Search results for "lipid droplet"

showing 10 items of 54 documents

Paraoxonase-1 deficiency is associated with severe liver steatosis in mice fed a high-fat high-cholesterol diet: a metabolomic approach.

2013

Oxidative stress is a determinant of liver steatosis and the progression to more severe forms of disease. The present study investigated the effect of paraoxonase-1 (PON1) deficiency on histological alterations and hepatic metabolism in mice fed a high-fat high-cholesterol diet. We performed nontargeted metabolomics on liver tissues from 8 male PON1-deficient mice and 8 wild-type animals fed a high-fat, high-cholesterol diet for 22 weeks. We also measured 8-oxo-20-deoxyguanosine, reduced and oxidized glutathione, malondialdehyde, 8-isoprostanes and protein carbonyl concentrations. Results indicated lipid droplets in 14.5% of the hepatocytes of wild-type mice and in 83.3% of the PON1-deficie…

Malemedicine.medical_specialtyBiologymedicine.disease_causeDiet High-FatBiochemistrychemistry.chemical_compoundMiceLipid dropletInternal medicineNonalcoholic fatty liver diseasemedicineAnimalsMetabolomicsAmino AcidsOrotic AcidTriglycerideLipid peroxideAryldialkylphosphataseGeneral Chemistrymedicine.diseaseMalondialdehydeLipid MetabolismGlutathioneFatty LiverMice Inbred C57BLOxidative StressEndocrinologyCholesterolGlucosechemistryLiverUrea cycleSteatosisOxidative stressBiomarkersJournal of proteome research
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Lipid droplet-associated proteins in high-fat fed mice with the effects of voluntary running and diet change

2014

Abstract Objective The relation between lipid accumulation and influence of exercise on insulin sensitivity is not straightforward. A proper balance between lipid droplet synthesis, lipolysis, and oxidative metabolism would ensure low local intramyocellular fatty acid levels, thereby possibly protecting against lipotoxicity-associated insulin resistance. This study investigated whether the accumulation of triglycerides and lipid droplets in response to high availability of fatty acids after high-fat feeding would parallel the abundance of intramyocellular perilipin proteins, especially PLIN5. The effects on these variables after diet change or voluntary running exercise intervention in skel…

Malemedicine.medical_specialtyEndocrinology Diabetes and MetabolismGlucose uptakePerilipin 2Blotting WesternDiet High-FatReal-Time Polymerase Chain ReactionRunningMicechemistry.chemical_compoundEndocrinologyInsulin resistanceInternal medicineLipid dropletmedicineAnimalsLipolysista315chemistry.chemical_classificationTriglyceridebiologyChemistryProteinsFatty acidta3141medicine.diseaseMice Inbred C57BLEndocrinologyPerilipinbiology.proteinMetabolism: Clinical and Experimental
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HPMA-based block copolymers promote differential drug delivery kinetics for hydrophobic and amphiphilic molecules.

2015

Abstract We describe a method how polymeric nanoparticles stabilized with (2-hydroxypropyl)methacrylamide (HPMA)-based block copolymers are used as drug delivery systems for a fast release of hydrophobic and a controlled release of an amphiphilic molecule. The versatile method of the miniemulsion solvent-evaporation technique was used to prepare polystyrene (PS) as well as poly-d/l-lactide (PDLLA) nanoparticles. Covalently bound or physically adsorbed fluorescent dyes labeled the particles’ core and their block copolymer corona. Confocal laser scanning microscopy (CLSM) in combination with flow cytometry measurements were applied to demonstrate the burst release of a fluorescent hydrophobic…

Materials sciencePolymersPolyestersBiomedical EngineeringNanoparticleFluorescent Antibody TechniqueNanotechnology02 engineering and technology010402 general chemistry01 natural sciencesBiochemistryBiomaterialschemistry.chemical_compoundSurface-Active AgentsDrug Delivery SystemsAmphiphileCopolymerMethacrylamideHumansMolecular BiologyDrug CarriersGeneral MedicineLipid Droplets021001 nanoscience & nanotechnologyControlled release0104 chemical sciencesMiniemulsionDrug LiberationKineticschemistryDrug deliveryBiophysicsMethacrylatesNanoparticlesPolystyrenesNanocarriers0210 nano-technologyHydrophobic and Hydrophilic InteractionsBiotechnologyHeLa CellsActa biomaterialia
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Lipid rafts: a signalling platform linking lipoprotein metabolism to atherogenesis.

2012

Lipid rafts are microdomains of the plasma membrane which are enriched in cholesterol and sphingolipids. They serve as a platform for signal transduction, in particular during immune and inflammatory responses. As hypercholesterolemia and inflammation are two key elements of atherogenesis, it is conceivable that the cholesterol and cholesterol oxide content of lipid rafts might influence the inflammatory signalling pathways, thus modulating the development of atherosclerosis. In support of this emerging view, lipid rafts have been shown to be involved in several key steps of atherogenesis, such as the oxysterol-mediated apoptosis of vascular cells, the blunted ability of high density lipopr…

OxysterolCholesterolLipoproteinsInflammationAtherosclerosisSphingolipidCell biologychemistry.chemical_compoundCholesterolMembrane MicrodomainschemistryLipid dropletmedicineAnimalsBlood VesselsHumanslipids (amino acids peptides and proteins)Signal transductionmedicine.symptomInflammation MediatorsCardiology and Cardiovascular MedicineLipid raftLipoproteinSignal TransductionAtherosclerosis
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Myoglobin, expressed in brown adipose tissue of mice, regulates the content and activity of mitochondria and lipid droplets

2021

Abstract The identification of novel physiological regulators that stimulate energy expenditure through brown adipose tissue (BAT) activity in substrate catalysis is of utmost importance to understand and treat metabolic diseases. Myoglobin (MB), known to store or transport oxygen in heart and skeletal muscles, has recently been found to bind fatty acids with physiological constants in its oxygenated form (i.e., MBO2). Here, we investigated the in vivo effect of MB expression on BAT activity. In particular, we studied mitochondrial function and lipid metabolism as essential determinants of energy expenditure in this tissue. We show in a MB-null (MBko) mouse model that MB expression in BAT i…

PalmitatesOxidative phosphorylationMitochondrion1307 Cell BiologyMiceAdipose Tissue BrownLipid dropletBrown adipose tissueRespiration1312 Molecular BiologymedicineAnimalsHumansPPAR alpha11434 Center for Clinical StudiesMuscle SkeletalMolecular BiologyUncoupling Protein 1Mice KnockoutMyoglobinChemistryProteinsThermogenesisLipid metabolismLipid DropletsCell BiologyMetabolism10081 Institute of Veterinary PhysiologyPeroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alphaMitochondriaCell biologyOxygenDisease Models AnimalAdipocytes Brownmedicine.anatomical_structure10076 Center for Integrative Human Physiology570 Life sciences; biologyApoptosis Regulatory ProteinsEnergy MetabolismThermogenesisBiochimica et Biophysica Acta (BBA) - Molecular and Cell Biology of Lipids
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Electron microscopic studies on skin and lymphocytes in early juvenile neuronal ceroid-lipofuscinosis.

1987

Skin and lymphocytes of three patients with early juvenile neuronal ceroid-lipofuscinosis (NCL) were ultras trueturally investigated. Fingerprint profiles (FPP), isolated and I or mixed with curvilinear profiles (CLP), in various dermal cells and large, usually single lipopigments delineated by a trilaminar membrane and filled with a granular matrix, FPP and occasionally lipid droplets in lymphocytes were observed in all three patients. Characteristic lipopigments in lymphocytes are an important feature to differentiate between early juvenile NCL and late infantile and juvenile NCL.

Pathologymedicine.medical_specialtyLymphocyteEarly juvenileInfantile neuronal ceroid lipofuscinosisMatrix (biology)BiologyLipofuscinDevelopmental NeuroscienceNeuronal Ceroid-LipofuscinosesLipid dropletmedicineJuvenileHumansLymphocytesChildSkinGeneral Medicinemedicine.diseaseMicroscopy Electronmedicine.anatomical_structureChild PreschoolPediatrics Perinatology and Child HealthUltrastructureNeuronal ceroid lipofuscinosisNeurology (clinical)Braindevelopment
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The Participation of the Complement System in Atherosclerotic Vascular Disease

1991

Atherosclerosis is a vascular disease of large and medium-sized arteries wherein the tunica intima becomes thickened due to lipid accumulation, mostly cholesterol and its esters, smooth muscle cell proliferation, and increased deposition of connective tissue matrix. A major risk factor in the development of this disease is hypercholesterolemia arising from elevated levels of low density lipoproteins (LDL). The earliest recognizable lesion, which may be a precursor to the fibrofatty plaque, is the fatty streak. It is predominantly composed of monocyte-derived macrophage foam cells, i.e. cells ladened with intracellular lipid droplets. Hence, a fundamental aspect of atherogenesis is the insud…

Pathologymedicine.medical_specialtyVascular smooth muscleChemistryMonocyteFatty streakConnective tissueTunica intimaLesionmedicine.anatomical_structureLipid dropletmedicineMacrophagelipids (amino acids peptides and proteins)medicine.symptom
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Studies on the fat body of oncopeltus fasciatus invaded by pseudomonas aeruginosa1

1977

A fatal disease in laboratory cultures of Oncopeltus fasciatus is caused by Pseudomonas aeruginosa , which invades the fat body and destroys it. The cellular breakdown was studied by electron microscopy. Infected fat body tissue shrinks, disintegrates, and dissolves. The nuclei, apparently, remain intact until immediately before cell death. Bacteria, which are found singularly in cytoplasm, form an expanding electron-lucent halo. Within this halo, free ribosomes and ribosomes of rough endoplasmic reticulum (ER) disappear. The changes are presumably caused by proteolytic activity of the parasite, which results in the destruction of the fat body and death of the host. Lipid droplets dissolve,…

Programmed cell deathbiologyEndoplasmic reticulumCellMatrix (biology)biology.organism_classificationRibosomeMicrobiologymedicine.anatomical_structureCytoplasmLipid dropletmedicineEcology Evolution Behavior and SystematicsBacteriaJournal of Invertebrate Pathology
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Low density lipoproteins and human serum albumin as the carriers of squalenoylated drugs: insights from molecular simulations

2018

We have studied the interaction of three clinically promising squalenoylated drugs (gemcitabine-squalene, adenine-squalene, and doxorubicin-squalene) with low-density lipoproteins (LDL) by means of atomistic molecular dynamics simulations. It is shown that all studied squalenoylated drugs accumulate inside the LDL particles. This effect is promoted by the squalene moiety, which acts as an anchor and drives the hydrophilic drugs into the hydrophobic core of the LDL lipid droplet. Our data suggest that LDL particles could be a universal carriers of squalenoylated drugs in the bloodstream. Interaction of gemcitabine-squalene with human serum albumin (HSA) was also studied by ensemble of dockin…

Squalene[PHYS.PHYS.PHYS-BIO-PH]Physics [physics]/Physics [physics]/Biological Physics [physics.bio-ph]Drug CompoundingPharmaceutical ScienceSerum Albumin Human02 engineering and technologyPlasma protein bindingMolecular Dynamics Simulation010402 general chemistry01 natural sciencesMolecular Docking SimulationDeoxycytidineSqualenechemistry.chemical_compound[ PHYS.PHYS.PHYS-BIO-PH ] Physics [physics]/Physics [physics]/Biological Physics [physics.bio-ph]Lipid dropletDrug DiscoverymedicineMoietyHumansComputingMilieux_MISCELLANEOUSDrug CarriersBinding SitesAdenine[SDV.SP]Life Sciences [q-bio]/Pharmaceutical sciences021001 nanoscience & nanotechnologyHuman serum albuminGemcitabine3. Good health0104 chemical sciences[CHIM.THEO]Chemical Sciences/Theoretical and/or physical chemistryLipoproteins LDLMolecular Docking Simulation[ SDV.SP ] Life Sciences [q-bio]/Pharmaceutical scienceschemistryDocking (molecular)Doxorubicin[ CHIM.THEO ] Chemical Sciences/Theoretical and/or physical chemistryBiophysicsMolecular MedicineNanoparticles0210 nano-technologyDrug carrierHydrophobic and Hydrophilic Interactionsmedicine.drugProtein Binding
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The Elovl5 enzyme in breast cancer : regulation of cancer cell proliferation and metastasis

2021

Breast cancer is the most diagnosed cancer and the leading cause of cancer death in women. The 5-year relative survival rate is less than 30% for metastatic breast cancer and metastases account for more than 75% of breast cancer deaths. Fatty acid metabolism is altered in cancer and contributes to tumor progression and metastasis. Here, we were interested in Elongation of very long chain fatty acids protein 5 (Elovl5) which catalyzes the elongation of long-chain fatty acids. We observed that the tumors from patients with a breast cancer had a lower expression of Elovl5 than paired normal breast tissue. However, low expression of Elovl5 is associated with a worse prognosis in breast cancer p…

TGF-ΒBreast cancer[SDV.MHEP] Life Sciences [q-bio]/Human health and pathologyFatty acid elongationÉlongationdes acides grasElovl5MetastasesLipid dropletsCancer du seinMetastasisGouttelettes lipidiques
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