Search results for "messenger"

showing 10 items of 1493 documents

Ontogeny of bradykinin B2 receptors in the rat kidney: Implications for segmental nephron maturation

1997

Ontogeny of bradykinin B 2 receptors in the rat kidney: Implications for segmental nephron maturation. Kinins modulate renal function, yet their role in the developing kidney is largely unknown. To explore the developmental role of the kallikrein-kinin system, we examined the postnatal ontogeny and intrarenal localization of B 2 receptors in the rat. Northern blot analysis and RT-PCR documented the expression of B 2 receptor mRNA in the kidney and extrarenal tissues of fetal, neonatal and adult animals. The abundance of B 2 receptor mRNA is 10- to 30-fold higher in neonatal than adult tissues in the following order: kidney > heart > aorta > lung > brain. Receptor autoradiography revealed a …

Malemedicine.medical_specialtyReceptor Bradykinin B2Receptor expressionMolecular Sequence DataBradykininNephronBiologyBradykininKidneyPolymerase Chain ReactionRats Sprague-Dawleychemistry.chemical_compoundParacrine signallingInternal medicinemedicineAnimalsTissue DistributionAmino Acid SequenceRNA MessengerNorthern blotReceptorBradykinin Receptor AntagonistsDNA PrimersKidneyBase Sequenceurogenital systemReceptors BradykininGene Expression Regulation DevelopmentalNephronsKininImmunohistochemistryPeptide FragmentsRatsmedicine.anatomical_structureEndocrinologyAnimals NewbornchemistryNephrologyAutoradiographyKidney International
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Expression of nicotinic acetylcholine receptors in the rat superior cervical ganglion on mRNA and protein level.

1994

The expression of nicotinic acetylcholine receptors (nAChR) in the rat superior cervical ganglion was investigated by Western blotting, immunohistochemistry and non-radioactive in situ hybridization applying probes for the alpha 4-1 and beta 2 subunit mRNA. Immunoblot analysis of homogenized ganglia using the anti-nAChRs antibody WF6 revealed a labeled protein band of apparent molecular weight of 40 kDa which is typical for the alpha subunit of nAChRs. Applying double-labeling immunofluorescence with antibodies against tyrosine hydroxylase, nAChR-like molecules were identified in most postganglionic neurons and in a subpopulation of small intensely fluorescent (SIF) cells. alpha 4-1 and bet…

Malemedicine.medical_specialtySuperior cervical ganglionBlotting WesternFluorescent Antibody TechniqueGene ExpressionNerve Tissue ProteinsSuperior Cervical GanglionBiologyReceptors NicotinicRats Sprague-DawleyCellular and Molecular NeuroscienceGanglion type nicotinic receptorInternal medicineMuscarinic acetylcholine receptormedicineAnimalsRNA MessengerMolecular BiologyIn Situ HybridizationG alpha subunitAcetylcholine receptorNeuronsMolecular biologyRatsEndocrinologyNicotinic agonistnervous systemSmall intensely fluorescent cellFemalesense organsAcetylcholinemedicine.drugBrain research. Molecular brain research
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Induction of trefoil factor (TFF)1, TFF2 and TFF3 by hypoxia is mediated by hypoxia inducible factor-1: implications for gastric mucosal healing

2008

Background and purpose:  Mucosal microcirculation is compromised during gastric damage induced by non-steroidal anti-inflammatory drugs, such as aspirin. Consequently, oxygen supply to epithelial cells is decreased. The trefoil factor (TFF) peptides are involved in mechanisms of defence and repair in the gastrointestinal tract but their regulation at sites of gastric injury is unknown. Experimental approach:  Hypoxia and expression of TFF genes and peptides were measured in the damaged stomach of aspirin-treated rats. In a human gastric cell line (AGS cells), the effects of hypoxia and of hypoxia inducible factor (HIF)-1 (through transient transfection of HIF-1α siRNA or over-expression of …

Malemedicine.medical_specialtyTFF peptidesBiologyCell LineRats Sprague-DawleyInternal medicineGene expressionmedicineGastric mucosaAnimalsHumansRNA MessengereducationPharmacologyeducation.field_of_studyAspirinhypoxiaStomachAnti-Inflammatory Agents Non-SteroidalHIF-1Trefoil factor 2Epithelial CellsTransfectionHypoxia (medical)TFF genesepithelial cellsCell HypoxiaRatsUp-RegulationEndocrinologymedicine.anatomical_structureHypoxia-inducible factorsCell cultureGastric Mucosagastric damageCancer researchHypoxia-Inducible Factor 1Trefoil Factor-2medicine.symptomPeptidesResearch Paper
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Identification of calcium sensing receptor (CaSR) mRNA-expressing cells in normal and injured rat brain

2009

Calcium sensing receptor (CaSR), isolated for the first time from bovine and human parathyroid, is a G-protein-coupled receptors that has been involved in diverse physiological functions. At present a complete in vivo work on the identification of CaSR mRNA-expressing cells in the adult brain lacks and this investigation was undertaken in order to acquire more information on cell type expressing CaSR mRNA in the rat brain and to analyse for the first time its expression in different experimental models of brain injury. The expression of CaSR mRNAs was found mainly in scattered cells throughout almost all the brain regions. A double labeling analysis showed a colocalization of CaSR mRNA expr…

Malemedicine.medical_specialtyTime FactorsCentral nervous systemHippocampusCell CountSettore BIO/11 - Biologia MolecolareBiologySettore BIO/09 - Fisiologiachemistry.chemical_compoundSeizuresInternal medicineSettore BIO/10 - BiochimicaCaSRmedicineAnimalsRNA MessengerRats WistarIbotenic AcidMolecular BiologyIn Situ HybridizationNeuronsKainic AcidGeneral NeuroscienceDentate gyrusBrainColocalizationImmunohistochemistryRatsOligodendrogliamedicine.anatomical_structureEndocrinologynervous systemchemistryBrain InjuriesNeurogliaNeurology (clinical)Pyramidal cellCaSR; BrainCalcium sensing receptor (CaSR) isolated for the first time from bovine and human parathyroid is a G-protein-coupled receptors that has been involved in diverse physiological functions. At present a complete in vivo work on the identification of CaSR mRNA-expressing cells in the adult brain lacks and this investigation was undertaken in order to acquire more information on cell type expressing CaSR mRNA in the rat brain and to analyse for the first time its expression in different experimental models of brain injury. The expression of CaSR mRNAs was found mainly in scattered cells throughout almost all the brain regions. A double labeling analysis showed a colocalization of CaSR mRNA expression in neurons and oligodendrocytes whereas it was not found expressed both in the microglia and in astrocytes. One week after kainate-induced seizure CaSR was found in the injured CA3 region of the hippocampus and very interestingly it was found up-regulated in the neurons of CA1-CA2 and dentate gyrus. Similarly 1 week following ibotenic acid injection in the hippocampus CaSR mRNA expression was increased in oligodendrocytes both in the lesioned area and in the contralateral CA1-CA3 pyramidal cell layers and dentate gyrus. One week after needle-induced mechanical lesion an increase of labeled cells expressing CaSR mRNA was observed along the needle track. In conclusion the present results contribute to extend available data on cell type-expressing CaSR in normal and injured brain and could spur to understand the role of CaSR in repairing processes of brain injury.Receptors Calcium-SensingIbotenic acidDevelopmental BiologyAstrocyte
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The impact of ovarian stimulation on the expression of candidate reprogramming genes in mouse preimplantation embryos.

2012

Ovarian stimulation with gonadotrophins is an integral part of assisted reproductive technologies in human subfertility/infertility treatment. Recent findings have associated ovarian stimulation with the increased incidence of imprinting disorders in humans as well as defects in genome-wide methylation reprogramming and, in particular, imprinting in mice. Here, we present the first study that determined the impact of ovarian stimulation on the expression of developmentally important reprogramming genes <i>(Apex1, Lig1, Lig3, Mbd2, Mbd3, Mbd4, </i>and<i> Polb)</i> in single early mouse morula embryos (16-cell stage). Using absolute quantification of mRNA by quantitati…

Malemedicine.medical_specialtyTime FactorsGonadotropins EquineDown-RegulationStimulationReproductive technologyBiologyChorionic GonadotropinMBD4AndrologyMiceOogenesisOvulation InductionInternal medicineGeneticsmedicineDNA-(Apurinic or Apyrimidinic Site) LyaseAnimalsHumansHorsesRNA MessengerMolecular BiologyGenetics (clinical)GametogenesisDNA Polymerase betaRegulation of gene expressionReverse Transcriptase Polymerase Chain ReactionEmbryogenesisGene Expression Regulation DevelopmentalEmbryoDNA-Binding ProteinsMice Inbred C57BLEndocrinologyBlastocystMicroscopy Fluorescenceembryonic structuresFemaleReprogrammingTranscription FactorsCytogenetic and genome research
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Postexercise myostatin and activin IIb mRNA levels: effects of strength training.

2007

ABSTRACTHULMI, J. J., J. P. AHTIAINEN, T. KAASALAINEN, E. PO¨LLA¨NEN, K. HA¨KKINEN, M. ALEN, H. SELA¨NNE, V. KOVANEN,and A. A. MERO. Postexercise Myostatin and Activin IIb mRNA Levels: Effects of Strength Training. Med. Sci. Sports Exerc., Vol.39, No. 2, pp. 289–297, 2007. Purpose: Muscle hypertrophy is likely to result from the cumulative effects of repeated bouts ofresistance exercise (RE) on postexercise molecular responses. Therefore, we determined muscle growth- and regeneration-relatedmRNA expression in response to a single RE bout both before and after a strength-training (ST) period. By means of this novellongitudinal setting, we examined whether postexercise gene expression at the …

Malemedicine.medical_specialtyTime FactorsWeight LiftingVastus lateralis muscleGene ExpressionPhysical Therapy Sports Therapy and RehabilitationPhysical exerciseMyostatinMyoDMuscle hypertrophyQuadriceps MuscleTransforming Growth Factor betaInternal medicinemedicineHumansOrthopedics and Sports MedicineRNA MessengerLeg pressMuscle SkeletalExerciseMyogeninAgedbiologybusiness.industryAge FactorsMiddle AgedMyostatinmusculoskeletal systemActivinsEndocrinologyCase-Control Studiesbiology.proteinmedicine.symptombusinessMuscle contractionMuscle ContractionMedicine and science in sports and exercise
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Delayed increase of S100A9 messenger RNA predicts hospital-acquired infection after septic shock.

2011

OBJECTIVE: Septic shock remains a serious disease with high mortality and increased risk of hospital-acquired infection. The prediction of outcome is of the utmost importance for selecting patients for therapeutic strategies aiming to modify the immune response. The aim of this study was to assess the capability of S100A9 messenger RNA in whole blood from patients with septic shock to predict survival and the occurrence of hospital-acquired infection. DESIGN: Cohort study. SETTING: Two intensive care units in a university hospital. SUBJECTS: The study included patients with septic shock (n = 166) and healthy volunteers (n = 44). INTERVENTIONS: None. MEASUREMENTS AND MAIN RESULTS: For the pa…

Malemedicine.medical_specialtyTime Factors[SDV]Life Sciences [q-bio]Critical Care and Intensive Care Medicine03 medical and health sciences0302 clinical medicinePredictive Value of TestsInternal medicineIntensive careHospital-acquired infectionmedicineCalgranulin BHumans030212 general & internal medicineRNA MessengerComputingMilieux_MISCELLANEOUS030304 developmental biologyAged0303 health sciencesCross InfectionSeptic shockbusiness.industryOdds ratioMiddle Agedmedicine.diseaseShock Septic3. Good health[SDV] Life Sciences [q-bio]Intensive Care Units[SDV.SPEE] Life Sciences [q-bio]/Santé publique et épidémiologieShock (circulatory)Predictive value of testsBiomarker (medicine)[SDV.SPEE]Life Sciences [q-bio]/Santé publique et épidémiologiemedicine.symptombusinessBiomarkersCohort studyCritical care medicine
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Group II metabotropic glutamate receptor activation by agonist LY379268 treatment increases the expression of brain derived neurotrophic factor in th…

2009

A number of in vitro and in vivo studies using selective agonists have indicated a neuroprotective role for group-II metabotropic glutamate (mGlu2/3) receptors in various models of neuronal injury. Although an interplay among neurotrophic factors and mGlu2/3 receptors signalling system has been suggested as possible mechanism involved on neuroprotection, at present poor information are available concerning the in vivo regulation by mGlu2/3 receptors activation of specific neurotrophic factors. By using in situ hybridization and western blotting methods the aim of present study was to analyse the potential regulatory role of selective mGluR2/3 agonist LY379268 treatment on brain derived neur…

Malemedicine.medical_specialtyTime Factorsmedicine.drug_classTropomyosin receptor kinase BBiologyReceptors Metabotropic GlutamateHippocampusSettore BIO/09 - FisiologiaMiceNeurotrophic factorsInternal medicineExcitatory Amino Acid AgonistsmedicineAnimalsRNA MessengerAmino Acidsneurotrophic factorNeuronsBrain-derived neurotrophic factorDose-Response Relationship DrughippocampuBrain-Derived Neurotrophic FactorGeneral NeuroscienceGlutamate receptorBrainLY379268Bridged Bicyclo Compounds HeterocyclicReceptor antagonistUp-RegulationMice Inbred C57BLMetabotropic receptorEndocrinologyBDNFXanthenesMetabotropic glutamate receptorcerebral cortexMetabotropic glutamate receptor 2Excitatory Amino Acid AntagonistsmGlu2/3 receptor
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Hepatocyte growth factor/hepatopoietin A is expressed in fat-storing cells from rat liver but not myofibroblast-like cells derived from fat-storing c…

1992

Hepatocyte growth factor/hepatopoietin A is a complete mitogen for parenchymal liver cells, and its expression is increased as an early response to acute liver injury. To identify the liver cell population responsible for hepatocyte growth factor gene expression, we investigated tissue sections and isolated and purified cell fractions from normal rat liver by in situ and Northern blot hybridization. Hepatocyte growth factor transcripts were present in sinusoidal liver cells, which were preferentially located in the periportal parenchyma. Northern hybridization analysis of RNA isolated from purified liver cell fractions demonstrated that HGF messenger RNA is present only in fat-storing cells…

Malemedicine.medical_specialtyTranscription Geneticmedicine.medical_treatmentGene ExpressionMESSENGER-RNA; MOLECULAR-CLONING; MARKED INCREASE; VITAMIN-A; PURIFICATION; COLLAGEN; SEQUENCE; STORAGE; GENESBiologyCell LineParacrine signallingInternal medicinemedicineAnimalsNorthern blotRNA MessengerGrowth SubstancesHepatologyHepatocyte Growth FactorGrowth factorLiver cellNucleic Acid HybridizationMuscle SmoothRats Inbred StrainsFibroblastsBlotting NorthernLipid MetabolismCell biologyRatsEndocrinologymedicine.anatomical_structureHepatocyte nuclear factor 4LiverCell cultureHepatocyteHepatocyte growth factormedicine.drug
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6-Hydroxydopamine lesioning differentially affects α-synuclein mRNA expression in the nucleus accumbens, striatum and substantia nigra of adult rats

2002

The effect of a unilateral 6-hydroxydopamine (6-OHDA) lesion and/or repeated administration of levodopa (L-DOPA) to normal and 6-OHDA-lesioned rats on alpha-synuclein mRNA expression was investigated by in situ hybridization histochemistry. A 6-OHDA lesion of the nigro-striatal pathway alone, confirmed by the loss of nigral tyrosine hydroxylase mRNA expression, markedly decreased alpha-synuclein mRNA in the lesioned substantia nigra (SN). In contrast, the levels of alpha-synuclein mRNA in the denervated striatum and nucleus accumbens were not altered. Chronic administration of L-DOPA to normal or 6-OHDA-lesioned rats had no effect on alpha-synuclein mRNA expression in the SN, striatum or nu…

Malemedicine.medical_specialtyTyrosine 3-MonooxygenaseDopamineanimal diseasesDopamine AgentsSynucleinsNerve Tissue ProteinsSubstantia nigraStriatumNucleus accumbensBiologyDrug Administration ScheduleNucleus Accumbenschemistry.chemical_compoundDopamineInternal medicineBasal gangliamedicineAnimalsTyrosine hydroxylase mRNARNA MessengerRats WistarOxidopamineNeuronsHydroxydopamineTyrosine hydroxylaseGeneral NeuroscienceParkinson Diseaseα-Synuclein mRNARatsnervous system diseasesNeostriatumSubstantia NigraEndocrinologynervous systemchemistrySympatholyticsalpha-SynucleinSettore BIO/14 - Farmacologia6-HydroxydopamineOxidopaminemedicine.drugNeuroscience Letters
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