Search results for "molecular targeted therapy"

showing 10 items of 163 documents

Imaging to study solid tumour origin and progression: lessons from research and clinical oncology

2017

Biomedical imaging in recent decades has clarified our understanding of normal and pathological cellular processes in vivo. In particular, this approach recently provided insights into processes occurring at a molecular or genetic level rather than at the anatomical level. The evolution of this discipline by engineering have led to its integration into biomedical research to (1) increase sensitivity and resolution imaging and to (2) improve tissue and cell specificity. Currently, imaging approaches are used in three different biomedical areas: (a) identification of cellular processes in physiological and disease state; (b) in vivo single-cell imaging; and (c) identification of new prognosti…

Diagnostic Imaging0301 basic medicineOncologymedicine.medical_specialtyPathologyeducationImmunologyBiologyMedical OncologyMultimodal Imaging03 medical and health sciences0302 clinical medicineCancer stem cellNeoplasmsInternal medicinemedicineAnimalsHumansImmunology and AllergyMolecular Targeted TherapySolid tumourClinical OncologyResearchOptical ImagingCell BiologyImaging cell biology cancer stem cellsMolecular ImagingCell Transformation Neoplastic030104 developmental biology030220 oncology & carcinogenesisDisease ProgressionNeoplastic Stem Cells
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Can NF-κB Be Considered a Valid Drug Target in Neoplastic Diseases? Our Point of View

2020

Multidrug resistance (MDR), of the innate and acquired types, is one of major problems in treating tumor diseases with a good chance of success. In this review, we examine the key role of nuclear factor-kappa B (NF-κB) to induce MDR in three tumor models characterized precisely by innate or acquired MDR, in particular triple negative breast cancer (TNBC), hepatocellular carcinoma (HCC), and acute myeloid leukemia (AML). We also present different pharmacological approaches that our group have employed to reduce the expression/activation of this transcriptional factor and thus to restore chemo-sensitivity. Finally, we examine the latest scientific evidence found by other groups, the most sign…

Drug targetAntineoplastic AgentsReviewCatalysisNF-κBdrug targetlcsh:ChemistryInorganic Chemistrychemistry.chemical_compoundNeoplasmsMDRmedicineBiomarkers TumorcancerAnimalsHumansMolecular Targeted TherapyPhysical and Theoretical Chemistrylcsh:QH301-705.5Molecular BiologySpectroscopyTriple-negative breast cancerbusiness.industryOrganic ChemistryNF-kappa BCancerMyeloid leukemiaNF-κBGeneral Medicinemedicine.diseaseComputer Science ApplicationsMultiple drug resistanceClinical trialCell Transformation Neoplasticlcsh:Biology (General)lcsh:QD1-999chemistryDrug Resistance NeoplasmHepatocellular carcinomaCancer researchSettore BIO/14 - FarmacologiaDisease SusceptibilitybusinessInternational Journal of Molecular Sciences
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Network pharmacology of cancer: From understanding of complex interactomes to the design of multi-target specific therapeutics from nature

2015

Despite massive investments in drug research and development, the significant decline in the number of new drugs approved or translated to clinical use raises the question, whether single targeted drug discovery is the right approach. To combat complex systemic diseases that harbour robust biological networks such as cancer, single target intervention is proved to be ineffective. In such cases, network pharmacology approaches are highly useful, because they differ from conventional drug discovery by addressing the ability of drugs to target numerous proteins or networks involved in a disease. Pleiotropic natural products are one of the promising strategies due to their multi-targeting and d…

EpigenomicsProteomics0301 basic medicineDrugmedia_common.quotation_subjectSystems biologyGene regulatory networkSynthetic lethalityDiseaseComputational biologyBiologyPharmacology03 medical and health sciencesNeoplasmsDrug DiscoveryBiomarkers TumormedicineAnimalsHumansMetabolomicsGene Regulatory NetworksMolecular Targeted TherapyProtein Interaction Mapsmedia_commonPharmacologyPlants MedicinalDrug discoveryGene Expression ProfilingSystems BiologyCancermedicine.diseaseAntineoplastic Agents PhytogenicGene Expression Regulation Neoplastic030104 developmental biologyBiological networkPhytotherapySignal TransductionPharmacological Research
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Colorectal cancer defeating? Challenge accepted!

2013

Colorectal tumours are actually considered as aberrant organs, within it is possible to notice a different stage of cell growth and differentiation. Their origin is reported to arise from a subpopulation of tumour cells endowed with, just like the healthy stem cells, self-renewal and aberrant multi-lineage differentiation capacity likely to be called colorectal cancer stem cells (CCSCs). Cancer stem cells (CSCs) fate, since their origin, reflects the influences from their microenvironment (or niche) both in the maintenance of stemness, in promoting their differentiation, and in inducing epithelial-mesenchymal transition, responsible of CSCs dissemination and subsequent formation of metastat…

Epithelial-Mesenchymal TransitionColorectal cancerClinical BiochemistryBiologyBiochemistryImmune systemCancer stem cellmedicineTumor MicroenvironmentAnimalsHumansMolecular Targeted TherapyCytotoxicityMolecular BiologyCell growthChemotaxisGeneral MedicineCell cyclemedicine.diseaseGene Expression Regulation NeoplasticDrug Resistance NeoplasmCancer stem cell Colorectal cancer Immune system Individualized therapy Targeting Tumour microenvironment.ImmunologyCancer researchNeoplastic Stem CellsMolecular MedicineStem cellColorectal NeoplasmsSignal TransductionMolecular aspects of medicine
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Metabolite Sensing GPCRs: Promising Therapeutic Targets for Cancer Treatment?

2020

G-protein-coupled receptors constitute the most diverse and largest receptor family in the human genome, with approximately 800 different members identified. Given the well-known metabolic alterations in cancer development, we will focus specifically in the 19 G-protein-coupled receptors (GPCRs), which can be selectively activated by metabolites. These metabolite sensing GPCRs control crucial processes, such as cell proliferation, differentiation, migration, and survival after their activation. In the present review, we will describe the main functions of these metabolite sensing GPCRs and shed light on the benefits of their potential use as possible pharmacological targets for cancer treat…

G-protein-coupled receptorMetaboliteReviewComputational biologyBiologyReceptors G-Protein-CoupledBile Acids and Saltschemistry.chemical_compoundNeoplasmsmetabolite sensing GPCRBiomarkers TumormedicinecancerAnimalsHumansMolecular Targeted TherapyAmino AcidsReceptorlcsh:QH301-705.5G protein-coupled receptorCell growthDisease ManagementCancerGeneral MedicineLipid Metabolismmedicine.diseaseCancer treatmentlcsh:Biology (General)Gene Expression RegulationchemistryHuman genomeDisease SusceptibilityCancer developmentEnergy MetabolismSignal TransductionCells
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The GABAergic System and the Gastrointestinal Physiopathology.

2015

Since the first report about the presence of γ-aminobutyric acid (GABA) within the gastrointestinal (GI) tract, accumulating evidence strongly supports the widespread representation of the GABAergic system in the enteric milieu, underlining its potential multifunctional role in the regulation of GI functions in health and disease. GABA and GABA receptors are widely distributed throughout the GI tract, constituting a complex network likely regulating the diverse GI behaviour patterns, cooperating with other major neurotransmitters and mediators for maintaining GI homeostasis in physiologic and pathologic conditions. GABA is involved in the circuitry of the enteric nervous system, controlling…

GABA AgentsGastrointestinal DiseasesPharmacologyBiologySettore BIO/09 - FisiologiaEnteric Nervous Systemgamma-Aminobutyric acidgaba enteric neuronParacrine signallingImmune systemReceptors GABAparasitic diseasesDrug DiscoverymedicineAnimalsHumansMolecular Targeted Therapygamma-Aminobutyric AcidPharmacologyGABAA receptorVisceral painsocial sciencesGastrointestinal TractGABA AgentsDrug Designpopulation characteristicsGABAergicEnteric nervous systemmedicine.symptomhuman activitiesNeurosciencegeographic locationsmedicine.drug
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Disparity between Inter-Patient Molecular Heterogeneity and Repertoires of Target Drugs Used for Different Types of Cancer in Clinical Oncology

2020

Inter-patient molecular heterogeneity is the major declared driver of an expanding variety of anticancer drugs and personalizing their prescriptions. Here, we compared interpatient molecular heterogeneities of tumors and repertoires of drugs or their molecular targets currently in use in clinical oncology. We estimated molecular heterogeneity using genomic (whole exome sequencing) and transcriptomic (RNA sequencing) data for 4890 tumors taken from The Cancer Genome Atlas database. For thirteen major cancer types, we compared heterogeneities at the levels of mutations and gene expression with the repertoires of targeted therapeutics and their molecular targets accepted by the current guideli…

Gene mutationMedical OncologychemotherapyGenomeTranscriptomelcsh:ChemistryDrug Delivery SystemsProstateNeoplasmstumor heterogeneityMedicineCluster AnalysisMolecular Targeted TherapyPathology MolecularPrecision Medicinelcsh:QH301-705.5targeted therapeuticscancer drugsSpectroscopyExome sequencingGeneral MedicineGenomicspersonalized medicineComputer Science ApplicationsDrug repositioningmedicine.anatomical_structureAntineoplastic AgentsComputational biologyCatalysisArticleInorganic Chemistrymolecular diagnosticsGenetic HeterogeneityDrug TherapyExome SequencingHumansPhysical and Theoretical ChemistryMolecular Biologygenomeclinical oncologybusiness.industryOrganic ChemistryMolecular diagnosticsmutationslcsh:Biology (General)lcsh:QD1-999MutationPersonalized medicinebusinesstranscriptomeInternational Journal of Molecular Sciences
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Immunotherapy in gastrointestinal cancer: Recent results, current studies and future perspectives

2016

The new therapeutic approach of using immune checkpoint inhibitors as anticancer agents is a landmark innovation. Early studies suggest that immune checkpoint inhibition might also be effective in patients with gastrointestinal cancer. To improve the efficacy of immunotherapy, different strategies are currently under evaluation. This review summarises the discussion during the European Organisation for Research and Treatment of Cancer Gastrointestinal Tract Cancer Translational Research Meeting in Mainz in November 2014 and provides an update on the most recent results of immune therapy in gastrointestinal cancers. Knowledge of potential relationships between tumour cells and their microenv…

Genetic Markers0301 basic medicineCancer Researchmedicine.medical_treatmentAntineoplastic AgentsTranslational researchContext (language use)Antibodies Monoclonal Humanized03 medical and health sciencesGastrointestinal cancer0302 clinical medicineImmune systemBiomarkers TumormedicineHumansMolecular Targeted TherapyGastrointestinal cancerGastrointestinal NeoplasmsOncolytic Virotherapybusiness.industryCancerCell Cycle CheckpointsImmunotherapymedicine.diseaseImmune checkpointOncolytic virusTreatment Outcome030104 developmental biologyOncology030220 oncology & carcinogenesisImmunologyCancer researchImmunotherapyEpidemiologic MethodsbusinessCheckpoint inhibitorsForecastingEuropean Journal of Cancer
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Cell uptake enhancement of folate targeted polymer coated magnetic nanoparticles.

2013

Dual targeted drug delivery systems represent a potential platform for developing efficient vector to tumor sites. In this study we evaluated a folate- and magnetic-targeted nanocarriers based on 10 nm iron oxide nanodomais coated with the properly synthesized and characterized folic acid (FA)-functionalized amphiphilic copolymer PHEA-PLA-PEG-FA. FA was chemically conjugated to one end of diamino-polyethylene glycol of 2000 Da, in order to ensure its exposition on the polymer coated magnetic nanoparticles (MNPs-FA). The prepared nanoparticles have been exhaustively characterized by different methods, including DLS, SEM, FT-IR and magnetic measurements. Magnetic nanoparticles showed dimensio…

IRON-OXIDE NANOPARTICLES; DRUG-DELIVERY; COPOLYMERSPolymersmedia_common.quotation_subjectBiomedical EngineeringPharmaceutical ScienceMedicine (miscellaneous)NanoparticleBioengineeringFolic AcidCoated Materials BiocompatibleCell Line TumorMaterials TestingHumansGeneral Materials ScienceViability assayMolecular Targeted TherapyInternalizationMagnetite Nanoparticlesmedia_commonChemistryNeoplasms Experimentalequipment and suppliesTreatment OutcomeTargeted drug deliveryCancer cellBiophysicsMCF-7 CellsMagnetic nanoparticlesNanocarriershuman activitiesFolate Targeting; Magnetic Nanoparticles; Cell Uptake; Ferrozine Assay; Polymer CoatingSuperparamagnetismJournal of biomedical nanotechnology
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HSP90 and HSP70: Implication in Inflammation Processes and Therapeutic Approaches for Myeloproliferative Neoplasms.

2015

Myeloproliferative neoplasms (MPN) are clonal stem cell disorders that lead to the excessive production of one or more blood cell lineages. It has been reported that, in most MPN, inflammatory cytokines are frequently increased, indicating that inflammation plays a crucial role in these disorders. Heat shock proteins (HSP) are induced in response to many stressful conditions from heat shock to hypoxia and inflammation. Besides their chaperone and cytoprotective functions, HSPs are key players during inflammation, hence the term “chaperokine.” Through their chaperone activity, HSP90, a stabilizer of many oncogenes (e.g., JAK2), and HSP70, a powerful antiapoptotic chaperone, tightly regulate …

ImmunologyInflammationReview ArticleBiologyModels BiologicalProinflammatory cytokineMyeloproliferative DisordersHeat shock proteinlcsh:PathologymedicineHumansHSP70 Heat-Shock ProteinsHSP90 Heat-Shock ProteinsMolecular Targeted TherapyMyeloproliferative DisordersCell BiologyHsp90Chaperone (protein)ImmunologyCancer researchbiology.proteinmedicine.symptomSignal transductionStem cellInflammation Mediatorslcsh:RB1-214Signal TransductionMediators of inflammation
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