Search results for "opioid receptor"

showing 10 items of 46 documents

Regional differences in mu-opioid receptor-dependent modulation of basal dopamine transmission in rat striatum

2016

Abstract The nigrostriatal dopamine system is implicated in the regulation of reward and motor activity. Dopamine (DA) release in dorsal striatum (DS) is controlled by the firing rate of DA neurons in substantia nigra pars compacta. However, influences at terminal level, such as those involving activation of mu opioid receptors (MORs), can play a key role in determining DA levels in striatum. Nonetheless, published data also suggest that the effect of opioid drugs on DA levels may differ depending on the DS subregion analyzed. In this study, in vivo microdialysis in rats was used to explore this regional dependence. Changes in basal DA levels induced by local retrodialysis application of DA…

Male0301 basic medicineAgonistmedicine.medical_specialtymedicine.drug_classDopamineMicrodialysisReceptors Opioid muSubstantia nigraStriatum03 medical and health scienceschemistry.chemical_compound0302 clinical medicineDopamineInternal medicinemedicineAnimalsRats WistarPars compactaGeneral NeuroscienceVentral striatumEnkephalin Ala(2)-MePhe(4)-Gly(5)-Corpus StriatumDAMGO030104 developmental biologyEndocrinologymedicine.anatomical_structurenervous systemchemistryμ-opioid receptorNeuroscience030217 neurology & neurosurgerymedicine.drugNeuroscience Letters
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Shell/core differences in mu- and delta-opioid receptor modulation of dopamine efflux in nucleus accumbens

2008

The mu- and delta-opioid receptors located at the terminal level in nucleus accumbens are involved in the opiate modulation of dopamine release in this brain area. However, recent studies suggest that the effects of opioid drugs on the core subregion of nucleus accumbens may completely differ from those observed in the shell. We used in vivo microdialysis to simultaneously apply selective mu- and delta-opioid receptor agonists and to measure extracellular levels of dopamine in three subregions of the accumbens, namely shell, core, and the transition zone between them. The regional analysis of these subregions of the accumbens demonstrated that basal levels of dopamine and its metabolites we…

MaleAgonistTime FactorsEnkephalinmedicine.drug_classDopamineMicrodialysisReceptors Opioid muPharmacologyNucleus accumbensNucleus Accumbensδ-opioid receptorCellular and Molecular Neurosciencechemistry.chemical_compoundDopamine receptor D1DopamineReceptors Opioid deltamedicineAnimalsRats WistarPharmacologyDopaminergicHomovanillic AcidEnkephalin Ala(2)-MePhe(4)-Gly(5)-RatsAnalgesics OpioidDAMGOchemistry34-Dihydroxyphenylacetic AcidEnkephalin D-Penicillamine (25)-medicine.drugNeuropharmacology
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A runner’s high depends on cannabinoid receptors in mice

2015

Exercise is rewarding, and long-distance runners have described a runner's high as a sudden pleasant feeling of euphoria, anxiolysis, sedation, and analgesia. A popular belief has been that endogenous endorphins mediate these beneficial effects. However, running exercise increases blood levels of both β-endorphin (an opioid) and anandamide (an endocannabinoid). Using a combination of pharmacologic, molecular genetic, and behavioral studies in mice, we demonstrate that cannabinoid receptors mediate acute anxiolysis and analgesia after running. We show that anxiolysis depends on intact cannabinoid receptor 1 (CB1) receptors on forebrain GABAergic neurons and pain reduction on activation of pe…

MaleCannabinoid receptormedicine.drug_classmedicine.medical_treatmentPharmacologyDepolarization-induced suppression of inhibitionRunningReceptor Cannabinoid CB2MiceReceptor Cannabinoid CB1Opioid receptorPhysical Conditioning AnimalmedicineCannabinoid receptor type 2AnimalsEndorphinsMultidisciplinaryBehavior AnimalBiological SciencesEndocannabinoid systemMice Inbred C57BLnervous systemOpioidlipids (amino acids peptides and proteins)CannabinoidPsychologyhuman activitiespsychological phenomena and processesmedicine.drug
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Improved effect of the combination naltrexone/D-penicillamine in the prevention of alcohol relapse-like drinking in rats

2014

Opioid antagonists are licensed drugs for treating alcohol use disorders; nonetheless, clinical studies have evidenced their limited effectiveness. Preclinical findings indicate that opioid receptor (OR) antagonists, such as naltrexone (NTX), reduce the alcohol deprivation effect (ADE). However, a detailed analysis of published data shows the existence of a delayed increase in ethanol consumption after continuous OR blockade, a phenomenon originally called as ‘delayed ADE’. We have recently reported that D-penicillamine (DP) is able to prevent ADE through a mechanism dependent on the inactivation of acetaldehyde, the main metabolite of ethanol. Hypothetically, OR activation would be trigge…

MaleCombination therapyAlcohol Drinkingmedicine.drug_classInjections SubcutaneousNarcotic AntagonistsPharmacologyInfusions SubcutaneousNaltrexoneethanol relapse preventionchemistry.chemical_compoundOpioid receptormedicineSecondary PreventionAnimalsPharmacology (medical)PharmacologyEthanolbusiness.industryPenicillaminePenicillamineD-penicillamineAcetaldehydeNaltrexoneRatsPsychiatry and Mental healthOpioidchemistrymu-opioid receptorDrug Therapy Combinationμ-opioid receptorbusinessnaltrexonehuman activitiesmedicine.drugAlcohol Deterrentsacetaldehyde
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Dose-dependent induction of CPP or CPA by intra-pVTA ethanol: Role of mu opioid receptors and effects on NMDA receptors.

2020

AbstractThe neurobiological mechanisms underlying alcohol motivational properties are still not fully understood, however, the mu-opioid receptors (MORs) have been evidenced as central elements in the manifestation of the alcohol reinforcing properties. Drug-associated environmental stimuli can trigger alcohol relapse and promote alcohol consumption whereby N-methyl-D-aspartate (NMDA) receptors play a pivotal role. Here we sought to demonstrate, for the first time, that ethanol induces conditioned place preference or aversion (CPP or CPA) when administered locally into the ventral tegmental area (VTA) and the associated role of MORs. We further analyzed the changes in the expression and mRN…

MaleMicroinjectionsReceptors Opioid muHippocampusNucleus accumbensPharmacologyReceptors N-Methyl-D-AspartateArticle03 medical and health scienceschemistry.chemical_compound0302 clinical medicineConditioning PsychologicalmedicineAvoidance LearningAnimalsRats WistarReceptorBiological Psychiatry030304 developmental biologyPharmacology0303 health sciencesEthanolDose-Response Relationship DrugEthanolChemistryVentral Tegmental AreaConditioned place preference030227 psychiatryRatsVentral tegmental areamedicine.anatomical_structureInfusions Intraventricularnervous systemNMDA receptorμ-opioid receptor030217 neurology & neurosurgeryProgress in neuro-psychopharmacologybiological psychiatry
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GHB differentially affects morphine actions on motor activity and social behaviours in male mice

2003

There are several reports suggesting that gamma-hydroxybutyric acid (GHB) influences the endogenous opioid system. The present study aimed to investigate the effects of GHB on motor and social activities and to examine its influence on morphine's actions on these behaviours. In a first experiment, several doses of GHB were studied but only the highest (200 and 400 mg/kg) produced a decrease in spontaneous motor activity measured in an actimeter cage. When hyperactivity induced by injecting 50 mg/kg of morphine was evaluated, all the GHB doses efficiently counteracted this morphine action. Using the paradigm of isolation-induced aggression, administration of 200 mg/kg of GHB significantly de…

MaleNarcoticsmedicine.drug_classClinical BiochemistryMale miceMotor ActivityPharmacologyToxicologyBiochemistryMiceBehavioral NeuroscienceOpioid receptormedicineAnimalsDrug InteractionsMotor activitySocial BehaviorBiological PsychiatryEndogenous opioidPharmacologyMorphineAggressionBiological activityAggressionOpioidExploratory BehaviorMorphinemedicine.symptomSodium OxybatePsychologyAnesthetics Intravenousmedicine.drugPharmacology Biochemistry and Behavior
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Catalase-independent early-gene expression in rat brain following acute ethanol exposure

2004

Early-gene expression evoked by acute ethanol treatment was studied in rat brain by quantitative immunocytochemistry, with reference to ethanol metabolism by the enzyme catalase. Colocalization with mu-opioid receptor (MOR) sites was also examined. Ethanol challenges [1, 2.5, and 4 g/kg intraperitoneally (i.p.)] evoked dose-dependent increases in c-Fos expression in several brain regions, but overlap with MOR-rich sites was only partial. Strong inhibition of brain catalase activity (ca. 60%) with 3-amino-1,2,4-triazole (AT, 1 g/kg i.p.) did not alter ethanol-induced c-Fos nor Krox-24 expression in any of the brain regions analyzed. This evidence demonstrates that catalase-mediated metabolis…

MaleNervous systemmedicine.medical_specialtyCentral nervous systemReceptors Opioid muGene ExpressionCell Countc-FosRats Sprague-DawleyInternal medicinemedicineAnimalsEnzyme InhibitorsEthanol metabolismMolecular BiologyAmitroleBrain ChemistryEthanolbiologyGeneral NeuroscienceBrainCentral Nervous System DepressantsColocalizationCatalaseImmunohistochemistryRatsmedicine.anatomical_structureEndocrinologyCatalasebiology.proteinNeurology (clinical)μ-opioid receptorProto-Oncogene Proteins c-fosImmediate early geneDevelopmental BiologyBrain Research
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Inflammatory Pain Promotes Increased Opioid Self-Administration: Role of Dysregulated Ventral Tegmental Area μ Opioid Receptors

2015

Pain management in opioid abusers engenders ethical and practical difficulties for clinicians, often resulting in pain mismanagement. Although chronic opioid administration may alter pain states, the presence of pain itself may alter the propensity to self-administer opioids, and previous history of drug abuse comorbid with chronic pain promotes higher rates of opioid misuse. Here, we tested the hypothesis that inflammatory pain leads to increased heroin self-administration resulting from altered mu opioid receptor (MOR) regulation of mesolimbic dopamine (DA) transmission. To this end, the complete Freund's adjuvant (CFA) model of inflammation was used to assess the neurochemical and functi…

MalePain ThresholdSucroseReceptors Opioid muAction PotentialsPainMesolimbic pathwayPharmacologyHeroinRats Sprague-DawleyQuinoxalinesThreshold of painmental disordersmedicineAnimalsInflammationNeuronsGeneral NeuroscienceVentral Tegmental AreaChronic painGlycine AgentsArticlesStrychnineEnkephalin Ala(2)-MePhe(4)-Gly(5)-medicine.diseaseRatsVentral tegmental areaAnalgesics OpioidHeroinDisease Models Animalmedicine.anatomical_structureOpioidInhibitory Postsynaptic PotentialsHyperalgesiaHyperalgesiaConditioning Operantμ-opioid receptormedicine.symptomPsychologyExcitatory Amino Acid Antagonistsmedicine.drug
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The rewarding effects of ethanol are modulated by binge eating of a high-fat diet during adolescence

2017

Abstract Binge-eating is considered a specific form of overeating characterized by intermittent and high caloric food intake in a short period of time. Epidemiologic studies support a positive relation between the ingestion of fat and ethanol (EtOH), specifically among adolescent subjects. The aim of this work was to clarify the role of the compulsive, limited and intermittent intake of a high-fat food during adolescence on the rewarding effects of EtOH. After binge-eating for 2 h, three days a week from postnatal day (PND) 29, the reinforcing effects of EtOH were tested with EtOH self-administration (SA), conditioned place preference (CPP) and ethanol locomotor sensitization procedures in …

Maleendocrine systemmedicine.medical_specialtyTime FactorsSelf AdministrationNucleus accumbensDiet High-FatMice03 medical and health sciencesCellular and Molecular Neuroscience0302 clinical medicineRewardInternal medicinemental disordersmedicineAnimalsIngestionBulimiaOvereatingreproductive and urinary physiologyPharmacologyEthanolBinge eatingDrug Administration RoutesCentral Nervous System DepressantsConditioned place preference030227 psychiatryVentral tegmental areaDisease Models Animalmedicine.anatomical_structureEndocrinologyAnimals NewbornAnesthesiaConditioning Operantmedicine.symptomμ-opioid receptorSelf-administrationPsychologyLocomotion030217 neurology & neurosurgeryNeuropharmacology
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Kappa opioid receptor blockade in the nucleus accumbens shell prevents sex-dependent alcohol deprivation effect induced by inflammatory pain.

2021

ABSTRACT Pain-induced negative affect reduces life quality of patients by increasing psychiatric comorbidities, including alcohol use disorders (AUDs). Indeed, clinical data suggest pain as a risk factor to suffer AUDs, predicting relapse drinking in abstinent patients. Here, we analyse the impact of pain on alcohol relapse and the role of kappa opioid receptor (KOR) activation in mediating these pain-induced effects because KORs play an important role in pain-driven negative affect and AUD. Female and male Sprague-Dawley rats underwent 2 alcohol intermittent access periods separated by a forced abstinence period. The complete Freund adjuvant model of inflammatory pain was introduced during…

Malemedicine.medical_specialtymedia_common.quotation_subjectPainAlcoholNucleus accumbensκ-opioid receptorNucleus AccumbensRats Sprague-Dawleychemistry.chemical_compoundInternal medicinemedicineAnimalsHumansRisk factormedia_commonbusiness.industryReceptors Opioid kappaAntagonistAbstinenceBlockadeRatsAlcoholismAnesthesiology and Pain MedicineEndocrinologyNeurologychemistryFemaleNeurology (clinical)Norbinaltorphiminebusinessmedicine.drugPain
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