Search results for "microsomes"

showing 10 items of 193 documents

Irreversible binding of acrylonitrile to nucleic acids

1983

1. [2,3-14C]Acrylonitrile was incubated with rat-liver microsomes, NADPH and either DNA, RNA or bovine serum albumin. Irreversible binding occurred to the macromolecular targets. Binding was lower when incubations were performed without microsomes. 2. Most of the 14C bound to DNA, RNA or polynucleotides (poly-A, poly-C, poly-G, poly-U) after incubation of [2,3-14C]acrylonitrile with rat-liver microsomes and 'conventional' re-isolation of the nucleic acids was removed from the macromolecular target when subsequently chromatographed on hydroxyapatite. 3. Radioactivity attached to DNA after prolonged non-enzymic incubations with [2,3-14C]acrylonitrile was also removed from the DNA by chromatog…

MaleAlkylationHealth Toxicology and MutagenesisIn Vitro TechniquesToxicologyBiochemistrychemistry.chemical_compoundNucleic AcidsNitrilesAnimalsCarbon RadioisotopesBovine serum albuminPharmacologyAcrylonitrilebiologyRNARats Inbred StrainsGeneral MedicineRatschemistryBiochemistryPolynucleotideMicrosomes Liverbiology.proteinMicrosomeNucleic acidAcrylonitrileDNAMacromoleculeXenobiotica
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Metabolism of tresperimus by rat aorta semicarbazide-sensitive amine oxidase (SSAO).

2002

Tresperimus (Cellimis), a new immunosuppressive agent, is mainly eliminated in the rat through metabolism, in which the oxidative deamination of the primary amine of the drug plays a major role. We have previously demonstrated in vivo the significant involvement of semicarbazide-sensitive amine oxidase (SSAO) in this reaction. Rat aorta, a tissue with one of the highest specific SSAO activities, was tested as a new in vitro model to elucidate tresperimus metabolism, using a combination of liquid chromatography/mass spectrometry (LC/MS) and high-performance liquid chromatography (HPLC) analyses. The metabolites resulting from the main metabolic pathway of the drug were formed in rat aorta ho…

MaleAmine oxidaseMonoamine oxidaseDeaminationLysyl oxidaseAorta ThoracicIn Vitro TechniquesGas Chromatography-Mass SpectrometryRats Sprague-DawleyMicrosomesAnimalsPharmacology (medical)Chromatography High Pressure LiquidPharmacologyChemistryAmine oxidase (copper-containing)Oxidative deaminationMetabolismHydrogen-Ion ConcentrationRatsBiochemistryDeaminationAminopropionitrileAmine Oxidase (Copper-Containing)CarbamatesDrug metabolismImmunosuppressive AgentsFundamentalclinical pharmacology
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Characterization of highly polar DNA adducts derived from dibenz[A,H]anthracene (DBA), 3,4-dihydroxy-3,4-dihydro-DBA, and 3,4,10,11-tetrahydroxy-3,4,…

1993

Two highly polar DNA adducts were found after metabolic activation of 3,4,10,11-tetrahydroxy-3,4,10,11-tetrahydrodibenz[ a,h]anthracene(DBA-3,4;10, 11-bisdiol) by liver microsomes isolated from male Sprague-Dawley rats pretreated with Aroclor 1254 in presence of calf thymus DNA. These DNA adducts could be assigned to the metabolites of dibenz[ a,h]anthracene (DBA), of 3R,4R,10R,11R-tetrahydroxy-3,4,10,11-tetrahydro-DBA and of 3R,4R,10S,US-tetrahydroxy-3,4,10,11-tetrahydro-DBA. DNA adducts derived from metabolites of 3S,4S,10S,11S-tetrahydroxy-3,4,10,11-tetrahydro-DBA were not found. These highly polar adducts also could be detected by reversed phase HPLC after incubation of dibenz[ a,h]ant…

MaleAnthraceneStereochemistryHealth Toxicology and MutagenesisPublic Health Environmental and Occupational HealthReversed-phase chromatographyDNAToxicologyIn vitroAdductRatsRats Sprague-Dawleychemistry.chemical_compoundchemistryBenz(a)AnthracenesMicrosomes LiverOrganic chemistryDibenz(ah)anthraceneAnimalsEnantiomerIncubationDNAToxicology and industrial health
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Antioxidant Profile of Mono-and Dihydroxylated Flavone Derivatives in Free Radical Generating Systems

1995

Abstract A number of free radical generating systems were used to investigate the antioxidant properties and structure-activity relationships of a series of monohydroxylated and dihydrox­ylated flavones. Ortho-dihydroxylated flavones showed the highest inhibitory activity on en­ zymic and non-enzymic microsomal lipid peroxidation as well as on peroxyl radical scaveng­ing. Most flavones were weak scavengers of hydroxyl radical, while ortho-dihydroxylated flavones interacted with superoxide anion generated by an enzymic system or by human neutrophils. This series of compounds did not exert cytotoxic effects on these cells. Scaveng­ing of superoxide and peroxyl radicals may determ ine the anti…

MaleAntioxidantFree RadicalsNeutrophilsStereochemistrymedicine.medical_treatmentIn Vitro TechniquesHydroxylationFlavonesAntioxidantsGeneral Biochemistry Genetics and Molecular BiologyLipid peroxidationStructure-Activity Relationshipchemistry.chemical_compoundSuperoxidesmedicineAnimalsHumansOrganic chemistryRats WistarFlavonoidschemistry.chemical_classificationMolecular StructureHydroxyl RadicalChemistrySuperoxideFlavone derivativesFree Radical ScavengersPeroxidesRatsPeroxyl radicalsMicrosomes LiverMicrosomeHydroxyl radicalLipid PeroxidationNADPZeitschrift für Naturforschung C
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Elevation of hepatic epoxide hydratase activity by ethoxyquin is due to increased synthesis of the enzyme.

1980

Abstract Feeding of the antioxidant ethoxyquin to rats leads to an increase of epoxide hydratase activity in liver microsomes. The apparent half life of the increase is 3–4 days. Elevation of epoxide hydratase activity is also obtained by intraperitoneal treatment of mice with ethoxyquin. This elevation is prevented by concomitant treatment with cycloheximide. When radiolabelled leucine is incorporated into microsomal protein by liver cell fractions from either ethoxyquin-fed or untreated rats, gel electrophoresis reveals that ethoxyquin feeding increases incorporation into epoxide hydratase. These results suggest that the elevation of epoxide hydratase activity by ethoxyquin is due to incr…

MaleAntioxidantmedicine.medical_treatmentBiophysicsCycloheximideBiochemistrySubstrate Specificitychemistry.chemical_compoundEthoxyquinmedicineAnimalsEnzyme inducerBenzopyrenesCycloheximideMolecular Biologychemistry.chemical_classificationEpoxide HydrolasesEthoxyquinbiologyLiver cellCell BiologyRatsEnzymechemistryBiochemistryEnzyme Inductionbiology.proteinMicrosomeMicrosomes LiverQuinolinesLeucineBiochemical and biophysical research communications
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Effects of a water-soluble extract of rosemary and its purified component rosmarinic acid on xenobiotic-metabolizing enzymes in rat liver

2001

The effects of a water-soluble extract (WSE) of rosemary and its purified antioxidant rosmarinic acid (RA) on xenobiotic metabolizing enzymes (XME) were studied in rat liver after dietary administration. The modulation of phase I enzymes such as cytochrome P450 (CYP) 1A, 2B, 2E1, 3A, and phase II enzymes such as glutathione S-transferase (GST), quinone reductase (QR) and UDP-glucuronosyltransferase (UGT) was evaluated by measuring enzyme activities with specific substrates. Protein levels of CYPs and rGST A1/A2, A3/A5, M1, M2 and P1 were measured using antibodies in Western blots. Caffeic acid was also studied because it results from RA biotransformation in rat after oral administration. Ma…

MaleAntioxidantmedicine.medical_treatment[SDV]Life Sciences [q-bio]ReductaseToxicologychemistry.chemical_compoundCytosol0302 clinical medicine[SDV.IDA]Life Sciences [q-bio]/Food engineeringCaffeic acidChromatography High Pressure LiquidComputingMilieux_MISCELLANEOUSchemistry.chemical_classification0303 health sciencesbiologyRosmarinic acidOrgan SizeGeneral Medicine[SDV.IDA] Life Sciences [q-bio]/Food engineeringStimulation Chemical3. Good health[SDV] Life Sciences [q-bio]LiverBiochemistry030220 oncology & carcinogenesisMicrosomes Liver[SPI.GPROC] Engineering Sciences [physics]/Chemical and Process EngineeringImmunoblottingDepsidesdigestive systemFlavonesXenobiotics03 medical and health sciencesmedicineAnimals[SPI.GPROC]Engineering Sciences [physics]/Chemical and Process EngineeringRats Wistar030304 developmental biologyFlavonoidsLamiaceaePlant ExtractsTerpenesBody WeightROMARINCytochrome P450GlutathioneDietRatsEnzymechemistryCinnamatesbiology.proteinRATSpectrophotometry UltravioletBiomarkersFood Science
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Metabolic Activation of the (+)-S,S- and (−)-R,R-Enantiomers of trans-11,12-Dihydroxy-11,12-dihydrodibenzo[a,l]pyrene:  Stereoselectivity, DNA Adduct…

1997

Polycyclic aromatic hydrocarbons require metabolic activation in order to exert their biological activity initiated by DNA binding. The metabolic pathway leading to bay or fjord region dihydrodiol epoxides as ultimate mutagenic and/or carcinogenic metabolites is thought to play a dominant role. For dibenzo[a,l]pyrene, considered as the most potent carcinogenic polycyclic aromatic hydrocarbon, the formation of the fjord region syn- and/or anti-11,12-dihydrodiol 13,-14-epoxide (DB[a,l]PDE) diastereomers has been found to be the principal metabolic activation pathway in cell cultures leading to DNA adducts. In order to further elucidate the stereoselectivity involved in this activation pathway…

MaleAroclorsStereochemistryToxicologyChinese hamsterDihydroxydihydrobenzopyrenesRats Sprague-DawleyDNA AdductsMicechemistry.chemical_compoundCricetulusCricetinaepolycyclic compoundsAnimalsBiotransformationCarcinogenchemistry.chemical_classificationCarcinogenic Polycyclic Aromatic HydrocarbonbiologyStereoisomerismGeneral MedicineChlorodiphenyl (54% Chlorine)biology.organism_classificationRatsMetabolic pathwayEnzymechemistryCarcinogensMicrosomes LiverMicrosomePyreneStereoselectivityMutagensChemical Research in Toxicology
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Metabolic activation of aflatoxin B1 to aflatoxin B1-8,9-epoxide in woodchucks undergoing chronic active hepatitis

1997

Chronic hepatitis B virus infection as well as consumption of food contaminated with the mycotoxin aflatoxin B1 are considered to be 2 major risk factors for the development of primary liver cancer in humans. Furthermore, epidemiological surveys indicate that hepatitis B virus and aflatoxin B1 might act synergistically to induce primary liver cancer. In the present study, we have tested the hypothesis that the metabolic activation of aflatoxin B1 to aflatoxin B1-8,9-epoxide, the ultimate mutagenic and carcinogenic mycotoxin metabolite, is enhanced in an experimental model of chronic hepatitis using woodchucks, chronically infected with the woodchuck hepatitis virus. Woodchuck liver microsom…

MaleCancer ResearchAflatoxinAflatoxin B1virusesBiologymedicine.disease_causeViruschemistry.chemical_compoundHepatitis B ChronicmedicineAnimalsHepatitis B Virus Woodchuckheterocyclic compoundsMycotoxinCarcinogenHepatitisHepatitis B virusWoodchuck hepatitis virustechnology industry and agriculturefood and beveragesCancermedicine.diseasebiology.organism_classificationVirologyOncologychemistryHepatitis Viral AnimalMarmotaMicrosomes LiverFemaleMutagensInternational Journal of Cancer
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Characterization of DNA adducts at the bay region of dibenz[a,h]anthracene formed in vitro

1993

Bay region diolepoxide-DNA adducts of dibenz[a,h]anthracene (DBA) formed in vitro were identified and their absolute stereochemistry was assigned. After activation of [5,12-14C]DBA with liver microsomes obtained from Aroclor 1254 treated male Sprague-Dawley rats in the presence of calf thymus DNA for 1 h, the amount of DNA adducts was found to be 9.9 +/- 2.4 pmol/mg DNA, calculated on the basis of the portion of radioactivity eluted from the HPLC reversed-phase column with a water/acetonitrile gradient. Bay region diolepoxide-DNA adducts represented 27.5% of radioactivity associated with DNA adducts. The absolute configuration of the various adducts was determined from the reaction of the (…

MaleCancer ResearchAnthraceneMetaboliteAbsolute configurationStereoisomerismDNAGeneral MedicineIn Vitro TechniquesHigh-performance liquid chromatographyMedicinal chemistryRatsAdductRats Sprague-DawleyDNA Adductschemistry.chemical_compoundchemistryBiochemistryDeoxyadenosineBenz(a)AnthracenesMicrosomes LiverAnimalsDeoxyguanosineDibenz(ah)anthraceneBiotransformationCarcinogenesis
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Biological activation of 1,3-butadiene to vinyl oxirane by rat liver microsomes and expiration of the reactive metabolite by exposed rats.

1983

When 1,3-butadiene is incubated with rat liver microsomes and NADPH both enantiomers of vinyl oxirane are formed, the amount of epoxide being dependent on incubation time, microsomal protein, and substrate concentration. Inhibition by SKF 525 A or dithiocarb as well as induction by pretreatment with phenobarbital or 20-methylcholanthrene suggest participation of cytochrome P-450 in this reaction. The amount of epoxide is enhanced by addition of 1,1,1-trichloropropene oxide and reduced by glutathione, especially in the presence of hepatic cytosol. When rats are exposed to 1,3-butadiene in a closed chamber (conditions of maximal metabolism) vinyl oxirane is exhaled and can be quantitatively d…

MaleCancer ResearchCytochromeMetaboliteEpoxideIn Vitro TechniquesAcetonechemistry.chemical_compoundEthers CyclicmedicineButadienesAnimalsBiotransformationbiology13-ButadieneRats Inbred StrainsStereoisomerismGeneral MedicineGlutathioneMetabolismRatsOncologychemistryBiochemistryMicrosomebiology.proteinMicrosomes LiverEpoxy CompoundsPhenobarbitalmedicine.drugMutagensJournal of cancer research and clinical oncology
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