Search results for "membrane potential"

showing 10 items of 327 documents

Cytotoxic activity of the histone deacetylase 3-Selective inhibitor Pojamide on MDA-MB-231 triple-negative breast cancer cells

2019

We examined the effects of the ferrocene-based histone deacetylase-3 inhibitor Pojamide (N1-(2-aminophenyl)-N8-ferrocenyloctanediamide) and its two derivatives N1-(2-aminophenyl)-N6-ferrocenyladipamide and N1-(2-aminophenyl)-N8-ferroceniumoctanediamide tetrafluoroborate on triple-negative MDA-MB-231 breast cancer cells. Viability/growth assays indicated that only the first two compounds at 70 &mu

0301 basic medicineQD0901Triple Negative Breast Neoplasmslcsh:Chemistry0302 clinical medicinebreast cancer cellmitochondrial transmembrane potentialCytotoxic T cellQDSettore BIO/06 - Anatomia Comparata E Citologialcsh:QH301-705.5SpectroscopyTriple-negative breast cancerreactive oxygen speciesCell DeathChemistryHistone deacetylase inhibitorQapoptosisGeneral MedicineCell cycle3. Good healthComputer Science Applications030220 oncology & carcinogenesisFemalecell cycleProgrammed cell deathautophagymedicine.drug_classCell SurvivalCatalysisArticleHistone DeacetylasesInorganic Chemistry03 medical and health sciencesCell Line TumormedicineBiomarkers TumorHumansViability assayPhysical and Theoretical ChemistryMolecular Biologyhistone deacetylase inhibitorcell viabilityOrganic ChemistryAutophagyapoptosiMatrix MetalloproteinasesHistone Deacetylase InhibitorsSettore BIO/18 - Genetica030104 developmental biologylcsh:Biology (General)lcsh:QD1-999ApoptosisCancer researchQD0146breast cancer cells
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Cytotoxicity of seven naturally occurring phenolic compounds towards multi-factorial drug-resistant cancer cells

2016

Abstract Introduction In medical oncology, multi-drug resistance (MDR) of cancer cells continues to be a major impediment. We are in quest of novel anti-proliferative agents to overcome drug-resistant tumor cells. Methods In the present study, we investigated the cytotoxicity of 7 naturally occurring phenolic compounds including two isoflavonoids alpinumisoflavone ( 1 ) and laburnetin ( 2 ), one biflavonoid amentoflavone ( 3) , three lignans pycnanthulignene A ( 4 ), pycnanthulignene B ( 5 ), and syringaresinol ( 7 ) and one xanthone, euxanthone ( 6 ) against 9 drug-sensitive and MDR cancer cell lines. The resazurin reduction assay was used to evaluate the cytotoxicity of these compounds, w…

0301 basic medicineSyringaresinolPharmaceutical SciencePharmacologyAmentoflavone03 medical and health scienceschemistry.chemical_compound0302 clinical medicinePhenolsIsoflavonoidCell Line TumorNeoplasmsOxazinesDrug DiscoveryHumansCytotoxic T cellCytotoxicityMembrane Potential MitochondrialPharmacologyCell Cycle CheckpointsAlpinumisoflavoneAntineoplastic Agents PhytogenicDrug Resistance MultipleEnzyme Activation030104 developmental biologyXanthenesComplementary and alternative medicinechemistryDrug Resistance NeoplasmApoptosisCaspases030220 oncology & carcinogenesisCancer cellMolecular MedicineReactive Oxygen SpeciesPhytomedicine
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2-methoxyestradiol impacts on amino acids-mediated metabolic reprogramming in osteosarcoma cells by interaction with NMDA receptor

2017

Deregulation of serine and glycine metabolism, have been identified to function as metabolic regulators in supporting tumor cell growth. The role of serine and glycine in regulation of cancer cell proliferation is complicated, dependent on concentrations of amino acids and tissue-specific. D-serine and glycine are coagonists of N-methyl-D-aspartate receptor subunit GRIN1. Importantly, NMDA receptors are widely expressed in cancer cells and play an important role in regulation of cell death, proliferation and metabolism of numerous malignancies. The aim of the present work was to associate the metabolism of glycine and D-serine with the anticancer activity of 2-methoxyestradiol. 2-methoxyest…

0301 basic medicineTime Factors2-methoxyestradiol neuronal nitric oxide synthase D-serine glycine osteosarcomaPhysiologyClinical BiochemistryNitric Oxide Synthase Type ISerine0302 clinical medicineCell MovementSerinechemistry.chemical_classificationMembrane Potential MitochondrialOsteosarcomaEstradiolTubulin ModulatorsAmino acidMolecular Docking Simulation030220 oncology & carcinogenesisMCF-7 CellsNMDA receptorOsteosarcomaFemalemedicine.drugProtein BindingSignal TransductionProgrammed cell deathGlycineAntineoplastic AgentsBone NeoplasmsBreast NeoplasmsNerve Tissue ProteinsBiologyMolecular Dynamics SimulationReceptors N-Methyl-D-Aspartate03 medical and health sciencesStructure-Activity RelationshipProtein DomainsmedicineHumans2-MethoxyestradiolCell ProliferationBinding SitesDose-Response Relationship DrugCell BiologyMetabolismmedicine.disease2-Methoxyestradiol030104 developmental biologychemistryCancer cellCancer researchEnergy Metabolism
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Loss of MCL1 function sensitizes the MDA-MB-231 breast cancer cells to rh-TRAIL by increasing DR4 levels.

2019

Triple-negative breast cancer (TNBC) is a form of BC characterized by high aggressiveness and therapy resistance probably determined by cancer stem cells. MCL1 is an antiapoptotic Bcl-2 family member that could limit the efficacy of anticancer agents as recombinant human tumor necrosis factor related apoptosis-inducing ligand (rh-TRAIL). Here, we investigated MCL1 expression in TNBC tissues and cells. We found MCL1 differentially expressed (upregulated or downregulated) in TNBC tissues. Furthermore, in comparison to the human mammary epithelial cells, we found that MDA-MB-231 cells show similar messenger RNA levels but higher MCL1 protein levels, whereas it resulted downregulated in MDA-MB-…

0301 basic medicinecancer stem cellIndolesPhysiologyCell SurvivalClinical BiochemistryCellPopulationApoptosisTNF-Related Apoptosis-Inducing Ligand03 medical and health sciences0302 clinical medicineCancer stem cellSettore BIO/10 - BiochimicaCell Line Tumormedicinerh-TRAILBiomarkers TumorGene silencingHumansViability assayGene SilencingeducationCell ShapeCell ProliferationMembrane Potential Mitochondrialeducation.field_of_studySulfonamidesChemistryCell growthCell CycleCell BiologyCell cycleRecombinant ProteinsGene Expression Regulation NeoplasticReceptors TNF-Related Apoptosis-Inducing Ligand030104 developmental biologymedicine.anatomical_structureMCL1ApoptosisDR4 receptor030220 oncology & carcinogenesisCancer researchtriple-negative breast cancerMyeloid Cell Leukemia Sequence 1 ProteinJournal of cellular physiology
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Cytoprotective Effects of Dinitrosyl Iron Complexes on Viability of Human Fibroblasts and Cardiomyocytes

2019

Nitric oxide (NO) is an important signaling molecule that plays a key role in maintaining vascular homeostasis. Dinitrosyl iron complexes (DNICs) generating NO are widely used to treat cardiovascular diseases. However, the involvement of DNICs in the metabolic processes of the cell, their protective properties in doxorubicin-induced toxicity remain to be clarified. Here, we found that novel class of mononuclear DNICs with functional sulfur-containing ligands enhanced the cell viability of human lung fibroblasts and rat cardiomyocytes. Moreover, DNICs demonstrated remarkable protection against doxorubicin-induced toxicity in fibroblasts and in rat cardiomyocytes (H9c2 cells). Data revealed t…

0301 basic medicinedonors nitric oxideCellOxidative phosphorylationdinitrosyl iron complexesheart diseaseMitochondrionNitric oxide03 medical and health scienceschemistry.chemical_compound0302 clinical medicinemedicinePharmacology (medical)Viability assayInner mitochondrial membranecell viabilityOriginal Researchchemistry.chemical_classificationPharmacologyReactive oxygen specieslcsh:RM1-950GlutathioneCell biology030104 developmental biologymedicine.anatomical_structurelcsh:Therapeutics. Pharmacologychemistry030220 oncology & carcinogenesismembrane potentialFrontiers in Pharmacology
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The Severity of Acute Stress Is Represented by Increased Synchronous Activity and Recruitment of Hypothalamic CRH Neurons

2016

The hypothalamo-pituitary-adrenocortical (HPA) axis regulates stress physiology and behavior. To achieve an optimally tuned adaptive response, it is critical that the magnitude of the stress response matches the severity of the threat. Corticotropin-releasing hormone (CRH) released from the paraventricular nucleus of the hypothalamus is a major regulator of the HPA axis. However, how CRH-producing neurons in an intact animal respond to different stressor intensities is currently not known. Using two-photon calcium imaging on intact larval zebrafish, we recorded the activity of CRH cells, while the larvae were exposed to stressors of varying intensity. By combining behavioral and physiologic…

0301 basic medicineendocrine systemmedicine.medical_specialtyHydrocortisoneCorticotropin-Releasing HormoneHypothalamusRegulatorMotor ActivityMembrane PotentialsAnimals Genetically Modified03 medical and health sciencesCorticotropin-releasing hormoneCalcium imagingStress PhysiologicalInternal medicineAvoidance LearningmedicineAnimalsZebrafishHeat-Shock ProteinsZebrafishHydrocortisoneNeuronsMembrane potentialbiologyGeneral NeuroscienceArticlesbiology.organism_classificationLuminescent Proteins030104 developmental biologymedicine.anatomical_structureEndocrinologyGene Expression Regulationnervous systemHypothalamusLarvaCalciumPsychologyNucleusNeurosciencehormones hormone substitutes and hormone antagonistsmedicine.drugThe Journal of Neuroscience
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The potassium channels TASK2 and TREK1 regulate functional differentiation of murine skeletal muscle cells.

2015

Two-pore domain potassium (K2P) channels influence basic cellular parameters such as resting membrane potential, cellular excitability, or intracellular Ca2+-concentration [Ca2+]i. While the physiological importance of K2P channels in different organ systems (e.g., heart, central nervous system, or immune system) has become increasingly clear over the last decade, their expression profile and functional role in skeletal muscle cells (SkMC) remain largely unknown. The mouse SkMC cell line C2C12, wild-type mouse muscle tissue, and primary mouse muscle cells (PMMs) were analyzed using quantitative PCR, Western blotting, and immunohistochemical stainings as well as functional analysis includin…

0301 basic medicinemedicine.medical_specialtyPhysiologyCellular differentiationMuscle Fibers SkeletalMedizinDown-RegulationBiologyCell LineMembrane Potentials03 medical and health sciencesMyoblast fusionMicePotassium Channels Tandem Pore DomainInternal medicinemedicineMyocyteAnimalsHumansPatch clampMuscle SkeletalMyogenesisSkeletal muscleCell DifferentiationCell BiologyPotassium channelCell biologyUp-Regulation030104 developmental biologyEndocrinologymedicine.anatomical_structurePotassiumC2C12American journal of physiology. Cell physiology
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Focal Cortical Lesions Induce Bidirectional Changes in the Excitability of Fast Spiking and Non Fast Spiking Cortical Interneurons

2014

A physiological brain function requires neuronal networks to operate within a well-defined range of activity. Indeed, alterations in neuronal excitability have been associated with several pathological conditions, ranging from epilepsy to neuropsychiatric disorders. Changes in inhibitory transmission are known to play a key role in the development of hyperexcitability. However it is largely unknown whether specific interneuronal subpopulations contribute differentially to such pathological condition. In the present study we investigated functional alterations of inhibitory interneurons embedded in a hyperexcitable cortical circuit at the border of chronically induced focal lesions in mouse …

500 Naturwissenschaften und Mathematik::570 Biowissenschaften; Biologie570Neural NetworksPostsynaptic CurrentExcitotoxicity610lcsh:MedicineNeurophysiologyAction PotentialsNeural Homeostasis600 Technik Medizin angewandte Wissenschaften::610 Medizin und Gesundheitmedicine.disease_causeInhibitory postsynaptic potentialMiceEpilepsyInterneuronsmedicineBiological neural networkAnimalslcsh:ScienceVisual CortexCerebral CortexMembrane potentialMultidisciplinarymusculoskeletal neural and ocular physiologylcsh:RNeurotransmissionBiology and Life SciencesExcitatory Postsynaptic Potentialsmedicine.diseaseVisual cortexmedicine.anatomical_structurenervous systemCellular NeuroscienceExcitatory postsynaptic potentiallcsh:QNeuroscienceResearch ArticleNeurosciencePLoS ONE
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Cannabinoid type 1 receptor modulates intestinal propulsion by an attenuation of intestinal motor responses within the myenteric part of the peristal…

2007

Cannabinoid-1 (CB1) receptor activation affects gastrointestinal propulsion in vivo. It was our aim to further characterize the involved myenteric mechanisms in vivo and in vitro. In CB1(-/-) mice and wild-type littermates we performed in vivo transit experiments by charcoal feeding and in vitro electrophysiological recordings in mouse small intestinal smooth muscle. Ascending neuronal contraction (ANC) following electrical field stimulation was studied in rat ileum in a partitioned organ bath separating the aboral stimulation site from the oral recording site. The knockout animals displayed an accelerated upper gastrointestinal transit compared to control animals. The CB1 receptor antagoni…

AM251Malemedicine.medical_specialtyCannabinoid receptorPhysiologyPolyunsaturated Alkamidesmedicine.medical_treatmentNeuromuscular JunctionMotilityStimulationArachidonic AcidsBiologyNeuromuscular junctionMembrane PotentialsMiceOrgan Culture TechniquesPiperidinesReceptor Cannabinoid CB1Internal medicineCannabinoid Receptor ModulatorsReflexmedicineAnimalsRNA MessengerIntestinal MucosaRats WistarReceptorMice KnockoutMyoelectric Complex MigratingEndocrine and Autonomic SystemsReverse Transcriptase Polymerase Chain ReactionGastroenterologyMuscle SmoothEndocannabinoid systemElectric StimulationRatsIntestinesEndocrinologymedicine.anatomical_structurePyrazolesPeristalsisCannabinoidmedicine.drugEndocannabinoidsNeurogastroenterology and motility : the official journal of the European Gastrointestinal Motility Society
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Factors affecting basket catheter detection of real and phantom rotors in the atria: A computational study

2018

[EN] Anatomically based procedures to ablate atrial fibrillation (AF) are often successful in terminating paroxysmal AF. However, the ability to terminate persistent AF remains disappointing. New mechanistic approaches use multiple-electrode basket catheter mapping to localize and target AF drivers in the form of rotors but significant concerns remain about their accuracy. We aimed to evaluate how electrode-endocardium distance, far-field sources and inter-electrode distance affect the accuracy of localizing rotors. Sustained rotor activation of the atria was simulated numerically and mapped using a virtual basket catheter with varying electrode densities placed at different positions withi…

Ablation TechniquesNormalization propertyTime FactorsPhysiologymedicine.medical_treatmentAction Potentials02 engineering and technology030204 cardiovascular system & hematologylaw.invention0302 clinical medicineModelslawHeart RateMedicine and Health SciencesMapping cathetersCardiac Atrialcsh:QH301-705.5Numerical AnalysisEcologyRotor (electric)HeartAblationElectrophysiologyComputational Theory and Mathematicsmedicine.veinModeling and SimulationRotorsPhysical SciencesInferior Vena CavaEngineering and TechnologyAnatomyBasket catheterArrhythmiaInterpolationResearch ArticleBiotechnologyMaterials scienceCatheters0206 medical engineeringPhase (waves)CardiologyInferior vena cavaModels BiologicalMembrane PotentialImaging phantomVeinsTECNOLOGIA ELECTRONICA03 medical and health sciencesCellular and Molecular NeuroscienceAblation Techniques; Action Potentials; Atrial Fibrillation; Computational Biology; Computer Simulation; Heart Atria; Heart Conduction System; Heart Rate; Humans; Models Biological; Time FactorsHeart Conduction SystemPhase analysisGeneticsmedicineHumansComputer SimulationHeart AtriaMolecular BiologyEcology Evolution Behavior and SystematicsMechanical EngineeringComputational BiologyBiology and Life SciencesBiological020601 biomedical engineeringAtrial fibrillationInterpolationlcsh:Biology (General)Cardiovascular AnatomyBlood VesselsMedical Devices and EquipmentMathematicsBiomedical engineeringEndocardium
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