Search results for "mito"

showing 10 items of 2513 documents

Impaired cellular immune responses in chronic renal failure: Evidence for a T cell defect

1986

Impaired cellular immune responses in chronic renal failure: Evidence for a T cell defect. Cellular immune responses in vitro were studied in 24 patients on chronic hemodialysis and 16 healthy volunteers with normal kidney function. Patients on maintenance hemodialysis had lymphopenia with diminished numbers of both T4 + and T8 + T-lymphocytes. The T4/T8 ratios were within the normal range. Peripheral blood lymphocytes (PBL) showed a diminished proliferative response upon stimulation with concanavalin A, phytohemagglutinin and poke weed mitogen. When cell surface antigens were used for stimulation (mixed lymphocyte culture) uremic lymphocytes also showed a lower proliferation rate. Although…

AdultMaleIsoantigensT-LymphocytesLymphocyteT cellLymphocyte CooperationRenal functionStimulationchemical and pharmacologic phenomenaLymphocyte ActivationLeukocyte CountImmune systemmedicineHumansLymphocytesImmunodeficiencyAgedUremiaB-LymphocytesImmunity Cellularbiologybusiness.industryAntibodies MonoclonalMiddle Agedmedicine.diseaseIn vitromedicine.anatomical_structureNephrologyConcanavalin AAntibody FormationImmunologybiology.proteinInterleukin-2Kidney Failure ChronicFemaleMitogensbusinessKidney International
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Oxidative stress markers at birth: Analyses of a neonatal population

2015

In order to further understand neonatal stress and, thus, control it efficaciously, there is a need for more information on the manifestations of stress at the molecular level in the newborn, with particular regard to oxidants, and anti-oxidant and anti-stress mechanisms, including mitochondrial heat shock protein-chaperones such as Hsp60. We investigated patterns of anti-oxidants, biomarkers of oxidative stress, and Hsp60 levels in sera from newborns and found significant associations between glutathione (GSH) levels and gestational age, delivery modality, and lipid hydroperoxydes (LOOH) level. LOOH levels and spontaneous (vaginal) delivery were independently associated with increased GSH …

AdultMaleLipid Peroxidesanimal structuresHistologyNeonatal stressPopulationNeonatal strePhysiologyOxidative-stress markerDiseaseBiologymedicine.disease_causeMitochondrial Proteinschemistry.chemical_compoundLipid hydroperoxydemedicineHumanseducationOxidative-stress markerseducation.field_of_studyfungiInfant NewbornAnti-stress moleculeGestational ageChaperonin 60Cell BiologyGeneral MedicineGlutathioneHsp60GlutathioneNeonatal stress; Oxidative-stress markers; Lipid hydroperoxydes; Anti-stress molecules; Glutathione; Hsp60Oxidative StressAdult lifeLipid hydroperoxydeschemistryAnti-stress moleculesImmunologyFemaleHSP60BiomarkersOxidative stressNeonatal stressActa Histochemica
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The link between mitochondrial DNA hypervariable segment I heteroplasmy and ageing among genetically unrelated Latvians

2011

International audience; Various studies have demonstrated that mitochondrial DNA (mtDNA) heteroplasmy tends to increase with age and that the observed frequency of heteroplasmy among populations mostly depends on the way it is measured. Therefore, we investigated age-related association on the presence of mtDNA heteroplasmy within the hypervariable segment 1 (HVS-I) in a selected study group. The study group consisted of 300 maternally unrelated Latvians ranging in age from 18 to over 90years. To determine the optimal method for mtDNA heteroplasmy detection, three approaches were used: (i) SURVEYORTM Mutation Detection Kit, (ii) sequencing and (iii) denaturing gradient-gel electrophoresis (…

AdultMaleMitochondrial DNAAgingAdolescentBiologyBiochemistryDNA MitochondrialBroad spectrumYoung AdultEndocrinologyLatviansGeneticsHumansMutation detectionheteroplasmydetection of mtDNA heteroplasmyMolecular BiologyAgedGeneticsAged 80 and overDenaturing Gradient Gel ElectrophoresismtDNA[SDV.MHEP.GEG]Life Sciences [q-bio]/Human health and pathology/Geriatry and gerontologyCell BiologySequence Analysis DNAMiddle AgedLatviaHeteroplasmyAgeingageingMutationFemaleHuman mitochondrial DNA haplogroup
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Deciphering exome sequencing data: Bringing mitochondrial DNA variants to light

2019

The expanding use of exome sequencing (ES) in diagnosis generates a huge amount of data, including untargeted mitochondrial DNA (mtDNA) sequences. We developed a strategy to deeply study ES data, focusing on the mtDNA genome on a large unspecific cohort to increase diagnostic yield. A targeted bioinformatics pipeline assembled mitochondrial genome from ES data to detect pathogenic mtDNA variants in parallel with the "in-house" nuclear exome pipeline. mtDNA data coming from off-target sequences (indirect sequencing) were extracted from the BAM files in 928 individuals with developmental and/or neurological anomalies. The mtDNA variants were filtered out based on database information, cohort …

AdultMaleMitochondrial DNAAtaxiaAdolescentDevelopmental Disabilities[SDV]Life Sciences [q-bio]BiologyDNA MitochondrialGenomeHaplogroupYoung Adult03 medical and health sciencesExome SequencingGeneticsmedicineHumansChildExomeGenetics (clinical)Exome sequencingComputingMilieux_MISCELLANEOUSAged030304 developmental biologyAged 80 and overGenetics0303 health sciences030305 genetics & heredityInfant NewbornComputational BiologyGenetic VariationInfantMiddle AgedPhenotypeEarly DiagnosisChild PreschoolFemaleNervous System Diseasesmedicine.symptom
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Epigenetic modifications precede molecular alterations and drive human hepatocarcinogenesis

2021

Development of primary liver cancer is a multistage process. Detailed understanding of sequential epigenetic alterations is largely missing. Here, we performed Infinium Human Methylation 450k BeadChips and RNA-Seq analyses for genome-wide methylome and transcriptome profiling of cirrhotic liver (n = 7), low- (n = 4) and high-grade (n = 9) dysplastic lesions, and early (n = 5) and progressed (n = 3) hepatocellular carcinomas (HCC) synchronously detected in 8 patients with HCC with chronic hepatitis B infection. Integrative analyses of epigenetically driven molecular changes were identified and validated in 2 independent cohorts comprising 887 HCCs. Mitochondrial DNA sequencing was further em…

AdultMaleMitochondrial DNACarcinoma HepatocellularCirrhosisMolecular biologyCarcinogenesisBiologyEpigenesis GeneticHepatitis B ChronicmedicineHumansEpigeneticsAgedHepatologyGene Expression ProfilingLiver NeoplasmsDNA NeoplasmGeneral MedicineMethylationDNA MethylationMiddle AgedHCCSmedicine.diseaseGene Expression Regulation NeoplasticOncologyApoptosisDNA methylationCancer researchEpigeneticsCalmodulin-Binding ProteinsFemaleLiver cancerLiver cancerResearch ArticleJCI Insight
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Could mitochondrial haplogroups play a role in sporadic amyotrophic lateral sclerosis?

2004

Mitochondrial impairment has been implicated in the pathogenesis of the amyotrophic lateral sclerosis (ALS). Furthermore, mitochondrial-specific polymorphisms were previously related to other neurodegenerative diseases, such as Parkinson, Friedreich and Alzheimer disease. To investigate if specific genetic polymorphisms within the mitochondrial genome (mtDNA) could act as susceptibility factors and contribute to the clinical expression of sporadic ALS (sALS), we have genotyped predefined European mtDNA haplogroups in 222 Italian patients with sALS and 151 matched controls. Individuals classified as haplogroup I demonstrated a significant decrease in risk of ALS versus individuals carrying t…

AdultMaleMitochondrial DNAPathologymedicine.medical_specialtyDiseaseBiologyDNA MitochondrialHaplogroupCohort StudiesDegenerative diseaseConfidence IntervalsOdds RatiomedicineHumansAmyotrophic lateral sclerosisAgedALS; Haplogroups; mtDNA;Polymorphism GeneticmtDNAGeneral NeuroscienceAmyotrophic Lateral SclerosisOdds ratioMiddle Agedmedicine.diseaseMitochondriaALS; mtDNA; HaplogroupsHaplotypesALS; Haplogroups; mtDNAImmunologyHaplogroupsFemaleAlzheimer's diseaseALSHuman mitochondrial DNA haplogroup
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Application of mtDNA sequence analysis in forensic casework for the identification of human remains

2000

Abstract In four forensic cases of unidentified skeletal remains investigated in the last year, we were able to attach three to missing persons. In one case we could show that the discovered bone sample did not fit to a missing child. The method for mitochondrial DNA analysis for the routine identification of skeletal remains was established in our institute by typing bone samples of defined age obtained from Frankfurt's cemetery. Reproducible results were obtained for bones up to 75 years old. For analysis the bone samples were pulverised to fine powder, decalcified and DNA was extracted. From the DNA we amplified a 404-bp fragment from HV-1 and a 379-bp fragment from HV-2 of the mtDNA con…

AdultMaleMitochondrial DNASequence analysisMinisatellite RepeatsBiologyDNA MitochondrialPolymerase Chain ReactionBone and BonesPathology and Forensic Medicinelaw.inventionlawAge Determination by SkeletonHumansChildPolymerase chain reactionGeneticsmtDNA control regionForensic anthropologySequence Analysis DNADNA FingerprintingHypervariable regionForensic identificationDNA profilingForensic AnthropologyFemaleLawForensic Science International
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Mitochondrial dysfunction, persistent oxidative damage, and catalase inhibition in immune cells of naïve and treated Crohnʼs disease

2009

Background: Oxidative stress is considered a potential etiological factor for Crohn's disease (CD). We characterized the reactive oxygen species (ROS) generated in immune peripheral cells of CD patients, as well as their antioxidant enzyme status and the presence of oxidative damage. In addition, mitochondrial function (ΔΨm) was analyzed to detect the possible origin of ROS. Methods: Cells were obtained from patients at the onset of disease, prior to any treatment. Experiments were repeated when patients were in clinical remission. A set of experiments was carried out in a group of CD patients in persistent morphological remission. Controls were healthy volunteers who were not receiving any…

AdultMaleMitochondrial DiseasesMitochondrionBiologyPharmacologymedicine.disease_causeInflammatory bowel diseaseAntioxidantsMonocytesNitric oxideSuperoxide dismutasechemistry.chemical_compoundCrohn DiseaseMalondialdehydemedicineHumansImmunology and AllergyLymphocytesMembrane Potential Mitochondrialchemistry.chemical_classificationReactive oxygen speciesSuperoxide DismutaseSuperoxideGastroenterologyDeoxyguanosineHydrogen PeroxideCatalaseOxidantsmedicine.diseaseOxidative Stresschemistry8-Hydroxy-2'-DeoxyguanosineCatalaseCase-Control StudiesImmunologybiology.proteinFemaleReactive Oxygen SpeciesOxidative stressGranulocytesInflammatory Bowel Diseases
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Uncoupling protein-2 (UCP2) induces mitochondrial proton leak and increases susceptibility of non-alcoholic steatohepatitis (NASH) liver to ischaemia…

2008

Background: The mechanisms of progression from fatty liver to steatohepatitis and cirrhosis are not well elucidated. Mitochondrial dysfunction represents a key factor in the progression of non-alcoholic steatohepatitis (NASH) as mitochondria are the main cellular site of fatty acid oxidation, ATP synthesis and reactive oxygen species (ROS) production. Aims: (1) To evaluate the role of the uncoupling protein 2 in controlling mitochondrial proton leak and ROS production in NASH rats and humans; and (2) to assess the acute liver damage induced by ischaemia–reperfusion in rats with NASH. Methods: Mitochondria were extracted from the livers of NASH humans and rats fed a methionine and choline de…

AdultMaleMitochondrial ROSmedicine.medical_specialtyMitochondria LiverMitochondrionBiologymedicine.disease_causeIon ChannelsMitochondrial ProteinsAdenosine TriphosphateInternal medicinemedicineAnimalsHumansUncoupling proteinUncoupling Protein 2Rats WistarBeta oxidationAdenosine TriphosphatasesMembrane Potential MitochondrialAldehydesFatty liverGastroenterologyMiddle Agedmedicine.diseaseRatsFatty LiverOxidative StressEndocrinologyMitochondrial respiratory chainLiverBiochemistryReperfusion InjuryAcute DiseaseDisease ProgressionFemaleSteatohepatitisReactive Oxygen SpeciesOxidative stressGut
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Gemcitabine plus metronomic 5-fluorouracil or capecitabine as a second-/third-line chemotherapy in advanced adrenocortical carcinoma: a multicenter p…

2010

Adrenocortical carcinoma (ACC) is a rare neoplasm characterized by poor prognosis. First-line systemic treatments in advanced disease include mitotane, either alone or in combination with chemotherapy. Studies evaluating second-line therapy options have obtained disappointing results. This trial assessed the activity and toxicity of gemcitabine plus metronomic fluoropyrimidines in heavily pretreated advanced ACC patients. From 1998 to 2008, 28 patients with advanced ACC progressing after mitotane plus one or two systemic chemotherapy lines were enrolled. They received a combination of i.v. gemcitabine (800 mg/m2, on days 1 and 8, every 21 days) and i.v. 5-fluorouracil protracted infusion (2…

AdultMaleMucositisOncologyAntimetabolites AntineoplasticCancer Researchmedicine.medical_specialtyAdrenocortical carcinomaSettore MED/06 - Oncologia MedicaEndocrinology Diabetes and Metabolismmedicine.medical_treatmentPhases of clinical researchDeoxycytidineGastroenterologyDrug Administration ScheduleCapecitabineEndocrinologyInternal medicineAntineoplastic Combined Chemotherapy ProtocolsmedicineMucositisHumansMitotaneCapecitabineAgedChemotherapybusiness.industryLeukopeniaMiddle Agedmedicine.diseaseThrombocytopeniaGemcitabineAdrenal Cortex NeoplasmsGemcitabineRegimenTreatment OutcomeOncologyFluorouracilDisease ProgressionFemaleFluorouracilbusinessmedicine.drug
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