Search results for "necrosis"

showing 10 items of 1354 documents

Apoptosis in liver disease

2001

A variety of biological functions are regulated through extracellular signals. Amongst the best studied examples is growth control, which is achieved by the regulatory function of growth factors. In recent years it has become apparent that cell death (apoptosis) is controlled in a similar fashion. Apoptosis, firstly a morphologically defined process, is a highly controlled type of cell death that plays a critical role in embryonic development, deletion of autoreactive T-cells and adult tissue homoeostasis. There is increasing evidence that derangement of the apoptotic program is the underlying cause of a series of diseases including liver diseases. The deadly program can be initiated by lig…

ProteasesProgrammed cell deathApoptosisLigandsReceptors Tumor Necrosis FactorFas ligandTransforming Growth Factor beta1Antigens CDTransforming Growth Factor betaExtracellularAnimalsHumansfas ReceptorCaspaseHepatologybiologyLiver DiseasesGastroenterologyFas receptorCell biologyBiochemistryReceptors Tumor Necrosis Factor Type IApoptosisCaspasesbiology.proteinIntracellularEuropean Journal of Gastroenterology & Hepatology
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Cellular effects of bacterial N-3-Oxo-dodecanoyl-L-Homoserine lactone on the sponge Suberites domuncula (Olivi, 1792): insights into an intimate inte…

2014

International audience; Sponges and bacteria have lived together in complex consortia for 700 million years. As filter feeders, sponges prey on bacteria. Nevertheless, some bacteria are associated with sponges in symbiotic relationships. To enable this association, sponges and bacteria are likely to have developed molecular communication systems. These may include molecules such as N-acyl-L-homoserine lactones, produced by Gram-negative bacteria also within sponges. In this study, we examined the role of N-3-oxododecanoyl-L-homoserine lactone (3-oxo-C12-HSL) on the expression of immune and apoptotic genes of the host sponge Suberites domuncula. This molecule seemed to inhibit the sponge inn…

ProteomicsApoptosisPathogenesisPathology and Laboratory MedicineBiochemistrycaspase 74-Butyrolactonecaspase 3lcsh:ScienceCytoskeletoncaspase like 7 gene0303 health sciencesToll-like receptorMarine Ecologytoll like receptorGenomicsproto oncogeneEndocytosisCell biologySuberites domunculaCellular Structures and Organellesalpha actininCell signalingtoll like receptor associated factor 6Gram negative bacteriumparacrine signalingMicrobiology03 medical and health sciencesGeneticsRNA Messengerhost pathogen interactionprotein expressiontwo dimensional electrophoresisBacteria030306 microbiologyEcology and Environmental Scienceslcsh:RBiology and Life SciencesComputational BiologyImmunity Innatecarrier proteinSpongebacterial membranelcsh:Qimmunological toleranceSuberitesProtein AbundanceSuberitessuberites domuncula[SDV]Life Sciences [q-bio]lcsh:MedicineMolecular Cell BiologyMedicine and Health Sciencesinnate immunityperforinMultidisciplinaryEcologybiologymessenger RNAarticlecell communicationAnimal Modelsmatrix assisted laser desorption ionization time of flight mass spectrometryunclassified drugPoriferaHost-Pathogen InteractionscytotoxicityactinTranscriptome Analysishormone actionResearch ArticleSymbiotic bacteriaprotein bcl 2Marine BiologycofilinResearch and Analysis Methodsn (3 oxododecanoyl)homoserine lactoneMicrobial EcologycogninModel OrganismsHomoserineAnimalscontrolled study14. Life underwatergeneSymbiosiscell viabilityadenosine triphosphatase030304 developmental biologynonhumanChemical EcologyMembrane ProteinsCell Biologytumor necrosis factor receptor associated factor 6Genome Analysisbiology.organism_classificationalpha tubulinGene Expression RegulationMembrane proteingene expressioncaspase like 3 geneGenome Expression AnalysisBacteriaPLoS ONE
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Complement proteins regulating macrophage polarisation on biomaterials

2019

[EN] One of the events occurring when a biomaterial is implanted in an host is the protein deposition onto its surface, which might regulate cell responses. When a biomaterial displays a compromised biocompatibility, distinct complement pathways can be activated to produce a foreign body reaction. In this article, we have designed different types of biomaterial surfaces to study the inflammation process. Here, we used different concentrations of (3-glycidoxypropyl)-trimethoxysilane (GPTMS), an organically-modified alkoxysilane as a precursor for the synthesis of various types of sol-gel materials functionalizing coatings for titanium implants to regulate biological responses. Our results sh…

ProteomicsCellBiocompatible Materials02 engineering and technology01 natural sciencesimmune responseMiceColloid and Surface ChemistryCIENCIA DE LOS MATERIALES E INGENIERIA METALURGICATitanium010304 chemical physicsChemistryhybrid sol-gelBiomaterialSurfaces and InterfacesGeneral MedicineSilanes021001 nanoscience & nanotechnologyInterleukin-10medicine.anatomical_structureReconstructive and regenerative medicine Radboud Institute for Molecular Life Sciences [Radboudumc 10]Rabbits0210 nano-technologyBiotechnologyComplement systemBiocompatibilitySurface PropertiesMacrophage polarizationmacrophage plasticityOsseointegrationHybrid sol-gelMacrophage plasticityImmune systemAll institutes and research themes of the Radboud University Medical Centerproteomicsdental implants0103 physical sciencesmedicineAnimalsSecretionParticle SizePhysical and Theoretical ChemistryImmune responsecomplement systemTibiaTumor Necrosis Factor-alphaMacrophagesDental implantsComplement System ProteinsComplement systemRAW 264.7 CellsBiophysics
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Pseudomonas corrugata contains a conserved N-acyl homoserine lactone quorum sensing system; its role in tomato pathogenicity and tobacco hypersensiti…

2007

Pseudomonas corrugata is a phytopathogenic bacterium, causal agent of tomato pith necrosis, yet it is an ubiquitous bacterium that is part of the microbial community in the soil and in the rhizosphere of different plant species. Although it is a very heterogeneous species, all the strains tested were able to produce short chain acyl homoserine lactone (AHL) quorum sensing signal molecules. The main AHL produced was N-hexanoyl-l-homoserine lactone (C6-AHL). An AHL quorum sensing system, designated PcoI/PcoR, was identified and characterized. The role of the quorum sensing system in the expression of a variety of traits was evaluated. Inactivation of pcoI abolished the production of AHLs. The…

Pseudomonas corrugateHypersensitivity responseVirulenceEcologyQuorum SensingSettore AGR/12 - Patologia VegetalePseudomonas corrugatatomato pith necrosisPseudomonaMicrobiologyApplied Microbiology and BiotechnologyPseudomonas corrugata; quorum sensing; tomato pith necrosisTomato pith necrosiSolanum lycopersicum4-ButyrolactonePseudomonasAcyl homoserine lactoneMutationTobaccoMicrobial antagonismLycopersicon esculentum
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Pterostilbene-induced tumor cytotoxicity: a lysosomal membrane permeabilization-dependent mechanism.

2012

The phenolic phytoalexin resveratrol is well known for its health-promoting and anticancer properties. Its potential benefits are, however, limited due to its low bioavailability. Pterostilbene, a natural dimethoxylated analog of resveratrol, presents higher anticancer activity than resveratrol. The mechanisms by which this polyphenol acts against cancer cells are, however, unclear. Here, we show that pterostilbene effectively inhibits cancer cell growth and stimulates apoptosis and autophagosome accumulation in cancer cells of various origins. However, these mechanisms are not determinant in cell demise. Pterostilbene promotes cancer cell death via a mechanism involving lysosomal membrane …

PterostilbeneCancer Treatmentlcsh:MedicineApoptosisResveratrolBiochemistryLung and Intrathoracic Tumorschemistry.chemical_compoundMolecular cell biologyRNA interferenceNeoplasmsPhagosomesStilbenesDrug DiscoveryBreast TumorsBasic Cancer Researchlcsh:ScienceCytotoxicitySkin TumorsApoptotic Signaling CascadeCellular Stress ResponsesMultidisciplinaryMicroscopy ConfocalCell DeathMalignant MelanomaFlow CytometryCellular StructuresSignaling CascadesCell biologyEukaryotic CellsOncologyCaspasesMedicineCellular TypesCell DivisionResearch ArticleSignal TransductionProgrammed cell deathDrugs and DevicesDrug Research and DevelopmentMitosisAntineoplastic AgentsBiologyPermeabilityCell GrowthInhibitory Concentration 50NecrosisComplementary and Alternative MedicineCell Line TumorGastrointestinal TumorsAutophagyHumansHSP70 Heat-Shock ProteinsBiologyCell ProliferationDose-Response Relationship DrugL-Lactate DehydrogenaseCell growthlcsh:RAutophagyProteinsCancers and NeoplasmsRegulatory ProteinschemistrySubcellular OrganellesApoptosisResveratrolCancer celllcsh:QGene expressionLysosomesCytometryPloS one
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Effect of cilomilast (Ariflo) on TNF-α, IL-8, and GM-CSF release by airway cells of patients with COPD

2003

Background: Inflammation in chronic obstructive pulmonary disease (COPD) is characterised by increased neutrophilic infiltration of the airways. Cilomilast, a novel selective phosphodiesterase 4 inhibitor in clinical development for COPD treatment, exerts anti-inflammatory effects. The ability of cilomilast to inhibit the release of neutrophil chemoattractants such as tumour necrosis factor (TNF)-α, interleukin (IL)-8, and granulocyte-macrophage colony stimulating factor (GM-CSF) by bronchial epithelial cells and sputum cells isolated from 10 patients with COPD, 14 normal controls, and 10 smokers was investigated. Methods: Bronchial epithelial cells obtained by bronchial brushing and sputum…

Pulmonary and Respiratory MedicineAdultMaleSputum CytologyCyclohexanecarboxylic AcidsPhosphodiesterase Inhibitorsmedicine.medical_treatmentChronic Obstructive Pulmonary DiseaseCarboxylic AcidsCell CountEnzyme-Linked Immunosorbent AssayRespiratory MucosaBronchial brushingPulmonary Disease Chronic ObstructiveNitrilesmedicineHumansInterleukin 8Cells CulturedAgedbusiness.industryTumor Necrosis Factor-alphaCilomilastInterleukin-8SputumGranulocyte-Macrophage Colony-Stimulating FactorEpithelial CellsMiddle Agedrespiratory tract diseasesBronchodilator AgentsChemotaxis LeukocyteCytokineGranulocyte macrophage colony-stimulating factorImmunologySputumTumor necrosis factor alphaFemalemedicine.symptombusinessmedicine.drug
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Mast cell-derived tumour necrosis factor is essential for allergic airway disease

2007

Mast cells are thought to contribute to allergic airway disease. However, the role of mast cell-produced mediators, such as tumour necrosis factor (TNF), for the development of allergic airway disease is unclear. In order to define the role of mast cells in acute allergic airway disease two strains of mast cell-deficient mice (Kit W/Wv and Kit W-sh/W-sh ) were studied. Compared with their wild-type littermates, Kit W/Wv and Kit W-sh/W-sh mice developed significantly lower airway responsiveness to methacholine and less airway inflammation and goblet cell metaplasia, following sensitisation in the absence of adjuvant and airway challenge. Transfer of bone marrow-derived mast cells (BMMCs) fro…

Pulmonary and Respiratory MedicineAllergyPathologymedicine.medical_specialtyNecrosisInflammationMiceMetaplasiamedicineAnimalsMast CellsInflammationMice KnockoutGoblet cellTumor Necrosis Factor-alphabusiness.industryAllergensrespiratory systemmedicine.diseaseMast cellAsthmarespiratory tract diseasesDisease Models Animalmedicine.anatomical_structureImmunologyImmunizationTumor necrosis factor alphaGoblet CellsBronchial Hyperreactivitymedicine.symptomAirwaybusinessEuropean Respiratory Journal
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Antitumour activity of mononuclear phagocytes: role of tumour necrosis factor alpha.

1992

Tumour necrosis factor alpha (TNF) is a cytokine produced by mononuclear phagocytes (MP) originally discovered for its cytotoxic activity on tumour cell targets. It was subsequently demonstrated that, in addition to its oncolytic potential, TNF exerts a wide variety of activities on the host defensive system against malignancies. This article briefly reviews the current concepts on the role of TNF in the antitumour activity of MP.

Pulmonary and Respiratory MedicineCytotoxicity ImmunologicPhagocytesbusiness.industryTumor Necrosis Factor-alphamedicine.medical_treatmentCellTumour necrosis factor alphaOncolytic virusKiller Cells NaturalMajor Histocompatibility ComplexCytokinemedicine.anatomical_structureNeoplasmsImmunologymedicineCytotoxic T cellHumansTumor necrosis factor alphabusinessRespiration; international review of thoracic diseases
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Lung-restricted activation of the alveolar macrophage/monocyte system in pulmonary sarcoidosis.

1992

An activation of T-cells that is restricted to the lung has been demonstrated in pulmonary sarcoidosis. The role of blood monocytes (MO) and alveolar macrophages (AM) in this concept of compartmentalized inflammation has not yet been evaluated. In order to elucidate this question, we measured the release of tumor necrosis factor alpha (TNF alpha) and interleukin-1 (IL-1) by peripheral blood mononuclear cells (PBMNC) and AM in 43 patients with sarcoidosis (32 with active, 11 with inactive disease) without therapy and correlated the spontaneous monokine release to parameters of the T-cell alveolitis and the course of the disease. TNF alpha as well as IL-1 were spontaneously released by AM of …

Pulmonary and Respiratory MedicineInterleukin 2Lung Diseasesmedicine.medical_specialtyTime FactorsSarcoidosisLung Diseases/metabolism610 MedizinInflammationSarcoidosis/metabolismLymphocyte ActivationMacrophages Alveolar/secretionPeripheral blood mononuclear cellMonocytesInterleukin-1/secretionInternal medicineMacrophages AlveolarmedicineMacrophageHumansddc:610Receptors Interleukin-2/metabolismTumor Necrosis Factor-alpha/secretionbusiness.industryTumor Necrosis Factor-alphaMonocyteLeukocytes Mononuclear/secretionMonocytes/immunologyReceptors Interleukin-2Macrophage ActivationMonokinemedicine.anatomical_structureEndocrinologyImmunologyAlveolar macrophageLeukocytes MononuclearInterleukin-2Tumor necrosis factor alphamedicine.symptomInterleukin-2/secretionbusinessmedicine.drugInterleukin-1The American review of respiratory disease
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Modulation of IL-1?, IL-6, IL-8, TNF-?, and TGF-? secretions by alveolar macrophages under NO2 exposure

2004

Activated alveolar macrophages (AMs) secrete interleukine (IL)1β, IL-6, IL-8, tumor necrosis factor-α (TNF-α), and transforming growth factor-β (TGF-β), whose inflammatory and fibroblast-activating characteristics may play a role in the maintenance of pulmonary inflammatory processes and subsequent fibrosis. Human AMs were transferred to a gas cylinder and exposed to NO2 in concentrations ranging from 0.1 to 0.5 ppm in synthetic air for 30 min at 37°C. AMs were fixed on a polycarbonate membrane and placed on culture medium. A culture was established, with the exposed AM (nonstimulated or stimulated with 1 μg/ml lipopolysaccharide [LPS]), and the remaining cells were used to determine the cy…

Pulmonary and Respiratory MedicineLipopolysaccharideBiologymedicine.diseaseAndrologychemistry.chemical_compoundchemistryFibrosisImmunologymedicinebiology.proteinTumor necrosis factor alphaTrypan blueInterleukin 8CytotoxicityInterleukin 6Transforming growth factorLung
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