Amylase (TA), Pancreatic Amylase (PA), and Lipase (L) activity in patients with chronic renal failure (CRF) on conservative therapy; CRF on periodical hemodialysis (HD); in renal transplant (RT) and in a control Group (C). Mean values were significantly higher in all groups than Group C for TA (p < 0.005), PA (p < 0.0001) and L (p < 0.0001). A statistically significant correlation was found between TA and L vs creatininemia values in CRF patients, but only up to a certain level (creatininemia < 6 mg %) (p < 0.03 and p < 0.05), above which there was no correlation. The enzyme most frequen…

AdultMalemedicine.medical_specialtyPancreatic diseasemedicine.medical_treatmentGastroenterologyNephropathychemistry.chemical_compoundEndocrinologyRenal DialysisInternal medicinemedicineHumansPancreasDialysisAgedKidneyCreatininebusiness.industryGastroenterologyLipaseMiddle Agedmedicine.diseaseKidney TransplantationEndocrinologymedicine.anatomical_structureOncologychemistryRenal pathologyCreatinineAmylasesPancreatitisKidney Failure ChronicFemaleHemodialysisbusinessInternational journal of pancreatology : official journal of the International Association of Pancreatology
researchProduct