Search results for "nuclear protein"

showing 10 items of 337 documents

Transcriptional regulation of nucleoredoxin-like genes takes place on a daily basis in the retina and pineal gland of rats.

2015

AbstractThe nucleoredoxin-like gene Nxnl1 (Txnl6) and its paralogue Nxnl2 encode the rod-derived cone viability factors (RdCVF and RdCVF2), which increase the resistance to photooxidative damage and have therapeutic potential for the survival of cones in retinitis pigmentosa. In this study, the transcription of Nxnl genes was investigated as a function of the day/night cycle in rats. The transcript levels of Nxnl1 and Nxnl2 were seen to display daily rhythms with steadily increasing values during the light phase and peak expression around dark onset in preparations of whole retina, photoreceptor cells and—but only in regard to Nxnl1—in photoreceptor-related pinealocytes. The cycling of Nxnl…

Malemedicine.medical_specialtyPhysiologyCircadian clockLaser Capture MicrodissectionBiologyPineal GlandRetinaPinealocyteRats Sprague-DawleyPineal glandInternal medicineRetinitis pigmentosamedicineAnimalsPhotoreceptor CellsCircadian rhythmRNA MessengerRegulation of gene expressionHomeodomain ProteinsRetinaGenes HomeoboxNuclear ProteinsDarknessmedicine.diseaseSensory SystemsCircadian RhythmRatsEndocrinologymedicine.anatomical_structureGene Expression RegulationDarknessFemalesense organsOxidoreductasesVisual neuroscience
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Mild phenotypes in a series of patients with Opitz GBBB syndrome with MID1 mutations

2004

Contains fulltext : 48815.pdf (Publisher’s version ) (Closed access) Opitz syndrome (OS; MIM 145410 and MIM 300000) is a congenital midline malformation syndrome characterized by hypertelorism, hypospadias, cleft lip/palate, laryngotracheoesophageal (LTE) abnormalities, imperforate anus, developmental delay, and cardiac defects. The X-linked form (XLOS) is caused by mutations in the MID1 gene, which encodes a microtubule-associated RBCC protein. In this study, phenotypic manifestations of patients with and without MID1 mutations were compared to determine genotype-phenotype correlations. We detected 10 novel mutations, 5 in familial cases, 2 in sporadic cases, and 3 in families for whom it …

Malemedicine.medical_specialtyUbiquitin-Protein LigasesBiologymedicine.disease_causeGastroenterologyG/BBB SYNDROMEFAMILIESGenomic disorders and inherited multi-system disorders [IGMD 3]Genotype-phenotype distinctionInternal medicineGeneticsmedicineHumansHypertelorismGeneGenetics (clinical)GeneticsFamily HealthX-linked Opitz syndromeMutationMID1Nuclear ProteinsGenetic Diseases X-LinkedExonsOpitz G/BBB Syndromemedicine.diseasePhenotypeGENEPedigreeSmith-Lemli-Opitz SyndromePhenotypeGenetic defects of metabolism [UMCN 5.1]HypospadiasMutationMicrotubule ProteinsFemalephenotypic variabilityXP22medicine.symptomImperforate anusFunctional Neurogenomics [DCN 2]BBBTranscription FactorsAmerican Journal of Medical Genetics. Part A
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Expanded CTG repeats trigger miRNA alterations in Drosophila that are conserved in myotonic dystrophy type 1 patients

2013

Myotonic dystrophy type 1 (DM1) is caused by the expansion of CTG repeats in the 3' untranslated region of the DMPK gene. Several missplicing events and transcriptional alterations have been described in DM1 patients. A large number of these defects have been reproduced in animal models expressing CTG repeats alone. Recent studies have also reported miRNA dysregulation in DM1 patients. In this work, a Drosophila model was used to investigate miRNA transcriptome alterations in the muscle, specifically triggered by CTG expansions. Twenty miRNAs were differentially expressed in CTG-expressing flies. Of these, 19 were down-regulated, whereas 1 was up-regulated. This trend was confirmed for thos…

Malemusculoskeletal diseasescongenital hereditary and neonatal diseases and abnormalitiesDown-RegulationGene ExpressionBiologyMyotonic dystrophyLife ExpectancyGeneticsmedicineAnimalsDrosophila ProteinsHumansMyotonic DystrophyMuscle SkeletalMolecular BiologyCells CulturedGenetics (clinical)Oligonucleotide Array Sequence AnalysisGeneticsBase SequenceLife spanNuclear ProteinsGeneral Medicinemedicine.diseaseMicroRNAsDrosophila melanogasterGene Expression RegulationFemaleTranscriptomeTrinucleotide Repeat Expansion
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Effects of triiodothyronine (T3) on differentiation of rat cortical neurons in primary cultures.

1991

Some of the events which characterize neuronal terminal differentiation have been studied in rat cortical neurons cultured in a selective synthetic medium for a period which corresponds to terminal brain maturation in vivo. In particular, we have studied the effect of T3 on the synthesis of nuclear proteins and the expression of the mRNAs which encode different variants of T3 nuclear receptors (c erb A proteins). We have shown that: a) T3 stimulates the turnover of nuclear proteins, with a more evident effect on the non-histone component; b) for the whole lifespan of cultures the predominant form of c erb Aα mRNA is the α2 variant (which encodes a protein unable to bind T3); whatever the fu…

Messenger RNATriiodothyroninePeriod (gene)Clinical BiochemistryBiomedical EngineeringBioengineeringCell BiologyCortical neuronsBiologyMolecular biologyNuclear receptorIn vivoNuclear proteinFunction (biology)BiotechnologyCytotechnology
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Conserved chromosomal clustering of genes governed by chromatin regulators in Drosophila

2008

Transcriptional analysis of chromatin regulator mutants in Drosophila melanogaster identified clusters of functionally related genes conserved in other insect species.

Model organismsanimal structuresTranscription GeneticEvolutionChromosomal Proteins Non-HistoneDrosòfila melanogasterGenome studiesDevelopmentBiologyNon-histone proteinAnimalsDrosophila ProteinsDrosòfila -- GenèticaTranscription factorGeneGeneticsMicroarray analysis techniquesResearchGene Expression ProfilingMutació (Biologia)fungiNuclear Proteinsbiology.organism_classificationChromatin Assembly and DisassemblyChromatinHistoneDrosophila melanogasterDrosofila melanogasterGene Expression RegulationMultigene Familybiology.proteinDrosophila melanogasterDrosophila ProteinGenètica del desenvolupamentTranscription FactorsGenome Biology
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HEXIM1 Diffusion in the Nucleus Is Regulated by Its Interactions with Both 7SK and P-TEFb

2019

International audience; How nuclear proteins diffuse and find their targets remains a key question in the transcription field. Dynamic proteins in the nucleus are classically subdiffusive and undergo anomalous diffusion, yet the underlying physical mechanisms are still debated. In this study, we explore the contribution of interactions to the generation of anomalous diffusion by the means of fluorescence spectroscopy and simulation. Using interaction-deficient mutants, our study indicates that HEXIM1 interactions with both 7SK RNA and positive transcription elongation factor b are critical for HEXIM1 subdiffusion and thus provides evidence of the effects of protein-RNA interaction on molecu…

Models MolecularAnomalous diffusion[SDV]Life Sciences [q-bio]PopulationBiophysicsPlasma protein bindingDiffusion03 medical and health sciences0302 clinical medicineCell Line Tumor7SK RNAmedicineHumansComputer SimulationPositive Transcriptional Elongation Factor BNuclear proteinP-TEFbeducationComputingMilieux_MISCELLANEOUS030304 developmental biologyCell Nucleus0303 health sciencesMolecular diffusioneducation.field_of_studyChemistryRNA-Binding ProteinsArticles[SDV] Life Sciences [q-bio][SDV.BBM.BP]Life Sciences [q-bio]/Biochemistry Molecular Biology/BiophysicsSpectrometry Fluorescencemedicine.anatomical_structureBiophysicsRNA Long NoncodingNucleus030217 neurology & neurosurgeryProtein BindingTranscription Factors
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Nocturnin in the demosponge Suberites domuncula: a potential circadian clock protein controlling glycogenin synthesis in sponges

2012

Sponges are filter feeders that consume a large amount of energy to allow a controlled filtration of water through their aquiferous canal systems. It has been shown that primmorphs, three-dimensional cell aggregates prepared from the demosponge Suberites domuncula and cultured in vitro , change their morphology depending on the light supply. Upon exposure to light, primmorphs show a faster and stronger increase in DNA, protein and glycogen content compared with primmorphs that remain in the dark. The sponge genome contains nocturnin, a light/dark-controlled clock gene, the protein of which shares a high sequence similarity with the related molecule of higher metazoans. The sponge nocturnin …

Models MolecularAryl hydrocarbon receptor nuclear translocatorGlycogeninPeriod (gene)Circadian clockGene ExpressionBiochemistry03 medical and health sciencesCryptochromeComplementary DNAAnimalsRNA Messenger14. Life underwaterMolecular BiologyDNA PrimersGlycoproteins030304 developmental biology0303 health sciencesBase SequencebiologyCircadian Rhythm Signaling Peptides and Proteins030302 biochemistry & molecular biologyNuclear ProteinsCell Biologybiology.organism_classificationCircadian RhythmSuberites domunculaCLOCKBiochemistryGlucosyltransferasesSuberitesTranscription FactorsBiochem. J.
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Homology modeling using simulated annealing of restrained molecular dynamics and conformational search calculations with CONGEN: application in predi…

1997

We have developed an automatic approach for homology modeling using restrained molecular dynamics and simulated annealing procedures, together with conformational search algorithms available in the molecular mechanics program CONGEN (Bruccoleri RE, Karplus M, 1987, Biopolymers 26:137-168). The accuracy of the method is validated by "predicting" structures of two homeodomain proteins with known three-dimensional structures, and then applied to predict the three-dimensional structure of the homeodomain of the murine Msx-1 transcription factor. Regions of the unknown protein structure that are highly homologous to the known template structure are constrained by "homology distance constraints,"…

Models MolecularSaccharomyces cerevisiae ProteinsProtein ConformationMSX1 Transcription FactorMolecular Sequence DataSaccharomyces cerevisiaeBiologyProtein EngineeringBiochemistryProtein Structure SecondaryMolecular dynamicsMiceProtein structureAnimalsComputer SimulationHomology modelingAmino Acid SequenceMolecular BiologyHomeodomain ProteinsMSX1 Transcription FactorSequence Homology Amino AcidNuclear ProteinsProtein engineeringProtein superfamilyengrailedRepressor ProteinsCrystallographyAntennapedia Homeodomain ProteinThreading (protein sequence)AlgorithmsInformation SystemsTranscription FactorsResearch ArticleProtein science : a publication of the Protein Society
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Specific expression patterns of epithelial to mesenchymal transition factors in gestational molar disease.

2015

Introduction The epithelial to mesenchymal transition, a well-known and re-emerging model in pathology, has not been completely investigated in the field of gestational pathology. This study aims at improving the comprehension of this process in molar disease, even looking for new possible immunohistochemical markers. Materials and methods We have analysed the immunohistochemical expression of Twist1 and Snai2, two of the most important transcription factors involved in epithelial to mesenchymal transition, in formalin-fixed paraffin-embedded samples of 23 spontaneous abortive pregnancies, 22 molar pregnancies (10 partial and 12 complete) and 7 term placentas. Results Twist1 and Snai2 were …

MolarPathologymedicine.medical_specialtyStromal cellEpithelial-Mesenchymal TransitionBiologyPregnancymedicineHumansEpithelial–mesenchymal transitionTwistClaudinComplete mole; EMT; Molar disease; Snai2; Twist; Twist1; Obstetrics and Gynecology; Reproductive Medicine; Developmental BiologyCadherinTwist-Related Protein 1EMTObstetrics and GynecologyTrophoblastNuclear ProteinsHydatidiform MoleImmunohistochemistryComplete mole; EMT; Molar disease; Snai2; Twist; Twist1Complete moleSNAI2Molar diseasemedicine.anatomical_structureReproductive MedicineSnai2Case-Control StudiesImmunohistochemistryFemaleSnail Family Transcription FactorsBiomarkersTwist1Developmental BiologyTranscription FactorsPlacenta
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In murine 3T3 fibroblasts, different second messenger pathways resulting in the induction of NO synthase II (iNOS) converge in the activation of tran…

1996

Transcription factor NF-kappaB is essential for the induction of nitric oxide synthase (NOS) II (iNOS) by bacterial lipopolysaccharide in murine macrophages (Xie, Q. W., Kashiwabara, Y., and Nathan, C. (1994) J. Biol. Chem. 269, 4705-4708). In 3T3 fibroblasts, agents other than cytokines are efficacious inducers of NOS II expression. In addition to cytokines such as interferon-gamma or tumor necrosis factor-alpha, protein kinase C-stimulating agents such as tetradecanoylphorbol-13-acetate, or cyclic AMP-elevating agents such as forskolin and 8-bromo-cAMP markedly increased NOS II mRNA (measured by Sl nuclease and RNase protection analyses), NOS II protein (determined by Western blotting), a…

Molecular Sequence DataBiochemistrySecond Messenger SystemsDexamethasoneGene Expression Regulation Enzymologicchemistry.chemical_compoundMicePyrrolidine dithiocarbamateTransforming Growth Factor betaAnimalsHumansAmino Acid SequenceRNA MessengerNuclear proteinProtein kinase AMolecular BiologyTranscription factorProtein Kinase CDNA PrimersForskolinbiologyBase SequenceNF-kappa BReceptor Protein-Tyrosine KinasesCell Biology3T3 CellsMolecular biologyCyclic AMP-Dependent Protein KinasesActinsNitric oxide synthasechemistryEnzyme InductionSecond messenger systembiology.proteinTumor necrosis factor alphaNitric Oxide SynthaseThe Journal of biological chemistry
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