Search results for "nap"

showing 10 items of 2226 documents

Serotonin Involvement in the Basal Ganglia Pathophysiology: Could the 5-HT2C Receptor be a New Target for Therapeutic Strategies?.

2006

The basal ganglia are a highly interconnected group of subcortical nuclei in the vertebrate brain that play a critical role not only in the control of movements but also in some cognitive and behavioral functions. Several recent studies have emphasized that serotonergic pathways in the central nervous system (CNS) are intimately involved in the modulation of the basal ganglia and in the pathophysiology of human involuntary movement disorders. These observations are supported by anatomical evidence demonstrating large serotonergic innervation of the basal ganglia. In fact, serotonergic terminals have been reported to make synaptic contacts with dopamine (DA)-containing neurons and gamma-amin…

Central Nervous SystemSerotoninmedicine.medical_specialtySubstantia nigraBiologyIndirect pathway of movementSerotonergicBiochemistrySerotonin AgentsBasal Ganglia DiseasesDopamineInternal medicineSerotonin AgentsDrug DiscoveryBasal gangliaReceptor Serotonin 5-HT2CmedicineAnimalsHumansBasal ganglia diseasegamma-Aminobutyric AcidNeuronsPharmacologyMovement DisordersOrganic ChemistryParkinson Diseasemedicine.diseasebasal ganglion pathophysiology Basal Ganglia Diseases pathophysiologyGlobus pallidusEndocrinologynervous systemSynapsesMolecular Medicinemedicine.drug
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Agrin in the Developing CNS: New Roles for a Synapse Organizer

2002

The heparan sulfate proteoglycan agrin is responsible for the formation, maintenance, and regeneration of the neuromuscular junction. In the central nervous system, agrin is widely expressed and concentrated at interneuronal synapses, but its function during synaptogenesis remains controversial. Instead, evidence for additional functions of agrin during axonal growth, establishment of the blood-brain barrier, and Alzheimer’s disease is accumulating.

Central Nervous Systemmedicine.medical_specialtyanimal structuresAgrinPhysiologyRegeneration (biology)Central nervous systemSynaptogenesisBiologyHeparan Sulfate ProteoglycansNeuromuscular junctionSynapsemedicine.anatomical_structureEndocrinologynervous systemAlzheimer DiseaseInternal medicineSynapsesmedicineAnimalsHumansAgrinNeurosciencePhysiology
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A novel cholinergic-specific antigen (Chol-2) in mammalian brain.

1993

Three new antisera have been raised in sheep against cholinergic electromotor presynaptic plasma membranes prepared from the electric organs of the electric ray, Torpedo marmorata. They all recognized one or more cholinergic-specific antigens in the mammalian nervous system by the following criteria: they sensitized the cholinergic subpopulation of rat-brain synaptosomes--and only this subpopulation--to lysis by the complement system and, in an immunocytochemical study, selectively stained choline acetyltransferase-positive cholinergic neurons in the rat spinal cord. However, two of the three antisera failed to recognize Chol-1 alpha and -beta, two closely related minor gangliosides already…

Central nervous systemBiologyTorpedoEpitopeAntigenParasympathetic Nervous SystemGangliosidesmedicineAnimalsCholinergic neuronAntigensMolecular BiologyAntiserumElectric OrganGangliosideSheepGeneral NeuroscienceImmune SeraCell MembraneBrainComplement System ProteinsImmunohistochemistryCell biologyComplement systemRatsmedicine.anatomical_structureImmunologyAntigens SurfaceSynapsesCholinergicNeurology (clinical)Developmental BiologySubcellular FractionsBrain research
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Nicotinic receptor function in the mammalian central nervous system.

1995

The diversity of neuronal nicotinic receptors (nAChRs) in addition to their possible involvement in such pathological conditions as Alzheimer's disease have directed our research towards the characterization of these receptors in various mammalian brain areas. Our studies have relied on electrophysiological, biochemical, and immunofluorescent techniques applied to cultured and acutely dissociated hippocampal neurons, and have been aimed at identifying the various subtypes of nAChRs expressed in the mammalian central nervous system (CNS), at defining the mechanisms by which CNS nAChR activity is modulated, and at determining the ion permeability of CNS nAChR channels. Our findings can be sum…

Central nervous systemHippocampal formationNeurotransmissionIn Vitro TechniquesReceptors NicotinicLigandsHippocampusSynaptic TransmissionGeneral Biochemistry Genetics and Molecular BiologyStructure-Activity RelationshipHistory and Philosophy of SciencemedicineAnimalsMagnesiumPhosphorylationReceptorCells CulturedMammalsMolecular StructureChemistryGeneral NeuroscienceAcetylcholineOlfactory bulbElectrophysiologyNicotinic agonistmedicine.anatomical_structurenervous systemCalciumSignal transductionNeuroscienceIon Channel GatingSignal TransductionAnnals of the New York Academy of Sciences
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Interplay between glutamatergic and GABAergic neurotransmission alterations in cognitive and motor impairment in minimal hepatic encephalopathy.

2015

The cognitive and motor alterations in hepatic encephalopathy (HE) are the final result of altered neurotransmission and communication between neurons in neuronal networks and circuits. Different neurotransmitter systems cooperate to modulate cognitive and motor function, with a main role for glutamatergic and GABAergic neurotransmission in different brain areas and neuronal circuits. There is an interplay between glutamatergic and GABAergic neurotransmission alterations in cognitive and motor impairment in HE. This interplay may occur: (a) in different brain areas involved in specific neuronal circuits; (b) in the same brain area through cross-modulation of glutamatergic and GABAergic neur…

CerebellumMinimal hepatic encephalopathyGlutamic AcidNeurotransmissionSynaptic TransmissionCellular and Molecular NeuroscienceGlutamatergicmedicineHyperammonemiaAnimalsHumansGlutamatergic neurotransmissionGABAergic neurotransmissionHepatic encephalopathygamma-Aminobutyric AcidHepatic encephalopathyGABAA receptorbusiness.industryGlutamate receptorHyperammonemiaCognitionCell Biologymedicine.diseaseMotor Skills Disordersmedicine.anatomical_structureHepatic EncephalopathyCognitive functionCognition DisordersbusinessNeuroscience
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Purkinje cell loss and motor coordination defects in profilin1 mutant mice.

2012

Profilin1 is an actin monomer-binding protein, essential for cytoskeletal dynamics. Based on its broad expression in the brain and the localization at excitatory synapses (hippocampal CA3-CA1 synapse, cerebellar parallel fiber (PF)-Purkinje cell (PC) synapse), an important role for profilin1 in brain development and synapse physiology has been postulated. We recently showed normal physiology of hippocampal CA3-CA1 synapses in the absence of profilin1, but impaired glial cell binding and radial migration of cerebellar granule neurons (CGNs). Consequently, brain-specific inactivation of profilin1 by exploiting conditional mutants and Nestin-mediated cre expression resulted in a cerebellar hyp…

CerebellumPatch-Clamp TechniquesPurkinje cellBiophysicsAction PotentialsParallel fiberMice TransgenicNerve Tissue ProteinsBiologyHippocampal formationIn Vitro TechniquesMotor ActivitySynapseNestinMiceProfilinsPurkinje CellsIntermediate Filament ProteinsmedicineAnimalsGeneral NeuroscienceAge FactorsBrainGene Expression Regulation DevelopmentalLong-term potentiationElectric StimulationDisease Models Animalmedicine.anatomical_structurenervous systemCytoarchitectureAnimals NewbornCerebellar cortexMutationDisease ProgressionPsychomotor DisordersNeuroscienceNeuroscience
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Electrophysiological Investigations of Retinogeniculate and Corticogeniculate Synapse Function

2019

The lateral geniculate nucleus is the first relay station for the visual information. Relay neurons of this thalamic nucleus integrate input from retinal ganglion cells and project it to the visual cortex. In addition, relay neurons receive top-down excitation from the cortex. The two main excitatory inputs to the relay neurons differ in several aspects. Each relay neuron receives input from only a few retinogeniculate synapses, which are large terminals with many release sites. This is reflected by the comparably strong excitation, the relay neurons receive, from retinal ganglion cells. Corticogeniculate synapses, in contrast, are simpler with few release sites and weaker synaptic strength…

Cerebral CortexGeneral Immunology and MicrobiologyOptic tractChemistryGeneral Chemical EngineeringGeneral NeuroscienceLateral geniculate nucleusRetinal ganglionGeneral Biochemistry Genetics and Molecular BiologySynapseMiceElectrophysiologymedicine.anatomical_structureVisual cortexSynapsesmedicineExcitatory postsynaptic potentialAnimalsVisual PathwaysNeuronNeuroscienceJournal of Visualized Experiments
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Early GABAergic circuitry in the cerebral cortex.

2013

In the cerebral cortex GABAergic signaling plays an important role in regulating early developmental processes, for example, neurogenesis, migration and differentiation. Transient cell populations, namely Cajal-Retzius in the marginal zone and thalamic input receiving subplate neurons, are integrated as active elements in transitory GABAergic circuits. Although immature pyramidal neurons receive GABAergic synaptic inputs already at fetal stages, they are integrated into functional GABAergic circuits only several days later. In consequence, GABAergic synaptic transmission has only a minor influence on spontaneous network activity during early corticogenesis. Concurrent with the gradual devel…

Cerebral CortexNeuronsGeneral NeuroscienceNeurogenesisNeurotransmissionBiologyInhibitory postsynaptic potentialSynaptic TransmissionCorticogenesismedicine.anatomical_structurenervous systemCerebral cortexSubplateSynapsesmedicineBiological neural networkGABAergicAnimalsHumansNerve NetNeurosciencegamma-Aminobutyric AcidCurrent opinion in neurobiology
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The role of the claustrum in the bilateral control of frontal oculomotor neurons in the cat.

1991

The effect of claustrum (CL) stimulation on the spontaneous unitary activity of ipsi and contralateral frontal oculomotor neurons, was studied in chloralose-anaesthetized cats. A total of 205 units was bilaterally recorded in the medial oculomotor area, homologous of the primate "frontal eye fields"; 127 neurons were identified as projecting to the superior colliculus; for 33 of these last units stimulation of the ipsilateral CL provoked an excitatory effect lasting 10-25 ms and appearing with a latency of 5-15 ms; on 8 units the excitatory effect was followed by an inhibition lasting 100-250 ms. Ninety-eight of the 127 neurons were also tested through activation of the contralateral CL: 13…

Cerebral CortexNeuronsSuperior ColliculiOculomotor nerveGeneral NeuroscienceSuperior colliculusElectroencephalographyAnatomyFrontal eye fieldsBiologyCorpus callosumClaustrumBasal GangliaElectric StimulationStereotaxic Techniquesmedicine.anatomical_structureOculomotor NerveCerebral cortexStereotaxic techniqueNeural PathwaysmedicineExcitatory postsynaptic potentialCatsAnimalsNeuroscienceExperimental brain research
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Genes, dopamine and cortical signal-to-noise ratio in schizophrenia.

2004

A large body of phenomenological evidence implicates abnormal connectivity of brain macrocircuitry and microcircuitry in schizophrenia. Recent discoveries of susceptibility genes for schizophrenia have zeroed in on the synaptic signaling machinery of cortical microcircuits as fundamental to disease causation and have militated for further revision of the role of dopamine in this illness. Dopamine, long implicated in psychosis and in antipsychotic drug effects, is crucial in optimizing signal-to-noise ratio of local cortical microcircuits. This action of dopamine is achieved principally by D1- and D2-receptor-mediated effects on pyramidal and local circuit neurons, which mediate neuronal exc…

Cerebral CortexPsychosisGeneral NeuroscienceDopamineCentral nervous systemGlutamate receptormedicine.diseasechemistry.chemical_compoundmedicine.anatomical_structurechemistryDopamineSchizophreniamedicineSchizophreniaHumansSynaptic signalingNerve NetPsychologyNeurotransmitterNeuroscienceDopamine hypothesis of schizophreniamedicine.drugTrends in neurosciences
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