Search results for "neuronal plasticity"

showing 10 items of 166 documents

Chronic restraint stress and chronic corticosterone treatment modulate differentially the expression of molecules related to structural plasticity in…

2004

Stress and stress-related hormones induce structural changes in neurons of the adult CNS. Neurons in the hippocampus, the amygdala and the prefrontal cortex undergo neurite remodeling after chronic stress. In the hippocampus some of these effects can be mimicked with chronic administration of adrenal steroids. These changes in neuronal structure may be mediated by certain molecules related to plastic events such as the polysialylated form of the neural cell adhesion molecule (PSA-NCAM). The expression of PSA-NCAM persists in the adult hippocampus and it is up-regulated after chronic stress. The piriform cortex also displays considerable levels of PSA-NCAM during adulthood and indirect evide…

MaleRestraint Physicalmedicine.medical_specialtyDoublecortin ProteinHippocampusNeural Cell Adhesion Molecule L1AmygdalaRats Sprague-Dawleychemistry.chemical_compoundCorticosteroneStress PhysiologicalInternal medicinePiriform cortexmedicineAnimalsChronic stressOlfactory memoryPrefrontal cortexCerebral CortexNeuronal PlasticitybiologyGeneral NeuroscienceDoublecortinRatsmedicine.anatomical_structureEndocrinologynervous systemchemistryGene Expression RegulationChronic Diseasebiology.proteinSialic AcidsCorticosteroneNeuroscienceNeuroscience
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Polysomnographic Findings in Fragile X Syndrome Children with EEG Abnormalities

2019

Fragile X syndrome (FXS) is a genetic syndrome with intellectual disability due to the loss of expression of the FMR1 gene located on chromosome X (Xq27.3). This mutation can suppress the fragile X mental retardation protein (FMRP) with an impact on synaptic functioning and neuronal plasticity. Among associated sign and symptoms of this genetic condition, sleep disturbances have been already described, but few polysomnographic reports in pediatric age have been reported. This multicenter case-control study is aimed at assessing the sleep macrostructure and at analyzing the presence of EEG abnormalities in a cohort of FXS children. We enrolled children with FXS and, as controls, children wit…

MaleSleep Wake Disorderscongenital hereditary and neonatal diseases and abnormalitiesPediatricsmedicine.medical_specialtyAdolescentArticle SubjectPolysomnographyNeurosciences. Biological psychiatry. NeuropsychiatryFragile X Mental Retardation Protein03 medical and health sciences0302 clinical medicinechildrenIntellectual disabilitymedicineHumansIctalCircadian rhythmChildEEG abnormalitiesPathologicalPSG030304 developmental biology0303 health sciencesNeuronal PlasticityFragile X syndrome; intellectual disability; polysomnographicbusiness.industryCase-control studyNeuropsychologyElectroencephalographyGeneral Medicinemedicine.diseasepolysomnographicFragile X syndromeNeuropsychology and Physiological PsychologyNeurologyintellectual disabilityCase-Control StudiesFragile X SyndromeCohortFemaleNeurology (clinical)FXSSleepbusiness030217 neurology & neurosurgeryRC321-571Research ArticleBehavioural Neurology
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Chronic antidepressant treatment induces contrasting patterns of synaptophysin and PSA-NCAM expression in different regions of the adult rat telencep…

2007

Structural modifications occur in the brain of severely depressed patients and they can be reversed by antidepressant treatment. Some of these changes do not occur in the same direction in different regions, such as the medial prefrontal cortex, the hippocampus or the amygdala. Differential structural plasticity also occurs in animal models of depression and it is also prevented by antidepressants. In order to know whether chronic fluoxetine treatment induces differential neuronal structural plasticity in rats, we have analyzed the expression of synaptophysin, a protein considered a marker of synaptic density, and the expression of the polysialylated form of the neural cell adhesion molecul…

MaleTelencephalonNeuropilNeuriteSynaptophysinHippocampusPrefrontal CortexNeural Cell Adhesion Molecule L1AmygdalaHippocampusRats Sprague-DawleyAnimal models of depressionFluoxetinemedicineAnimalsPharmacology (medical)Prefrontal cortexBiological PsychiatryPharmacologyNeuronal PlasticitybiologyCerebrumAmygdalaImmunohistochemistryAntidepressive AgentsRatsPsychiatry and Mental healthmedicine.anatomical_structurePhenotypenervous systemNeurologySynaptophysinbiology.proteinSialic AcidsAntidepressive Agents Second-GenerationNeural cell adhesion moleculeNeurology (clinical)NeuroscienceEuropean neuropsychopharmacology : the journal of the European College of Neuropsychopharmacology
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A critical period for experience-dependent remodeling of adult-born neuron connectivity.

2015

Neurogenesis in the dentate gyrus (DG) of the adult hippocampus is a process regulated by experience. To understand whether experience also modifies the connectivity of new neurons, we systematically investigated changes in their innervation following environmental enrichment (EE). We found that EE exposure between 2-6 weeks following neuron birth, rather than merely increasing the number of new neurons, profoundly affected their pattern of monosynaptic inputs. Both local innervation by interneurons and to even greater degree long-distance innervation by cortical neurons were markedly enhanced. Furthermore, following EE, new neurons received inputs from CA3 and CA1 inhibitory neurons that w…

MaleTime FactorsCIENCIAS MÉDICAS Y DE LA SALUDNeurogenesisNeuroscience(all)NeurocienciasSynaptogenesisHippocampusMice TransgenicHippocampal formationEnvironmentMotor ActivityInhibitory postsynaptic potentialTransfectionCRITICAL PERIODADULT NEUROGENESISArticleMiceNeural PathwaysmedicineAnimalsSYNAPTOGENESISCells CulturedNeuronsEnvironmental enrichmentNeuronal PlasticityGeneral NeuroscienceDentate gyrusCritical Period PsychologicalNeurogenesisBrain//purl.org/becyt/ford/3.1 [https]Embryo MammalianMice Inbred C57BLENRICHED ENVIRONMENTLuminescent ProteinsMedicina Básicamedicine.anatomical_structurenervous system//purl.org/becyt/ford/3 [https]NeuronNerve NetPsychologyNeuroscience
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Ipsilateral versus contralateral spontaneous post-stroke neuroplastic changes: involvement of BDNF?

2013

International audience; Stroke is a leading cause of death and disability in industrialized countries. Although surviving patients exhibit a certain degree of restoration of function attributable to brain plasticity, the majority of stroke survivors has to struggle with persisting deficits. In order to potentiate post-stroke recovery, several rehabilitation therapies have been undertaken and many experimental studies have reported that brain-derived neurotrophic factor (BDNF) is central to many facets of neuroplastic processes. However, although BDNF role in brain plasticity is well characterized through strategies that manipulate its content, the involvement of this neurotrophin in spontan…

MaleTime FactorsSynaptophysinHippocampusTropomyosin receptor kinase BHippocampal formationBrain Ischemia03 medical and health sciences0302 clinical medicineNeurotrophic factorsNeuroplasticitymedicineAnimalsRats WistarStroke030304 developmental biology0303 health sciencesNeuronal PlasticitybiologyGeneral NeuroscienceBrain-Derived Neurotrophic Factor[SCCO.NEUR]Cognitive science/NeuroscienceBrainmedicine.diseaseRatsStrokenervous system[ SCCO.NEUR ] Cognitive science/Neurosciencebiology.proteinSynaptophysinPsychologyNeuroscience030217 neurology & neurosurgeryNeurotrophin
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Temporal refinement of sensory-evoked activity across layers in developing mouse barrel cortex.

2019

Rhythmic whisking behavior in rodents fully develops during a critical period about 2 weeks after birth, in parallel with the maturation of other sensory modalities and the onset of exploratory locomotion. How whisker-related sensory processing develops during this period in the primary somatosensory cortex (S1) remains poorly understood. Here, we characterized neuronal activity evoked by single- or dual-whisker stimulation patterns in developing S1, before, during and after the occurrence of active whisking. Employing multi-electrode recordings in all layers of barrel cortex in urethane-anesthetized mice, we find layer-specific changes in multi-unit activity for principal and neighboring b…

Maleanimal structuresStimulationSensory systemStimulus (physiology)Somatosensory system03 medical and health sciencesMice0302 clinical medicineEvoked Potentials SomatosensorymedicinePremovement neuronal activityAnimals030304 developmental biologyNeurons0303 health sciencesNeuronal PlasticityChemistryGeneral NeuroscienceWhisking in animalsSomatosensory CortexBarrel cortexmedicine.anatomical_structureCerebral cortexVibrissaeFemaleNeuroscience030217 neurology & neurosurgeryThe European journal of neuroscienceREFERENCES
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Role of mTOR-regulated autophagy in spine pruning defects and memory impairments induced by binge-like ethanol treatment in adolescent mice.

2021

Abstract Adolescence is a brain maturation developmental period during which remodeling and changes in synaptic plasticity and neural connectivity take place in some brain regions. Different mechanism participates in adolescent brain maturation, including autophagy that plays a role in synaptic development and plasticity. Alcohol is a neurotoxic compound and its abuse in adolescence induces neuroinflammation, synaptic and myelin alterations, neural damage and behavioral impairments. Changes in synaptic plasticity and its regulation by mTOR have also been suggested to play a role in the behavioral dysfunction of binge ethanol drinking in adolescence. Therefore, by considering the critical ro…

MaleautophagyDendritic spineSynaptic pruningPeriod (gene)synaptic pruningBiologyPathology and Forensic MedicineBinge Drinkingbinge ethanol treatmentMyelinMicemedicineAnimalsPI3K/AKT/mTOR pathwayNeuroinflammationResearch Articlescognitive functionMemory DisordersNeuronal PlasticityGeneral NeuroscienceTOR Serine-Threonine KinasesAutophagyBraindendritic spinesMice Inbred C57BLmedicine.anatomical_structureSynaptic plasticitymTORFemaleadolescenceNeurology (clinical)NeuroscienceResearch ArticleBrain pathology (Zurich, Switzerland)
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Priming the Motor Cortex With Anodal Transcranial Direct Current Stimulation Affects the Acute Inhibitory Corticospinal Responses to Strength Trainin…

2019

Frazer, AK, Howatson, G, Ahtiainen, JP, Avela, J, Rantalainen, T, and Kidgell, DJ. Priming the motor cortex with anodal transcranial direct current stimulation affects the acute inhibitory corticospinal responses to strength training. J Strength Cond Res 33(2): 307-317, 2019-Synaptic plasticity in the motor cortex (M1) is associated with strength training (ST) and can be modified by transcranial direct current stimulation (tDCS). The M1 responses to ST increase when anodal tDCS is applied during training due to gating. An additional approach to improve the M1 responses to ST, which has not been explored, is to use anodal tDCS to prime the M1 before a bout of ST. We examined the priming effe…

Malecorticospinal silent periodmedicine.medical_treatmentstrength exercisePyramidal TractsIsometric exercise030204 cardiovascular system & hematologyTranscranial Direct Current Stimulation0302 clinical medicineElbowOrthopedics and Sports Medicineta315Cross-Over StudiesNeuronal PlasticityTranscranial direct-current stimulationMotor CortexGeneral Medicinemedicine.anatomical_structurestimulointiFemalecorticospinal excitabilityvoimaharjoitteluPriming (psychology)Motor cortexAdultmedicine.medical_specialtyStrength trainingkeskushermostoneuroplasticityeducationB100Physical Therapy Sports Therapy and RehabilitationInhibitory postsynaptic potentialta311203 medical and health sciencesYoung AdultPhysical medicine and rehabilitationDouble-Blind MethodIsometric ContractionNeuroplasticitymedicineHumansneuroplastisuusbusiness.industryResistance Training030229 sport sciencesEvoked Potentials MotorTranscranial magnetic stimulationaivokuoriNeuroplasticitytranscranial direct current stimulationbusinessJournal of strength and conditioning research
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Neurofunctional correlates of attention rehabilitation in Parkinson's disease: an explorative study

2014

The effectiveness of cognitive rehabilitation (CR) in Parkinson’s disease (PD) is in its relative infancy, and nowadays there is insufficient information to support evidence-based clinical protocols. This study is aimed at testing a validated therapeutic strategy characterized by intensive computer-based attention-training program tailored to attention deficits. We further investigated the presence of synaptic plasticity by means of functional magnetic resonance imaging (fMRI). Using a randomized controlled study, we enrolled eight PD patients who underwent a CR program (Experimental group) and seven clinically/demographically-matched PD patients who underwent a placebo intervention (Contro…

Malemedicine.medical_specialtyBrain activity and meditationPrefrontal CortexDermatologyNeuropsychological TestsStatistical parametric mappingPhysical medicine and rehabilitationParietal LobemedicineHumansAttentionSingle-Blind MethodEffects of sleep deprivation on cognitive performanceCognitive rehabilitation therapyPrincipal Component AnalysisNeuronal PlasticityResting state fMRImedicine.diagnostic_testCognitionParkinson DiseaseGeneral MedicineMiddle AgedMagnetic Resonance ImagingDorsolateral prefrontal cortexPsychiatry and Mental healthParkinson disease rehabilitationmedicine.anatomical_structureMemory Short-TermPhenotypeSpace PerceptionTherapy Computer-AssistedVisual PerceptionFemaleSettore MED/26 - NeurologiaNeurology (clinical)Nerve NetFunctional magnetic resonance imagingPsychologyCognition DisordersNeurosciencePsychomotor Performance
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Dopamine acting through D2 receptors modulates the expression of PSA-NCAM, a molecule related to neuronal structural plasticity, in the medial prefro…

2008

A "neuroplastic" hypothesis proposes that changes in neuronal structural plasticity may underlie the aetiology of depression and the action of antidepressants. The medial prefrontal cortex (mPFC) is affected by this disorder and shows an intense expression of the polysialylated form of the neural cell adhesion molecule (PSA-NCAM), a plasticity-associated molecule, which is expressed mainly in interneurons. The monoamines serotonin, dopamine and noradrenaline are the principal targets of antidepressant action. Pharmacological manipulation of serotonin levels regulates synaptophysin and PSA-NCAM expression in the adult mPFC. However, the involvement of structural plasticity on the antidepress…

Malemedicine.medical_specialtyInterneuronDopamineSynaptophysinPrefrontal CortexNeural Cell Adhesion Molecule L1Synaptic TransmissionDopamine agonistRats Sprague-DawleyDevelopmental NeuroscienceDopamineDopamine receptor D2Internal medicinePhenethylaminesmedicineAnimalsNeuronsAnalysis of VarianceMicroscopy ConfocalNeuronal PlasticityGlutamate DecarboxylaseReceptors Dopamine D2ChemistryDopaminergicDopamine antagonistImmunohistochemistryRatsmedicine.anatomical_structureEndocrinologynervous systemNeurologyDopamine receptorDopamine AgonistsSialic AcidsDopamine AntagonistsHaloperidolNeural cell adhesion moleculeNeurosciencemedicine.drugExperimental Neurology
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