Search results for "medicine.drug_class"

showing 10 items of 2726 documents

Reslizumab (RES) in asthma patients (pts) with severe eosinophilic asthma stratified by GINA asthma steps 4 and 5: Analysis of two phase 3, placebo (…

2016

Background: RES reduced exacerbation frequency and improved lung function and asthma control in pts with inadequately controlled asthma and elevated blood eosinophils (EOS) in two PBO-controlled, phase 3 trials (Castro et al. Lancet Resp Med 2015). Aims/objectives: To determine the efficacy of RES in asthma pts stratified by GINA Step 4 (G4) and Step 5 (G5) categories of asthma severity. Methods: Pooled data from two 52-week, PBO-controlled trials of RES (3mg/kg IV Q4W) in pts with inadequately controlled asthma and blood EOS ≥400/µL on inhaled corticosteroid (ICS) ± another controller; primary results have been reported. Current analysis includes pts stratified by G4 (medium/high-dose ICS …

medicine.medical_specialtyPediatricsExacerbationbusiness.industrymedicine.drug_classEosinophilic asthmaPlaceboRate ratiomedicine.diseaserespiratory tract diseases03 medical and health sciences0302 clinical medicine030228 respiratory systemReslizumabInternal medicineAsthma controlMedicineCorticosteroid030212 general & internal medicinebusinessmedicine.drugAsthma5.1 Airway Pharmacology and Treatment
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Long-term outcome of antiandrogen monotherapy in advanced prostate carcinoma: 12-year results of a phase II study

2003

OBJECTIVE To present the long-term outcome of patients with locally advanced or metastatic prostate carcinoma treated by first-line antiandrogen monotherapy. PATIENTS AND METHODS From 1983 to 1990, 41 patients with advanced prostate carcinoma were treated with flutamide monotherapy until progression or the appearance of toxicity. Twenty-five patients (61%) had T3-T4N0M0 and 16 (39%) T2–4N0–3M1 prostate carcinoma. Consensus criteria were adopted to evaluate the response. Plasma testosterone and sexual function were recorded for the first 3 years. RESULTS Flutamide was administered for up to 147 months; seven patients (17%) interrupted the treatment because of toxicity. There was an objective…

medicine.medical_specialtyPerformance statusmedicine.drug_classbusiness.industryUrologyUrologyAntiandrogenmedicine.diseaseSurgeryFlutamideProstate-specific antigenchemistry.chemical_compoundProstate cancerchemistrymedicineCarcinomaHormonal therapymedicine.symptomBone painbusinessBJU International
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Clinical evaluation of an Er:YAG laser for nonsurgical treatment of peri-implantitis: a pilot study

2004

The aim of this controlled, parallel design clinical study was to compare the effectiveness of an Er:YAG laser (ERL) to that of mechanical debridement using plastic curettes and antiseptic therapy for nonsurgical treatment of peri-implantitis. Twenty patients with moderate to advanced peri-implantitis lesions were randomly treated with either (1) an ERL using a cone-shaped glass fiber tip at an energy setting of 100 mJ/pulse and 10 pps (ERL), or (2) mechanical debridement using plastic curettes and antiseptic therapy with chlorhexidine digluconate (0.2%) (C). The following clinical parameters were measured at baseline, 3 and 6 months after treatment by one blinded and calibrated examiner: P…

medicine.medical_specialtyPeri-implantitisbusiness.industrymedicine.drug_classmedicine.medical_treatmentChlorhexidineBleeding on probingDentistrySurgerylaw.inventionRandomized controlled trialAntisepticlawDebridement (dental)medicineOral Surgerymedicine.symptombusinessGingival recessionEr:YAG lasermedicine.drugClinical Oral Implants Research
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Combination of hormone replacement therapy and high physical activity is associated with differences in Achilles tendon size in monozygotic female tw…

2009

Estrogen concentration has been suggested to play a role in tendon abnormalities and injury. In physically active postmenopausal women, hormone replacement therapy (HRT) has been suggested to decrease tendon diameter. We hypothesized that HRT use and physical activity are associated with Achilles tendon size and tissue structure. The study applied cotwin analysis of fourteen 54- to 62-yr-old identical female twin pairs with current discordance for HRT use for an average of 7 yr. Achilles tendon thickness and cross-sectional areas were determined by ultrasonography, and tendon structural organization was analyzed from the images using linear discriminant analysis (LDA). Maximal voluntary and…

medicine.medical_specialtyPhysiologymedicine.drug_classEstroneHormone Replacement TherapyAnterior cruciate ligamentPhysical activityMonozygotic twinPhysical exerciseMotor ActivityAchilles Tendon03 medical and health sciences0302 clinical medicinePhysiology (medical)Internal medicineSex Hormone-Binding GlobulinMedicineHumansTestosteroneMuscle StrengthAgedUltrasonographyAchilles tendonbusiness.industryEstrogens030229 sport sciencesTwins MonozygoticMiddle AgedTendonmedicine.anatomical_structureEndocrinologyCholesterolTransgender hormone therapyEstrogenFemaleMenopausebusiness030217 neurology & neurosurgeryMuscle ContractionJournal of applied physiology (Bethesda, Md. : 1985)
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Early, but not late onset estrogen replacement therapy prevents oxidative stress and metabolic alterations caused by ovariectomy.

2014

Aims: The usefulness of estrogen replacement therapy (ERT) in preventing oxidative stress associated with menopause is controversial. We aimed to study if there is a critical time window for effective treatment of the effects of ovariectomy with estrogens at the molecular, metabolic, and cellular level. Results: Our main finding is that early, but not late onset of ERT prevents an ovariectomy-associated increase in mitochondrial hydrogen peroxide levels, oxidative damage to lipids and proteins, and a decrease in glutathione peroxidase and catalase activity in rats. This may be due to a change in the estrogen receptor (ER) expression profile: ovariectomy increases the ER α/β ratio and immedi…

medicine.medical_specialtyPhysiologymedicine.drug_classGlucose uptakeOvariectomyClinical BiochemistryGlucose Transport Proteins FacilitativeEstrogen receptorMitochondria LiverBiologymedicine.disease_causeBiochemistryAntioxidantsInternal medicinemedicineAnimalsMetabolomicsMolecular BiologyGeneral Environmental Sciencechemistry.chemical_classificationEstradiolGlutathione peroxidaseEstrogen Replacement TherapyGlucose transporterBrainCell BiologyHydrogen Peroxidemedicine.diseaseRatsMenopauseOxidative StressOriginal Research CommunicationsEndocrinologyGlucosechemistryEstrogenCatalasebiology.proteinGeneral Earth and Planetary SciencesFemaleOxidative stressAntioxidantsredox signaling
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Renal protection by antihypertensive drugs

1998

During the last few years there has been a renewed interest in blood-pressure (BP)-induced kidney damage, owing to a progressive increase in the incidence and prevalence of hypertension and vascular diseases as a cause of end-stage renal disease (ESRD). The need to prevent ESRD demands continued efforts so as to identify early those people with hypertension who are at risk and to provide them with effective antihypertensive therapy. This review analyses what is needed in terms of surrogate endpoints for monitoring kidney damage and what is known about the impact of antihypertensive treatments in reducing the BP burden on the kidney in non-diabetic subjects. Although glomerular filtration ra…

medicine.medical_specialtyPhysiologymedicine.drug_classRenal functionBlood PressureKidneyurologic and male genital diseasesEssential hypertensionNephropathyRisk FactorsInternal medicineInternal MedicinemedicineAlbuminuriaHumansAntihypertensive drugAntihypertensive AgentsKidneyProteinuriabusiness.industrySurrogate endpointmedicine.diseasefemale genital diseases and pregnancy complicationsEndocrinologymedicine.anatomical_structureHypertensionCardiologyKidney Failure ChronicMicroalbuminuriamedicine.symptomCardiology and Cardiovascular MedicinebusinessJournal of Hypertension
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A 35-year effective treatment of catecholaminergic polymorphic ventricular tachycardia with propafenone

2018

Key Teaching Points • Despite proven catecholaminergic polymorphic ventricular tachycardia (CPVT) with pathogen RyR2 mutation and recurrent syncope, patients could have a favorable long-term outcome over 35 years under treatment. • Propafenone could be effective for treatment of patients with CPVT. • The beneficial effect of the monotherapy with propafenone in our patient may result from the combined antiarrhythmic effect of this drug with Na+ channel blockade and beta blocker capabilities.

medicine.medical_specialtyPolymorphic premature ventricular beatsmedicine.drug_classCase ReportPropafenone030204 cardiovascular system & hematologyCatecholaminergic polymorphic ventricular tachycardiaRyanodine receptor 203 medical and health sciences0302 clinical medicinePropafenoneInternal medicineMedicineEffective treatment030212 general & internal medicineExercise-induced syncopeBeta blockerbusiness.industrymedicine.diseaseBlockadeExercise-induced syncopeCatecholaminergic polymorphic ventricular tachycardiacardiovascular systemCardiologyAntiarrhythmic effectCardiology and Cardiovascular MedicinebusinessRyanodine receptor mutationmedicine.drugHeartRhythm Case Reports
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Artrial Natriuretic Peptide Concentrations in Preeclampsia

1989

During pregnancy various humoral and functional mechanisms were recognized, that regulate the water and electrolyte household. In preeclampsia, increased water-sodium retention results from a variety of pathophysiological changes in the endocrinological system. There are also organ-specific changes, such as renal clearance reduction up to 30% in comparison to normal pregnancy. For many years, physiologists expected to find additional cardial mechanisms that influence homeostasis. In 1981, de Bold [3] identified ANP a new humoral factor that leads to increased water and sodium excretion. The influence of ANP in preeclampsia has been the subject of controversial discussion.

medicine.medical_specialtyPregnancybusiness.industrymedicine.drug_classNormal pregnancymedicine.diseasePathophysiologyPreeclampsiaEndocrinologySodium excretionInternal medicinemedicineNatriuretic peptidesense organsbusinessHomeostasisClearance
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Acute estradiol protects CA1 neurons from ischemia-induced apoptotic cell death via the PI3K/Akt pathway

2010

Global ischemia arising during cardiac arrest or cardiac surgery causes highly selective, delayed death of hippocampal CA1 neurons. Exogenous estradiol ameliorates global ischemia-induced neuronal death and cognitive impairment in male and female rodents. However, the molecular mechanisms by which a single acute injection of estradiol administered after the ischemic event intervenes in global ischemia-induced apoptotic cell death are unclear. Here we show that acute estradiol acts via the phosphoinositide 3-kinase (PI3K)/protein kinase B (Akt) signaling cascade to protect CA1 neurons in ovariectomized female rats. We demonstrate that global ischemia promotes early activation of glycogen syn…

medicine.medical_specialtyProgrammed cell deathmedicine.drug_classOvariectomyBlotting WesternIschemiaApoptosisHippocampusArticleBrain IschemiaBrain ischemiaPhosphatidylinositol 3-KinasesInternal medicinemedicineAnimalsMolecular BiologyProtein kinase BPI3K/AKT/mTOR pathwayCaspaseNeuronsbiologyEstradiolGeneral NeuroscienceEstrogensmedicine.diseaseRatsEndocrinologyEstrogenApoptosisNerve DegenerationCancer researchbiology.proteinFemaleNeurology (clinical)Proto-Oncogene Proteins c-aktDevelopmental BiologySignal Transduction
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Estrogen regulates cytokine production and apoptosis in PMA-differentiated, macrophage-like U937 cells

2003

We have investigated the effects of sex steroids, estradiol (E2), and testosterone (T) on the synthesis of tumor necrosis factor alpha (TNF-alpha) and interleukin-10 (IL-10) in phorbol-myristate-acetate (PMA)-differentiated human monoblastic U937 cells. The ability of both hormones to modulate the viability and programmed cell death of macrophage-like PMA-differentiated U937 cells was also inspected. E2 increased TNF-alpha synthesis, whereas T had no effect on the production of this cytokine. The combination of E2 and its antagonist tamoxifen or ICI-182,789 completely abolished the induction of TNF-alpha, while combination of T and its antagonist Casodex (CSDX) did not significantly affect …

medicine.medical_specialtyProgrammed cell deathmedicine.drug_classmedicine.medical_treatmentCell BiologyBiologyBiochemistryCell biologyInterleukin 10CytokineEndocrinologyEstrogenApoptosisInternal medicinemedicineMacrophageTumor necrosis factor alphaIL-2 receptorMolecular BiologyJournal of Cellular Biochemistry
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