Search results for "neuropeptide Y"

showing 7 items of 47 documents

Distribution of Met-enkephalyl-Arg-Gly-Leu in rat larynx: partial coexistence with vasoactive intestinal polypeptide, peptide histidine isoleucine an…

1990

Abstract Using light microscopic (LM) enzyme-immunohistochemistry on deparaffinized adjacent sections Met-enkephalyl-Arg-Gly-Leu (ME-RGL) immunoreactivity was found to partially coexist with immunoreactive neuropeptide Y (NPY), vasoactive intestinal polypeptide (VIP) and peptide histidine isoleucine (PHI) in intrinsic laryngeal neurons of the rat. Further ME-RGL-immunoreactive (ir) fibres were found around glands in the subepithelium, in connective tissue of striated muscle and in the perichondrium, as well as around arterial and venous blood vessels. They frequently contacted mast cells and macrophages. The presence of ME-RGL indicates pro-enkephalin-related origin of this novel laryngeal …

medicine.medical_specialtyEnkephalin MethionineVasoactive intestinal peptideConnective tissueNeuropeptideCalcitonin gene-related peptideBiologyNerve FibersPeptide PHIInternal medicinemedicineAnimalsNeuropeptide YTissue DistributionGeneral NeuroscienceNeuropeptidesLaryngeal NervesPeptide PHINeuropeptide Y receptorRatsmedicine.anatomical_structureEndocrinologyCalcitoninPeripheral nervous systemGangliaLarynxVasoactive Intestinal PeptideNeuroscience letters
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Molecular anatomy of the neuro-immune connection.

1991

Light microscopic immunohistochemistry was employed to elucidate and compare the presence, distribution, and coexistence of various peptides, neuroendocrine markers and enzymes of the catecholamine pathway in nerves supplying lymphoid tissues in a variety of mammalian species. All lymphoid organs and tissues receive innervation by fibers containing dopamine-beta-hydroxylase and/or tyrosine hydroxylase, neural markers like protein gene product 9.5, synaptophysin and neurofilament and a varied spectrum of peptides. The prominent peptides were tachykinins (substance P, neurokinin A), calcitonin gene-related peptide (CGRP), neuropeptide Y (NPY), and vasoactive intestinal polypeptide/peptide his…

medicine.medical_specialtyNeurofilamentLymphoid TissueNeuroimmunomodulationSwineVasoactive intestinal peptideGuinea PigsPalatine TonsilFluorescent Antibody TechniqueSubstance PThymus GlandCalcitonin gene-related peptidechemistry.chemical_compoundMiceDogsBone MarrowInternal medicineNeural PathwaysmedicineAnimalsHumansNeuronsSheepTyrosine hydroxylasebiologyChemistryGeneral NeuroscienceGeneral MedicineAnatomyNeuropeptide Y receptorImmunohistochemistryRatsEndocrinologySynaptophysinbiology.proteinCatsNeurokinin ALymph NodesPeptidesSpleenThe International journal of neuroscience
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Agouti-related peptide-expressing neurons are mandatory for feeding.

2005

Multiple hormones controlling energy homeostasis regulate the expression of neuropeptide Y (NPY) and agouti-related peptide (AgRP) in the arcuate nucleus of the hypothalamus. Nevertheless, inactivation of the genes encoding NPY and/or AgRP has no impact on food intake in mice. Here we demonstrate that induced selective ablation of AgRP-expressing neurons in adult mice results in acute reduction of feeding, demonstrating direct evidence for a critical role of these neurons in the regulation of energy homeostasis.

medicine.medical_specialtyPro-OpiomelanocortinTime FactorsPeptideCell CountBiologyEnergy homeostasisEatingMiceArcuate nucleusInternal medicinemedicineAnimalsAgouti-Related ProteinDiphtheria ToxinNeuropeptide YRegulation of gene expressionchemistry.chemical_classificationMice KnockoutNeuronsGeneral Neurosciencedigestive oral and skin physiologyBody WeightArcuate Nucleus of HypothalamusProteinsFeeding BehaviorNeuropeptide Y receptorbeta-GalactosidaseAnorexiaEndocrinologynervous systemchemistryGene Expression RegulationHypothalamusIntercellular Signaling Peptides and ProteinsAgouti-related peptidehormones hormone substitutes and hormone antagonistsHormoneNature neuroscience
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Neurochemistry of identified motoneurons of the tensor tympani muscle in rat middle ear

2008

The objective of the present study was to identify efferent and afferent transmitters of motoneurons of the tensor tympani muscle (MoTTM) to gain more insight into the neuronal regulation of the muscle. To identify MoTTM, we injected the fluorescent neuronal tracer Fluoro-Gold (FG) into the muscle after preparation of the middle ear in adult rats. Upon terminal uptake and retrograde neuronal transport, we observed FG in neurons located lateral and ventrolateral to the motor trigeminal nucleus ipsilateral to the injection site. Immunohistochemical studies of these motoneurons showed that apparently all contained choline acetyltransferase, demonstrating their motoneuronal character. Different…

medicine.medical_specialtyStilbamidinesEar MiddleNeuropeptideSubstance PCalcitonin gene-related peptideInjections IntramuscularTensor tympani muscleCholine O-AcetyltransferaseRats Sprague-Dawleychemistry.chemical_compoundInternal medicinemedicineAnimalsNeuronal transportFluorescent DyesMotor NeuronsStaining and LabelingTyrosine hydroxylaseChemistryNeuropeptidesNeurochemistryNeuropeptide Y receptorImmunohistochemistryCholine acetyltransferaseSensory SystemsRatsEndocrinologynervous systemFemaleTensor TympaniHearing Research
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Critical role of dipeptidyl peptidase IV in neuropeptide Y-mediated endothelial cell migration in response to wounding

2001

Recently, we have discovered that neuropeptide Y (NPY), a sympathetic neurotransmitter, is also present in human umbilical endothelial cells (HUVECs), and is potently chemotactic and angiogenic by acting on one or several of Y1-Y5 receptors. In HUVECs, NPY is co-localized with dipeptidyl peptidase IV (DPPIV) which cleaves Tyr(1)-Pro(2) from NPY(1-36) to form NPY(3-36) resulting in the formation of a non-Y1 receptor agonist, which remains angiogenic. Presently we studied the effects of DPPIV's blockade using monoclonal antibodies (mAbs) on migration of HUVECs in response to NPY(1-36) or NPY(3-36) following cell wounding. Both peptides caused similar dose-dependent increases in cell migration…

medicine.medical_specialtyTime FactorsEndotheliumPhysiologyDipeptidyl Peptidase 4Blotting WesternImmunoblottingBiologyBiochemistryDipeptidyl peptidaseUmbilical CordCellular and Molecular NeuroscienceEndocrinologyWestern blotCell MovementInternal medicinemental disordersmedicineHumansNeuropeptide YReceptormedicine.diagnostic_testChemotaxisNeuropeptide Y receptorhumanitiesCell biologyBlotEndothelial stem cellEndocrinologymedicine.anatomical_structureWounds and InjuriesEndothelium VascularPeptides
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Effect of Acetaldehyde Intoxication and Withdrawal on NPY Expression: Focus on Endocannabinoidergic System Involvement

2014

Acetaldehyde (ACD), the first alcohol metabolite, plays a pivotal role in the rewarding, motivational and addictive properties of the parental compound. Many studies have investigated the role of ACD in mediating neurochemical and behavioral effects induced by alcohol administration, but very little is known about the modulation of neuropeptide systems following ACD intoxication and withdrawal. Indeed the neuropeptide Y (NPY) system is altered during alcohol withdrawal in key regions for cerebrocortical excitability and neuroplasticity. The primary goal of this research was to investigate the effects of ACD intoxication and withdrawal by recording rat behavior and by measuring neuropeptide …

medicine.medical_specialtylcsh:RC435-571hippocampusnucleus accumbensHippocampusNeuropeptidePhysical dependenceNucleus accumbensendocannabinoidergic systemNeurochemicallcsh:PsychiatryInternal medicinemental disordersmedicineendocannabinoid systemneuropeptide Y expressionOriginal ResearchPsychiatryacetaldehyde withdrawal neuropeptide Y expression endocannabinoidergic system hippocampus nucleus accumbensKindlingAlcohol dependenceacetaldehyde withdrawal neuropeptide Y expression endocannabinoidergic system hippocampusnucleus accumbensNeuropeptide Y receptorPsychiatry and Mental healthEndocrinologyacetaldehyde withdrawalmedicine.symptomPsychologyFrontiers in Psychiatry
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Neuropeptide Y: una overview sugli effetti di neuro- ed immuno-modulazione

2002

neuromodulazioneSettore BIO/14 - FarmacologiaNeuropeptide Yimmunomodulazione
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