Search results for "optic nerve"

showing 10 items of 108 documents

Betulinic Acid Protects from Ischemia-Reperfusion Injury in the Mouse Retina

2021

Ischemia/reperfusion (I/R) events are involved in the pathophysiology of numerous ocular diseases. The purpose of this study was to test the hypothesis that betulinic acid protects from I/R injury in the mouse retina. Ocular ischemia was induced in mice by increasing intraocular pressure (IOP) to 110 mm Hg for 45 min, while the fellow eye served as a control. One group of mice received betulinic acid (50 mg/kg/day p.o. once daily) and the other group received the vehicle solution only. Eight days after the I/R event, the animals were killed and the retinal wholemounts and optic nerve cross-sections were prepared and stained with cresyl blue or toluidine blue, respectively, to count cells in…

Malemedicine.medical_specialtyretinagenetic structuresQH301-705.5ischemia-reperfusion injuryarteriolesVideo microscopyProtective AgentsArticlechemistry.chemical_compoundMicebetulinic acidInternal medicineBetulinic acidmedicineAnimalsBiology (General)AxonGanglion cell layerreactive oxygen speciesRetinaAnti-Inflammatory Agents Non-SteroidalRetinalGeneral Medicinemedicine.diseaseeye diseasesMice Inbred C57BLmedicine.anatomical_structureEndocrinologychemistryReperfusion InjuryOptic nervesense organsPentacyclic TriterpenesReperfusion injuryCells
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Tocilizumab as first treatment option in optic neuropathy secondary to Graves' orbitopathy.

2018

Dysthyroid optic neuropathy (DON) is one of the complications that can affect Graves' orbitopathy (GO) patients. Its prevalence is estimated at less than 5%. It is usually treated with intravenous steroids, radiotherapy or orbital decompression. Tocilizumab has been proposed as a treatment option in cases of GO refractory to steroid treatment, with good clinical results. Our aim is to report the case of a patient with optic neuropathy secondary to GO treated with tocilizumab as primary treatment option.

Malemusculoskeletal diseasesDecompressionmedicine.medical_specialtyDecompressionmedicine.medical_treatmentVisual Acuity030209 endocrinology & metabolismdysthyroid optic neuropathyAntibodies Monoclonal HumanizedOptic neuropathy03 medical and health scienceschemistry.chemical_compoundGraves' orbitopathytocilizumab0302 clinical medicineTocilizumabRefractoryOptic Nerve DiseasesmedicineHumansbusiness.industryTreatment optionsMiddle Agedmedicine.diseaseeye diseasesSurgeryRadiation therapyGraves OphthalmopathyOphthalmologychemistry030221 ophthalmology & optometryPrimary treatmentVisual FieldsIntravenous steroidsbusinessTomography Optical Coherencesteroids
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Central retinal artery occlusion-A new, provisional treatment approach.

2019

The retinal ganglion cells infarcted in central retinal artery occlusion (CRAO) are the somata of the optic nerve axons, part of the central nervous system. Consequently, CRAO with inner retinal infarction is a small vessel stroke, usually with the devastating consequence of severe visual loss in the affected eye. At present, there is no generally accepted, evidence-based therapy of nonarteritic CRAO in contrast to ischemic cerebral stroke that has well-accepted treatment protocols. Widely divergent and controversial therapeutic options for CRAO reflect the desperation of treating physicians and disparate conflicting studies. We examine reasons why treatment of nonarteritic CRAO remains pro…

MassageRetinal Ganglion Cellsmedicine.medical_specialtybusiness.industryRetinal Artery OcclusionAnticoagulantsCherry-red spotCerebral strokemedicine.diseaseRetinal ganglionOphthalmologyFibrinolytic AgentsOphthalmologyOptic nerveMedicineCentral retinal artery occlusionHumansSmall vesselmedicine.symptombusinessStrokeAntihypertensive AgentsIntraocular PressureRetinal infarctionSurvey of ophthalmology
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Drug connectivity mapping and functional analysis reveal therapeutic small molecules that differentially modulate myelination

2022

Disruption or loss of oligodendrocytes (OLs) and myelin has devastating effects on CNS function and integrity, which occur in diverse neurological disorders, including Multiple Sclerosis (MS), Alzheimer’s disease and neuropsychiatric disorders. Hence, there is a need to develop new therapies that promote oligodendrocyte regeneration and myelin repair. A promising approach is drug repurposing, but most agents have potentially contrasting biological actions depending on the cellular context and their dose-dependent effects on intracellular pathways. Here, we have used a combined systems biology and neurobiological approach to identify compounds that exert positive and negative effects on olig…

MyelinMiceMyelin SheathNSC Neural stem cellSystems BiologyOPC Oligodendrocyte progenitor cellHigh-Throughput Nucleotide SequencingLINCS The Library of Integrated Network-based Cellular SignaturesCell DifferentiationGeneral MedicineCNS Central Nervous SystemOligodendrogliamedicine.anatomical_structureOligodendrogenesisNFOL Newly formed oligodendrocyteOL OligodendrocyteSignal TransductionSubventricular zoneOptic nerveIn silicoSystems biologyMorpholinesSVZ subventricular zoneContext (language use)RM1-950BiologyArticlemedicinePharmacogenomics The Library of Integrated Network-Based Cellular Signatures/LINCSAnimalsH-LY29 High concentration of LY294002Computer SimulationPI3K/AKT/mTOR pathwayL-LY29 Low concentration of LY294002PharmacologyPI3K/AktTCN TriciribineDose-Response Relationship DrugRegeneration (biology)Multiple sclerosismedicine.diseaseOligodendrocyteOligodendrocyteiNSCs iPSC-derived NSCsTAPs Transiently amplifying progenitorsMice Inbred C57BLMS Multiple SclerosisiPCS induced Pluripotent Stem CellChromonesPharmacogeneticsTherapeutics. PharmacologyMOL Myelinating oligodendrocyteNeuroscienceBiomedicine & Pharmacotherapy
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The role of glia, mitochondria, and the immune system in glaucoma.

2009

Author(s): Tezel, Gulgun; Fourth ARVO/Pfizer Ophthalmics Research Institute Conference Working Group

Nerve degenerationRetinal Ganglion Cellsmedicine.medical_specialtyMitochondrial Diseasesbusiness.industryGlaucomaGlaucomaMitochondrionmedicine.diseaseAxonsMitochondriaImmune systemOphthalmologyImmune SystemOptic Nerve DiseasesmedicineHumansbusinessOptic nerve diseasesNeurogliaInvestigative ophthalmologyvisual science
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Comprehensive Anatomic Assessment of Ipsilateral Pterional Versus Contralateral Subfrontal Approaches to the Internal Carotid Ophthalmic Segment: A C…

2019

Objective Medially pointing aneurysms of the ophthalmic segment of the internal carotid artery (oICA) represent a neurosurgical challenge. Conventional ipsilateral approaches require internal carotid artery and optic nerve (ON) mobilization as well as anterior clinoidectomy (AC), all associated with increased surgical risk. Contralateral approaches could provide a better exposure of the superomedial aspect of the oICA, ophthalmic artery, and superior hypophyseal artery, sparing AC and internal carotid artery or ON mobilization. However, the microsurgical anatomy of this approach has not been systematically studied. In the present work, we exhaustibly analyzed the anatomic and morphometric c…

Olfactory NerveSuperior Hypophyseal ArteryDissection (medical)Neurosurgical ProceduresOphthalmic Artery03 medical and health sciencesImaging Three-Dimensional0302 clinical medicinemedicine.arteryCadavermedicineHumansbusiness.industryVirtual RealityIntracranial AneurysmOptic NerveOrgan Sizemedicine.diseaseThree dimensional simulationFrontal lobe030220 oncology & carcinogenesisOphthalmic arteryOptic nerveSurgeryNeurology (clinical)Internal carotid arteryCadaveric spasmNuclear medicinebusinessCarotid Artery Internal030217 neurology & neurosurgeryWorld Neurosurgery
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Peripapillary Staphyloma and Asymmetric Morning Glory Anomaly

2006

We report a patient with concomitant occurrence of peripapillary staphyloma and morning glory anomaly. The morning glory anomaly presented with an optic nerve head with oblique entrance, located on one side of the peripapillary staphyloma, giving an unusually asymmetric and previously undescribed appearance to the abnormality. The optical coherence tomography provided useful information about the shape and deepness of the peripapillary staphyloma and disclosed a retinal detachment not appreciated ophthalmoscopically.

Ophthalmologybusiness.industryMorning glory anomalyOptic nervemedicineStaphylomaRetinal detachmentNeurology (clinical)Anatomymedicine.diseasebusinessNeuro-Ophthalmology
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Angioma retiniano como síndrome mascarada de tumor del nervio óptico en la enfermedad de Von Hippel-Lindau

2006

espanolCaso clinico: Varon de 35 anos con antecedentes familiares de enfermedad de von Hippel-Lindau (VHL) diagnosticado de dos hemangiomas retinianos en ojo derecho (OD) y uno en ojo izquierdo. Las lesiones se trataron con crioterapia en OD y laserterapia en OI. A pesar de una aparente buena evolucion de las lesiones retinianas hubo una progresiva disminucion de la AV. Se realizo RM donde se evidencio lesiones tumorales del nervio optico (NO) bilaterales compatibles con glioma, meningioma o hemangioblastoma. Actualmente: AV (OD): movimiento de manos y AV(OI): 0,6. Discusion: La presencia de los hemangiomas retinianos en este paciente hizo retrasar el diagnostico de un tumor en el NO, infre…

Optic nerve tumorhemangioblastoma del nervio ópticoVisual acuitybusiness.industrymedicine.diseaseeye diseasesOphthalmologyLeft eyeHemangioblastomaEnfermedad de von Hippel-LindauOptic nervemedicinesense organsmedicine.symptomNuclear medicinebusinessneoplasmstumor del nervio ópticohemangioma retiniano
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Cutting edge: Multiple sclerosis-like lesions induced by effector CD8 T cells recognizing a sequestered antigen on oligodendrocytes.

2008

Abstract CD8 T cells are emerging as important players in multiple sclerosis (MS) pathogenesis, although their direct contribution to tissue damage is still debated. To assess whether autoreactive CD8 T cells can contribute to the pronounced loss of oligodendrocytes observed in MS plaques, we generated mice in which the model Ag influenza hemagglutinin is selectively expressed in oligodendrocytes. Transfer of preactivated hemagglutinin-specific CD8 T cells led to inflammatory lesions in the optic nerve, spinal cord, and brain. These lesions, associating CD8 T cell infiltration with focal loss of oligodendrocytes, demyelination, and microglia activation, were very reminiscent of active MS le…

Pathologymedicine.medical_specialtyMultiple SclerosisImmunologyMice TransgenicBiologyCD8-Positive T-LymphocytesPathogenesisMiceAntigenmedicineImmunology and AllergyCytotoxic T cellAnimalsAntigensMyelin SheathMicrogliaMultiple sclerosismedicine.diseaseOligodendrocyteCell biologyOligodendrogliamedicine.anatomical_structureHemagglutininsOptic nerveInfiltration (medical)Journal of immunology (Baltimore, Md. : 1950)
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The Neuroepithelium Disruption Could Generate Autoantibodies against AQP4 and Cause Neuromyelitis Optica and Hydrocephalus

2014

Neuromyelitis optica is an inflammatory disease characterized by neuritis and myelitis of the optic nerve. Its physiopathology is connected with the aquaporin-4 water channel, since antibodies against aquaporin-4 have been found in the cerebrospinal fluid and blood of neuromyelitis optica patients. The seropositivity for aquaporin-4 antibodies is used for the diagnosis of neuromyelitis optica or neuromyelitis optica spectrum disease. On the other hand, aquaporin-4 is expressed in astrocyte feet in the brain-blood barrier and subventricular zones of the brain ventricles. Aquaporin-4 expression is high in cerebrospinal fluid in hydrocephalus. Furthermore, neuroepithelial denudation precedes n…

Pathologymedicine.medical_specialtyNeuromyelitis opticabusiness.industryurogenital systemNeuritisMyelitisReview Articlemedicine.diseasePathophysiologyeye diseasesHydrocephalusNeuroepithelial cellCerebrospinal fluidmedicineOptic nervebusinessInternational Scholarly Research Notices
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