Search results for "pero"

showing 10 items of 3365 documents

Comparison of diffusion, cytotoxicity and tissue inflammatory reactions of four commercial bleaching products against human dental pulp stem cells

2018

AbstractMultiple side effects related to bleaching were found to occur in the dental pulp tissue, including decreased cell metabolism and viability. In this work we evaluated the in vitro diffusion capacity, cytotoxicity and biocompatibility of four commercial bleaching products on stem cells from human dental pulp (hDPSCs). Two commercial bleaching gels hydrogen peroxide-based (HP), Norblanc Office 37.5% (Nor-HP) and Opalescence Boost 40% (Opal-HP) were applied for 30 min to enamel/dentine discs. Another two gels from the same manufacturers, 16% carbamide peroxide-based (CP), Norblanc Home (Nor-CP) and Opalescence CP 16% (Opal-CP), were applied for 90 min. The diffusion of HP was analysed …

AdultMale0301 basic medicineNecrosisBiocompatibilityScienceCarbamide PeroxideArticleDiffusionTooth whiteningYoung Adult03 medical and health scienceschemistry.chemical_compoundDental biomaterials0302 clinical medicineAnti-Infective Agentsstomatognathic systemDental pulp stem cellsTooth BleachingmedicineAnimalsHumansRats WistarTooth Bleaching AgentsHydrogen peroxideCytotoxicityDental PulpInflammationMultidisciplinaryEnamel paintStem CellsQRHydrogen PeroxideMolecular biologyPeroxidesRatsstomatognathic diseases030104 developmental biologychemistryvisual_artToxicityMicroscopy Electron Scanningvisual_art.visual_art_mediumMedicinePulp (tooth)Femalemedicine.symptom030217 neurology & neurosurgeryScientific Reports
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Active paraplegics are protected against exercise-induced oxidative damage through the induction of antioxidant enzymes

2016

Exercise improves functional capacity in spinal cord injury (SCI). However, exhaustive exercise, especially when sporadic, is linked to the production of reactive oxygen species that may have a detrimental effect on SCI. We aimed to study the effect of a single bout of exhaustive exercise on systemic oxidative stress parameters and on the expression of antioxidant enzymes in individuals with paraplegia. The study was conducted in the Physical Therapy department and the Physical Education and Sports department of the University of Valencia. Sixteen paraplegic subjects were submitted to a graded exercise test (GET) until volitional exhaustion. They were divided into active or non-active group…

AdultMale0301 basic medicinePathologymedicine.medical_specialtyAntioxidantNeurologyEnzimasmedicine.medical_treatmentEnzimaPharmacologyAntioxidantsProtein CarbonylationOxidative damage03 medical and health sciences0302 clinical medicineMalondialdehydeAccelerometrymedicineHumansRNA MessengerExerciseSpinal cord injuryAgedParaplegiachemistry.chemical_classificationGlutathione PeroxidaseParaplejíaSuperoxide Dismutasebusiness.industryGeneral MedicineMiddle AgedCatalasemedicine.disease030104 developmental biologyEnzymeGene Expression RegulationNeurologychemistryExercise TestLeukocytes MononuclearFemaleLipid PeroxidationNeurology (clinical)ParaplegiabusinessEnfermedad030217 neurology & neurosurgery
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Stimulatory TSH-Receptor Antibodies and Oxidative Stress in Graves Disease

2018

CONTEXT: We hypothesized that TSH-receptor (TSHR) stimulating antibodies (TSAbs) are involved in oxidative stress mechanisms in patients with Graves disease (GD). METHODS: Nicotinamide adenine dinucleotide phosphate oxidase, isoform 2 (NOX2); oxidative parameters; and oxidative burst were measured in serum, urine, and whole blood from patients with GD and control subjects. Superoxide production was investigated in human embryonic kidney (HEK)-293 cells stably overexpressing the TSHR. Lipid peroxidation was determined by immunodot-blot analysis for protein-bound 4-hydroxy-2-nonenal (4-HNE) in human primary thyrocytes and HEK-293–TSHR cells. RESULTS: Serum NOX2 levels were markedly higher in …

AdultMale0301 basic medicineendocrine systemmedicine.medical_specialtyendocrine system diseasesEndocrinology Diabetes and MetabolismGraves' diseaseClinical Biochemistry030209 endocrinology & metabolismContext (language use)medicine.disease_causeBiochemistryLipid peroxidation03 medical and health scienceschemistry.chemical_compound0302 clinical medicineEndocrinologyInternal medicinemedicineHumansEuthyroidClinical Research ArticlesTriiodothyroninebusiness.industryBiochemistry (medical)Toxic nodular goiterReceptors ThyrotropinMiddle AgedPrognosismedicine.diseaseGraves DiseaseRespiratory burstOxidative StressHEK293 Cells030104 developmental biologyEndocrinologychemistryFemaleLipid PeroxidationbusinessBiomarkershormones hormone substitutes and hormone antagonistsOxidative stressFollow-Up StudiesImmunoglobulins Thyroid-StimulatingThe Journal of Clinical Endocrinology & Metabolism
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Novel findings in patients with primary hyperoxaluria type III and implications for advanced molecular testing strategies

2012

Identification of mutations in the HOGA1 gene as the cause of autosomal recessive primary hyperoxaluria (PH) type III has revitalized research in the field of PH and related stone disease. In contrast to the well-characterized entities of PH type I and type II, the pathophysiology and prevalence of type III is largely unknown. In this study, we analyzed a large cohort of subjects previously tested negative for type I/II by complete HOGA1 sequencing. Seven distinct mutations, among them four novel, were found in 15 patients. In patients of non-consanguineous European descent the previously reported c.700+5G>T splice-site mutation was predominant and represents a potential founder mutation, w…

AdultMaleAdolescentIn silicoCell Culture TechniquesMedizinGene ExpressionContext (language use)Biologymedicine.disease_causeArticlePrimary hyperoxaluriaKidney CalculiGeneticsmedicineHumansGenetic TestingGeneGenetics (clinical)Genetic testingGeneticsMutationmedicine.diagnostic_testGenetic heterogeneityOxo-Acid-LyasesMiddle Agedmedicine.diseasePhenotypePedigreeHyperoxaluria PrimaryMutationFemale
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Role of Circulating miRNAs as Biomarkers in Idiopathic Pulmonary Arterial Hypertension: Possible Relevance of miR-23a

2015

Idiopathic pulmonary hypertension (IPAH) is a rare disease characterized by a progressive increase in pulmonary vascular resistance leading to heart failure. MicroRNAs (miRNAs) are small noncoding RNAs that control the expression of genes, including some involved in the progression of IPAH, as studied in animals and lung tissue. These molecules circulate freely in the blood and their expression is associated with the progression of different vascular pathologies. Here, we studied the expression profile of circulating miRNAs in 12 well-characterized IPAH patients using microarrays. We found significant changes in 61 miRNAs, of which the expression of miR23a was correlated with the patients’ …

AdultMaleAgingArticle SubjectNF-E2-Related Factor 2Idiopathic Pulmonary HypertensionBiologyBiochemistryPulmonary function testingmicroRNAmedicineGene silencingHumansFamilial Primary Pulmonary Hypertensionlcsh:QH573-671Cells CulturedAgedlcsh:CytologySuperoxide DismutaseGene Expression ProfilingCytochromes cCell BiologyGeneral MedicineMiddle Agedmedicine.diseasePeroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alphaGene expression profilingMicroRNAsmedicine.anatomical_structureHeart failureImmunologyVascular resistanceBiomarker (medicine)FemaleBiomarkersHeme Oxygenase-1Research ArticleTranscription FactorsOxidative Medicine and Cellular Longevity
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Impaired plasma nitric oxide availability and extracellular superoxide dismutase activity in healthy humans with advancing age

2006

This study is aimed to verify the modifications of extracellular superoxide dismutase (EC-SOD) activity and its potential involvement on the mechanism responsible for the impairment of plasma nitric oxide (NO) availability occurring with advancing age in healthy humans. For this purpose, plasma samples were drawn from 40 healthy men, aged 20-92 years, in fasting state and used for measurements of stable end-product nitrite/nitrate (NOx), as expression of NO availability, EC-SOD activity, thiobarbituric acid reactive substances (TBARS) as marker of lipid peroxidation, Trolox equivalent antioxidant capacity (TEAC) as a measure of plasma total antioxidant capacity, and in vitro susceptibility …

AdultMaleAgingmedicine.medical_specialtyAntioxidantThiobarbituric acidextracellular superoxide dismutasemedicine.medical_treatmentTrolox equivalent antioxidant capacitymedicine.disease_causeAntioxidantsGeneral Biochemistry Genetics and Molecular BiologyNitric oxideLipid peroxidationchemistry.chemical_compoundNitric oxide Extracellular superoxide dismutase ; Oxidative stress; Advancing ageadvancing agenitric oxideInternal medicinemedicineTBARSAnimalsHumansoxidative stressGeneral Pharmacology Toxicology and PharmaceuticsAgedAged 80 and overSuperoxide DismutaseFastingGeneral MedicineMiddle AgedLipidsEndocrinologychemistryBiochemistryLow-density lipoproteinLipid PeroxidationExtracellular SpaceOxidative stressLife Sciences
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PGC-1α Induction in Pulmonary Arterial Hypertension

2012

Idiopathic Pulmonary arterial hypertension (IPAH) is characterized by the obstructive remodelling of pulmonary arteries, and a progressive elevation in pulmonary arterial pressure (PAP) with subsequent right-sided heart failure and dead. Hypoxia induces the expression of peroxisome proliferator activated receptorγcoactivator-1α(PGC-1α) which regulates oxidative metabolism and mitochondrial biogenesis. We have analysed the expression of PGC-1α, cytochrome C (CYTC), superoxide dismutase (SOD), the total antioxidant status (TAS) and the activity of glutathione peroxidase (GPX) in blood samples of IPAH patients. Expression of PGC-1αwas detected in IPAH patients but not in healthy volunteers. Th…

AdultMaleAgingmedicine.medical_specialtyArticle SubjectHypertension PulmonaryPeroxisome proliferator-activated receptorBiologyBiochemistrySuperoxide dismutaseChloridesInternal medicinemedicineHumansFamilial Primary Pulmonary Hypertensionlcsh:QH573-671Heat-Shock ProteinsAgedchemistry.chemical_classificationGlutathione Peroxidaselcsh:CytologySuperoxide DismutaseGlutathione peroxidaseAge FactorsCytochromes cCell BiologyGeneral MedicineHypoxia (medical)Middle Agedmedicine.diseasePulmonary hypertensionPeroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alphaEndocrinologymedicine.anatomical_structurechemistryMitochondrial biogenesisHeart failurebiology.proteinVascular resistanceFemaleVascular Resistancemedicine.symptomTranscription FactorsResearch ArticleOxidative Medicine and Cellular Longevity
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Lipid Peroxidation, Nitric Oxide Metabolites, and Their Ratio in a Group of Subjects with Metabolic Syndrome

2014

Our aim was to evaluate lipid peroxidation, expressed as thiobarbituric acid-reactive substances (TBARS), nitric oxide metabolites (nitrite + nitrate) expressed asNOx, and TBARS/NOxratio in a group of subjects with metabolic syndrome (MS). In this regard we enrolled 106 subjects with MS defined according to the IDF criteria, subsequently subdivided into diabetic (DMS) and nondiabetic (NDMS) and also into subjects with a low triglycerides/HDL-cholesterol (TG/HDL-C) index or with a high TG/HDL-C index. In the entire group and in the four subgroups of MS subjects we found an increase in TBARS andNOxlevels and a decrease in TBARS/NOxratio in comparison with normal controls. Regarding all these …

AdultMaleAgingmedicine.medical_specialtySettore MED/09 - Medicina InternaArticle SubjectInflammationNitric OxideThiobarbituric Acid Reactive SubstancesBiochemistryNitric oxideLipid peroxidationchemistry.chemical_compoundInsulin resistanceInternal medicinemedicineTBARSHumanslcsh:QH573-671NitriteNitritesTriglyceridesNOxMetabolic SyndromeNitrateslcsh:CytologyCell BiologyGeneral MedicineMiddle Agedmedicine.diseaseEndocrinologyBiochemistrychemistryLipid Peroxidation Nitric Oxide Metabolites Metabolic SyndromeFemaleLipid Peroxidationmedicine.symptomMetabolic syndromeLipoproteins HDLResearch ArticleOxidative Medicine and Cellular Longevity
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Disease-associated polymorphisms in ERAP1 do not alter endoplasmic reticulum stress in patients with ankylosing spondylitis

2014

The mechanism by which human leukocyte antigen B27 (HLA-B27) contributes to ankylosing spondylitis (AS) remains unclear. Genetic studies demonstrate that association with and interaction between polymorphisms of endoplasmic reticulum aminopeptidase 1 (ERAP1) and HLA-B27 influence the risk of AS. It has been hypothesised that ERAP1-mediated HLA-B27 misfolding increases endoplasmic reticulum (ER) stress, driving an interleukin (IL) 23-dependent, pro-inflammatory immune response. We tested the hypothesis that AS-risk ERAP1 variants increase ER-stress and concomitant pro-inflammatory cytokine production in HLA-B27(+) but not HLA-B27(-) AS patients or controls. Forty-nine AS cases and 22 healthy…

AdultMaleAnkylosing Spondylitismedicine.medical_treatmentImmunologyInflammationSingle-nucleotide polymorphismDiseaseBiologyERAP1AminopeptidasesPolymorphism Single NucleotideMinor Histocompatibility AntigensYoung AdultGene expressionGeneticsmedicineHumansSpondylitis AnkylosingERAP1 Ankylosing SpondylitisEndoplasmic Reticulum Chaperone BiPSpondylitisHLA-B27 AntigenGenetics (clinical)InflammationAnkylosing spondylitisEndoplasmic reticulumMiddle AgedEndoplasmic Reticulum Stressmedicine.diseaseCytokineImmunologyFemalemedicine.symptomGenes & Immunity
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Antioxidant Activities and Oxidative Stress Byproducts in Human Hypertension

2003

The objective was to study oxidative status, antioxidant activities, and reactive oxygen species byproducts in whole blood and mononuclear peripherals cells and their relationship with blood pressure. Sixty-six hypertensive patients and 16 normotensive volunteers as a control group were studied. In both, whole blood and peripheral mononuclear cells oxidized/reduced glutathione ratio and malondialdehyde was significantly higher, and the activity of superoxide dismutase, catalase, and glutathione peroxidase was significantly lower in hypertensive patients when compared with normal subjects. The content of damaged base 8-oxo-2′-deoxyguanosine in nuclear and mitochondrial deoxyribonucleoprotein…

AdultMaleAntioxidantmedicine.medical_treatmentBlood PressureOxidative phosphorylationPharmacologymedicine.disease_causeDNA MitochondrialAntioxidantsMalondialdehydeInternal MedicinemedicineHumansWhole bloodchemistry.chemical_classificationGlutathione PeroxidaseReactive oxygen speciesGlutathione DisulfideSuperoxide DismutaseChemistryDeoxyguanosine8-Hydroxy-2'-deoxyguanosineDNAMetabolismMiddle AgedCatalaseGlutathioneDNA metabolismOxidative StressBiochemistry8-Hydroxy-2'-DeoxyguanosineHypertensionFemaleReactive Oxygen SpeciesOxidative stressHypertension
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