Search results for "phenotype"

showing 10 items of 1875 documents

Inhibitors of β-catenin affect the immuno-phenotype and functions of dendritic cells in an inhibitor-specific manner

2015

Many tumors are characterized by mutation-induced constitutive activation of β-catenin which promotes tumor growth and survival. Consequently, the development of specific β-catenin inhibitors for tumor therapy has come into the focus of drug development. β-Catenin was also shown to contribute to the tolerance-promoting function of unstimulated dendritic cells (DCs). In response to activation, DCs acquire potent T cell stimulatory capacity and induce profound tumor antigen-specific immune responses. Here we asked for effects of pre-clinically established β-catenin inhibitors (CCT-031374, iCRT-5, PNU-75654) on mouse bone marrow-derived (BM)DCs. All three inhibitors moderately increased surfac…

CD4-Positive T-LymphocytesLipopolysaccharides0301 basic medicineCell SurvivalOvalbuminT cellImmunologyPopulationAntineoplastic AgentsBone Marrow CellsMice Transgenicchemical and pharmacologic phenomena03 medical and health sciencesImmune systemmedicineAnimalsImmunology and AllergyeducationCells Culturedbeta CateninCell ProliferationPharmacologyCD86education.field_of_studyCD40biologyFollicular dendritic cellsCell growthhemic and immune systemsDendritic CellsCell biologyMice Inbred C57BLPhenotype030104 developmental biologymedicine.anatomical_structurebiology.proteinCytokinesCD80International Immunopharmacology
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WHOLE BODY IRRADIATION INDUCES IFN-γ PRODUCTION IN BALB/c MICE BY PREVENTING THE APPEARANCE OF A Vα14+NK T DOWNREGULATORY POPULATION

2000

Lymph node cells from TNCB-immune BALB/c mice fail to produce IFN-gamma when exposed to antigen in vitro. Conversely, lymph node cells of irradiated (550 rads) BALB/c mice produce IFN-gamma. Transfer experiments show that normal BALB/c mice contain cells which suppress IFN-gamma production. These downregulatory cells are CD4(+)alpha beta(+)and rearrange the invariant V alpha 14-J alpha 281 T cell receptor alpha chain, thus belonging to the NK T cell subset. Downregulatory cells probably act by producing IL-4 as their effect is blocked by mAb to IL-4.

CD4-Positive T-LymphocytesMalemedicine.drug_classCD8 AntigensReceptors Antigen T-Cell alpha-betaImmunologyPopulationWhole body irradiationAntigen-Presenting CellsDown-RegulationAntigens ProtozoanEnzyme-Linked Immunosorbent AssayCell SeparationMonoclonal antibodyBiochemistryBALB/cInterferon-gammaMiceAntigenmedicineAnimalsImmunology and AllergyeducationMolecular BiologyLymph nodeLeishmania majorMice Inbred BALB Ceducation.field_of_studybiologyChemistryT-cell receptorAntibodies MonoclonalHematologyFlow Cytometrybiology.organism_classificationMolecular biologyIn vitroKiller Cells NaturalPhenotypemedicine.anatomical_structureCD4 AntigensImmunologyInterleukin-4Lymph NodesSpleenCytokine
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PD-1 signalling in CD4+T cells restrains their clonal expansion to an immunogenic stimulus, but is not critically required for peptide-induced tolera…

2010

Summary The ultimate outcome of T-cell recognition of peptide–major histocompatibility complex (MHC) complexes is determined by the molecular context in which antigen presentation is provided. The paradigm is that, after exposure to peptides presented by steady-state dendritic cells (DCs), inhibitory signals dominate, leading to the deletion and/or functional inactivation of antigen-reactive T cells. This has been utilized in a variety of models providing peptide antigen in soluble form in the absence of adjuvant. A co-inhibitory molecule of considerable current interest is PD-1. Here we show that there is the opportunity for the PD-1/PD-L1 interaction to function in inhibiting the T-cell r…

CD4-Positive T-LymphocytesOvalbuminTransgeneProgrammed Cell Death 1 ReceptorImmunologyAntigen presentationMice TransgenicCell SeparationCD8-Positive T-LymphocytesBiologyLymphocyte ActivationMajor histocompatibility complexMiceImmune systemBlocking antibodyImmune ToleranceAnimalsImmunology and AllergyT-cell receptorOriginal ArticlesFlow CytometryAntigens DifferentiationPeptide FragmentsCell biologyMice Inbred C57BLTolerance inductionPhenotypeImmunologybiology.proteinCD8Signal TransductionImmunology
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Effect of female genital schistosomiasis and anti-schistosomal treatment on monocytes, CD4+ T-cells and CCR5 expression in the female genital tract

2014

Published version of an article from the journal: PLoS One. Also available from the publisher: http://dx.doi.org/10.1371/journal.pone.0098593 BACKGROUND: Schistosoma haematobium is a waterborne parasite that may cause female genital schistosomiasis (FGS), characterized by genital mucosal lesions. There is clinical and epidemiological evidence for a relationship between FGS and HIV. We investigated the impact of FGS on HIV target cell density and expression of the HIV co-receptor CCR5 in blood and cervical cytobrush samples. Furthermore we evaluated the effect of anti-schistosomal treatment on these cell populations. DESIGN: The study followed a case-control design with post treatment follow…

CD4-Positive T-LymphocytesViral DiseasesGynecologic InfectionsVDP::Medical disciplines: 700::Clinical medical disciplines: 750::Tropical medicine: 761Gene Expressionlcsh:MedicineGlobal HealthMonocytesPraziquantelWhite Blood CellsImmunodeficiency VirusesAnimal CellsMedicine and Health SciencesSchistosomiasisPublic and Occupational Healthlcsh:ScienceT CellsCoinfectionObstetrics and GynecologyGenitalia FemaleAIDSInfectious DiseasesPhenotypeMedical MicrobiologyHelminth InfectionsViral PathogensSchistosoma haematobiumFemaleCellular TypesResearch ArticleNeglected Tropical DiseasesAdultAdolescentReceptors CCR5Immune CellsUrologyImmunologySexually Transmitted DiseasesMicrobiologyImmunophenotypingYoung AdultParasitic DiseasesAnimalsHumansMicrobial PathogensBlood CellsGenitourinary Infectionslcsh:RBiology and Life SciencesHIVCell BiologyTropical DiseasesCase-Control StudiesWomen's HealthClinical Immunologylcsh:QGenital Diseases Female
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Atypical Human Effector/Memory CD4(+) T Cells With a Naive-Like Phenotype

2018

The induction of adaptive immunological memory, mediated by T and B cells, plays an important role in protective immunity to pathogens induced by previous infections or vaccination. Naive CD4+ T cells that have been primed by antigen develop into memory or effector cells, which may be distinguished by their capability to exert a long-term and rapid response upon re-challenge by antigen, to produce distinct cytokines and surface marker expression phenotypes such as CD45RA/RO, CD27, CD62L, and CCR7. Moreover, a distinct lineage of memory T cells populates tissues (tissue-resident memory T cells or TRM cells) which orchestratea the response to pathogens re encountered at tissue sites. Recent e…

CD4-Positive T-Lymphocyteslcsh:Immunologic diseases. Allergy0301 basic medicineNaive T cellMini Reviewmedicine.medical_treatmentT cellImmunologyBiologyTranscriptomeimmunological memoryM. tuberculosis infectionCD4+ T cell03 medical and health scienceseffector T cellsnaive T cellImmune systemAntigenT-Lymphocyte Subsetseffector T cellCD4(+) T cellscytokinemedicineAnimalsHumansImmunology and AllergyEffectorCell DifferentiationPhenotypeCD4+ T cellscytokinesinfection3. Good healthCell biologynaive T cellsPhenotype030104 developmental biologyCytokinemedicine.anatomical_structurelcsh:RC581-607Immunologic MemoryBiomarkers
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Yeasts vectored by migratory birds collected in the Mediterranean island of Ustica and description of Phaffomyces usticensis f.a. sp. nov., a new spe…

2014

Nine yeast species belonging to genera Candida , Cryptococcus , Phaffomyces , Rhodotorula and Wickerhamomyces , and one species of Aureobasidium genus were isolated from the cloaca of migratory birds. Candida glabrata and C. inconspicua were the species most frequently isolated and Wickerhamomyces sylviae , which has recently been described as a new species isolated from bird cloaca, was again found. The majority of isolates showed the ability to grow up to 40 °C and/or at pH 3.0, two environmental conditions typical of the digestive tract of birds. The phylogenetic analysis of the D1/D2 domain of 26S rRNA gene placed the cultures of Phaffomyces in a new lineage that differed from the close…

Cactaceaephenotypic characterizationMolecular Sequence DataCryptococcusAureobasidiumRhodotorulaApplied Microbiology and BiotechnologyMicrobiologyNew species Yeast birdsBirdscactus-yeastMediterranean IslandsWickerhamomycesAscomycotaGenusPhaffomyces usticensis sp. novBotanyAnimalsPhylogenyRibosomalPhylogenetic treebiologyCandida glabrataphylogenetic analysisGeneral Medicinebiology.organism_classificationnovel speciesDelichon urbicumSettore AGR/11 - Entomologia Generale E ApplicataPhenotypeRNA RibosomalCactusRNADelichon urbicum; Phaffomyces usticensis sp. nov.; cactus-yeast; novel species; phenotypic characterization; phylogenetic analysis; Animals; Ascomycota; Birds; Cactaceae; Mediterranean Islands; Molecular Sequence Data; Phenotype; Phylogeny; RNA RibosomalSettore AGR/16 - Microbiologia Agraria
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The Role of Cadherins in Ca2+-Mediated Cell Adhesion and Inherited Photoreceptor Degeneration

2002

Cadherins are Ca2+-binding, transmembrane proteins involved in cell adhesion. Recently, three cadherin molecules, cadherin-23, protocadherin-15, and cadherin-3, were found to be defective in various human diseases, many of them with photoreceptor degeneration and/or sensorineural hearing loss as major features such Usher syndrome type 1D (USH1D), USH1F, and hypotrichosis with juvenile macular dystrophy. The process, by which mutations lead to photoreceptor degeneration is still not fully understood. Data from the inner ear phenotype of USH1 mouse models suggest that loss of cell adhesion is a crucial event.

CadherinUsher syndromeMacular dystrophyBiologymedicine.diseasePhenotypeTransmembrane proteinCell biologyRetinitis pigmentosaotorhinolaryngologic diseasesmedicineHypotrichosissense organsCell adhesion
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Localization of quantitative trait loci for diapause and other photoperiodically regulated life history traits important in adaptation to seasonally …

2015

Seasonally changing environments at high latitudes present great challenges for the reproduction and survival of insects, and photoperiodic cues play an important role in helping them to synchronize their life cycle with prevalent and forthcoming conditions. We have mapped quantitative trait loci (QTL) responsible for the photoperiodic regulation of four life history traits, female reproductive diapause, cold tolerance, egg-to-eclosion development time and juvenile body weight in Drosophila montana strains from different latitudes in Canada and Finland. The F2 progeny of the cross was reared under a single photoperiod (LD cycle 16:8), which the flies from the Canadian population interpret a…

CanadaGenotypeGenetic LinkagePhotoperioddevelopment timeQuantitative Trait Locijuvenile body weightBiologyDiapauseQuantitative trait locusPolymorphism Single NucleotideLife history theoryGene interactionDrosophila montanaGenetic linkageGeneticsJuvenileAnimalsEcology Evolution Behavior and SystematicsCrosses GeneticFinlandGeneticsphotoperiodismta1184food and beveragescold tolerancediapauseGenetics PopulationPhenotypeEvolutionary biologyta1181EpistasisDrosophilaFemaleSeasonsAdaptation
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Functional characterisation of alpha-galactosidase a mutations as a basis for a new classification system in fabry disease.

2013

Fabry disease (FD) is an X-linked hereditary defect of glycosphingolipid storage caused by mutations in the gene encoding the lysosomal hydrolase α-galactosidase A (GLA, α-gal A). To date, over 400 mutations causing amino acid substitutions have been described. Most of these mutations are related to the classical Fabry phenotype. Generally in lysosomal storage disorders a reliable genotype/phenotype correlation is difficult to achieve, especially in FD with its X-linked mode of inheritance. In order to predict the metabolic consequence of a given mutation, we combined in vitro enzyme activity with in vivo biomarker data. Furthermore, we used the pharmacological chaperone (PC) 1-deoxygalacto…

Cancer Research1-Deoxynojirimycinlcsh:QH426-470Nonsense mutationMutantBiologymedicine.disease_causeGeneticsmedicineHumansBiologyMolecular BiologyGenetics (clinical)Ecology Evolution Behavior and SystematicsGeneticsSphingolipidsMutationAlpha-galactosidasePoint mutationmedicine.diseasePhenotypeFabry diseasePharmacological chaperoneProtein Transportlcsh:GeneticsPhenotypeAmino Acid Substitutionalpha-GalactosidaseMutationComputer Sciencebiology.proteinFabry DiseaseMedicineGlycolipidsResearch Articlemedicine.drugPLoS Genetics
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CD90+ liver cancer cells modulate endothelial cell phenotype through the release of exosomes containing H19 lncRNA

2015

Background CD90+ liver cancer cells have been described as cancer stem-cell-like (CSC), displaying aggressive and metastatic phenotype. Using two different in vitro models, already described as CD90+ liver cancer stem cells, our aim was to study their interaction with endothelial cells mediated by the release of exosomes. Methods Exosomes were isolated and characterized from both liver CD90+ cells and hepatoma cell lines. Endothelial cells were treated with exosomes, as well as transfected with a plasmid containing the full length sequence of the long non-coding RNA (lncRNA) H19. Molecular and functional analyses were done to characterize the endothelial phenotype after treatments. Results …

Cancer ResearchAngiogenesisAngiogenesis; CD90+ liver cancer cells; Exosomes; Long-non-coding RNA H19; Antigens Thy-1; Cell Adhesion; Cell Line Tumor; Endothelial Cells; Exosomes; Human Umbilical Vein Endothelial Cells; Humans; Liver Neoplasms; RNA Long Noncoding; Phenotype; Molecular Medicine; Oncology; Cancer ResearchBiologyCD90+ liver cancer cellsExosomesCell LineSettore BIO/13 - Biologia ApplicataCancer stem cellCell Line TumormedicineCell AdhesionHuman Umbilical Vein Endothelial CellsHumansCD90AntigensThy-1TumorExosomes Long-non-coding RNA H19 CD90+ liver cancer cells AngiogenesisResearchLiver NeoplasmsCancerEndothelial Cellsmedicine.diseaseMicrovesiclesCell biologyEndothelial stem cellPhenotypeOncologyembryonic structuresThy-1 AntigensRNAMolecular MedicineRNA Long NoncodingLong NoncodingAngiogenesisStem cellLiver cancerLong-non-coding RNA H19Molecular Cancer
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