Search results for "nitrite"

showing 10 items of 227 documents

Silibinin improves hepatic and myocardial injury in mice with nonalcoholic steatohepatitis

2012

Abstract Background Nonalcoholic fatty liver disease is a chronic metabolic disorder with significant impact on cardiovascular and liver mortality. Aims In this study, we examined the effects of silibinin on liver and myocardium injury in an experimental model of nonalcoholic fatty liver disease. Methods A four-week daily dose of silibinin (20 mg/kg i.p.) was administrated to db/db mice fed a methionine–choline deficient diet. Hepatic and myocardial histology, oxidative stress and inflammatory cytokines were evaluated. Results Silibinin administration decreased HOMA-IR, serum ALT and markedly improved hepatic and myocardial damage. Silibinin reduced isoprostanes, 8-deoxyguanosine and nitrit…

MaleGene ExpressionIsoprostanesmedicine.disease_causeGastroenterologyAntioxidantsMicechemistry.chemical_compoundMethionineNon-alcoholic Fatty Liver DiseaseNonalcoholic fatty liver diseasePhosphorylationGastroenterologyAlanine TransaminaseGlutathioneCholine DeficiencyMitochondrial respiratory chainLiverHeart Inflammation NAFLD Oxidative stress SilibininCytokinesmedicine.symptomSilymarinmedicine.medical_specialtySilibininInflammationStatistics NonparametricProinflammatory cytokineInsulin resistanceInternal medicinemedicineAnimalsNitritesAnalysis of VarianceNitratesHepatologySettore BIO/16 - Anatomia Umanabusiness.industryMyocardiumJNK Mitogen-Activated Protein KinasesGlutathionemedicine.diseaseDietFatty LiverOxidative StressEndocrinologychemistrySilybinInsulin ResistancebusinessOxidative stressDigestive and Liver Disease
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Increased Circulating Levels of 3-Nitrotyrosine Autoantibodies

2012

3-nitrotyrosine formation is an oxidative protein modification that was first discovered in vivo in the early 1990s by Beckman and colleagues.1,2 The biological relevance of this process was extensively investigated in the subsequent years and further facilitated by the development of 3-nitrotyrosine–specific antibodies.3 Protein tyrosine nitration is mainly mediated by 3 biochemical processes (Figure): (1) by peroxynitrite (ONOO−) formation,4–6 the reaction product of nitric oxide (•NO) and superoxide (•O2−); (2) by a (myelo)peroxidase-catalyzed nitrogen dioxide radical (•NO2) formation from hydrogen peroxide and nitrite;7,8 and (3) by a nonspecific formation of the nitrogen dioxide radica…

MaleImmunoglobulinsProstacyclinCoronary Artery DiseasePharmacologyArticleProstacyclin synthaseNitric oxideEpitopeschemistry.chemical_compoundPhysiology (medical)medicineHumansbiologySuperoxidebusiness.industryNitric Oxide Synthase Type IIIPeroxynitrous acidchemistryBiochemistryMyeloperoxidasebiology.proteinTyrosineFemaleCardiology and Cardiovascular MedicinebusinessPeroxynitritemedicine.drugCirculation
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Suppression of leukotriene B4 and tumour necrosis factor alpha release in acute inflammatory responses by novel prenylated hydroquinone derivatives.

1998

A series of prenyl hydroquinone derivatives synthesized as structural analogs of marine products were tested for their effects on inflammatory responses in vitro and in vivo. 2-Prenyl-1,4-hydroquinone (H1), 2-diprenyl-1,4-hydroquinone (H2), 2-triprenyl-1,4-hydroquinone (H3) and 2-tetraprenyl-1,4-hydroquinone (H4) scavenged reactive oxygen species and inhibited 5-lipoxygenase (5-LO) activity in human neutrophils. The inhibition of 5-LO activity was demonstrated in vivo in the mouse air pouch injected with zymosan and arachidonic acid-induced ear inflammation. The four compounds suppressed the production of tumour necrosis factor alpha (TNFalpha) in J774 cells stimulated with lipopolysacchari…

MaleNecrosisLipopolysaccharideLeukotriene B4Anti-Inflammatory AgentsPharmacologyLeukotriene B4Dinoprostonechemistry.chemical_compoundMiceIn vivomedicineAnimalsEdemaHumansCells CulturedNitritesPharmacologyInflammationArachidonic AcidbiologyTumor Necrosis Factor-alphaZymosanGeneral MedicineHydroquinonesNitric oxide synthasechemistryBiochemistryDepression ChemicalArachidonate 5-lipoxygenaseLuminescent Measurementsbiology.proteinTumor necrosis factor alphamedicine.symptomNaunyn-Schmiedeberg's archives of pharmacology
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Nitric oxide synthase 2 (NOS2) expression in histologically normal margins of oral squamous cell carcinoma

2014

The activity of Nitric Oxide Synthase 2 (NOS2) was found in oral squamous cell carcinomas (OSCC) but not in normal mucosa. Molecular changes associated to early carcinogenesis have been found in mucosa near carcinomas, which is considered a model to study field cancerization. The aim of the present study is to analyze NOS2 expression at the histologically normal margins of OSCC. Study Design: Eleven biopsy specimens of OSCC containing histologically normal margins (HNM) were analyzed. Ten biopsies of normal oral mucosa were used as controls. The activity of NOS2 was determined by immunohistochemistry. Salivary nitrate and nitrite as well as tobacco and alcohol consumption were also analyzed…

MalePathologyNitric Oxide Synthase Type IImedicine.disease_causechemistry.chemical_compoundField cancerizationMedicineNitriteMouth neoplasmAged 80 and overbiologymedicine.diagnostic_testCarcinoma oral de células escamosasNitric oxide synthase 2Patología//purl.org/becyt/ford/3.1 [https]Middle Aged:CIENCIAS MÉDICAS [UNESCO]Ciencias de la saludMedicina BásicaOral squamous cell carcinomaUNESCO::CIENCIAS MÉDICASCarcinoma Squamous CellImmunohistochemistryFemaleMouth Neoplasms//purl.org/becyt/ford/3 [https]Adultmedicine.medical_specialtyCIENCIAS MÉDICAS Y DE LA SALUDOdontologíaBiopsyparasitic diseasesCarcinomaHumansNitric Oxide Synthase 2 (NOS2)Cancerización de campoGeneral DentistryAgedMarcadores de malignidadOral Medicine and Pathologybusiness.industryResearchmedicine.diseasestomatognathic diseasesCross-Sectional StudiesOtorhinolaryngologychemistrybiology.proteinSurgeryField cancerizationbusinessCarcinogenesisMalignity markers
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Nitric Oxide Mediates Natural Polyphenol-induced Bcl-2 Down-regulation and Activation of Cell Death in Metastatic B16 Melanoma

2007

Intravenous administration to mice of trans-pterostilbene (t-PTER; 3,5-dimethoxy-4'-hydroxystilbene) and quercetin (QUER; 3,3',4',5,6-pentahydroxyflavone), two structurally related and naturally occurring small polyphenols, inhibits metastatic growth of highly malignant B16 melanoma F10 (B16M-F10) cells. t-PTER and QUER inhibit bcl-2 expression in metastatic cells, which sensitizes them to vascular endothelium-induced cytotoxicity. However, the molecular mechanism(s) linking polyphenol signaling and bcl-2 expression are unknown. NO is a potential bioregulator of apoptosis with controversial effects on Bcl-2 regulation. Polyphenols may affect NO generation. Short-term exposure (60 min/day) t…

MaleProgrammed cell deathCeramideEndotheliumDown-RegulationBiologyNitric OxideBiochemistryMicechemistry.chemical_compoundPhenolsCell Line TumorCell AdhesionmedicineAnimalsRNA MessengerNeoplasm MetastasisCytotoxicityMelanomaMolecular BiologyNitritesFlavonoidsNitratesCell DeathReverse Transcriptase Polymerase Chain ReactionPolyphenolsHydrogen PeroxideCell BiologyGenes bcl-2Cell biologyMice Inbred C57BLEndothelial stem cellmedicine.anatomical_structureProto-Oncogene Proteins c-bcl-2Mitochondrial permeability transition porechemistryCell cultureApoptosisMitochondrial MembranesCancer researchEndothelium VascularJournal of Biological Chemistry
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Pre-clinical assessment of a water-in-fluorocarbon emulsion for the treatment of pulmonary vascular diseases

2019

Abstract Hypoxic pulmonary vasoconstriction (HPV) is a well-characterized vascular response to low oxygen pressures and is involved in life-threatening conditions such as high-altitude pulmonary edema (HAPE) and pulmonary arterial hypertension (PAH). While the efficacy of oral therapies can be affected by drug metabolism, or dose-limiting systemic toxicity, inhaled treatment via pressured metered dose inhalers (pMDI) may be an effective, nontoxic, practical alternative. We hypothesized that a stable water-in-perfluorooctyl bromide (PFOB) emulsion that provides solubility in common pMDI propellants, engineered for intrapulmonary delivery of pulmonary vasodilators, reverses HPV during acute h…

MalePulmonary CirculationDrug Evaluation PreclinicalPharmaceutical Science02 engineering and technologyPharmacology030226 pharmacology & pharmacyRats Sprague-DawleyDrug Delivery Systems0302 clinical medicineHypoxic pulmonary vasoconstrictionHigh-altitude pulmonary edemapulmonary hypertensionMedicineFluorocarbonsPhenylpropionatesmedicine.diagnostic_testGeneral Medicine021001 nanoscience & nanotechnologyPulmonary edema3. Good healthPyridazinesTreatment Outcomemedicine.anatomical_structureEmulsions0210 nano-technologyendothelinResearch Articlemedicine.drugpulmonary pressuresAmbrisentanambrisentanHypertension PulmonaryPulmonary Edema03 medical and health sciencesmedicine.arteryAnimalsAntihypertensive AgentsLungsodium nitriteperfluorocarbonbusiness.industrylcsh:RM1-950Watermedicine.diseasePulmonary hypertensionRatsBronchoalveolar lavagelcsh:Therapeutics. PharmacologyPulmonary arteryhigh altitude pulmonary edemabusinessDrug Delivery
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Inducible NO synthase confers chemoresistance in head and neck cancer by modulating survivin

2009

The dual role of the inducible NO synthase (iNOS) and NO signaling in head and neck squamous cell carcinoma (HNSCC) is a complex and can both promote or inhibit tumor progression. However, the underlying molecular mechanisms are not yet resolved in detail. We show for the first time that conditions, favoring low NO levels conferred resistance against cisplatin/taxol-induced apoptosis in HNSCC cell lines. Cytoprotection was mediated by survivin, because we observed its upregulation subsequent to low doses of the NO donors S-nitroso-N-acetyl-penicillamine (SNAP) and sodium nitroprusside (SNP) or ectopic expression of physiologic amounts of iNOS. Also, RNAi-mediated depletion of survivin block…

MaleUmbilical VeinsCancer ResearchSurvivinFluorescent Antibody TechniqueNitric Oxide Synthase Type IIApoptosisp38 Mitogen-Activated Protein KinasesInhibitor of Apoptosis ProteinsImmunoenzyme TechniquesPhosphatidylinositol 3-Kinaseschemistry.chemical_compoundLY294002Enzyme InhibitorsRNA Small InterferingAged 80 and overReverse Transcriptase Polymerase Chain ReactionCell CycleMiddle AgedCell cycleOncologyHead and Neck NeoplasmsCarcinoma Squamous CellFemaleMicrotubule-Associated ProteinsNitroprussidePaclitaxelImmunoblottingAntineoplastic AgentsS-Nitroso-N-AcetylpenicillamineBiologyCell LineDownregulation and upregulationSurvivinmedicineHumansNitric Oxide DonorsRNA MessengerneoplasmsProtein kinase BNitritesPI3K/AKT/mTOR pathwayAgedmedicine.diseaseAntineoplastic Agents PhytogenicHead and neck squamous-cell carcinomachemistryDrug Resistance NeoplasmTumor progressionImmunologyCancer researchEndothelium VascularCisplatinProto-Oncogene Proteins c-aktInternational Journal of Cancer
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Nitric oxide production is increased in severely obese children and related to markers of oxidative stress and inflammation

2011

Nitric oxide (NO) is the major endothelium-derived relaxing factor. The aim of the present study was to evaluate NO synthesis and metabolism in severely obese children with different degrees of metabolic risk and to ascertain their relation with the parameters of oxidative stress and inflammation.The study involved 60 obese children evaluated with respect to metabolic risk factors (MRFs) (324 MRFs and 28 ≥ 4 MRFs) and 50 normal weight children between 7 and 14 years of age. Nutritional status was assessed by clinical and anthropometric examination. MRFs (serum lipid profile, insulin resistance indexes, blood pressure) in addition to uric acid, homocysteine, leptin, and inflammatory markers …

Malemedicine.medical_specialtyAdolescentInflammationNitric Oxidemedicine.disease_causeNitric oxidechemistry.chemical_compoundInsulin resistanceInternal medicinemedicineHumansObesityNitriteChildNitritesTriglyceridesAbdominal obesityInflammationNitratesTumor Necrosis Factor-alphabusiness.industrymedicine.diseaseObesityUric AcidOxidative StressEndocrinologychemistryUric acidFemaleInsulin ResistanceWaist Circumferencemedicine.symptomCardiology and Cardiovascular MedicinebusinessBiomarkersOxidative stressAtherosclerosis
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Plasma concentrations of nitric oxide and asymmetric dimethylarginine in human alcoholic cirrhosis.

2004

The liver plays a prominent role in the metabolism of asymmetric dimethyl-l-arginine (ADMA), an endogenous inhibitor of nitric oxide (NO) synthase. This study was designed to determine whether plasma levels of ADMA and NO production are altered in patients with compensated and decompensated alcoholic cirrhosis.Plasma levels of l-arginine, ADMA, symmetric dimethylarginine (SDMA) and NO (nitrite plus nitrate, NOx) were measured in nine patients with compensated alcoholic cirrhosis (Child-Pugh A) and 11 patients with advanced cirrhosis (Child-Pugh B-C). Seven healthy volunteers served as controls.ADMA and NOx concentrations in decompensated cirrhosis were higher than in the compensated group a…

Malemedicine.medical_specialtyAlcoholic liver diseaseCirrhosisVasodilationArginineNitric OxideNitric oxidechemistry.chemical_compoundLiver Cirrhosis AlcoholicInternal medicineBlood plasmamedicineHumansNitriteNitritesNitratesHepatologybusiness.industryMetabolismMiddle Agedmedicine.diseaseVasodilationEndocrinologychemistryFemaleEndothelium VascularbusinessAsymmetric dimethylarginineJournal of hepatology
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Hydralazine is a powerful inhibitor of peroxynitrite formation as a possible explanation for its beneficial effects on prognosis in patients with con…

2005

The hemodynamic and anti-ischemic effects of nitroglycerin (GTN) are rapidly blunted as a result of the development of nitrate tolerance. Hydralazine has been shown to prevent tolerance in experimental and clinical studies, all of which may be at least in part secondary to antioxidant properties of this compound. The antioxidant effects of hydralazine were tested in cell free systems, cultured smooth muscle cells, isolated mitochondria, and isolated vessels. Inhibitory effects on the formation of superoxide and/or peroxynitrite formation were tested using lucigenin and L-012 enhanced chemiluminescence as well as DHE-fluorescence. The peroxynitrite scavenging properties were also assessed by…

Malemedicine.medical_specialtyAntioxidantmedicine.medical_treatmentBiophysicsMitochondrionBiochemistryAntioxidantsMitochondrial ProteinsNitroglycerinchemistry.chemical_compoundPeroxynitrous AcidInternal medicinemedicineAnimalsHumansLucigeninRats WistarMolecular BiologyHeart FailureSuperoxideAldehyde Dehydrogenase MitochondrialMicrofilament ProteinsDrug ToleranceFree Radical ScavengersCell BiologyAldehyde DehydrogenaseHydralazineHydralazinePhosphoproteinsPrognosismedicine.diseaseReactive Nitrogen SpeciesMitochondriaRatsOxidative StressEndocrinologychemistryHeart failureIsosorbide dinitrateReactive Oxygen SpeciesCell Adhesion MoleculesPeroxynitritemedicine.drugBiochemical and Biophysical Research Communications
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