Search results for "nmda"

showing 10 items of 147 documents

Electrophysiological and Iontophoretic Aspects of the Habenular Influence on Hippocampal Neurones

1995

In previous experimental studies, carried out on cats, we demonstrated that electrical stimulation of lateral habenula (LH) at 0.5-3.0 Hz or 5-20 Hz had a double effect (low frequency-excitation; high frequency-inhibition) on the spontaneous firing rate of single hippocampal neurones. Our results, in agreement with similar case studies, allowed us to hypothesise that in the habenular modulation of the hippocampus the raphe nucleus is probably involved. In fact, all the effects of LH stimulation were antagonised by the iontophoretic intrahippocampal application of methysergide. In the present series of experiments, performed on rats, it was possible to demonstrate that LH stimulation at 1-10…

MaleSerotoninN-MethylaspartatePhysiologyMethysergideHippocampusStimulationHippocampal formationHippocampusPhysiology (medical)medicineAnimalsRats WistarNeuronsIontophoresisMethysergideChemistryGeneral MedicineIontophoresisElectric StimulationCochleaRatsElectrophysiologyElectrophysiologyRaphe NucleiNMDA receptorRaphe nucleiNeurosciencemedicine.drugArchives of Physiology and Biochemistry
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The effects of nitric oxide on striatal serotoninergic transmission involve multiple targets: an in vivo microdialysis study in the awake rat

2004

Abstract The role of endogenous nitric oxide (NO) in N -methyl- d -aspartate (NMDA)-induced modulation of serotonin (5-HT) release in the striatum of freely moving rats has been studied using microdialysis technique. NMDA-induced increase in 5-HT release was significantly inhibited by selective nitric oxide synthase (nNOS) inhibitor S -methylthiocitrulline (S-Me-TC), ONOO − scavenger l -cysteine ( l -cys), and guanylate cyclase (GC) inhibitor 1 H [1,2,4]oxadiazolo[4,3- a ]quinoxalin-1-one (ODQ). These data suggest that modulation of 5-HT levels is linked to the formation of NO produced by NMDA receptor activation and that endogenously produced NO increases 5-HT concentrations both by stimul…

MaleSerotoninmedicine.medical_specialtyMicrodialysisN-MethylaspartateMicrodialysisNitric Oxide Synthase Type IPharmacologyNitric OxideSerotonergicSynaptic TransmissionNitric oxidechemistry.chemical_compoundSuperoxidesPeroxynitrous AcidInternal medicinemedicineAnimalsEnzyme InhibitorsRats WistarNeurotransmitterCyclic GMPMolecular Biologyneurotransmitters; modulators; transporters; and receptors; nitric oxide; serotonin; striatumbiologyGeneral NeuroscienceFree Radical ScavengersRatsNeostriatumNitric oxide synthasePeroxynitrous acidEndocrinologychemistryGuanylate Cyclasebiology.proteinNMDA receptorNeurology (clinical)SerotoninNitric Oxide SynthaseSignal TransductionDevelopmental Biology
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Differential effect of beta-N-oxalylamino-L-alanine, the Lathyrus sativus neurotoxin, and (+/-)-alpha-amino-3-hydroxy-5-methylisoxazole-4-propionate …

2000

We studied the effect of beta-oxalylamino-L-alanine, a glutamate analog present in Lathyrus sativus seeds and implicated in the etiopathogenesis of neurolathyrism, and (+/-)-alpha-amino-3-hydroxy-5-methylisoxazole-4-propionate on the extracellular levels of aspartate, glutamate and taurine in the primary motor cortex of freely moving rats. We found that while both neurotoxins increase the level of aspartate and glutamate, only (+/-)-alpha(-amino-3-hydroxy-5-methylisoxazole-4-propionate is able to modulate the level of taurine. GYKI-52466, a non-competitive non-NMDA antagonist, inhibited beta-oxalylamino-L-alanine-induced increase of aspartate, but not that of glutamate. Conversely, this ant…

MaleTaurineTaurineMicrodialysisGlutamic AcidTetrodotoxinReceptors N-Methyl-D-AspartateRats Sprague-DawleyCellular and Molecular Neurosciencechemistry.chemical_compoundGlutamate aspartate transporterNeurotoxinAnimalsNeurotransmitteralpha-Amino-3-hydroxy-5-methyl-4-isoxazolepropionic AcidAlaninechemistry.chemical_classificationAspartic AcidbiologyGlutamate receptorMotor CortexAmino Acids DiaminoBrainCell BiologyCorpus StriatumAmino acidRatschemistryBiochemistrybiology.proteinPotassiumbeta-AlanineNMDA receptorExtracellular SpaceExcitatory Amino Acid AntagonistsNeurochemistry international
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Chronic Moderate Hyperammonemia Impairs Active and Passive Avoidance Behavior and Conditional Discrimination Learning in Rats

2000

Abstract The cerebral dysfunction associated with hepatic encephalopathy is generally considered to have hyperammonemia as one of its main causes. Hyperammonemia impairs the neuronal glutamate–nitric oxide–cyclic GMP pathway and the induction of NMDA receptor-dependent long-term potentiation in the hippocampus. We studied the performance of pre/neonatally and postnatally exposed rats to hyperammonemia on active avoidance, passive avoidance, and conditional discrimination tasks. Pre/neonatal hyperammonemia slowed learning of active avoidance behaviors and impaired memory for the passive avoidance task while postnatal hyperammonemia impaired learning on the conditional discrimination task. Hy…

MaleTime FactorsHippocampusAcetatesMotor ActivityDiscrimination LearningDevelopmental NeuroscienceAmmoniaPregnancyAvoidance LearningmedicineAnimalsRats WistarHepatic encephalopathyAnalysis of VarianceHyperammonemiaLong-term potentiationCognitionImpaired memorymedicine.diseaseAnimal FeedRatsAnimals NewbornNeurologyPrenatal Exposure Delayed EffectsAnesthesiaNMDA receptorFemalePassive avoidancePsychologyNeuroscienceExperimental Neurology
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Homogenous glycine receptor expression in cortical plate neurons and cajal-retzius cells of neonatal rat cerebral cortex

2004

Glycinergic membrane responses have been described in cortical plate neurons (CPn) and Cajal-Retzius cells (CRc) during early neocortical development. In order to elucidate the functional properties and molecular identity of glycine receptors in these two neuronal cell types, we performed whole-cell patch-clamp recordings and subsequent single-cell multiplex reverse transcriptase-polymerase chain reaction (RT-PCR) analyses on visually identified neurons in tangential and coronal slices as well as in situ hybridizations of coronal slices from neonatal rat cerebral cortex (postnatal days 0-4). In both CPn and CRc the glycinergic agonists glycine, beta-alanine and taurine induced inward curren…

Malemedicine.drug_classBiologyMembrane PotentialsGABA Antagonistschemistry.chemical_compoundReceptors GlycinemedicineAnimalsPatch clampRats WistarReceptorGlycine receptorCerebral CortexNeuronsDose-Response Relationship DrugGeneral NeuroscienceGene Expression Regulation DevelopmentalStrychnineBicucullineReceptor antagonistMolecular biologyRatsAnimals NewbornchemistryBiochemistryGlycineNMDA receptorExcitatory Amino Acid Antagonistsmedicine.drugNeuroscience
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Behavioural parameters in aged rats are related to LTP and gene expression of ChAT and NMDA-NR2 subunits in the striatum.

2004

Striatal parameters were assessed for their relevance to age-related behavioural decline. Forty aged rats (28-30 months) were tested in the water maze and open field. Of these, seven superior and seven inferior learners were compared with each other in terms of levels of in vitro short- and long-term potentiation (STP and LTP), and gene expression of choline acetyltransferase (ChAT) as well as of the NMDA-NR2A-C subunits assessed by quantitative RT-PCR. Results revealed that the superior as compared with the inferior learners had higher levels of ChAT mRNA in the striatum. For the superior group, ChAT mRNA was correlated with escape on to the cued platform in the water maze, whereas level o…

Malemedicine.medical_specialtyAgingeducationLong-Term PotentiationStriatumWater mazeReceptors N-Methyl-D-AspartateOpen fieldCholine O-AcetyltransferaseInternal medicineGene expressionmedicineAnimalsRNA MessengerRats WistarMaze LearningGeneral NeuroscienceLong-term potentiationCholine acetyltransferaseCorpus StriatumRatsEndocrinologynervous systemGene Expression RegulationSynaptic plasticityExploratory BehaviorNMDA receptorPsychologyNeuroscienceThe European journal of neuroscience
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Acute ammonia toxicity is mediated by the NMDA type of glutamate receptors

1992

AbstractPrevious experiments in our laboratory suggested that ammonium toxicity could be mediated by the NMDA type of glutamate receptors. To assess this hypothesis we tested if MK-801, a specific antagonist of the NMDA receptor, is able to prevent ammonium toxicity. Mice and rats were injected i.p. with 12 and 7 mmol/kg of ammonium acetate, respectively, 73% of the mice and 70% of the rats died. However, when the animals were injected i.p. with 2 mg/kg of MK-801, 15 min before ammonium injection, only 5% of the mice and 15% of the rats died. The remarkable protection afforded by MK-801 indicates that ammonia toxicity is mediated by the NMDA receptor.

Malemedicine.medical_specialtyBiophysicsGlutamic AcidReceptors N-Methyl-D-AspartateBiochemistryAmmonia toxicityMicechemistry.chemical_compoundGlutamatesAmmoniaStructural BiologyInternal medicineGeneticsmedicineAnimalsHyperammonemiaNeurotransmitter metabolismAmmoniumReceptorMolecular BiologyMK-801Glutamate receptorRats Inbred StrainsValineCell BiologyGlutamic acidNMDA receptorRatsReceptors NeurotransmitterEndocrinologyReceptors GlutamatechemistryToxicityNMDA receptorDizocilpine MaleateAmmonium acetateFEBS Letters
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Role of N-methyl-D-aspartate receptors in the long-term effects of repeated social defeat stress on the rewarding and psychomotor properties of cocai…

2019

Abstract Exposure to social stress increases the vulnerability of experimental animals to the rewarding effects of cocaine and it has been suggested that the glutamatergic system could be involved in these effects of stress. The aim of this work is to determine the role of N-methyl- d -aspartate (NMDA) glutamate receptors in the influence of social stress on the conditioned place preference and locomotor sensitization induced by cocaine. Mice treated with saline or NMDA antagonist memantine (5 or 10 mg/kg) underwent repeated social defeat or were kept in the exploration control condition. After three weeks, all groups (SAL + RSD, M5 + RSD, M10 + RSD, SAL + EXP, M5 + EXP and M10 + EXP) were …

Malemedicine.medical_specialtyConditioning ClassicalMice Inbred StrainsReceptors N-Methyl-D-AspartateSocial defeat03 medical and health sciencesBehavioral NeuroscienceGlutamatergicMice0302 clinical medicineCocaineRewardMemantineInternal medicinemedicineAnimalsSocial BehaviorSensitization030304 developmental biologySocial stress0303 health sciencesBehavior Animalbusiness.industryMemantineGlutamate receptorConditioned place preferencemedicine.anatomical_structureEndocrinologyPsychological DistanceNMDA receptorbusiness030217 neurology & neurosurgeryStress Psychologicalmedicine.drugBehavioural brain research
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NMDA receptor antagonist felbamate reduces behavioral deficits and blood-brain barrier permeability changes after experimental subarachnoid hemorrhag…

2007

Increased levels of glutamate and aspartate have been detected after subarachnoid hemorrhage (SAH) that correlate with neurological status. The NMDA receptor antagonist felbamate (FBM; 2-phenyl-1,3-propanediol dicarbamate) is an anti-epileptic drug that elicits neuroprotective effects in different experimental models of hypoxia-ischemia. The aim of this dose-response study was to evaluate the effect of FBM after experimental SAH in rats on (1) behavioral deficits (employing a battery of assessment tasks days 1-5 post-injury) and (2) blood-brain barrier (BBB) permeability changes (quantifying microvascular alterations according to the extravasation of protein-bound Evans Blue by a spectropho…

Malemedicine.medical_specialtyExcitotoxicityPhenylcarbamatesBehavioral deficitsmedicine.disease_causeCisterna magnaBlood–brain barrierNeuroprotectionReceptors N-Methyl-D-AspartateFelbamateRats Sprague-Dawleychemistry.chemical_compoundInternal medicinemedicineAnimalsAnimals; Blood-Brain Barrier; Rats; Subarachnoid Hemorrhage; Evans Blue; Behavioral deficits; Cognitive deficits; NMDA receptor; FelbamatePostural BalanceEvans BlueBehavior AnimalDose-Response Relationship Drugbusiness.industryCognitive deficitsMicrocirculationBody WeightGlutamate receptorSubarachnoid HemorrhageNMDA receptorFelbamateRatsEndocrinologymedicine.anatomical_structureNeuroprotective AgentsSpectrometry FluorescencechemistryBlood-Brain BarrierPropylene GlycolsAnesthesiaCerebrovascular CirculationNMDA receptorNeurology (clinical)businessmedicine.drugEvans BlueJournal of neurotrauma
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Nitric oxide and glutamate interaction in the control of cortical and hippocampal excitability.

1999

Summary: Purpose: We investigated the role of nitric oxide (NO) as a new neurotransmitter in the control of excitability of the hippocampus and the cerebral cortex, as well as the possible functional interaction between NO and the glutamate systems. Methods: The experiments were performed on anesthetized rats. The bioelectrical activities of the somatosensory cortex and the CA1 region of the hippocampus of these rats were recorded. Pharmacologic inhibition of NO synthase (NOS) through the nonselective and brain-selective inhibitors, N-nitro-L-arginine methyl ester (l-NAME) and 7-nitroindazole (7-NI), was performed. Results: The treatments caused the appearance of an interictal discharge act…

Malemedicine.medical_specialtyHippocampusGlutamic AcidHippocampal formationNeurotransmissionBiologyNitric OxideHippocampusReceptors N-Methyl-D-Aspartatechemistry.chemical_compoundInternal medicinemedicineAnimalsRats WistarNeurotransmitterCerebral CortexEpilepsyGlutamate receptorSomatosensory CortexRatsEndocrinologyNeurologychemistryCNQXExcitatory postsynaptic potentialNMDA receptorNeurology (clinical)Epilepsia
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