Search results for "nitric-oxide"

showing 10 items of 30 documents

Candidate genetic pathways for attention-deficit/hyperactivity disorder (ADHD) show association to hyperactive/impulsive symptoms in children with AD…

2013

Item does not contain fulltext OBJECTIVE: Because multiple genes with small effect sizes are assumed to play a role in attention-deficit/hyperactivity disorder (ADHD) etiology, considering multiple variants within the same analysis likely increases the total explained phenotypic variance, thereby boosting the power of genetic studies. This study investigated whether pathway-based analysis could bring scientists closer to unraveling the biology of ADHD. METHOD: The pathway was described as a predefined gene selection based on a well-established database or literature data. Common genetic variants in pathways involved in dopamine/norepinephrine and serotonin neurotransmission and genes involv…

Malegenetic pathwaysMedizinSocial SciencesGenome-wide association studyDCN PAC - Perception action and controlSeverity of Illness Index/dk/atira/pure/sustainabledevelopmentgoals/clean_water_and_sanitationneuritic outgrowth2738 Psychiatry and Mental HealthDOPAMINE0302 clinical medicineDevelopmental and Educational PsychologyIsraelChildQUANTITATIVE TRAITS10058 Department of Child and Adolescent PsychiatryGenomic disorders and inherited multi-system disorders [DCN PAC - Perception action and control IGMD 3]EuropePsychiatry and Mental healthattention-deficit/hyperactivity disorder symptomsPhenotypeChild PreschoolFemalemedicine.symptomPsychologySDG 6 - Clean Water and Sanitationmedicine.drugClinical psychologymedicine.medical_specialtyDIAGNOSTIC-APPROACHAdolescentGenotypeLARGE MULTICENTER ADHDIDENTIFIES ASSOCIATIONDEFICIT HYPERACTIVITY DISORDERDCN MP - Plasticity and memoryneurotransmitter systems610 Medicine & healthQuantitative trait locusHyperkinesisImpulsivityMental health [NCEBP 9]Genomic disorders and inherited multi-system disorders DCN MP - Plasticity and memory [IGMD 3]03 medical and health sciencesDopamineRating scalemedicineCriterion validityddc:61Attention deficit hyperactivity disorderHumansddc:610Medizinische Fakultät » Universitätsklinikum Essen » LVR-Klinikum Essen » Klinik für Psychiatrie Psychosomatik und Psychotherapie des Kindes- und JugendaltersDCN PAC - Perception action and control NCEBP 9 - Mental healthGENOME-WIDE ASSOCIATIONNITRIC-OXIDE SYNTHASEPsychiatryGenetic Association StudiesMETAANALYSISPsychiatric Status Rating Scales3204 Developmental and Educational Psychologymedicine.disease030227 psychiatryAttention Deficit Disorder with HyperactivityImpulsive BehaviorEtiologyCRITERION VALIDITY030217 neurology & neurosurgery
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Homoarginine Levels are Regulated by L-Arginine: Glycine Amidinotransferase and Affect Stroke Outcome: Results from Human and Murine Studies

2013

Background— Endogenous arginine homologues, including homoarginine, have been identified as novel biomarkers for cardiovascular disease and outcomes. Our studies of human cohorts and a confirmatory murine model associated the arginine homologue homoarginine and its metabolism with stroke pathology and outcome. Methods and Results— Increasing homoarginine levels were independently associated with a reduction in all-cause mortality in patients with ischemic stroke (7.4 years of follow-up; hazard ratio for 1-SD homoarginine, 0.79 [95% confidence interval, 0.64–0.96]; P =0.019; n=389). Homoarginine was also independently associated with the National Institutes of Health Stroke Scale+age score …

Malegenetics [Homoarginine]AmidinotransferasesArginineGenome-wide association studyCohort StudiesMicesingle nucleotide polymorphismMedicinehomoarginineProspective StudiesStrokegenetics [Arginine]CARDIOVASCULAR RISKHazard ratioMOUSE MODELMiddle Ageddiagnosis [Stroke]strokeDEFICIENCYTreatment OutcomeISCHEMIC-STROKEgenetics [Stroke]genetics [Amidinotransferases]genetics [Polymorphism Single Nucleotide]FemaleCardiology and Cardiovascular MedicineAdultmedicine.medical_specialtySingle-nucleotide polymorphismMASS-SPECTROMETRIC DETERMINATIONArginineGUANIDINO COMPOUNDSPhysiology (medical)Internal medicineglycine amidinotransferaseAnimalsHumansCREATINEddc:610Translational research Energy and redox metabolism [ONCOL 3]AgedNITRIC-OXIDEBLOOD-FLOWbusiness.industryVascular diseasemedicine.diseaseHomoarginineCEREBRAL-ARTERY OCCLUSIONL-arginine:glycine amidinotransferaseMice Inbred C57BLDisease Models AnimalEndocrinologyHEK293 CellsGlycinegenome-wide association studiesHuman medicineArginine:glycine amidinotransferasebusinessGenome-Wide Association StudyCirculation
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Brain BDNF Levels Elevation Induced by Physical Training is Reduced after Unilateral Common Carotid Artery Occlusion in Rats

2014

International audience; We investigated the contribution of blood flow elevation in the cerebrovasculature to physical training-induced brain-derived neurotrophic factor (BDNF) levels elevation in the brain. Brain-derived neurotrophic factor protein levels were measured in the motor cortex 24 h after the last session of a forced treadmill walking (30 minutes a day, 18 m/minute for 7 consecutive days). Unilateral common carotid artery occlusion and modulation of exercise intensity (0 versus -10% inclination of the treadmill) were used as strategies to reduce the (normal) elevation of flow in the cerebrovasculature occurring during exercise. Administration of N-nitro-L-arginine methyl ester (…

Malemedicine.medical_specialtyCarotid Artery CommonINHIBITIONHemodynamicsArterial Occlusive DiseasesNitric OxideVascular occlusionNEUROTROPHIC FACTOR EXPRESSIONPhysical Conditioning AnimalRats Inbred SHRmedicine.arteryInternal medicineOcclusionmedicineAnimalsDYNAMIC EXERCISECommon carotid arteryEnzyme InhibitorsRats WistarTreadmillSPONTANEOUSLY HYPERTENSIVE-RATSBrain-derived neurotrophic factorNITRIC-OXIDEbusiness.industryBrain-Derived Neurotrophic FactorHemodynamicsBrainVELOCITYRatsNG-Nitroarginine Methyl EsterNeurologyCerebral blood flowHIPPOCAMPAL BDNFAnesthesiaHypertensionCEREBRAL-BLOOD-FLOWCardiologyExercise intensityOriginal ArticleEXERCISE-INDUCED INCREASESACTIVATION PATTERNS[ SCCO ] Cognitive scienceNeurology (clinical)medicine.symptomCardiology and Cardiovascular MedicinebusinessJournal of Cerebral Blood Flow & Metabolism
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Fungal-associated NO is involved in the regulation of oxidative stress during rehydration in lichen symbiosis

2010

[EN] Background Reactive oxygen species (ROS) are normally produced in respiratory and photosynthetic electron chains and their production is enhanced during desiccation/rehydration. Nitric oxide (NO) is a ubiquitous and multifaceted molecule involved in cell signaling and abiotic stress. Lichens are poikilohydrous organisms that can survive continuous cycles of desiccation and rehydration. Although the production of ROS and NO was recently demonstrated during lichen rehydration, the functions of these compounds are unknown. The aim of this study was to analyze the role of NO during rehydration of the lichen Ramalina farinacea (L.) Ach., its isolated photobiont partner Trebouxia sp. and Ast…

Microbiology (medical)TrebouxiaII reaction centerLichensDesiccation toleranceBOTANICAlcsh:QR1-502Nitric Oxidemedicine.disease_causeMicrobiologylcsh:MicrobiologyMicrobiologyRamalina farinaceaDesiccation tolerancePhotosystem-IINitric-oxideChlorophytaBotanymedicineSymbiosisLichenBIOLOGIA VEGETALchemistry.chemical_classificationReactive oxygen speciesbiologyAbiotic stressFungiWaterbiology.organism_classificationOxidative StresschemistryPhotosynthetic electron-transportReactive Oxygen SpeciesDesiccationNon-heme ironOxidative stressResearch ArticleBMC Microbiology
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2019 ESC Guidelines for the diagnosis and management of acute pulmonary embolism developed in collaboration with the European Respiratory Society (ER…

2020

Guidelines summarize and evaluate available evidence with the aim of assisting health professionals in proposing the best management strategies for an individual patient with a given condition. Guidelines and their recommendations should facilitate decision making of health professionals in their daily practice. However, the final decisions concerning an individual patient must be made by the responsible health professional(s) in consultation with the patient and caregiver as appropriate.

ORAL ANTICOAGULANT-THERAPYdiagnosis[SDV]Life Sciences [q-bio]Medizin030204 cardiovascular system & hematologyEmbolectomyGuidelineRECURRENT VENOUS THROMBOEMBOLISM0302 clinical medicinePregnancyDaily practiceDiagnosisPulmonary medicineVenous thrombosisPulmonary MedicineThrombolytic TherapyDEEP-VEIN THROMBOSISDisease management (health)Societies MedicalComputingMilieux_MISCELLANEOUShealth care economics and organizationsRisk assessmentddc:616RIGHT-VENTRICULAR DYSFUNCTIONDisease ManagementShockMOLECULAR-WEIGHT HEPARINThrombolysishumanities3. Good healthPulmonary embolismEuropeAnticoagulation; Biomarkers; Diagnosis; Dyspnoea; Echocardiography; Embolectomy; Guidelines; Heart failure; Pregnancy; Pulmonary embolism; Right ventricle; Risk assessment; Shock; Thrombolysis; Treatment; Venous thromboembolism; Venous thrombosismedicine.veinEchocardiographyAcute DiseaseRight ventricleMedical emergencyCardiology and Cardiovascular MedicineRisk assessmentguidelines; pulmonary embolism; venous thrombosis; shock dyspnoea; heart failure: right ventricle: diagnosis; risk assessment: echocardiography; biomarkers; treatment; anticoagulation; thrombolysis; pregnancy; venous thromboembolism; embolectomyDiagnosiVenous thromboembolismthrombolysiseducationCardiologyMEDLINEThrombolysiHeart failure610 Medicine & healthGuidelinesInferior vena cava2705 Cardiology and Cardiovascular MedicineEXTRACORPOREAL MEMBRANE-OXYGENATION03 medical and health sciencesAnticoagulationMedicalDyspnoeamedicineHumansRIGHT HEART THROMBIVENTILATION-PERFUSION SCINTIGRAPHYshock dyspnoeaHealth professionalsbusiness.industryINFERIOR VENA-CAVA10031 Clinic for Angiologyheart failure: right ventricle: diagnosisPulmonary embolismAnticoagulantsbiomarkersrisk assessment: echocardiography030229 sport sciencesBiomarkermedicine.diseaseTreatmentINHALED NITRIC-OXIDESocietiesbusinessBiomarkers
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7-Nitroindazole protects striatal neurons against MPP+ -induced degeneration.

2006

The neuropathological hallmark of Parkinson's disease (PD) is the selective degeneration of dopaminergic (DAergic) neurons in the substantia nigra pars compacta (SNc). In this study, using a microdialysis technique, we investigated whether an inhibitor of neuronal nitric oxide synthase (nNOS), 7-nitrindazole (7-NI), could protect against DAergic neuronal damage induced by in vivo infusion of 1-methyl-4-phenylpiridinium iodide (MPP+) in freely moving rats. Experiments were performed over 2 days in three groups of rats: (a) nonlesioned, (b) MPP+-lesioned, and (c) 7-NI pretreated MPP+-lesioned rats. On day 1, control rats were perfused with an artificial CSF, while 1 mM MPP+ was infused into t…

PARKINSONS-DISEASERAT STRIATUMMONOAMINE-OXIDASENITRIC-OXIDE SYNTHASEBRAINNEURODEGENERATIVE DISEASESSODIUM-SALICYLATEMPTP NEUROTOXICITYTRANSPORTER ACTIVITYLIPID-PEROXIDATION
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Temporal phase control of bound-bound and bound-free two-photon transitions in NO with two time-delayed cross-polarized pulses

2001

0953-4075; Temporal phase control over the multi-photoionization of NO is investigated experimentally The coherent excitation of the molecular system is achieved by a sequence of two phase-related non-Fourier-transform-limited laser pulses delivered by a nanosecond dye laser, The phase-locked pulses are produced by making use of an actively stabilized interferometer with a tunable pathlength difference. As the coherence time of the laser is comparable with the inhomogenous dephasing time of the medium, the measurements are performed with temporally overlapped pulses. In order to avoid the modulation of the ionic signal coming from optical interference, the linear polarizations of the two pu…

PhysicsINTERFERENCECoherence timeDye laserNITRIC-OXIDESPECTROSCOPYbusiness.industryDephasingNanosecondCondensed Matter PhysicsLaserAtomic and Molecular Physics and Opticslaw.inventionMOLECULESOpticslawExcited stateIonizationAUTOIONIZATIONAtomic physicsCOHERENT CONTROLFIELDbusiness2-PHOTON TRANSITIONSExcitation
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eNOS Activation by HDL Is Impaired in Genetic CETP Deficiency.

2014

Mutations in the CETP gene resulting in defective CETP activity have been shown to cause remarkable elevations of plasma HDL-C levels, with the accumulation in plasma of large, buoyant HDL particles enriched in apolipoprotein E. Genetic CETP deficiency thus represents a unique tool to evaluate how structural alterations of HDL impact on HDL atheroprotective functions. Aim of the present study was to assess the ability of HDL obtained from CETP-deficient subjects to protect endothelial cells from the development of endothelial dysfunction. HDL isolated from one homozygous and seven heterozygous carriers of CETP null mutations were evaluated for their ability to down-regulate cytokine-induced…

Settore MED/09 - Medicina InternaCHOLESTEROL EFFLUXApolipoprotein BEpidemiologylcsh:MedicineANTIINFLAMMATORY PROPERTIESmedicine.disease_causeBiochemistryVascular Medicinechemistry.chemical_compoundHigh-density lipoproteinEnosMedicine and Health SciencesEndothelial dysfunctionlcsh:ScienceMutationMultidisciplinarybiologyHomozygoteCETP; eNOS; HDL;NeurochemistryLipidsGenetic EpidemiologyeNOSlipids (amino acids peptides and proteins)AnatomyNeurochemicalsLipoproteins HDLResearch Articlemedicine.medical_specialtyDrug Research and DevelopmentHDLNitric Oxide Synthase Type IIILipoproteinsENDOTHELIAL FUNCTIONINHIBITIONCardiologyDown-RegulationVascular Cell Adhesion Molecule-1Nitric OxideCELL-ADHESION MOLECULE-1Lipid Metabolism Inborn ErrorsESTER TRANSFER PROTEINInternal medicineCETPCholesterylester transfer proteinHuman Umbilical Vein Endothelial CellsmedicineHumansNITRIC-OXIDE SYNTHASEInflammationClinical GeneticsPharmacologyCholesterollcsh:RTorcetrapibEndothelial CellsBiology and Life SciencesProteinsnutritional and metabolic diseasesLipid MetabolismAtherosclerosismedicine.diseasebiology.organism_classificationCholesterol Ester Transfer Proteinscarbohydrates (lipids)MetabolismEndocrinologychemistryOther Clinical MedicineMutationImmunologyCardiovascular Anatomybiology.proteinlcsh:QTORCETRAPIBClinical MedicineHIGH-DENSITY-LIPOPROTEINSCAVENGER RECEPTOR BI
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ERYTHROPOIETIN FOR THE TREATMENT OF SUBARACHNOID HEMORRAGE: A FEASIBLE INGREDIENT FOR A SUCCESS MEDICAL RECIPE

2015

Subarachnoid hemorrhage (SAH) following aneurysm bleeding accounts for 6% to 8% of all cerebrovascular accidents. Although an aneurysm can be effectively managed by surgery or endovascular therapy, delayed cerebral ischemia is diagnosed in a high percentage of patients resulting in significant morbidity and mortality. Cerebral vasospasm occurs in more than half of all patients after aneurysm rupture and is recognized as the leading cause of delayed cerebral ischemia after SAH. Hemodynamic strategies and endovascular procedures may be considered for the treatment of cerebral vasospasm. In recent years, the mechanisms contributing to the development of vasospasm, abnormal reactivity of cerebr…

Subarachnoid hemorrhagebusiness.industrySettore MED/27 - NeurochirurgiaCerebral arteriesIschemiaHemodynamicsVasospasmReview Articlemedicine.diseasenervous system diseasesCerebral vasospasmAneurysmErythropoietinAnesthesiaRECOMBINANT-HUMAN-ERYTHROPOIETIN NITRIC-OXIDE SYNTHASE ENDOTHELIAL NO SYNTHASE BLOOD-BRAIN-BARRIER IN-VIVO EVIDENCE CEREBRAL-ISCHEMIA DARBEPOETIN-ALPHA TISSUE PROTECTION DOUBLE-BLIND RECEPTORGeneticscardiovascular systemMolecular MedicineMedicinecardiovascular diseasesbusinessMolecular BiologyGenetics (clinical)medicine.drugsubarachnoid hemorrage erytropoietin
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Nitric Oxide Reductase (P450nor) from Fusarium oxysporum

2008

biologyChemistryNitric-oxide reductaseFusarium oxysporumbiology.organism_classificationMicrobiology
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