Search results for "pero"

showing 10 items of 3365 documents

PPAR in Cardiovascular Disorders

2016

Peroxisome proliferation-activated receptors (PPARs) are ligand-inducible transcription factors that, upon binding their ligands, translocate into the nucleus, where they regulate transcription of numerous genes that have the peroxisome proliferator response element (PPRE) in the promoter region [1]. In humans, there are 3 PPAR isoforms: PPAR-α, PPAR-β/δ, and PPAR-γ. The isoforms have partially overlapping spectra of activity and are differently expressed in organs and tissues [2]. PPAR-α is expressed mostly in tissues characterized by high catabolic activity, including skeletal muscle, liver, proximal tubular cells in kidneys, and brown fat. This PPAR isoform regulates components of β-oxid…

medicine.medical_specialtyArticle SubjectPeroxisome proliferator-activated receptor030209 endocrinology & metabolism030204 cardiovascular system & hematologyBiology03 medical and health scienceschemistry.chemical_compound0302 clinical medicineDownregulation and upregulationInternal medicineDrug DiscoverymedicineGlucose homeostasisPharmacology (medical)Beta oxidationlcsh:QH301-705.5chemistry.chemical_classificationFatty acid metabolismLipid metabolismPeroxisomeEndocrinologyEditorialchemistrylcsh:Biology (General)Rosiglitazonemedicine.drugPPAR Research
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Hypolipidaemic effects of fenofibrate and fasting in the herbivorous grass carp (Ctenopharyngodon idella) fed a high-fat diet

2008

We investigated whether the hypolipidaemic effect of fenofibrate and fasting observed in most omnivorous mammals may also apply to herbivorous fish. Grass carp (Ctenopharyngodon idella) fed a high-fat (8 %) diet exhibited a marked increase in blood lipids and body fat after 6 weeks. They were then treated with fenofibrate (100 mg/kg body weight) in the same high-fat diet for 2 weeks, followed by fasting for 1 week. Plasma lipid concentration, body fat amount, fatty acid composition, plasma thiobarbituric acid-reactive substances and some parameters related to hepatic fatty acid oxidation were measured, and liver samples were stained for histological examination. Fenofibrate treatment decrea…

medicine.medical_specialtyCarpsmedicine.drug_classMedicine (miscellaneous)Blood lipidsHyperlipidemiasFibrateBiologyThiobarbituric Acid Reactive SubstancesLipid peroxidationFish Diseaseschemistry.chemical_compoundFenofibrateInternal medicinemedicineAnimalsBeta oxidationHypolipidemic AgentsNutrition and DieteticsFenofibrateCholesterolFatty AcidsLipid metabolismFastingLipid Metabolismbiology.organism_classificationCombined Modality TherapyDietary FatsGrass carpEndocrinologyLiverchemistryAnimal Nutritional Physiological Phenomenalipids (amino acids peptides and proteins)Lipid PeroxidationOxidation-Reductionmedicine.drugBritish Journal of Nutrition
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Dihydrocucurbitacin B, isolated from Cayaponia tayuya, reduces damage in adjuvant-induced arthritis

2005

23,24-Dihydrocucurbitacin B, from the anti-rheumatic plant Cayaponia tayuya, was tested on arthritis induced by adjuvant to corroborate the anti-inflammatory properties of this plant. Arthritis was induced in Lewis rats; the resulting arthritic rats were then treated with dihydrocucurbitacin B (1 mg/kg orally, daily, 1 week). The effect of dihydrocucurbitacin B on the synthesis, release, and activity of pro-inflammatory enzymes (elastase, cyclooxygenase-2, and nitric oxide synthase-2) as well as its effect on different mediators (tumor necrosis factor-alpha and interleukin-1beta) were determined. Dihydrocucurbitacin B modified the evolution of the clinical symptoms, reducing the swelling an…

medicine.medical_specialtyCell Survivalmedicine.medical_treatmentAnti-Inflammatory AgentsAdministration OralNitric Oxide Synthase Type IIPainArthritisPlant RootsDinoprostoneCell LineNitric oxidechemistry.chemical_compoundSuperoxidesInternal medicinemedicineAnimalsLymphocytesNitritesPharmacologyDose-Response Relationship DrugPancreatic ElastasebiologyPlant Extractsbusiness.industryMacrophagesElastasemedicine.diseasebiology.organism_classificationArthritis ExperimentalTriterpenesCayaponia tayuyaRatsEnzyme ActivationNitric oxide synthaseCucurbitaceaeEndocrinologyCytokinechemistryCyclooxygenase 2Rats Inbred LewAntirheumatic AgentsToxicitybiology.proteinCytokinesFemaleTumor necrosis factor alphabusinessPhytotherapyEuropean Journal of Pharmacology
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Medical significance of peroxisome proliferator-activated receptors.

1999

Peroxisome proliferator-activated receptors (PPAR) were discovered in 1990, ending 25 years of uncertainty about the molecular mechanisms of peroxisome proliferation. Subsequently, PPARs have improved our understanding of adipocyte differentiation. But there is more to PPARs than solving a puzzle about an organelle (the peroxisome) long considered an oddity, and their medical significance goes beyond obesity too. Enhanced PPAR type alpha expression protects against cardiovascular disorders though the role of enhanced PPARgamma expression seems less favourable. PPAR mechanisms, mainly via induction of more differentiated cell phenotypes, protect against some cancers. The differentiation of m…

medicine.medical_specialtyCellular differentiationPeroxisome ProliferationPeroxisome proliferator-activated receptorReceptors Cytoplasmic and NuclearBiologyMicrobodiesInternal medicineNeoplasmsmedicineAdipocytesAnimalsHumansReceptorRegulation of gene expressionchemistry.chemical_classificationResearchFatty AcidsCell DifferentiationGeneral MedicinePeroxisomeEndocrinologychemistryNuclear receptorGene Expression RegulationCardiovascular DiseasesCancer researchPeroxisome proliferator-activated receptor alphaOxidation-ReductionTranscription FactorsLancet (London, England)
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Chelation of synaptic zinc induces overexcitation in the hilar mossy cells of the rat hippocampus.

2004

Complete removal of synaptic zinc by the chelator dietyldithiocarbamate (DEDTC; 500 mg/kg i.p.) in rat was followed by convulsive behaviour including wet dog shakes alternating immobility. Histological analysis 1 day after DEDTC administration detected expression of heat shock protein in the hippocampus restricted to hilar cells. These cells colocalize the marker for neurons and the glutamate receptor GluR2/3 showing that they are excitatory neurons. Additionally, they projected to the contralateral dentate gyrus. Therefore, they correspond to hilar mossy cells. These data show that the synaptic zinc has a role in normal hippocampus avoiding overexcitation, that would impair functionality e…

medicine.medical_specialtyCentral nervous systemPresynaptic TerminalsWheat Germ Agglutinin-Horseradish Peroxidase ConjugateHippocampusAction PotentialsHSP72 Heat-Shock Proteinsc-FosHippocampusSynaptic TransmissionSeizuresInternal medicineNeural PathwaysmedicineAnimalsReceptors AMPAHeat-Shock ProteinsChelating AgentsbiologyGeneral NeuroscienceDentate gyrusGlutamate receptorColocalizationImmunohistochemistryRatsZincEndocrinologymedicine.anatomical_structurenervous systemDentate GyrusMossy Fibers Hippocampalbiology.proteinExcitatory postsynaptic potentialDitiocarbImmediate early geneProto-Oncogene Proteins c-fosNeuroscience letters
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Tumor Oxygenation Under Normobaric and Hyperbaric Hyperoxia

1997

Tumor hypoxia is an important factor limiting the efficiency of sparsely ionizing ra-diation and O2-dependent chemotherapy. Since the tumor pO2 is the result of a dynamic steady state between oxygen supply and O2 consumption of the tumor tissue, hypoxia could be reduced either by increasing the O2-supply or by reducing the O2 demand of the tumor cells. The O2 supply can be improved for instance by (i) increasing the arterial oxy-gen partial pressure, (ii) improving (and homogenizing) the tumor perfusion, or (iii) en-hancing the O2 release from blood into the tissue by right-shifting the HbO2 dissociation curve. Theoretically, it should also be possible to improve tumor oxygenation by a rela…

medicine.medical_specialtyChemotherapyTumor hypoxiabusiness.industryHyperbaric oxygenationmedicine.medical_treatmentOxygen–haemoglobin dissociation curveTumor OxygenationHyperbaric hyperoxiaInternal medicineTumor perfusionCardiologyMedicineRadiosensitivitybusiness
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Complement activation by oxidatively modified low-density lipoproteins

1999

Background Oxidatively modified low-density lipoproteins (LDLs) have been implicated in the pathogenesis of atherosclerosis and are found in human vascular lesions. There is increasing evidence that complement activation may also play a role in atherogenesis. Activated complement proteins have been demonstrated to be present in early atherosclerotic lesions, and lipids isolated from lesions have been shown to activate complement, hence their designation as lesion complement activator (LCA). The question now arose whether oxidized LDLs would also activate complement. Material and methods The complement-activating capacity of a lesion complement activator preparation and of minimally as well …

medicine.medical_specialtyClinical BiochemistryInflammationImmunoelectrophoresis030204 cardiovascular system & hematologyBiochemistryLipid peroxidationPathogenesisLesion03 medical and health scienceschemistry.chemical_compound0302 clinical medicineInternal medicinemedicine030304 developmental biology0303 health sciencesmedicine.diagnostic_testChemistryVascular diseaseGeneral Medicinemedicine.diseaseComplement systemComplement (complexity)EndocrinologyBiochemistrylipids (amino acids peptides and proteins)medicine.symptomEuropean Journal of Clinical Investigation
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Sjøgren's syndrome-associated oxidative stress and mitochondrial dysfunction: Prospects for chemoprevention trials

2012

An involvement of oxidative stress (OS) was found in recent studies of Sjøgren's syndrome (SS) that reported significant changes in protein oxidation, myeloperoxidase activity, TNF-α, nitrotyrosine, and GSH levels in plasma from SS patients. Excess levels of OS markers, as oxidative DNA damage and propanoyl-lysine, were reported in saliva from SS patients. Previous reports concurred with a role of OS in SS pathogenesis, by showing a decreased expression of antioxidant activities in conjunctival epithelial cells of SS patients and in parotid gland tissue samples from SS patients. A link between OS and mitochondrial dysfunction (MDF) is recognized both on the grounds of the established role o…

medicine.medical_specialtyDNA damageMitochondrionBiologyProtein oxidationmedicine.disease_causeChemopreventionBiochemistryPathogenesischemistry.chemical_compoundInternal medicinemedicineHumansSalivaPeroxidasechemistry.chemical_classificationReactive oxygen speciesTumor Necrosis Factor-alphaNitrotyrosineAutoantibodyGeneral MedicineGlutathioneMitochondriaOxidative StressSjogren's SyndromeEndocrinologychemistryTyrosineBiomarkersOxidative stressDNA DamageFree Radical Research
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PATHOPHYSIOLOGY AND CLINICAL ASPECTS OF URINARY LITHIASIS

2007

Urine is a complex balanced solution containing dissociated and non-dissociated solutes. Any variation in urine saturation grade (number of crystals dissolved in a volume of urine), urinary pH and the concentration of crystallization inhibitors can break the normal existing balance and lead to urolithiasis. In the present article we analyze the principal mechanisms (absorptive, renal, resorptive) of hypercalciuria. It will be also shown how heredity directly influences the clinical aspects of cystine, xanthine and oxalate lithiasis and how diet, in association with metabolic disorders, interferes in uric acid and oxalate stone formation. Finally, we report on the roles of urinary tract malf…

medicine.medical_specialtyDrug-Related Side Effects and Adverse ReactionsUrologyUrinary systemHypercalciuriaCystineUrologyHyperuricemiaUrinenKidneyXanthineOxalatechemistry.chemical_compoundUrolithiasisHumansMedicineHypercalciuriaCalcium metabolismHyperoxaluriaCystinuriabusiness.industryOxalic AcidXanthinemedicine.diseaseUric AcidchemistryUrogenital AbnormalitiesUrinary Tract InfectionsCystineUric acidCalciumbusiness
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Effects of alcohol-free beer on lipid profile and parameters of oxidative stress and inflammation in elderly women

2009

We assessed the influence of alcohol-free beer on factors implicated in atherosclerosis, such as lipid profile, oxidative stress parameters, and proinflammatory cytokines, in postmenopausal women, a population particularly at risk for atherosclerotic disease.The study was carried out in 29 nuns, 58 to 73 y old, who live in a convent with a disciplined, regular, and homogeneous lifestyle. The nuns maintained their habits and diet routine, but their meals were supplemented with 500 mL/d of alcohol-free beer (0.0%) divided into two doses over a 45-d period. Lipid profile, inflammatory markers such as C-reactive protein, interleukins 1 and 6, and tumor necrosis factor-alpha, and parameters of o…

medicine.medical_specialtyEndocrinology Diabetes and Metabolismmedicine.medical_treatmentPopulationInflammationmedicine.disease_causeThiobarbituric Acid Reactive SubstancesProinflammatory cytokineBeveragesLipid peroxidationchemistry.chemical_compoundInternal medicineHumansMedicineeducationInflammationeducation.field_of_studyNutrition and DieteticsEthanolmedicine.diagnostic_testTumor Necrosis Factor-alphabusiness.industryCholesterolInterleukinsVitamin EBeerMiddle AgedLipidsPostmenopauseOxidative StressC-Reactive ProteinEndocrinologychemistryCardiovascular DiseasesFemaleLipid PeroxidationInflammation Mediatorsmedicine.symptombusinessLipid profileOxidation-ReductionBiomarkersOxidative stressNutrition
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