Search results for "pancrea"

showing 10 items of 814 documents

Blockade of the trans-sulfuration pathway in acute pancreatitis due to nitration of cystathionine β-synthase

2019

© 2019 Published by Elsevier B.V.

Male0301 basic medicineS-AdenosylmethionineHomocysteineClinical BiochemistryNitric Oxide Synthase Type IINitrosative stressBiochemistryMicechemistry.chemical_compound0302 clinical medicineEdemaMedicineAcute inflammationHomocysteinelcsh:QH301-705.5lcsh:R5-920biologyGlutathioneUp-Regulationmedicine.anatomical_structureAcute pancreatitismedicine.symptomPancreaslcsh:Medicine (General)CeruletideResearch Papermedicine.medical_specialtyCystathionine beta-Synthase03 medical and health sciencesCystathionineInternal medicineAnimalsCysteineCystathionine β-synthaseS-adenosylmethionineMethioninebusiness.industryOrganic ChemistryGlutathionemedicine.diseaseCystathionine beta synthaseDisease Models Animal030104 developmental biologyEndocrinologyPancreatitischemistrylcsh:Biology (General)biology.proteinbusiness030217 neurology & neurosurgeryCysteineRedox Biology
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Pancreatic Protein Tyrosine Phosphatase 1B Deficiency Exacerbates Acute Pancreatitis in Mice

2016

Acute pancreatitis (AP) is a common and devastating gastrointestinal disorder that causes significant morbidity. The disease starts as local inflammation in the pancreas that may progress to systemic inflammation and complications. Protein tyrosine phosphatase 1B (PTP1B) is implicated in inflammatory signaling, but its significance in AP remains unclear. To investigate whether PTP1B may have a role in AP, we used pancreas PTP1B knockout (panc-PTP1B KO) mice and determined the effects of pancreatic PTP1B deficiency on cerulein- and arginine-induced acute pancreatitis. We report that PTP1B protein expression was increased in the early phase of AP in mice and rats. In addition, histological an…

Male0301 basic medicineWistarSystemic inflammationMedical and Health SciencesOral and gastrointestinalMicePathology2.1 Biological and endogenous factorsAetiologyNon-Receptor Type 1CancerMice KnockoutProtein Tyrosine Phosphatase Non-Receptor Type 1Pancreatitis Acute NecrotizingReverse Transcriptase Polymerase Chain ReactionRegular Articlemedicine.anatomical_structureAcute NecrotizingGastrointestinal disorderAcute pancreatitisTumor necrosis factor alphamedicine.symptomPancreashormones hormone substitutes and hormone antagonistsmedicine.medical_specialtyKnockoutInflammationPathology and Forensic MedicineProinflammatory cytokinePancreatic Cancer03 medical and health sciencesRare DiseasesInternal medicinemedicineAnimalsRats WistarAnimalbusiness.industrymedicine.diseaseRatsDisease Models Animal030104 developmental biologyEndocrinologyPancreatitisDisease ModelsPancreatitisProtein Tyrosine PhosphataseDigestive DiseasesbusinessThe American Journal of Pathology
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Glucagon-like peptide-2 treatment improves glucose dysmetabolism in mice fed a high fat diet

2016

Previous studies suggested that endogenous glucagon-like peptide 2 (GLP-2) is dispensable for the regulation of glucose homeostasis under normal conditions, while it can play a beneficial role in obesity conditions. The purpose of the present study was to investigate whether chronic treatment with Gly2-GLP-2, a stable analogue of GLP-2, can have an impact on glycaemic and lipid control in mice fed a high-fat diet (HFD), an animal model of human obesity and insulin resistance. HFD mice were treated once a day with Gly2-GLP-2 for 4 weeks. Body weight, food intake, fasting glucose, intraperitoneal glucose tolerance, insulin-induced glucose clearance, glucose-stimulated insulin secretion, β-cel…

Male0301 basic medicinemedicine.medical_specialtyEndocrinology Diabetes and MetabolismDrug Evaluation PreclinicalMicrovesicular SteatosisCarbohydrate metabolismDiet High-FatSettore BIO/09 - FisiologiaRandom Allocation03 medical and health sciencesEndocrinologyInsulin resistanceInternal medicineDiabetes mellitusGlucagon-Like Peptide 2medicineAnimalsGlucose homeostasisObesityPancreasPancreatic islets.Glucose Metabolism Disordersbusiness.industrydigestive oral and skin physiologyInsulin resistancemedicine.diseaseGlucagon-like peptide-2LipidsObesityMice Inbred C57BL030104 developmental biologyEndocrinologyLiverPeptidesbusinessGLP-2Dyslipidemia
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Pancreatic polypeptide stimulates mouse gastric motor activity through peripheral neural mechanisms

2016

Background Pancreatic polypeptide (PP) is supposed to be one of the major endogenous agonists of the neuropeptide Y4 receptor. Pancreatic polypeptide can influence gastrointestinal motility, acting mainly through vagal mechanisms, but whether PP acts directly on the stomach has not been explored yet. The aims of this study were to investigate the effects of PP on mouse gastric emptying, on spontaneous tone of whole stomach in vitro and to examine the mechanism of action. Methods Gastric emptying was measured by red phenol method after i.p. PP administration (1–3 nmol per mouse). Responses induced by PP (1–300 mmol L−1) on gastric endoluminal pressure were analyzed in vitro in the presence o…

Male0301 basic medicinemedicine.medical_specialtyPhysiologyGastric emptyingMuscarinic AntagonistsBiologyEndocrine and Autonomic SystemMice03 medical and health sciencesOrgan Culture Techniques0302 clinical medicineInternal medicineMuscarinic acetylcholine receptormedicineAnimalsPancreatic polypeptidePeripheral NervesPancreatic polypeptideReceptorAntrumDose-Response Relationship DrugGastric emptyingEndocrine and Autonomic SystemsStomachGastroenterologyMotilityAcetylcholineReceptors Neuropeptide YMice Inbred C57BL030104 developmental biologyEndocrinologymedicine.anatomical_structureMechanism of actionTachykininmedicine.symptomEnteric nervous systemGastrointestinal Motility030217 neurology & neurosurgeryAcetylcholinemedicine.drug
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Rapid progression of a splenic aneurysm due to segmental arterial mediolysis: a rare cause of acute pancreatitis.

2013

Abstract Background The etiology of acute pancreatitis can be manifold, beside the usual causes. We are reporting an unusual cause that triggered acute pancreatitis. Patient & results A 50 year-old male experienced attacks of acute pancreatitis (abdominal pain and elevated amylase and lipase) during sexual arousal. Serial imaging showed a rapidly-progressing, partly-thrombosed splenic artery aneurysm, with local compression of the pancreas. After angiographic coiling, the attacks subsided. Further angiography revealed additional aneurysms consistent with segmental arterial mediolysis at other sites of the body. Molecular analysis regarding Ehlers–Danlos-syndrome and genetic factors for panc…

MaleAbdominal painmedicine.medical_specialtyEndocrinology Diabetes and MetabolismMedizinBlood PressureAneurysm RupturedRadiology InterventionalAneurysmInternal medicinemedicineHumansSexual Dysfunctions PsychologicalHepatologymedicine.diagnostic_testbusiness.industryGastroenterologymedicine.diseaseAneurysmAbdominal PainRadiographymedicine.anatomical_structureBlood pressurePancreatitisAngiographyCardiologyEtiologyAcute pancreatitisPancreatitisRadiologymedicine.symptombusinessPancreasSplenic ArteryPancreatology : official journal of the International Association of Pancreatology (IAP) ... [et al.]
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Age and Protein Restriction Followed by Balanced Refeeding Affect Pancreatic Digestive Enzyme Outputs and Turnover Times in Rats

1991

Outputs and turnover times of trypsinogen 2, chymotrypsinogen 1, lipase and amylase were determined in pancreatic juice of growing male Wistar rats at various times during protein restriction (5% protein) followed by balanced refeeding (20% protein). In control rats fed a 20% protein diet, trypsinogen 2, chymotrypsinogen 1 and amylase outputs increased progressively with age, those of lipase remained constant and the turnover times of the four hydrolases were shortened. With time, protein restriction induced the most rapid decrease in trypsinogen 2 output, followed by that of amylase, then by those of trypsinogen 1 and lipase. Compared with controls, protein restriction enhanced specific ra…

MaleAgingmedicine.medical_specialtyTrypsinogenMedicine (miscellaneous)ChymotrypsinogenBiologydigestive systemCholecystokinin receptorchemistry.chemical_compoundInternal medicinemedicineAnimalsAmylaseLipasePancreasCholecystokininNutrition and DieteticsBody WeightRats Inbred StrainsLipaseRatsEndocrinologychemistryFoodAmylasesPancreatic juiceDigestive enzymeTrypsinogenbiology.proteinDietary ProteinsIsoelectric FocusingFood DeprivationThe Journal of Nutrition
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Senile amyloidosis: Principles of localization in a heterogeneous form of amyloidosis

1983

In order to identify amyloid deposits in patients over 60 years of age (so-called senile amyloid), the following five tissues were investigated under the light and electron microscope : 1. pituitary gland, 2. pancreatic islets of Langerhans, 3. heart, 4. aorta, and 5. brain. In all an increasing incidence of amyloid deposits was found with increasing age, and in the brain a significant quantitative increase in amyloid deposits with increasing age was observed. Despite the biochemical heterogeneity of amyloid found in old age, all the deposits seen in tissues examined were morphologically similar. Typical amyloid fibrils were always found (diameter 60–100 A), and these were invariably deposi…

MaleAmyloidPathologymedicine.medical_specialtyPituitary glandAmyloidBiologyBasement Membranelaw.inventionIslets of Langerhanslawmedicine.arterymental disordersmedicineHumansSenile plaquesAortaAgedAortaMyocardiumPancreatic isletsAmyloidosisAge FactorsBrainAmyloidosisMiddle Agedmedicine.diseaseMicroscopy Electronmedicine.anatomical_structurePituitary GlandFemaleSenile amyloidosisElectron microscopeVirchows Archiv B Cell Pathology Including Molecular Pathology
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Obese Rats Exhibit High Levels of Fat Necrosis and Isoprostanes in Taurocholate-Induced Acute Pancreatitis

2012

BACKGROUND: Obesity is a prognostic factor for severity in acute pancreatitis in humans. Our aim was to assess the role of oxidative stress and abdominal fat in the increased severity of acute pancreatitis in obese rats. METHODOLOGY: Taurocholate-induced acute pancreatitis was performed in lean and obese Zucker rats. Levels of reduced glutathione, oxidized glutathione, L-cysteine, cystine, and S-adenosylmethionine were measured in pancreas as well as the activities of serine/threonine protein phosphatases PP1 and PP2A and tyrosin phosphatases. Isoprostane, malondialdehyde, triglyceride, and free fatty acid levels and lipase activity were measured in plasma and ascites. Lipase activity was m…

MaleAnatomy and PhysiologyNecrosislcsh:MedicineAdipose tissueIsoprostanesmedicine.disease_causeBiochemistrychemistry.chemical_compoundMalondialdehydeMolecular Cell Biologylcsh:ScienceMultidisciplinaryPancreatitis Acute Necrotizingmusculoskeletal neural and ocular physiologyAnimal ModelsMalondialdehydeGlutathioneLipidsEnzymesBlood ChemistryMedicineAcute pancreatitismedicine.symptomResearch ArticleTaurocholic AcidCell Physiologymedicine.medical_specialtyBlotting WesternImmunologyGastroenterology and Hepatologymacromolecular substancesModel OrganismsInternal medicineChemical BiologymedicineAnimalsFat necrosisObesityPancreasBiologyTriglyceridesbusiness.industrylcsh:Rmedicine.diseaseObesityRatsRats ZuckerOxidative StressMetabolismEndocrinologyPancreatitisnervous systemchemistrySmall MoleculesRatPancreatitislcsh:QbusinessOxidative stressPLoS ONE
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Endoscopic cytology in biliary strictures. Personal experience

2008

The differential diagnosis between malignant and benign biliary strictures is the cornerstone of the management of jaundiced patients. The aim of our study is to define the role of cytology of the bile withdrawn during endoscopic retrograde cholangiopancreatography (ERCP), to reach a diagnosis of the nature of biliary stricture.This retrospective study was conducted on 67 consecutive patients affected of ingravescent obstructive jaundice who underwent ERCP+/-PTE (percutaneous transhepatic endoscopic)+bile withdrawn+stenting. We founded hilar stricture in 21 patients (31.3%), middle third the common duct stricture in 17 (25.3%), and lower third stricture in 28 patients (41.4%). In one patien…

MaleBile Duct DiseasesConstriction PathologicSensitivity and SpecificityCholangiocarcinomaDiagnosis DifferentialPredictive Value of TestsBileHumansAgedRetrospective StudiesAged 80 and overCholangiopancreatography Endoscopic RetrogradeCitology biliary cancerChi-Square DistributionBiopsy NeedleMiddle AgedPancreatic NeoplasmsJaundice ObstructiveSettore MED/18 - Chirurgia GeneraleTreatment OutcomeBile Duct NeoplasmsDrainageFemaleStentsFollow-Up Studies
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Amylase release from streptolysin O-permeabilized pancreatic acinar cells. Effects of Ca2+, guanosine 5'-[gamma-thio]triphosphate, cyclic AMP, tetanu…

1992

The molecular requirements for amylase release and the intracellular effects of botulinum A toxin and tetanus toxin on amylase release were investigated using rat pancreatic acinar cells permeabilized with streptolysin O. Micromolar concentrations of free Ca2+ evoked amylase release from these cells. Maximal release was observed in the presence of 30 microM free Ca2+. Ca(2+)-stimulated, but not basal, amylase release was enhanced by guanosine 5′-[gamma-thio]triphosphate (GTP[S]) (3-4 fold) or cyclic AMP (1.5-2 fold). Neither the two-chain forms of botulinum A toxin and tetanus toxin, under reducing conditions, nor the light chains of tetanus toxin, inhibited amylase release triggered by Ca2…

MaleBotulinum ToxinsCell Membrane PermeabilityClostridium tetanimedicine.disease_causeBiochemistryNorepinephrineBacterial ProteinsTetanus ToxinAcinar cellmedicineCyclic AMPNeurotoxinAnimalsAmylaseMolecular BiologyPancreasbiologyToxinProteolytic enzymesRats Inbred StrainsCell BiologyRatsBiochemistryGuanosine 5'-O-(3-Thiotriphosphate)AmylasesStreptolysinsbiology.proteinClostridium botulinumStreptolysinCalciumResearch Article
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