Search results for "pero"

showing 10 items of 3365 documents

Antenatal Steroids and Antioxidant Enzyme Activity in Preterm Infants: Influence of Gender and Timing

2009

Antenatal steroids have improved the survival of preterm infants; however, the mechanism of action is not fully understood. We aimed to establish an association between antenatal steroids and antioxidant activity and postnatal oxidative stress. In a prospective cohort study, extremely preterm neonates receiving antenatal steroids (CORT) or not (NOCORT) were enrolled. An association between antenatal steroids and activities of antioxidant enzymes and glutathione cycle enzymes in cord blood was found. In addition, reduced oxidative stress (GSH/GSSG ratio, CORT vs. NOCORT, 35.68 + or - 12.20 vs. 28.38 + or - 9.92; p < 0.01) and, decreased oxidation of proteins (ortho-tyrosine/phenylalanine rat…

AdultMalemedicine.medical_specialtyAntioxidantAdolescentPhysiologymedicine.medical_treatmentClinical BiochemistryBiologymedicine.disease_causeBiochemistryAntioxidantsYoung Adultchemistry.chemical_compoundSex FactorsAdrenal Cortex HormonesPregnancyInternal medicinemedicineHumansProspective StudiesProspective cohort studyMolecular BiologyGeneral Environmental SciencePregnancySuperoxide DismutaseInfant NewbornRetinopathy of prematurityCell BiologyGlutathioneCatalasemedicine.diseaseOxidative StressEndocrinologyBronchopulmonary dysplasiachemistryCord bloodGeneral Earth and Planetary SciencesFemaleInfant PrematureOxidative stressAntioxidants &amp; Redox Signaling
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Nitric oxide metabolites and oxidative stress in mild essential hypertension

2010

Many papers have showed non univocal data about oxidative stress status and nitric oxide metabolites in essential hypertension. Considering this preamble we examined the total antioxidant status (TAS), the lipid peroxidation (LP), expressed as thiobarbituric acid-reactive substances (TBARS), the stable end products of nitric oxide (NOx) and LP/NOx ratio in 25 subjects with untreated mild essential hypertension. The obtained data show a significant increase in TBARS (p < 0.001) and NOx (p < 0.001) in hypertensives and no variation in TAS and in TBARS/NOx ratio. None of these parameters was statistically related to the metabolic parameters or to the blood pressure values. The high level of li…

AdultMalemedicine.medical_specialtyAntioxidantPhysiologymedicine.medical_treatmentBlood PressureEssential hypertensionmedicine.disease_causeThiobarbituric Acid Reactive SubstancesAntioxidantsNitric oxideEssential hypertension.Lipid peroxidationchemistry.chemical_compoundPhysiology (medical)Internal medicinemedicineTBARSHumansNOxOxidate streNitric oxideHematologyMiddle Agedmedicine.diseaseOxidative StressBlood pressureEndocrinologychemistryBiochemistryHypertensionFemaleLipid PeroxidationCardiology and Cardiovascular MedicineOxidative stressClinical Hemorheology and Microcirculation
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Lipid peroxidation and nitric oxide metabolites in sedentary subjects and sportsmen before and after a cardiopulmonary test.

2012

Our aim was to investigate the effects of an exercise test on some indices of oxidative status and endothelial function, in trained and untrained subjects. We examined lipid peroxidation, nitric oxide metabolites (NOx) and their ratio before and after a cardiopulmonary test, using a cycloergometer. We enrolled 60 male subjects who practiced sport unprofessionally, subdivided in two groups (A and B) according to the values of VO2max. Group A included sportsmen with poor or fair aerobic fitness (VO2max 39 ml/Kg/min). The control group included 19 male sedentary subjects. Lipid peroxidation was evaluated by detection of the thiobarbituric acid-reactive substances (TBARS); the NOx were evaluate…

AdultMalemedicine.medical_specialtyAntioxidantPhysiologymedicine.medical_treatmentOxidative phosphorylationmedicine.disease_causeNitric OxideThiobarbituric Acid Reactive SubstancesNitric oxideLipid peroxidationchemistry.chemical_compoundGriess testPhysiology (medical)Internal medicineTBARSMedicineAerobic exerciseHumansNO exercise cardiopulmonary testbusiness.industryHematologyOxygenOxidative StressEndocrinologychemistryPhysical therapyExercise TestLipid PeroxidationSedentary BehaviorCardiology and Cardiovascular MedicinebusinessOxidative stressSportsClinical hemorheology and microcirculation
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Factors related to the impact of antihypertensive treatment in antioxidant activities and oxidative stress by-products in human hypertension

2004

The objective was to study factors related to the changes induced by antihypertensive treatment on oxidative status, antioxidant activities, and reactive oxygen species by-products in whole blood and mononuclear peripheral cells. Eighty-nine hypertensive patients (mean age 46 years, 46 men, average 24-h blood pressure 139/88 mm Hg, body mass index 29) were included. After 3 months of nonrandomized allocation to antihypertensive treatment (20 nonpharmacologic, 36 beta-blockers, 33 angiotensin receptor blocker), oxidized/reduced glutathione ratio and malondialdehyde were significantly reduced, and the activity of superoxide dismutase, catalase, and glutathione peroxidase was significantly inc…

AdultMalemedicine.medical_specialtyAntioxidantmedicine.medical_treatmentAngiotensin-Converting Enzyme Inhibitorsmedicine.disease_causeBenzoatesAntioxidantsSuperoxide dismutasechemistry.chemical_compoundInternal medicineInternal MedicinemedicineHumansTelmisartanAntihypertensive AgentsWhole bloodchemistry.chemical_classificationReactive oxygen speciesbiologybusiness.industryGlutathione peroxidaseGlutathioneMiddle AgedMalondialdehydeOxidative StressHydrochlorothiazideTreatment OutcomeEndocrinologyAtenololchemistryHypertensionbiology.proteinBenzimidazolesDrug Therapy CombinationFemalebusinessOxidative stressDNA DamageAmerican Journal of Hypertension
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Peripheral Oxidative Stress Markers in Patients with Bipolar Disorder during Euthymia and in Siblings.

2020

Aims:Oxidative stress is increased during the acute phases of bipolar disorder (BD). Our aim here was to analyze oxidative stress biomarkers in patients with BD during euthymia and their siblings.Method:A cross-sectional study was performed in euthymic patients with BD-I (n=48), unaffected siblings (n=23) and genetically unrelated healthy controls (n=21). Protein carbonyl content (PCC), total antioxidant capacity (TRAP), lipid peroxidation (TBARS) and uric acid were measured as biomarkers of oxidative stress in blood.Results:The antioxidant capacity (TRAP) was lower (p&lt;0.001) in patients with BD compared to their siblings and controls, whereas no differences were observed in PCC, TBARS o…

AdultMalemedicine.medical_specialtyBipolar DisorderEndocrinology Diabetes and MetabolismProtein Carbonyl Contentmedicine.disease_causeThiobarbituric Acid Reactive SubstancesLipid peroxidationProtein Carbonylation03 medical and health scienceschemistry.chemical_compound0302 clinical medicineAntimanic AgentsInternal medicineTBARSImmunology and AllergyMedicineHumansBipolar disorderValproic Acidbusiness.industrySiblingsValproic AcidMiddle Agedmedicine.disease030227 psychiatryUric AcidOxidative StressEndocrinologyCross-Sectional StudieschemistryCase-Control StudiesBiomarker (medicine)Uric acidFemaleLipid Peroxidationbusiness030217 neurology & neurosurgeryOxidative stressBiomarkersmedicine.drugEndocrine, metabolicimmune disorders drug targets
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Underexpressed Coactivators PGC1α AND SRC1 Impair Hepatocyte Nuclear Factor 4α Function and Promote Dedifferentiation in Human Hepatoma Cells

2006

Hepatocyte nuclear factor 4alpha (HNF4alpha) plays critical roles during liver development and in the transcriptional regulation of many hepatic genes in adult liver. Here we have demonstrated that in human hepatoma HepG2 cells, HNF4alpha is expressed at levels as high as in human liver but its activity on target genes is very low or absent. We have discovered that the low expression of key coactivators (PGC1alpha, SRC1, SRC2, and PCAF) might account for the lack of function of HNF4alpha in HepG2 cells. Among them, PGC1alpha and SRC1 are the two most important HNF4alpha coactivators as revealed by reporter assays with an Apo-CIII promoter construct. Moreover, the expression of these two coa…

AdultMalemedicine.medical_specialtyCarcinoma HepatocellularDown-RegulationBiologyBiochemistryNuclear Receptor Coactivator 1Cell Line TumorInternal medicinemedicineTranscriptional regulationHomeostasisHumansMolecular BiologyPsychological repressionHeat-Shock ProteinsAgedHistone AcetyltransferasesLiver NeoplasmsCell DifferentiationCell BiologyMiddle AgedPeroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alphaPhenotypeCell biologyNuclear receptor coactivator 1Hepatocyte nuclear factorsEndocrinologyHepatocyte Nuclear Factor 4LiverPCAFCell cultureFemaleHomeostasisTranscription FactorsJournal of Biological Chemistry
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Breathing 100% oxygen during water immersion improves postimmersion cardiovascular responses to orthostatic stress

2016

Abstract Physiological compensation to postural stress is weakened after long‐duration water immersion (WI), thus predisposing individuals to orthostatic intolerance. This study was conducted to compare hemodynamic responses to postural stress following exposure to WI alone (Air WI), hyperbaric oxygen alone in a hyperbaric chamber (O 2 HC), and WI combined with hyperbaric oxygen (O 2 WI), all at a depth of 1.35 ATA, and to determine whether hyperbaric oxygen is protective of orthostatic tolerance. Thirty‐two healthy men underwent up to 15 min of 70° head‐up tilt (HUT) testing before and after a single 6‐h resting exposure to Air WI ( N  = 10), O 2 HC ( N  = 12), or O 2 WI ( N  = 10). Heart …

AdultMalemedicine.medical_specialtyCardiac outputPhysiologyDivingOrthostatic intoleranceHemodynamicsBlood PressureNeurological Conditions Disorders and Treatments03 medical and health sciencesOrthostatic vital signs0302 clinical medicinePhysiology (medical)Internal medicineHeart rateImmersionMedicineAutonomic nervous systemblood flowHumansOriginal ResearchPresyncopeRespiratory Conditions Disorder and DiseasesHyperbaric Oxygenationbusiness.industryRespirationheart rate variabilityHeartStroke Volume030229 sport sciencesmedicine.diseaseSurgeryOxygenmedicine.anatomical_structureBlood pressureAutonomic nervous system; Blood flow; Blood pressure; Heart rate variability; Hyperoxia; Orthostatic tolerance; Water immersion; Adult; Diving; Humans; Hyperbaric Oxygenation; Immersion; Male; Orthostatic Intolerance; Oxygen; Vascular Resistance; Blood Pressure; Respiration; Stroke Volume; Physiology; Physiology (medical)Settore ING-INF/06 - Bioingegneria Elettronica E InformaticaVascular resistanceCardiologyOrthostatic Intolerancehyperoxiawater immersionorthostatic toleranceVascular Resistancebusiness030217 neurology & neurosurgeryHumanPhysiological Reports
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AZT treatment induces molecular and ultrastructural oxidative damage to muscle mitochondria. Prevention by antioxidant vitamins.

1998

AIDS patients who receive zidovudine (AZT) frequently suffer from myopathy. This has been attributed to mitochondrial (mt) damage, and specifically to the loss of mtDNA. This study examines whether AZT causes oxidative damage to DNA in patients and to skeletal muscle mitochondria in mice, and whether this damage may be prevented by supranutritional doses of antioxidant vitamins. Asymptomatic HIV-infected patients treated with AZT have a higher urinary excretion (355+/-100 pmol/kg/d) of 8-oxo-7, 8-dihydro-2'-deoxyguanosine (8-oxo-dG) (a marker of oxidative damage to DNA) than untreated controls (asymptomatic HIV-infected patients) (182+/-29 pmol/kg/d). This was prevented (110+/-79 pmol/kg/d)…

AdultMalemedicine.medical_specialtyDNA damageAnti-HIV Agentsmedicine.medical_treatmentAscorbic AcidBiologyDNA MitochondrialAntioxidantsZidovudinechemistry.chemical_compoundMiceInternal medicinemedicineDeoxyguanosineAnimalsHumansVitamin Eheterocyclic compoundsMyopathyVitamin ESkeletal musclevirus diseasesDeoxyguanosineGeneral MedicineGlutathioneHydrogen PeroxideAscorbic acidMitochondria Musclemedicine.anatomical_structureEndocrinologychemistryBiochemistry8-Hydroxy-2'-Deoxyguanosinemedicine.symptomZidovudinemedicine.drugDNA DamageResearch ArticleThe Journal of clinical investigation
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Electrically stimulated axon reflexes are diminished in diabetic small fiber neuropathies.

2004

Axon reflex mediated flare depends on the density and the function of cutaneous C-fibers and may be impaired in diabetic neuropathy. We induced neurogenic axon reflex flare by intracutaneous electrical stimulation and analyzed size and intensity of the flare on the dorsum of the foot and ventral thigh with laser Doppler imaging (LDI). We investigated 12 diabetic subjects with small fiber neuropathies (SFNs), 5 diabetic subjects without neuropathy (NO-Ns), and 14 healthy control subjects. Five of the normal subjects were reassessed after 12 months. In comparing patients with SFN to control subjects, we found that SFN flare size but not the intensity of vasodilation (flux) was reduced on the …

AdultMalemedicine.medical_specialtyDiabetic neuropathyEndocrinology Diabetes and MetabolismNeural ConductionPainStimulationVasodilationNerve FibersDiabetic NeuropathiesReference ValuesDiabetes mellitusInternal medicineReflexInternal MedicinemedicineHumansNeurons AfferentAxonSkinbusiness.industryPeroneal NerveMiddle Agedmedicine.diseaseAxonsElectric StimulationIntensity (physics)medicine.anatomical_structureEndocrinologyReflexAxon reflexFemalebusinessDiabetes
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Liraglutide Reduces Oxidative Stress And Restores Heme Oxygenase-1 and Ghrelin Levels in Patients with Type 2 Diabetes: A Prospective Pilot Study

2014

Abstract Context: Liraglutide is a glucagon-like peptide-1 analog and glucose-lowering agent whose effects on cardiovascular risk markers have not been fully elucidated. Objective: We evaluated the effect of liraglutide on markers of oxidative stress, heme oxygenase-1 (HO-1), and plasma ghrelin levels in patients with type-2 diabetes mellitus (T2DM). Design and Setting: A prospective pilot study of 2 months' duration has been performed at the Unit of Diabetes and Cardiovascular Prevention at University of Palermo, Italy. Patients and Intervention(s): Twenty subjects with T2DM (10 men and 10 women; mean age: 57 ± 13 y) were treated with liraglutide sc (0.6 mg/d for 2 wk, followed by 1.2 mg/d…

AdultMalemedicine.medical_specialtyEndocrinology Diabetes and MetabolismClinical Biochemistry030209 endocrinology & metabolismContext (language use)Pilot ProjectsType 2 diabetes030204 cardiovascular system & hematologymedicine.disease_causeBiochemistry03 medical and health scienceschemistry.chemical_compound0302 clinical medicineEndocrinologyGlucagon-Like Peptide 1Internal medicineDiabetes mellitusMedicineHumansHypoglycemic AgentsProspective StudiesAgedbusiness.industryLiraglutideBiochemistry (medical)Liraglutide Heme oxygenase Lipid peroxidation Ghrelin Type-2 diabetesOriginal ArticlesLiraglutideMiddle Agedmedicine.diseaseGhrelinMetformin3. Good healthMetforminOxidative StressEndocrinologychemistryDiabetes Mellitus Type 2GhrelinDrug Therapy CombinationFemaleGlycated hemoglobinbusinessOxidative stressHeme Oxygenase-1medicine.drug
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