Search results for "neuron"

showing 10 items of 2611 documents

Leprosy: report of a case with severe peripheral neuropathy

2009

Leprosy (Hansen's disease) is a chronic granulomatous infectious disease, caused by Mycobacterium leprae, with cutaneous and neurological manifestations. Leprosy is very rare in Europe but some cases are reported, especially among people coming from endemic areas. Here, we report a case of Hansen's disease and emphasize the importance of a prompt diagnosis and treatment also in non-endemic areas.

Malemedicine.medical_specialtyNeurologyAdolescentSensory Receptor CellsChronic granulomatousLeprosy neuropathyNeural ConductionDermatologyDiseaseLeprosymedicineHumansMuscle SkeletalSicilyMycobacterium lepraeMotor NeuronsbiologyElectromyographybusiness.industryPeripheral Nervous System DiseasesGeneral Medicinebiology.organism_classificationmedicine.diseaseDermatologySenegalPsychiatry and Mental healthPeripheral neuropathyInfectious disease (medical specialty)ImmunologySettore MED/26 - NeurologiaNeurology (clinical)LeprosyNeurosurgerybusinessNeurological Sciences
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REM sleep behavior disorder and periodic leg movements during sleep in ALS

2017

Objective To assess sleep characteristics and the occurrence of abnormal muscle activity during sleep, such as REM sleep without atonia (RSWA), REM sleep behavior disorder (RBD), and periodic leg movements during sleep (PLMS), in patients with amyotrophic lateral sclerosis (ALS). Methods A total of 41 patients with ALS and 26 healthy subjects were submitted to clinical interview and overnight video-polysomnography. Results A total of 22 patients with ALS (53.6%) reported poor sleep quality. Polysomnographic studies showed that patients with ALS had reduced total sleep time, increased wakefulness after sleep onset, shortened REM and slow-wave sleep, and decreased sleep efficiency, compared t…

Malemedicine.medical_specialtyNeurologyperiodic leg movementPolysomnography[SDV.NEU.NB]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]/NeurobiologySleep StagePolysomnographyREM Sleep Behavior Disorderrapid eye movement sleep without atoniaNon-rapid eye movement sleepREM sleep behavior disorder03 medical and health sciences0302 clinical medicineInsomniamedicineHumansamyotrophic lateral sclerosisleepComputingMilieux_MISCELLANEOUSSlow-wave sleepAgedmedicine.diagnostic_test[SDV.NEU.PC]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]/Psychology and behavior[SCCO.NEUR]Cognitive science/NeuroscienceAmyotrophic Lateral Sclerosis[SDV.NEU.SC]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]/Cognitive SciencesGeneral MedicineMiddle Agedmedicine.diseaseSleep in non-human animals3. Good healthNocturnal Myoclonus Syndrome030228 respiratory systemItalyNeurologyAnesthesiarapid eye movement sleep behavior disorderFemaleSettore MED/26 - NeurologiaSleep StagesNeurology (clinical)medicine.symptomSleep onsetPsychology030217 neurology & neurosurgeryHuman
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Acute neuromuscular responses to short and long roundhouse kick striking paces in professional Muay Thai fighters.

2019

International audience; BACKGROUND: Muay Thai fighters strongly rely on the use of the roundhouse kick due to its effectiveness (i.e. power) and implications on the final score. Therefore, different striking tempos at full power are used during training as a method to enhance kicking power. However, the neuromuscular responses are unknown. Thus, the goal of this study was to investigate neuromuscular responses to a single bout of shorter (every second = H1) and longer (every 3s = H3) kick striking time intervals, measured with the countermovement jump (CMJ).METHODS: Nine professional Muay Thai fighters participated in this randomized, cross-over trial. CMJs were measured on force plates bef…

Malemedicine.medical_specialtyNeuromuscular fatiguePhysical Therapy Sports Therapy and Rehabilitation61279603 medical and health sciences0302 clinical medicinePhysical medicine and rehabilitationThai-boxingMiddle kickMedicineHumansOrthopedics and Sports MedicineForce platform030212 general & internal medicineMuscle SkeletalCountermovement jumpCross-Over Studiesbusiness.industry030229 sport sciencesThailandCrossover studyNeuromuscular fatigueLower ExtremityMuscle FatiguePost activation potentiationCountermovement jumpPhysical Endurance[SDV.NEU]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]businessMartial ArtsThe Journal of sports medicine and physical fitness
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Plasticity of synaptic ribbons of the rat pineal gland in vitro — Minor effects of electrical stimulation

2003

Synaptic ribbons (SRs) of mammalian pinealocytes exhibit day/night changes in number and size, changes that are apparently regulated by the suprachiasmatic nucleus via postganglionic sympathetic nerve fibres. Since the neural control of SR changes is far from clear and as pinealocytes produce action potentials, we undertook to investigate whether electrical stimulation affects SR changes. Isolated rat pineal glands removed during the daytime were kept in vitro for 0, 30, 60, 90 or 120 min, with or without continuous electrical stimulation (1 mA, 1 Hz), followed by the quantification of SR profiles (SRPs) by transmission electron microscopy. SRs were categorised as to whether they lay less t…

Malemedicine.medical_specialtyNeuronal PlasticitySuprachiasmatic nucleusStimulationGeneral MedicineBiologyPlasticityPineal GlandElectric StimulationIn vitroRatsPinealocyteRats Sprague-DawleyRat Pineal GlandPineal glandmedicine.anatomical_structureEndocrinologyInternal medicineSynapsesNeuroplasticitymedicineAnimalsAnatomyDevelopmental BiologyAnnals of Anatomy - Anatomischer Anzeiger
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Nicotine receptors do not modulate the 3H-noradrenaline release from the isolated rat heart evoked by sympathetic nerve stimulation.

1982

Isolated rat hearts with the right sympathetic nerves attached were perfused at a constant flow rate of 7 ml/min with Tyrode's solution. (-)-3H-Noradrenaline (final concentration 10–13.9 nM) was infused for 10 min to label the noradrenaline stores. After wash-out the sympathetic nerves were stimulated electrically (3 Hz, 180 impulses, 1 ms, 20–30 mA) three times (S1–S3) at intervals of 15 min. 3H-Noradrenaline and its metabolites were determined by liquid scintillation counting according to Graefe et al. (1973). Both, nicotine 50 μM and p-aminophenethyltrimethylammonium (PAPETA) 30 μM, enhanced the 3H-noradrenaline overflow in the absence of nerve stimulation. The effect of PAPETA was bipha…

Malemedicine.medical_specialtyNicotineSympathetic Nervous SystemSympathetic nerveStimulationIn Vitro TechniquesReceptors NicotinicTritiumReuptakeMethoxyhydroxyphenylglycol3h noradrenalineNicotinechemistry.chemical_compoundNorepinephrineInternal medicinemedicineAnimalsReceptors CholinergicReceptorPharmacologyNeuronsMyocardiumHeartRats Inbred StrainsGeneral MedicineRat heartElectric StimulationRatsQuaternary Ammonium CompoundsEndocrinologychemistryMandelic AcidsHexamethoniumFemalemedicine.drugNaunyn-Schmiedeberg's archives of pharmacology
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Differential effects of calcium channel antagonists (omega-conotoxin GVIA, nifedipine, verapamil) on the electrically-evoked release of [3H]acetylcho…

1990

Electrically-evoked release of [3H]acetylcholine from autonomic neurons (myenteric plexus), motoneurons (phrenic nerve) and the central nervous system (neocortex) was investigated in the presence and absence of the calcium channel antagonists omega-conotoxin GVIA, nifedipine and verapamil, whereby the same species (rat) was used in all experiments. Release of [3H]acetylcholine was measured after incubation of the tissue with [3H]choline. omega-Conotoxin GVIA markedly reduced (70%) the evoked release of [3H]acetylcholine from the myenteric plexus of the small intestine (IC50: 0.7 nmol/l) with a similar potency at 3 and 10 Hz stimulation. An increase in the extracellular calcium concentration…

Malemedicine.medical_specialtyNifedipinechemistry.chemical_elementMollusk VenomsMyenteric PlexusCalciumAutonomic Nervous Systemcomplex mixturesNifedipineomega-Conotoxin GVIAInternal medicinemedicineAnimalsMyenteric plexusPhrenic nervePharmacologyCerebral CortexMotor NeuronsVoltage-dependent calcium channelCalcium channelRats Inbred StrainsGeneral MedicineCalcium Channel BlockersAcetylcholineElectric StimulationRatsPhrenic NerveEndocrinologynervous systemchemistryVerapamilAnesthesiaVerapamilFemaleAcetylcholinemedicine.drugNaunyn-Schmiedeberg's archives of pharmacology
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Uptake of [3H]dopamine into dopaminergic and noradrenergic neurones of the isolated neurointermediate lobe of the rat hypophysis. Effects of desipram…

1983

The isolated neurointermediate lobe (NIL) of the rat hypophysis accumulates [3H]dopamine from the incubation medium. Column chromatographic analysis showed that 92% of the tissue radioactivity was contained in the catecholamine fraction. [3H]Dopamine represented 70% and [3H]noradrenaline 30% of the [3H]catecholamines. Desipramine (1 microM) prevented the formation of [3H]noradrenaline without affecting the storage of [3H]dopamine. Nomifensine (10 microM) blocked the storage of [3H]dopamine and [3H]noradrenaline. Thus, in the NIL, [3H]dopamine is taken up into dopaminergic and noradrenergic neurones. In the latter, [3H]dopamine is converted to [3H]noradrenaline, indicating a significant dopa…

Malemedicine.medical_specialtyNomifensineDopamineBiochemistryCellular and Molecular NeuroscienceNorepinephrineNorepinephrinePituitary Gland PosteriorDopamineInternal medicineDesipraminemedicineAnimalsCatecholamine uptakeNeuronsChemistryDopaminergicDesipramineNeurointermediate lobeIsoquinolinesRatsNomifensineEndocrinologyCatecholaminemedicine.drugJournal of neurochemistry
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Functional and neurochemical changes of the gastrointestinal tract in a rodent model of Parkinson's disease.

2009

Patients with Parkinson’s disease develop motor disturbances often accompanied by peripheral autonomic dysfunctions, including gastrointestinal disorders, such as dysphagia, gastric stasis and constipation. While the mechanisms subserving enteric autonomic dysfunctions are not clearly understood, they may involve the enteric dopaminergic and/or nitrergic systems. In the present study, we demonstrate that rats with unilateral 6-hydroxydopamine lesion of nigrostriatal dopaminergic neurons develop a marked inhibition of propulsive activity compared to sham-operated controls, as indicated by a 60% reduction of daily fecal output at the 4th week of observation. Immunohistochemical data revealed …

Malemedicine.medical_specialtyParkinson's diseaseColonGastrointestinal DiseasesCONSTIPATIONENTERIC NEURONSIleumBiologyNitric OxideEnteric Nervous SystemNOLesionRats Sprague-Dawleychemistry.chemical_compoundParkinsonian DisordersIleumInternal medicineNitrergic NeuronsmedicineAnimalsOxidopamineGUT DYSFUNCTIONNeuronal PlasticityGeneral NeuroscienceDopaminergicParkinson Diseasemedicine.diseaseImmunohistochemistryRatsDisease Models Animalmedicine.anatomical_structureEndocrinologychemistryCONSTIPATION; ENTERIC NEURONS; GUT DYSFUNCTION; NEUROPLASTICITY; NITRIC OXIDEEnteric nervous systemNeuronmedicine.symptomConstipation Enteric neurons Gut dysfunction Neuroplasticity Nitric oxideGastrointestinal MotilityNitrergic NeuronNEUROPLASTICITYOxidopamineNeuroscience letters
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Elevation of striatal urate in experimental models of Parkinson's disease: a compensatory mechanism triggered by dopaminergic nigrostriatal degenerat…

2014

Epidemiological studies have indicated an inverse association between high uricemia and incidence of Parkinson's disease (PD). To investigate the link between endogenous urate and neurotoxic changes involving the dopaminergic nigrostriatal system, this study evaluated the modifications in the striatal urate levels in two models of PD. To this end, a partial dopaminergic degeneration was induced by 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine (MPTP) in mice, while a severe dopaminergic degeneration was elicited by unilateral medial forebrain bundle infusion of 6-hydroxydopamine (6-OHDA) in rats. Urate levels were measured by in vivo microdialysis at 7 or 14 days from toxin exposure. The resu…

Malemedicine.medical_specialtyParkinson's diseaseDopamineStriatumBiochemistryNeuroprotectionRats Sprague-DawleyCellular and Molecular Neurosciencechemistry.chemical_compoundHydroxydopaminesMiceDopamineInternal medicinemedicineAnimalsParkinson Disease SecondaryMedial forebrain bundleMPTPDopaminergic NeuronsNeurodegenerationDopaminergicMPTP Poisoningmedicine.diseaseRatsUric AcidMice Inbred C57BLNeostriatumSubstantia NigraEndocrinologynervous systemchemistryNeurosciencemedicine.drugJournal of neurochemistry
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Pigment variant of neuronal ceroid-lipofuscinosis

1995

A 6-year-old girl had progressive ataxia, and visual disturbances resulting in blindness. She died in her sleep at age 22 years. She shared with her sister and paternal relatives bilateral pes cavus deformities and impaired deep-tendon reflexes which suggested Charcot-Marie-Tooth disease. Her sister, who also had both polyneuropathy and a progressive central nervous system (CNS) disease, did not have pigmentary retinopathy. At autopsy, the patient was found to have neuronal ceroid-lipofuscinosis (NCL) marked by intraneuronal accumulation of autofluorescent granular lipopigments in ballooned perikarya and conspicuous extraneuronal pigmentation of subcortical grey matter, but without axonal s…

Malemedicine.medical_specialtyPathologyPostmortem studiesNeurologyCentral nervous systemAutopsyBiologyGrey matterEpitheliumNuclear FamilyDiagnosis DifferentialCharcot-Marie-Tooth DiseaseNeuronal Ceroid-LipofuscinosesmedicineNeuropilHumansChildGenetics (clinical)Cerebral CortexNeuronsPigmentationPigments BiologicalAnatomymedicine.diseaseMicroscopy ElectronKidney Tubulesmedicine.anatomical_structureSpinal CordFemaleNeuronal ceroid lipofuscinosisPolyneuropathyAmerican Journal of Medical Genetics
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