Search results for "microenvironment"

showing 10 items of 369 documents

Extracellular heat shock proteins in cancer: From early diagnosis to new therapeutic approach

2021

In cancer, human cells lose the ability to properly control the series of events that occur constantly during cell growth and division, including protein expression, stability, and dynamics. Heat shock proteins (Hsps) are key molecules in these events, constitutively expressed at high levels and could furthermore be induced by the response to cancer-induced stress. In tumor cells, Hsps have been shown to be implicated in the regulation of apoptosis, immune responses, angiogenesis and metastasis; in some cases, they can be overexpressed and dysregulated, representing important cancer hallmarks. In the past few years, it has been demonstrated that Hsps can be released by tumor cells through s…

endocrine systemCancer ResearchAngiogenesisApoptosischemical and pharmacologic phenomenaBiologyMetastasisImmune systemNeoplasmsHeat shock proteinBiomarkers TumorTumor MicroenvironmentmedicineExtracellularHumansHeat-Shock ProteinsEarly Detection of CancerTumor microenvironmentSettore BIO/16 - Anatomia UmanaCell growthCancerextracellular vesicles Heat shock proteins Liquid biopsyMolecular chaperones.Cancerhemic and immune systemsmedicine.diseaseCell biologybiological sciencesSeminars in Cancer Biology
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New Frontiers in Cancer Imaging and Therapy Based on Radiolabeled Fibroblast Activation Protein Inhibitors: A Rational Review and Current Progress.

2021

Over the past decade, the tumor microenvironment (TME) has become a new paradigm of cancer diagnosis and therapy due to its unique biological features, mainly the interconnection between cancer and stromal cells. Within the TME, cancer-associated fibroblasts (CAFs) demonstrate as one of the most critical stromal cells that regulate tumor cell growth, progression, immunosuppression, and metastasis. CAFs are identified by various biomarkers that are expressed on their surfaces, such as fibroblast activation protein (FAP), which could be utilized as a useful target for diagnostic imaging and treatment. One of the advantages of targeting FAP-expressing CAFs is the absence of FAP expression in q…

fibroblast activation proteincongenital hereditary and neonatal diseases and abnormalitiesStromal cellmedicine.medical_treatmentcancer-associated fibroblastPharmaceutical Science610 Medicine & healthCancer imagingReviewfibroblast activation protein inhibitorMetastasisPharmacy and materia medicaFibroblast activation protein alphaDrug Discoverymedicinetumor microenvironment610 Medicine & healthneoplasmsradiotherapynuclear imagingTumor microenvironmentbusiness.industryRCancerImmunosuppressionmedicine.diseasedigestive system diseasesRadiation therapyRS1-441Cancer researchMolecular MedicineMedicinebusiness
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The Ferroxidase Hephaestin in Lung Cancer: Pathological Significance and Prognostic Value

2021

AbstractIron is a fundamental nutrient utilized by living cells to support several key cellular processes. Despite its paramount role to sustain cell survival, excess of labile iron availability can inflict severe cell damage via reactive oxygen species generation which, in turn, can promote neoplastic transformation. The lung is particularly sensitive to iron-induced oxidative stress, given the high oxygen tensions herein present. Moreover, cigarette smoke as well as air pollution particulate can function as vehicles of iron supply, leading to an iron dysregulation condition shown to be crucial in the pathogenesis of several respiratory diseases including lung cancer. Hephaestin (HEPH) bel…

hephaestinCancer ResearchHephaestinSettore MED/08 - Anatomia Patologicamedicine.disease_causeironmedicineSettore MED/05 - Patologia ClinicaNeoplastic transformationLung cancerCell damageRC254-282Original ResearchTumor microenvironmentbiologybioinformaticCancerNeoplasms. Tumors. Oncology. Including cancer and carcinogensbioinformaticsmedicine.diseaseGene expression profilingbioinformatics; hephaestin; immunohistochemistry; iron; lung cancerlung cancerOncologyimmunohistochemistrybiology.proteinCancer researchAdenocarcinomaCeruloplasminCarcinogenesisOxidative stressFrontiers in Oncology
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C1q–Ha matrix regulates the local synthesis of hyaluronan in malignant pleural mesothelioma by modulating has3 expression

2021

Increased hyaluronic acid (HA) production is often associated with cancer progression. In malignant pleural mesothelioma (MPM), HA is found at elevated levels in pleural effusions and sera of patients, and it has been widely debated whether MPM cells are able to produce HA by themselves or through the release of growth factors stimulating other cells. Another key component of the MPM microenvironment is C1q, which can act as a pro-tumorigenic factor favoring cell adhesion, migration and proliferation. The aim of the current study was to prove that MPM primary cells are able to synthesize HA and to inquire the stimulus given by C1q&ndash

hyaluronan synthaseCancer ResearchComplement systemHyaluronic acidMalignant pleural mesotheliomahyaluronan synthasesMatrix (biology)lcsh:RC254-282Articlechemistry.chemical_compoundImmune systemHyaluronan synthaseHyaluronic acidhyaluronic acidmalignant pleural mesotheliomacancertumor microenvironmentC1q; Cancer; Complement system; HAS3; Hyaluronan synthases; Hyaluronic acid; Immune system; Malignant pleural mesothelioma; Tumor microenvironmenttumor microenvironment.Cell adhesioncomplement systemC1qCancerTumor microenvironmentMessenger RNAChemistrylcsh:Neoplasms. Tumors. Oncology. Including cancer and carcinogensComplement systemimmune systemHAS3Immune systemOncologyTumor microenvironmentCancer researchIntracellular
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Molecular Pathways Implicated in Radioresistance of Glioblastoma Multiforme: What Is the Role of Extracellular Vesicles?

2023

Glioblastoma multiforme (GBM) is a primary brain tumor that is very aggressive, resistant to treatment, and characterized by a high degree of anaplasia and proliferation. Routine treatment includes ablative surgery, chemotherapy, and radiotherapy. However, GMB rapidly relapses and develops radioresistance. Here, we briefly review the mechanisms underpinning radioresistance and discuss research to stop it and install anti-tumor defenses. Factors that participate in radioresistance are varied and include stem cells, tumor heterogeneity, tumor microenvironment, hypoxia, metabolic reprogramming, the chaperone system, non-coding RNAs, DNA repair, and extracellular vesicles (EVs). We direct our a…

hypoxiatheranosticOrganic Chemistrynon-coding RNADNA repairGeneral Medicinepersonalized medicineCatalysisComputer Science ApplicationsInorganic Chemistryradioresistancestem cellglioblastoma multiformechaperone systemtumor heterogeneityintercellular communicationtumor microenvironmentmetabolic reprogrammingextracellular vesiclePhysical and Theoretical ChemistryMolecular BiologySpectroscopy
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Effects of B-Cell Lymphoma on the Immune System and Immune Recovery after Treatment: The Paradigm of Targeted Therapy

2022

B-cell lymphoma and lymphoproliferative diseases represent a heterogeneous and complex group of neoplasms that are accompanied by a broad range of immune regulatory disorder phenotypes. Clinical features of autoimmunity, hyperinflammation, immunodeficiency and infection can variously dominate, depending on the immune pathway most involved. Immunological imbalance can play a role in lymphomagenesis, also supporting the progression of the disease, while on the other hand, lymphoma acts on the immune system to weaken immunosurveillance and facilitate immunoevasion. Therefore, the modulation of immunity can have a profound effect on disease progression or resolution, which makes the immune syst…

immunosenescenceLymphoma B-CellimmunosuppressionLymphomaB-cell lymphomaOrganic ChemistryGeneral Medicineimmune recoverychemotherapytargeted therapyImmunotherapy AdoptiveLymphoproliferative DisordersCatalysisComputer Science ApplicationsCAR-TSettore MED/15 - Malattie Del SangueInorganic ChemistryImmune Systemimmune therapyTumor MicroenvironmentimmunoevasionHumansPhysical and Theoretical ChemistryMolecular BiologySpectroscopy
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Osteoclast Immunosuppressive Effects in Multiple Myeloma: Role of Programmed Cell Death Ligand 1

2018

Immunomodulatory drugs and monoclonal antibody-based immunotherapies have significantly improved the prognosis of the patients with multiple myeloma (MM) in the recent years. These new classes of reagents target malignant plasma cells (PCs) and further modulate the immune microenvironment, which prolongs anti-MM responses and may prevent tumor occurrence. Since MM remains an incurable cancer for most patients, there continues to be a need to identify new tumor target molecules and investigate alternative cellular approaches using gene therapeutic strategies and novel treatment mechanisms. Osteoclasts (OCs), as critical multi-nucleated large cells responsible for bone destruction in >80% …

lcsh:Immunologic diseases. Allergy0301 basic medicineCarcinogenesisAngiogenesismedicine.medical_treatmentOsteoimmunologyT cellPlasma CellsProgrammed Cell Death 1 ReceptorImmunologyOsteoclastsCell CommunicationReviewB7-H1 AntigenImmune tolerance03 medical and health sciencesImmune systemAntigens NeoplasmImmune ToleranceTumor MicroenvironmentmedicineAnimalsHumansImmunology and AllergyBone ResorptionImmunologic Surveillancebone marrow microenvironmentTumor microenvironmentbusiness.industryprogrammed cell death ligand 1Immunotherapymultiple myeloma030104 developmental biologymedicine.anatomical_structureprogrammed cell death 1osteoclastosteoblastCancer researchimmunotherapylcsh:RC581-607businessB7-H1 AntigenSignal TransductionFrontiers in Immunology
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Role of Type I and II Interferons in Colorectal Cancer and Melanoma

2017

Cancer can be considered an aberrant organ with a hierarchical composition of different cell populations. The tumor microenvironment, including the immune cells and related cytokines, is crucial during all the steps of tumor development. In particular, type I and II interferons are involved in a plethora of mechanisms that regulate immune responses in cancer, thus balancing immune escape versus immune surveillance. Interferons are involved in both the direct and indirect regulation of cancer cell proliferation and metastatic potential. The mutational background of genes involved in interferons signaling could serve as a prognostic biomarker and a powerful tool to screen cancer patients elig…

lcsh:Immunologic diseases. Allergy0301 basic medicineColorectal cancerCellImmunologyContext (language use)ReviewBiology03 medical and health sciencesImmune systemInterferonmedicineAnti-cancer therapy; Cancer; Cancer progression; Colon; Interferon; Melanoma; Tumor immunology; Immunology and Allergy; ImmunologymelanomaImmunology and Allergycancertumor immunologyTumor microenvironmentcolonMelanomaCancerinterferonmedicine.diseasecancer progression030104 developmental biologymedicine.anatomical_structureImmunologyCancer researchlcsh:RC581-607anti-cancer therapymedicine.drugFrontiers in Immunology
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Hyaluronic Acid Present in the Tumor Microenvironment Can Negate the Pro-apototic Effect of a Recombinant Fragment of Human Surfactant Protein D on B…

2020

Copyright © 2020 Murugaiah, Agostinis, Varghese, Belmonte, Vieni, Alaql, Alrokayan, Khan, Kaur, Roberts, Madan, Bulla and Kishore. Human surfactant protein D (SP-D) belongs to the family of collectins that is composed of a characteristic amino-terminal collagenous region and a carboxy-terminal C-type lectin domain. Being present at the mucosal surfaces, SP-D acts as is a potent innate immune molecule and offers protection against non-self and altered self-such as pathogens, allergens, and tumour. Here, we examined the effect of a recombinant fragment of human SP-D (rfhSP-D) on a range of breast cancer lines. Breast cancer has four molecular subtypes characterised by varied expression of oes…

lcsh:Immunologic diseases. Allergy0301 basic medicinesurfactant protein DImmunologyCollectinApoptosisBreast Neoplasms03 medical and health sciencesbreast cancer0302 clinical medicineEpidermal growth factorCell Line Tumorhyaluronic acidTumor MicroenvironmentHumansImmunology and Allergyskin and connective tissue diseasesinnate immunityOriginal ResearchTumor microenvironmentChemistryimmune surveillanceIntrinsic apoptosisCell cyclePulmonary Surfactant-Associated Protein DRecombinant Proteins030104 developmental biologyApoptosisCell cultureSKBR3Cancer researchFemalelcsh:RC581-607030215 immunologyFrontiers in Immunology
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Is the Complement Protein C1q a Pro- or Anti-tumorigenic Factor? Bioinformatics Analysis Involving Human Carcinomas

2019

C1q is the first subcomponent of the classical pathway of the complement system and belongs to the C1q/Tumor Necrosis Factor superfamily. C1q can perform a diverse range of immune and non-immune functions in a complement-dependent as well as -independent manner. Being a pattern recognition molecule of the innate immunity, C1q can recognize a number of self, non-self and altered-self ligands and bring about effector mechanisms designed to clear pathogens via opsonisation and inflammatory response. C1q is locally synthesized by macrophages and dendritic cells, and thus, can get involved in a range of biological processes, such as angiogenesis and tissue remodeling, immune modulation, and immu…

lcsh:Immunologic diseases. Allergy0301 basic medicinetumorLung NeoplasmsMicroenvironmentPrognosiImmunologyComplementBreast Neoplasmschemical and pharmacologic phenomenaKaplan-Meier EstimateBiology03 medical and health sciencesClassical complement pathway0302 clinical medicineImmune systemimmune system diseasesmedicineHumansImmunology and Allergycomplementclassical pathwayskin and connective tissue diseasesC1qOriginal ResearchTumorInnate immune systemEffectorComplement C1qComputational BiologyCancerPrognosismedicine.diseasemicroenvironmentKidney NeoplasmsComplement systemClear cell renal cell carcinomaC1q; Classical pathway; Complement; Microenvironment; Prognosis; Tumor030104 developmental biologyClassical pathwayCancer researchAdenocarcinomaprognosislcsh:RC581-607030215 immunologyFrontiers in Immunology
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