Search results for "neuron"

showing 10 items of 2611 documents

Biallelic mutations in neurofascin cause neurodevelopmental impairment and peripheral demyelination

2019

See Karakaya and Wirth (doi:10.1093/brain/awz273) for a scientific commentary on this article. Neurofascin (NFASC) isoforms are immunoglobulin cell adhesion molecules involved in node of Ranvier assembly. Efthymiou et al. identify biallelic NFASC variants in ten unrelated patients with a neurodevelopmental disorder characterized by variable degrees of central and peripheral involvement. Abnormal expression of Nfasc155 is accompanied by severe loss of myelinated fibres.

Male[SDV]Life Sciences [q-bio][SDV.NEU.NB]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]/NeurobiologyNerve Fibers MyelinatedGene FrequencyNeurodevelopmental Disorder[SDV.BC.IC]Life Sciences [q-bio]/Cellular Biology/Cell Behavior [q-bio.CB]Nerve Growth FactorProtein IsoformsChildComputingMilieux_MISCELLANEOUSMyelin Sheathneurofascin; neurodevelopment; peripheral demyelinationAlleleneurodevelopmentDemyelinating DiseaseGenomicsneurodevelopment neurofascin peripheral demyelinationSettore MED/39 - Neuropsichiatria InfantilePedigree[SDV.IMM.IA]Life Sciences [q-bio]/Immunology/Adaptive immunologyChild PreschoolPeripheral Nerve[SDV.NEU]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]Femaleneurodevelopment; neurofascin; peripheral demyelinationNeurogliaHumanAdultAdolescentNervous System MalformationsGuillain-Barre SyndromeAxonNervous System MalformationneurofascinRanvier's NodesHumansNerve Growth FactorsPeripheral NervesAllelesAutoantibodiesperipheral demyelinationInfantProtein IsoformOriginal ArticlesAxonsnervous systemNeurodevelopmental DisordersCell Adhesion MoleculeMutationCell Adhesion MoleculesDemyelinating Diseases
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Olores, aprendizaje y periodos sensibles durante el desarrollo

2010

traduction simultanée du titre; absent

[ SDV.NEU.PC ] Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]/Psychology and behavior[SDV.AEN] Life Sciences [q-bio]/Food and Nutrition[SDV.NEU.PC]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]/Psychology and behavior[ SDV.AEN ] Life Sciences [q-bio]/Food and Nutrition[SDV.NEU.PC] Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]/Psychology and behaviorodeur[SDV.AEN]Life Sciences [q-bio]/Food and Nutritionapprentissagedéveloppement
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CRISPR-Cas9 screen reveals a MYCN-amplified neuroblastoma dependency on EZH2.

2018

Pharmacologically difficult targets, such as MYC transcription factors, represent a major challenge in cancer therapy. For the childhood cancer neuroblastoma, amplification of the oncogene MYCN is associated with high-risk disease and poor prognosis. Here, we deployed genome-scale CRISPR-Cas9 screening of MYCN-amplified neuroblastoma and found a preferential dependency on genes encoding the polycomb repressive complex 2 (PRC2) components EZH2, EED, and SUZ12. Genetic and pharmacological suppression of EZH2 inhibited neuroblastoma growth in vitro and in vivo. Moreover, compared with neuroblastomas without MYCN amplification, MYCN-amplified neuroblastomas expressed higher levels of EZH2. ChIP…

0301 basic medicineCellular differentiationMedical and Health SciencesNeuroblastomaSUZ12Oncogene MYCNCRISPR-Cas SystemCancerPediatricNeuronsN-Myc Proto-Oncogene ProteinTumorEZH2EpigeneticCell DifferentiationGeneral MedicineUp-RegulationGene Expression Regulation NeoplasticOncology5.1 PharmaceuticalsEpigeneticsDevelopment of treatments and therapeutic interventionsHumanResearch ArticlePediatric Research InitiativePediatric CancerImmunologymacromolecular substancesBiologyN-Myc Proto-Oncogene ProteinCell Line03 medical and health sciencesRare DiseasesNeuroblastomaCell Line TumormedicineGeneticsHumansEnhancer of Zeste Homolog 2 ProteinTranscription factorneoplasmsNeoplasticHuman GenomeNeurosciencesGene AmplificationNeuronmedicine.disease030104 developmental biologyGene Expression RegulationCancer researchHistone deacetylaseCRISPR-Cas SystemsThe Journal of clinical investigation
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Quantification and automatized adaptive detection of in vivo and in vitro neuronal bursts based on signal complexity.

2015

In this paper, we propose employing entropy values to quantify action potential bursts in electrophysiological measurements from the brain and neuronal cultures. Conventionally in the electrophysiological signal analysis, bursts are quantified by means of conventional measures such as their durations, and number of spikes in bursts. Here our main aim is to device metrics for burst quantification to provide for enhanced burst characterization. Entropy is a widely employed measure to quantify regularity/complexity of time series. Specifically, we investigate the applicability and differences of spectral entropy and sample entropy in the quantification of bursts in in vivo rat hippocampal meas…

Computer scienceQuantitative Biology::Tissues and OrgansAstrophysics::High Energy Astrophysical PhenomenaEntropyCell Culture TechniquesElectrophysiological PhenomenaAction Potentialsta3112HippocampusEntropy (classical thermodynamics)In vivoEntropy (information theory)AnimalsEntropy (energy dispersal)Rats WistarEntropy (arrow of time)ta217NeuronsSignal processingQuantitative Biology::Neurons and Cognitionta213Entropy (statistical thermodynamics)Signal Processing Computer-Assistedadaptive detectionelectrophysiological signal analysisquantificationneuronal burstsElectrophysiological PhenomenaSample entropyElectrophysiologyElectrophysiologyMicroelectrodeBiological systemNeuroscienceMicroelectrodesEntropy (order and disorder)Annual International Conference of the IEEE Engineering in Medicine and Biology Society. IEEE Engineering in Medicine and Biology Society. Annual International Conference
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More than a pore: How voltage-gated calcium channels act on different levels of neuronal communication regulation.

2021

ABSTRACT Voltage-gated calcium channels (VGCCs) represent key regulators of the calcium influx through the plasma membrane of excitable cells, like neurons. Activated by the depolarization of the membrane, the opening of VGCCs induces very transient and local changes in the intracellular calcium concentration, known as calcium nanodomains, that in turn trigger calcium-dependent signaling cascades and the release of chemical neurotransmitters. Based on their central importance as concierges of excitation-secretion coupling and therefore neuronal communication, VGCCs have been studied in multiple aspects of neuronal function and malfunction. However, studies on molecular interaction partners …

0301 basic medicineα2δ subunitsBiophysicschemistry.chemical_elementReviewNeurotransmissionCalciumBiochemistrySynaptic TransmissionCalcium in biology03 medical and health sciencesvoltage-induced calcium releasealternative splicing0302 clinical medicinevoltage-gated calcium channelsCavβ subunitsVGCC auxiliary subunitsCalcium SignalingIon channelNeuronssynaptogenesisVoltage-dependent calcium channelChemistryRyanodine receptorDepolarization030104 developmental biologyIon channelsCalciumgene regulationNeuroscience030217 neurology & neurosurgeryIntracellularResearch ArticleChannels (Austin, Tex.)
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The aristaless (Arx) gene: one gene for many "interneuronopathies".

2009

The ARX (Aristaless-related (X-linked) homeobox) gene is not only present in arthropods and their ancestors, but also in vertebrates including humans (ARX orthologs). The gene is composed of 5 coding exons and it is expressed predominantly in foetal and adult brain and skeletal muscle. In this review we report on our experience and review the existing literature on the genotype and phenotype heterogeneity associated with ARX abnormalities in humans ranging from severe neuronal migration defects (e.g., lissencephaly), to mild forms of X-linked mental retardation without apparent brain abnormalities. The ARX-related disorders are reviewed focusing on their clinical features and on the role of…

Doublecortin ProteinGenotypeLissencephalyBiologyNeuronal migration defectsGeneral Biochemistry Genetics and Molecular BiologyExonMiceGenotype-phenotype distinctionSettore MED/38 - Pediatria Generale E SpecialisticaInterneuronsmedicineAnimalsHumansAbnormalities MultipleGeneZebrafishGeneticsHomeodomain ProteinsGeneral Immunology and MicrobiologyARX homeoboxmedicine.diseasePhenotypeCranial Nerve DiseasesPhenotypeMultigene FamilyMental Retardation X-LinkedHomeoboxAbnormalityTranscription FactorsFrontiers in bioscience (Elite edition)
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GABAC receptors are functionally expressed in the intermediate zone and regulate radial migration in the embryonic mouse neocortex

2010

Radial neuronal migration in the cerebral cortex depends on trophic factors and the activation of different voltage- and ligand-gated channels. To examine the func- tional role of GABAC receptors in radial migration we ana- lyzed the effects of specific GABAA and GABAC receptor antagonists on the migration of BrdU-labeled neurons in vitro using organotypic neocortical slice cultures. These experi- ments revealed that the GABAA specific inhibitor bicuculline methiodide facilitated neuronal migration, while the GABAC specific inhibitor (1,2,5,6-tetrahydropyridine-4-yl) methylphos- phinic-acid (TPMPA) impeded migration. Co-application of TPMPA and bicuculline methiodide or the unspecific ionot…

PyridinesNeocortexIn Vitro TechniquesBiologyBicucullineGABAA-rho receptorGABA AntagonistsMicechemistry.chemical_compoundReceptors GABACell MovementmedicineAnimalsPicrotoxinGABA-A Receptor AntagonistsRNA MessengerReceptorGABA AgonistsNeuronsNeocortexGABAA receptorGeneral NeuroscienceGABA receptor antagonistReceptors GABA-APhosphinic AcidsCell biologyMice Inbred C57BLmedicine.anatomical_structurechemistryCrotonatesGABAergicNeurosciencePicrotoxinIonotropic effectNeuroscience
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The neurobiological bases for the pharmacotherapy of nicotine addiction.

2007

Nicotine, the major psychoactive agent present in tobacco, acts as a potent addictive drug both in humans and laboratory animals, whose locomotor activity is also stimulated. A large body of evidence indicates that the locomotor activation and the reinforcing effects of nicotine may be related to its stimulatory effects on the mesolimbic dopaminergic function. Thus, it is now well established that nicotine can increase in vivo DA outflow in the nucleus accumbens and the corpus striatum. The stimulatory effect of nicotine on DA release most probably results from its ability to excite the neuronal firing rate and to increase the bursting activity of DA neurons in the substantia nigra pars com…

RAT STRIATAL SYNAPTOSOMESNicotineINDUCED BEHAVIORAL SENSITIZATIONmedia_common.quotation_subjectSubstantia nigraStriatumNicotinic AntagonistsBiologyNucleus accumbensPharmacologyReceptors NicotinicNicotineDrug DiscoverySUSTAINED-RELEASE BUPROPIONmedicineLOCOMOTOR STIMULANT ACTIONAnimalsHumansNicotinic Agonistsmedia_commonPharmacologyMIDBRAIN DOPAMINE NEURONSPars compactaAddictionNIGRA PARS COMPACTAFACILITATES SMOKING CESSATIONTobacco Use DisorderSUBUNIT MESSENGER-RNAAntidepressive AgentsVentral tegmental areaVENTRAL TEGMENTAL AREANicotinic agonistmedicine.anatomical_structurenervous systemmedicine.drugSEROTONIN(2C) RECEPTORS BLOCKSCurrent pharmaceutical design
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Opposing Effects of CREBBP Mutations Govern the Phenotype of Rubinstein-Taybi Syndrome and Adult SHH Medulloblastoma

2018

Recurrent mutations in chromatin modifiers are specifically prevalent in adolescent or adult patients with Sonic hedgehog-associated medulloblastoma (SHH MB). Here, we report that mutations in the acetyltransferase CREBBP have opposing effects during the development of the cerebellum, the primary site of origin of SHH MB. Our data reveal that loss of Crebbp in cerebellar granule neuron progenitors (GNPs) during embryonic development of mice compromises GNP development, in part by downregulation of brain-derived neurotrophic factor (Bdnf). Interestingly, concomitant cerebellar hypoplasia was also observed in patients with Rubinstein-Taybi syndrome, a congenital disorder caused by germline mu…

0301 basic medicineCerebellumCrebbp protein mousemetabolism [Cerebellar Neoplasms]acetyltransferase; cerebellum; CREBBP; development; Rubinstein-Taybi syndrome; SHH medulloblastomagenetics [Hedgehog Proteins]MiceNeurotrophic factorsmetabolism [CREB-Binding Protein]Mice KnockoutNeuronsRubinstein-Taybi Syndromepathology [Rubinstein-Taybi Syndrome]CREBBPCREB-Binding ProteinPhenotypegenetics [CREB-Binding Protein]3. Good healthpathology [Cerebellar Neoplasms]acetyltransferasePhenotypemedicine.anatomical_structuregenetics [Rubinstein-Taybi Syndrome]Femalemetabolism [Hedgehog Proteins]Signal TransductionSHH medulloblastomaAdultcerebellumBiologyGeneral Biochemistry Genetics and Molecular BiologyCREBBP; Rubinstein-Taybi syndrome; SHH medulloblastoma; acetyltransferase; cerebellum; development.03 medical and health sciencesGermline mutationAcetyltransferasesmetabolism [Medulloblastoma]medicineAnimalsHumansgenetics [Cerebellar Neoplasms]Hedgehog Proteinsddc:610Cerebellar NeoplasmsdevelopmentMolecular BiologyMedulloblastomaRubinstein–Taybi syndromegenetics [Medulloblastoma]metabolism [Rubinstein-Taybi Syndrome]pathology [Medulloblastoma]Cell Biologymedicine.disease030104 developmental biologyMutationphysiology [CREB-Binding Protein]Cancer researchSHH protein humanCerebellar hypoplasia (non-human)metabolism [Acetyltransferases]CREBBP protein humanMedulloblastomaDevelopmental BiologyCongenital disorderDevelopmental Cell
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The Origin, Location, and Projections of the Embryonic Abdominal Motorneurons ofDrosophila

1997

We have used a retrograde labeling technique to identify motorneurons for each of the 30 body wall muscles of an abdominal hemisegment in the late stage 16Drosophilaembryo. Each motorneuron has a characteristic cell body position, dendritic arborization, and axonal projection. In addition, we have determined the neuroblasts of origin for most of the motorneurons we describe. Some organizational principles for the neuromuscular system have become apparent: (1) There is no obvious topographic relationship between the cell body positions of motorneurons and the position or orientation of the muscles they innervate; (2) motorneurons that innervate muscles of similar position and orientation are…

Motor Neuronsanimal structuresMusclesGeneral NeuroscienceMorphological typefungiBody positionLate stageArticlesDendritesAnatomyBiologybiology.organism_classificationNervous SystemEmbryonic stem cellGanglia InvertebrateDendritic ArborizationNeuroblastLarvaAnimalsCell LineageDrosophilaDrosophila (subgenus)NeuroscienceAbdominal MusclesThe Journal of Neuroscience
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