Search results for "peripheral"

showing 10 items of 1028 documents

Generation of proteoliposomes from subcellular fractions.

1998

Intracellular membranes are highly dynamic, yet they retain their identity and functional characteristics. Integral membrane proteins, which must confer this specific membrane identity, remain poorly characterized at the biochemical level, largely because detergent-mediated solubilization is required for purification and analysis, and several properties of integral membrane proteins can only be investigated when the molecule is properly embedded in a lipid bilayer. We present a method for the efficient reconstitution into proteoliposomes of integral membrane proteins from subcellular fractions. Integral membrane proteins were identified on high-resolution two-dimensional gels after selectiv…

ProteolipidsClinical BiochemistryPeripheral membrane proteinMembrane ProteinsBiological membraneIntracellular MembranesBiologyBiochemistryTransmembrane proteinAnalytical ChemistryCell LineMembrane proteinBiochemistryCricetinaeLiposomesMembrane fluidityAnimalsProtein–lipid interactionLipid bilayerIntegral membrane proteinSubcellular FractionsElectrophoresis
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Soot-exposed mononuclear cells increase inflammatory cytokine mRNA expression and protein secretion in cocultured bronchial epithelial cells.

2000

<i>Background:</i> Soot particles are air pollutants capable of inducing airway and lung parenchymal injury. Mononuclear and bronchial epithelial cells are central to the maintenance of homeostasis and inflammation in the airways. <i>Objectives:</i> The aim of this study was to evaluate the contribution of mononuclear cells to the release of inflammatory mediators by bronchial epithelial cells. <i>Methods:</i> To model the in vivo situation, an in vitro system of cocultured blood monocytes and BEAS-2B cells was established in a transwell system. Blood monocytes were exposed to soot particles (FR 101) at concentrations of up to 100 μg/10<sup>6</su…

Pulmonary and Respiratory MedicineAdultMaleInflammationBronchiEnzyme-Linked Immunosorbent AssayBiologycomplex mixturesPeripheral blood mononuclear cellSensitivity and SpecificityMonocytesAir pollutantsParenchymamedicineHumansRNA MessengerSoot particlesCells CulturedAir PollutantsLungInterleukin-6Reverse Transcriptase Polymerase Chain ReactionInterleukin-8Epithelial CellsBlood Proteinsrespiratory systemCarbonCoculture Techniquesrespiratory tract diseasesCell biologymedicine.anatomical_structureSecretory proteinCytokinesCytokine mrnaFemalemedicine.symptomInflammation MediatorsRespiration; international review of thoracic diseases
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Efficacy of C1 esterase inhibitor concentrate in treatment of cutaneous attacks of hereditary angioedema.

2015

BACKGROUND Although treatment with C1 esterase inhibitor (C1-INH) concentrate is well established for hereditary angioedema (HAE) attacks in general, data that assess its efficacy for cutaneous attack treatment are sparse. OBJECTIVE To assess efficacy of plasma-derived, nanofiltered C1-INH concentrate for cutaneous attack treatment by comparing treated attacks from the uncontrolled I.M.P.A.C.T.2 study with historical data for untreated attacks. METHODS Cutaneous attack data from patients with HAE who were treated for cutaneous edema with 20 IU/kg body weight C1-INH concentrate in the uncontrolled I.M.P.A.C.T.2 study (38 patients) were compared with data from untreated patients from an histo…

Pulmonary and Respiratory MedicineAdultMalemedicine.medical_specialtyAdolescentUntreated groupPeripheral edemaGastroenterologySeverity of Illness IndexYoung AdultInternal medicineSeverity of illnessmedicineImmunology and AllergyHumansYoung adultChildAgedEnd pointbusiness.industryAngioedemas HereditaryGeneral MedicineArticlesMiddle Agedmedicine.diseasePeripheralC1 esteraseTreatment OutcomeHereditary angioedemaDisease ProgressionFemalemedicine.symptombusinessComplement C1 Inhibitor ProteinAllergy and asthma proceedings
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Impact of chronic obstructive pulmonary disease on the outcomes of patients with peripheral artery disease.

2019

Peripheral artery disease (PAD) and chronic obstructive pulmonary disease (COPD) are both related with high in-hospital mortality. We aimed to investigate the impact of COPD on the in-hospital outcomes in PAD.PAD patients were selected based on ICD-code I70.2 of the German nationwide database, stratified for COPD and compared regarding adverse in-hospital outcomes.Between 01/2005-12/2015, 5,611,827 inpatients (64.8% males) were diagnosed with PAD; of those, 13.6% were coded additionally with COPD. Overall, 277,894 PAD patients (5.0%) died during in-hospital course. Prevalence of cardiovascular diseases as well as cancer (12.1% vs. 7.0%, P  0.001) was higher in PAD patients with COPD compare…

Pulmonary and Respiratory MedicineAdultMalemedicine.medical_specialtyArterial diseasemedicine.medical_treatmentMyocardial InfarctionPulmonary diseaseDiseaseIndependent predictorAmputation Surgical03 medical and health sciencesPeripheral Arterial DiseasePulmonary Disease Chronic Obstructive0302 clinical medicineRisk FactorsInternal medicineNeoplasmsmedicinePrevalenceHumans030212 general & internal medicineHospital MortalityAgedAged 80 and overCOPDbusiness.industryNationwide databaseCancerMiddle Agedmedicine.diseaserespiratory tract diseases030228 respiratory systemAmputationCardiovascular DiseasesFemalebusinessPulmonary EmbolismRespiratory medicine
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Characterization of the interstitial lung and peripheral blood T cell receptor repertoire in cigarette smokers.

2005

T lymphocytes modulate the pulmonary inflammatory response. The aim of this study was to evaluate the clonality within the interstitial lung and peripheral blood T cell receptor (TCR) repertoire in smokers. Interstitial T lymphocytes were isolated from surplus tissue of 16 patients (63 +/- 9 [+/- SD] yr old, 11 male) undergoing surgery due to lung cancer (n = 15) or emphysema. TCR clonality was assessed by PCR amplification followed by spectratyping. Nearly all TCR of interstitial lung lymphocytes showed oligoclonal bands (CD4(+) subset 13/16 patients, 81%; CD8(+) 100%) indicating a specific differentiation. Peripheral blood T lymphocytes (PBL) TCR (especially CD4(+)) had less oligoclonal b…

Pulmonary and Respiratory MedicineCD4-Positive T-LymphocytesMalePathologymedicine.medical_specialtyLung NeoplasmsCellular differentiationClinical BiochemistryReceptors Antigen T-Cellchemical and pharmacologic phenomenaBiologyCD8-Positive T-LymphocytesPolymerase Chain ReactionmedicineHumansIntraindividual comparisonCell LineageLung cancerMolecular BiologyLungAgedLungT-cell receptorSmokinghemic and immune systemsCell DifferentiationCell BiologyMiddle Agedmedicine.diseasePeripheral bloodT-Cell Receptor Repertoiremedicine.anatomical_structureBloodPulmonary EmphysemaFemaleCD8American journal of respiratory cell and molecular biology
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Indoor air pollutants stimulate interleukin-8-specific mRNA expression and protein secretion of alveolar macrophages.

1998

Indoor air pollutants may cause inflammatory changes of the airways and adjacent pulmonary tissue. After phagocytosis of inhaled particles alveolar macrophages (AM) release chemotactic mediators capable of attracting inflammatory cells into the lung tissue. To evaluate these mechanisms further peripheral blood mononuclear cells (PBMNC) and human AM (freshly recovered from the lower respiratory tract) were exposed to the indoor particles Soot FR 101 and Printex 90, the asbestos fiber Chrysotile B, and titanium dioxide (TiO2) at concentrations of 10 or 50 microg/10(6) cells for up to 8 h. The migration of granulocytes into the conditioned supernatants of AM and PBMNC was quantified by chemota…

Pulmonary and Respiratory MedicineGranulocyte migrationAdultMaleChemokineBiologyGranulocytePeripheral blood mononuclear cellPolymerase Chain ReactionMicrobiologyMacrophages AlveolarmedicineHumansInterleukin 8RNA MessengerAgedAir PollutantsLungInterleukin-8InterleukinMolecular biologyChemotaxis Leukocytemedicine.anatomical_structureAir Pollution Indoorbiology.proteinLeukocytes MononuclearFemaleBronchoalveolar Lavage FluidChemotaxis assayLung
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Lung-restricted activation of the alveolar macrophage/monocyte system in pulmonary sarcoidosis.

1992

An activation of T-cells that is restricted to the lung has been demonstrated in pulmonary sarcoidosis. The role of blood monocytes (MO) and alveolar macrophages (AM) in this concept of compartmentalized inflammation has not yet been evaluated. In order to elucidate this question, we measured the release of tumor necrosis factor alpha (TNF alpha) and interleukin-1 (IL-1) by peripheral blood mononuclear cells (PBMNC) and AM in 43 patients with sarcoidosis (32 with active, 11 with inactive disease) without therapy and correlated the spontaneous monokine release to parameters of the T-cell alveolitis and the course of the disease. TNF alpha as well as IL-1 were spontaneously released by AM of …

Pulmonary and Respiratory MedicineInterleukin 2Lung Diseasesmedicine.medical_specialtyTime FactorsSarcoidosisLung Diseases/metabolism610 MedizinInflammationSarcoidosis/metabolismLymphocyte ActivationMacrophages Alveolar/secretionPeripheral blood mononuclear cellMonocytesInterleukin-1/secretionInternal medicineMacrophages AlveolarmedicineMacrophageHumansddc:610Receptors Interleukin-2/metabolismTumor Necrosis Factor-alpha/secretionbusiness.industryTumor Necrosis Factor-alphaMonocyteLeukocytes Mononuclear/secretionMonocytes/immunologyReceptors Interleukin-2Macrophage ActivationMonokinemedicine.anatomical_structureEndocrinologyImmunologyAlveolar macrophageLeukocytes MononuclearInterleukin-2Tumor necrosis factor alphamedicine.symptomInterleukin-2/secretionbusinessmedicine.drugInterleukin-1The American review of respiratory disease
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Anatomic and flow dynamic considerations for safe right axillary artery cannulation.

2013

Objectives Neuroprotection is of paramount interest in cardiac surgery. Right axillary artery cannulation is well established in aortic surgery because it significantly improves survival and outcome, but malperfusion of the right brain after direct cannulation has been reported. Anatomically, 4 vessel segments are potentially amenable for cannulation of the subclavian and axillary arteries. Clinical studies vary widely in dissection sites and cannulation techniques. We investigated critical flow dynamics in the right brain caused by arterial inflow after direct cannulation and specified cannulation positions that provide optimal cerebral perfusion. Methods Distances from the lateral margin …

Pulmonary and Respiratory MedicineMalemedicine.medical_specialtyVertebral arterySubclavian ArteryHemodynamicsDissection (medical)Axillary arterymedicine.arteryCatheterization PeripheralmedicineCadaverHumansComputer SimulationCerebral perfusion pressureSubclavian arteryVertebral Arterybusiness.industryModels Cardiovascularmedicine.diseaseCannulaCardiac surgerySurgeryRegional Blood FlowCerebrovascular CirculationAxillary ArterySurgeryFemaleRadiologyAnatomic LandmarksbusinessCardiology and Cardiovascular MedicineBlood Flow VelocityThe Journal of thoracic and cardiovascular surgery
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Mononuclear Cells in Exudative Malignant Pleural Effusions

1994

The aims of this study were to develop a methodology for the isolation of highly enriched mononuclear phagocyte populations from exudative malignant pleural effusions (EMPE) and to characterize the phenotype and functional properties of these cells. Pleural effusion mononuclear cells (PEMC) were isolated by Ficoll centrifugation of EMPE and transudative pleural effusions and allowed to adhere to plastic for 1 h to obtain a pleural effusion mononuclear adherent cell (PEMAC) fraction. Only 66.0±4.2 percent of PEMAC ingested latex particles, indicating that a significant proportion of PEMAC were not phagocytic cells. Latex-positive PEMAC had the morphologic appearance of macrophages and staine…

Pulmonary and Respiratory MedicinePathologymedicine.medical_specialtybusiness.industryPleural effusionCD14InterleukinMononuclear phagocyte systemCritical Care and Intensive Care Medicinemedicine.diseasePeripheral blood mononuclear cellProinflammatory cytokineAntigenMedicineTumor necrosis factor alphaCardiology and Cardiovascular MedicinebusinessChest
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The murine bronchopulmonary microcirculation in hapten-induced inflammation

2006

ObjectiveThe clinical observation of central bronchial artery hypertrophy in chronic lung inflammation suggests the possibility that the bronchial circulation may also participate in adaptive responses in peripheral lung inflammation.MethodsTo investigate the potential role of the bronchial microcirculation in peripheral lung inflammation, we developed a murine model of lung inflammation using the intratracheal instillation of the peptide-hapten trinitrophenol in presensitized mice.ResultsClinical parameters indicated a peak inflammatory response at 96 hours. Similarly, gross and microscopic evidence of inflammation was observed 96 hours after antigen instillation. Using a forced oscillatio…

Pulmonary and Respiratory MedicinePulmonary CirculationPathologymedicine.medical_specialtyBronchiInflammationBronchial ArteriesCorrosion CastingMicrocirculationMuscle hypertrophyMicePicratesAntigenmedicine.arterymedicineAnimalsLungMice Inbred BALB CLungbusiness.industryMicrocirculationBronchial circulationPneumoniarespiratory systemrespiratory tract diseasesPeripheralPulmonary Alveolimedicine.anatomical_structureImmunologyMicroscopy Electron ScanningSurgerymedicine.symptomCardiology and Cardiovascular MedicinebusinessBronchial arteryHaptensThe Journal of Thoracic and Cardiovascular Surgery
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