Search results for "molecolare"

showing 10 items of 303 documents

Bioactive molecules from soil and marine bacteria: new potential applications

2015

bioactive moleculeSettore BIO/11 - Biologia Molecolarebioactive molecules; soil; sea urchinsoilsea urchin
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Isolation of organophosphorus-degrading bacteria from agricultural mediterranean soils

2010

biodegradationorganophosphorus pesticides solid phase micro-extractionSettore AGR/13 - Chimica AgrariaSettore BIO/11 - Biologia Molecolare
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Metodologie Biomolecolari per la Conservazione e il Restauro

2011

biodeteriogeni biotecnologie molecolari DNA microbico PCR DNA microarraySettore BIO/11 - Biologia Molecolare
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Il Tabulario della Cattedrale. Studio Conoscitivo e Restauro di Documenti in Carta e Pergamena

2008

biodeterioramento biotecnologie carta pergamenaSettore BIO/11 - Biologia Molecolare
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Caratterizzaione di consorzi microbici causa di deterioramento dei beni culturali e potenziali patogeni per l'uomo mediante analisi della molecola de…

2008

biodeterioramento biotecnologie patogeni PCR DNA microarraySettore BIO/11 - Biologia Molecolare
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Indagini Biomolecolari per la identificazione di specie batteriche deteriogene sui dipinti

2008

biodeterioramento biotecnologie restauroSettore BIO/11 - Biologia Molecolare
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Intragenic G-quadruplex structure formed in the human CD133 and its biological and translational relevance.

2016

Cancer stem cells (CSCs) have been identified in several solid malignancies and are now emerging as a plausible target for drug discovery. Beside the questionable existence of CSCs specific markers, the expression of CD133 was reported to be responsible for conferring CSC aggressiveness. Here, we identified two G-rich sequences localized within the introns 3 and 7 of the CD133 gene able to form G-quadruplex (G4) structures, bound and stabilized by small molecules. We further showed that treatment of patient-derived colon CSCs with G4-interacting agents triggers alternative splicing that dramatically impairs the expression of CD133. Interestingly, this is strongly associated with a loss of C…

cancer stem cells0301 basic medicineDNA damageSettore BIO/11 - Biologia MolecolareTumor initiationBiologyG-quadruplex03 medical and health sciencesCancer stem cellAntigens CDCell Line TumorG-QuadruplexeGeneticsHumansNeoplasm InvasivenessAC133 AntigenGeneGlycoproteinsCell ProliferationSettore MED/04 - Patologia GeneraleNeoplasm InvasiveneG-quadruplexProtein BiosynthesiDrug discoveryGene regulation Chromatin and EpigeneticsAlternative splicingIntroncd133Molecular biologyG-QuadruplexesGene Expression Regulation Neoplastic030104 developmental biologyCell Transformation NeoplasticDrug Resistance NeoplasmProtein BiosynthesisPeptideNeoplastic Stem CellsCancer researchNeoplastic Stem CellSettore MED/46 - Scienze Tecniche Di Medicina Di LaboratorioGlycoproteinPeptidesHuman
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CeRNA and interactome bioinformatic analyses on human telomerase

2014

We present a classic interactome bioinformatic analysis and a study on competing endogenous (ce) RNAs for hTERT. The hTERT gene codes for the catalytic subunit and limiting component of the human telomerase complex. Human telomerase reverse transcriptase (hTERT) is essential for the integrity of telomeres. Telomere dysfunctions have been widely reported to be involved in aging, cancer, and cellular senescence. The hTERT gene network has been analyzed using the BioGRID interaction database (http://thebiogrid.org/) and related analysis tools such as Osprey (http://biodata.mshri.on.ca/osprey/servlet/Index) and GeneMANIA (http://genemania.org/). The network of interaction of hTERT transcripts h…

ceRNA hTERT HSP90Settore BIO/11 - Biologia MolecolareSettore MED/13 - Endocrinologia
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Effetti di mutazioni diverse di ras in cellule di carcinoma colorettale (HT29)

2011

cellule di carcinoma colorettale (HT29).mutazioni diverseSettore BIO/11 - Biologia Molecolarera
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Epigenetic involvement in Hutchinson-Gilford progeria syndrome: a mini-review.

2013

Hutchinson-Gilford progeria syndrome (HGPS) is a rare human genetic disease that leads to a severe premature ageing phenotype, caused by mutations in the <i>LMNA</i> gene. The <i>LMNA</i> gene codes for lamin-A and lamin-C proteins, which are structural components of the nuclear lamina. HGPS is usually caused by a de novo <i>C1824T</i> mutation that leads to the accumulation of a dominant negative form of lamin-A called progerin. Progerin also accumulates physiologically in normal ageing cells as a rare splicing form of lamin-A transcripts. From this perspective, HGPS cells seem to be good candidates for the study of the physiological mechanisms of ageing…

congenital hereditary and neonatal diseases and abnormalitiesAgingEuchromatinSettore BIO/11 - Biologia MolecolarecernaBiologySettore MED/13 - EndocrinologiaEpigenesis GeneticLMNAHistonesAdenosine TriphosphateProgeriaHGPS Progeria; epigenetics; chromatin; cernamedicineHumansEpigeneticsProtein PrecursorsChildEpigenesisGeneticsCell NucleusProgeriaintegumentary systemnutritional and metabolic diseasesNuclear ProteinsDNA Methylationmedicine.diseaseProgerinChromatin Assembly and DisassemblyLamin Type AChromatinCell biologySettore BIO/18 - GeneticaMicroRNAsSettore MED/03 - Genetica MedicaMutationHGPS ProgeriachromatinNuclear laminaGeriatrics and GerontologyepigeneticMi-2 Nucleosome Remodeling and Deacetylase ComplexGerontology
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