Search results for "pathogen"

showing 10 items of 1657 documents

Cellular, physiological, and molecular adaptive responses of Erwinia amylovora to starvation.

2013

Erwinia amylovora causes fire blight, a destructive disease of rosaceous plants distributed worldwide. This bacterium is a nonobligate pathogen able to survive outside the host under starvation conditions, allowing its spread by various means such as rainwater. We studied E. amylovora responses to starvation using water microcosms to mimic natural oligotrophy. Initially, survivability under optimal (28 °C) and suboptimal (20 °C) growth temperatures was compared. Starvation induced a loss of culturability much more pronounced at 28 °C than at 20 °C. Natural water microcosms at 20 °C were then used to characterize cellular, physiological, and molecular starvation responses of E. amylovora. Ch…

StarvationMicrobial ViabilityEcologybiologyVirulenceMotilityVirulenceGene ExpressionErwiniabiology.organism_classificationApplied Microbiology and BiotechnologyMicrobiologyAdaptation PhysiologicalViable but nonculturableMicrobiologyFire blightmedicineErwinia amylovoramedicine.symptomWater MicrobiologyPathogenBacteriaFEMS microbiology ecology
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Spatial data of Ixodes ricinus instar abundance and nymph pathogen prevalence, Scandinavia, 2016-2017.

2020

ticks carry pathogens that can cause disease in both animals and humans, and there is a need to monitor the distribution and abundance of ticks and the pathogens they carry to pinpoint potential high risk areas for tick-borne disease transmission. In a joint Scandinavian study, we measured Ixodes ricinus instar abundance at 159 sites in southern Scandinavia in August-September, 2016, and collected 29,440 tick nymphs at 50 of these sites. We additionally measured abundance at 30 sites in August-September, 2017. We tested the 29,440 tick nymphs in pools of 10 in a Fluidigm real-time PCR chip to screen for 17 different tick-associated pathogens, 2 pathogen groups and 3 tick species. We present…

Statistics and ProbabilityNymphIxodes ricinus030231 tropical medicineZoologyLibrary and Information SciencesTickScandinavian and Nordic CountriesEducation03 medical and health sciences0302 clinical medicineAbundance (ecology)parasitic diseasesAnimalsNymphlcsh:ScienceAuthor CorrectionPathogenEcosystemEcological epidemiology0303 health sciencesEcologybiologyIxodes030306 microbiologybiology.organism_classificationComputer Science ApplicationsHabitatInstarlcsh:QStatistics Probability and UncertaintyBacterial infectionDisease transmissionEntomologyAnimal DistributionInformation SystemsVDP::Matematikk og Naturvitenskap: 400::Zoologiske og botaniske fag: 480Scientific data
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Endothelial function and pathogenesis of endothelial dysfunction

2018

Background: The crucial role of endothelium is due to the ability of endothelial cells to receive and concurrently respond to humoral and hemodynamic stimuli. The mechanisms that mediate these reactions are: the production of endothelium-derived factors and metabolizing enzymes; the expression of binding proteins and adhesive molecules; and the consequential shape changes. In fact, a wide range of anti-atherosclerotic action substances is produced by the endothelial cells with the objective of maintaining the balance between vasoconstriction and vasodilation, and inhibit or stimulate the proliferation and migration of smooth muscle cells, thrombogenesis and fibrinolysis. Smoke, age, hyperch…

StimuliPathologymedicine.medical_specialtyEndotheliumbusiness.industryImmunologymedicine.diseaseEndothelialPathogenesismedicine.anatomical_structureEndothelial cellDysfunctionPathogenesiImmunology and AllergyMedicineEndotheliumEndothelial dysfunctionbusinessFunction (biology)
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The Swedish dilemma - the almost exclusive use of APPswe-based mouse models impedes adequate evaluation of alternative β-secretases.

2022

Abstract Alzheimer's disease (AD) is the most common form of dementia, however incurable so far. It is widely accepted that aggregated amyloid β (Aβ) peptides play a crucial role for the pathogenesis of AD, as they cause neurotoxicity and deposit as so-called Aβ plaques in AD patient brains. Aβ peptides derive from the amyloid precursor protein (APP) upon consecutive cleavage at the β- and γ-secretase site. Hence, mutations in the APP gene are often associated with autosomal dominant inherited AD. Almost thirty years ago, two mutations at the β-secretase site were observed in two Swedish families (termed Swedish APP (APPswe) mutations), which led to early-onset AD. Consequently, APPswe was …

SwedenProteasesbiologyBACE1-ASNeurotoxicityMice TransgenicCell Biologymedicine.diseaseCathepsin BPathogenesisAmyloid beta-Protein PrecursorDisease Models AnimalADAMTS4Alzheimer Diseasemental disordersbiology.proteinAmyloid precursor proteinmedicineAnimalsHumansAmyloid Precursor Protein SecretasesMolecular BiologyAmyloid precursor protein secretaseNeuroscienceBiochimica et biophysica acta. Molecular cell research
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Sympatric diversification vs. immigration: deciphering host-plant specialization in a polyphagous insect, the stolbur phytoplasma vector Hyalesthes o…

2012

The epidemiology of vector transmitted plant diseases is highly influenced by dispersal and the host-plant range of the vector. Widening the vector's host range may increase transmission potential, whereas specialization may induce specific disease cycles. The process leading to a vector's host shift and its epidemiological outcome is therefore embedded in the frameworks of sympatric evolution vs. immigration of preadapted populations. In this study, we analyse whether a host shift of the stolbur phytoplasma vector, Hyalesthes obsoletus from field bindweed to stinging nettle in its northern distribution range evolved sympatrically or by immigration. The exploitation of stinging nettle has l…

SympatryPhytoplasmaPopulationZoologyHemipteraGeneticsAnimalseducationEcology Evolution Behavior and SystematicsPhylogenyPlant Diseaseseducation.field_of_studyGenetic diversitybiologyEcologyHost (biology)Genetic VariationUrtica dioicaCixiidaebiology.organism_classificationBiological EvolutionSympatrySympatric speciationVector (epidemiology)Host-Pathogen InteractionsBiological dispersalMicrosatellite RepeatsMolecular ecology
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One is not enough: On the effects of reference genome for the mapping and subsequent analyses of short-reads.

2020

Mapping of high-throughput sequencing (HTS) reads to a single arbitrary reference genome is a frequently used approach in microbial genomics. However, the choice of a reference may represent a source of errors that may affect subsequent analyses such as the detection of single nucleotide polymorphisms (SNPs) and phylogenetic inference. In this work, we evaluated the effect of reference choice on short-read sequence data from five clinically and epidemiologically relevant bacteria (Klebsiella pneumoniae, Legionella pneumophila, Neisseria gonorrhoeae, Pseudomonas aeruginosa and Serratia marcescens). Publicly available whole-genome assemblies encompassing the genomic diversity of these species…

Systematic errorSingle Nucleotide PolymorphismsPathology and Laboratory MedicineGenomeKlebsiella PneumoniaeDatabase and Informatics MethodsData sequencesKlebsiellaMedicine and Health SciencesBiology (General)CladePhylogenyData ManagementEcologyPhylogenetic treeBacterial GenomicsMicrobial GeneticsChromosome MappingHigh-Throughput Nucleotide SequencingPhylogenetic AnalysisGenomicsBacterial PathogensPhylogeneticsLegionella PneumophilaComputational Theory and MathematicsMedical MicrobiologyModeling and SimulationPathogensSequence AnalysisResearch ArticleComputer and Information SciencesBioinformaticsQH301-705.5LegionellaSequence alignmentSingle-nucleotide polymorphismGenomicsComputational biologyMicrobial GenomicsBiologyResearch and Analysis MethodsPolymorphism Single NucleotideMicrobiologyCellular and Molecular NeurosciencePhylogeneticsGeneticsSNPBacterial GeneticsEvolutionary SystematicsMolecular BiologyMicrobial PathogensEcology Evolution Behavior and SystematicsTaxonomyEvolutionary BiologyBacteriaOrganismsBiology and Life SciencesBacteriologySequence AlignmentGenome BacterialReference genomePLoS Computational Biology
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Role of serum interleukin-6 in deciding therapy for multidrug resistant oral lichen planus

2015

Background: Oral lichen planus (OLP) is a T cell mediated immune response. T cells locally present in the involved tissues release cytokines like interleukin-6 (IL-6), which contributes to pathogenesis of OLP. Also IL-6 has been associated with multidrug resistance protein (MRP) expression by keratinocytes. Correspondingly, upregulation of MRP was found in OLP. We conducted this study to evaluate the effects of various drugs on serum IL-6 in OLP; and correlation of these effects with the nature of clinical response and resistance pattern seen in OLP lesions with various therapeutic modalities. Thus we evaluated the role of serum IL-6 in deciding therapy for multidrug resistant OLP. Material…

T cellOdontologíaPathogenesisPimecrolimusImmune systemstomatognathic systemMedicineInterleukin 6General DentistryOral Medicine and Pathologybiologybusiness.industryResearchmedicine.disease:CIENCIAS MÉDICAS [UNESCO]Ciencias de la saludstomatognathic diseasesmedicine.anatomical_structureImmunologyUNESCO::CIENCIAS MÉDICASbiology.proteinBiomarker (medicine)MethotrexateOral lichen planusbusinessmedicine.drugJournal of Clinical and Experimental Dentistry
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Intrahepatic myeloid-cell aggregates enable local proliferation of CD8+T cells and successful immunotherapy against chronic viral liver infection

2013

Chronic infection is difficult to overcome because of exhaustion or depletion of cytotoxic effector CD8(+) T cells (cytotoxic T lymphoytes (CTLs)). Here we report that signaling via Toll-like receptors (TLRs) induced intrahepatic aggregates of myeloid cells that enabled the population expansion of CTLs (iMATEs: 'intrahepatic myeloid-cell aggregates for T cell population expansion') without causing immunopathology. In the liver, CTL proliferation was restricted to iMATEs that were composed of inflammatory monocyte-derived CD11b(+) cells. Signaling via tumor-necrosis factor (TNF) caused iMATE formation that facilitated costimulation dependent on the receptor OX40 for expansion of the CTL popu…

T cellmedicine.medical_treatmentImmunologyPopulationGreen Fluorescent ProteinsMice TransgenicBiologyCD8-Positive T-LymphocytesLymphocytic ChoriomeningitisMicemedicineImmunology and AllergyCytotoxic T cellAnimalsLymphocytic choriomeningitis virusMyeloid CellseducationCell ProliferationMice Knockouteducation.field_of_studyLiver infectionCD11b AntigenMicroscopy ConfocalLiver DiseasesImmunotherapyReceptors OX40Flow CytometryMice Inbred C57BLCTL*Chronic infectionmedicine.anatomical_structureAnimals NewbornLiverToll-Like Receptor 9ImmunologyChronic DiseaseHost-Pathogen InteractionsImmunotherapyCD8Signal TransductionT-Lymphocytes CytotoxicNature Immunology
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Role of IFN-gamma in immune responses to Candida albicans infections

2014

Candida albicans is the most frequent etiologic agent that causes opportunistic fungal infections called candidiasis, a disease whose systemic manifestation could prove fatal and whose incidence is increasing as a result of an expanding immunocompromised population. Here we review the role of interferon-gamma (IFN-γ) in host protection against invasive candidiasis. This cytokine plays an essential role in both the innate and adaptive arms of the immune response to candidiasis. We focus on recent progress on host-pathogen interactions leading to the production of IFN-γ by host cells. IFN-γ is produced by CD4 Th1, CD8, γδ T, and natural killer (NK) cells, essentially in response to both IL-12…

T-LymphocytesFarmacologíamedicine.medical_treatmentPopulationReviewDiseaseMicrobiologyIFN-gammaInterferon-gammaImmune systemCandida albicansmedicineHumansInterferon gammaeducationCandida albicanseducation.field_of_studybiologyCandidiasisbiology.organism_classificationCorpus albicansKiller Cells NaturalCytokineImmune SystemHost-Pathogen InteractionsImmunologyTh17 CellsInfectionCD8medicine.drug
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Activation of complement by the alternative pathway as a factor in the pathogenesis of periodontal disease.

1976

Dental plaque and a bacterium, Actinomyces viscosus, isolated from plaque that can reproduce periodontal disease in germ-free rats, are activators of complement by the alternative pathway. It is suggested that this process is involved in the pathogenesis of chronic inflammatory periodontal disease.

T-LymphocytesGuinea PigsDental PlaqueAntigen-Antibody ComplexDental plaquePathogenesisstomatognathic systemPeriodontal diseasemedicineActinomycesAnimalsHumansActinomyces viscosusBone ResorptionPeriodontitisGlycoproteinsB-LymphocytesEnzyme Precursorsbusiness.industryMacrophagesGlobulinsGeneral MedicineComplement C3Complement System Proteinsmedicine.diseaseCathepsinsComplement (complexity)RatsEndotoxinsstomatognathic diseasesMicrobial CollagenaseImmunologyAlternative complement pathwaybusinessLancet (London, England)
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