Search results for "poisoning"

showing 10 items of 76 documents

The oxygen status of arterial human blood

1990

The oxygen status of arterial human blood is described at least by four variables: Oxygen partial pressure (pO2, mmHg), oxygen saturation (sO2, %), hemoglobin content (cHb, g/dL) and oxygen content (cO2, mL/dL). Beside perfusion, however, the oxygen supply of all organs is decisively determined by the mean capillary pO2 which itself is primarily dependent on the arterial cO2. Therefore, the oxygen availability (cardiac output x caO2, mL/min) may be described by the cO2 value in arterial blood or those variables who determine the latter one. The diagnostic significance of the O2 variables of the oxygen status consequently increases in the order of pO2, sO2 (cHb) and cO2. In arterial blood, o…

Lung Diseasesmedicine.medical_specialtyPartial PressureClinical BiochemistryPoison controlchemistry.chemical_elementOxygenMethemoglobinHypoxemiaCarbon Monoxide PoisoningHemoglobinsInternal medicinemedicineHumansOximetryCardiac OutputOxygen saturationMethemoglobinCarbon monoxide poisoningbusiness.industryAnemiaArteriesGeneral Medicinemedicine.diseaseCapillariesrespiratory tract diseasesOxygenchemistryAnesthesiaCardiologyArterial bloodHemoglobinBlood Gas Analysismedicine.symptombusinessScandinavian Journal of Clinical and Laboratory Investigation
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A fatal case of a paint thinner ingestion: Comparison between toxicological and histological findings

2010

Toluene and xylene are aromatic hydrocarbons commonly used as an industrial solvent for the manufacturing of pharmaceuticals, paints, and chemicals. The Occupational Safety and Health Administration has determined that toluene levels of 2000 parts per million (ppm) are considered dangerous to life and health. Several studies have examined the absorption of toluene and xylene following inhalation and oral ingestion in humans. Volatile organic compounds that are absorbed into the blood are distributed throughout the body; in particular, distribution of absorbed toluene and xylene in humans and rodents is characterized by preferential uptake in well-perfused and lipophil tissues such as the br…

MaleAdolescent2734XylenePoison controlPaint thinnerBrain EdemaHemorrhagePulmonary EdemaAbsorption (skin)XylenesEsophaguKidneyGas Chromatography-Mass SpectrometryPathology and Forensic Medicinetoluene xilene paint thinner ingestion self poisoning varnish-diluting solventsToxicologychemistry.chemical_compoundForensic ToxicologyEsophagusSettore MED/43 - Medicina LegalePaint thinner ingestionSelf poisoningIngestionHumansGastrointestinal ContentForensic PathologyLungSolid Phase MicroextractionChromatographyChemistryXyleneForensic toxicologyBrainTolueneGastrointestinal ContentsSuicideLiverSolventSolventsPaint thinner ingestion; Self poisoning; Toluene; Varnish-diluting solvents; Xylene; Adolescent; Brain; Brain Edema; Esophagus; Forensic Pathology; Forensic Toxicology; Gas Chromatography-Mass Spectrometry; Gastrointestinal Contents; Hemorrhage; Humans; Kidney; Liver; Lung; Male; Pulmonary Edema; Solid Phase Microextraction; Solvents; Toluene; Xylenes; Suicide; 2734Varnish-diluting solventGas chromatography–mass spectrometryHumanToluene
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The influence of erdosteine administration on lead-induced oxidative stress in rat muscle.

2019

Lead-exposure is known to disrupt the redox balance of tissues leading to oxidative stress. Due to the fact that a mucolytic drug, erdosteine, exerts also antioxidant properties, we decided to perform a pilot study on rats to evaluate its therapeutic potency in lead poisoning. Male Wistar rats were divided randomly into the following seven groups having 10 animals in each. Group I served as the control group. During 8-week period, rats in groups II-IV, except standard alimentation, received: erdosteine in a dose 350 mg/kg (collateral control group), 1200 ppm of lead acetate in drinking water and placebo, as well as the same doses of lead and erdosteine, respectively. Rats in group V-VII rec…

MaleAntioxidantErdosteinemuscleHealth Toxicology and Mutagenesismedicine.medical_treatmentErdosteinePilot ProjectsThiophenes010501 environmental sciencesPharmacologyToxicologymedicine.disease_cause01 natural sciencesLead poisoningAntioxidants03 medical and health sciences0302 clinical medicineMalondialdehydemedicineAnimalsRats WistarLead (electronics)0105 earth and related environmental sciencesPharmacologyChemical Health and SafetyChemistrySuperoxide DismutaseMusclesPublic Health Environmental and Occupational Healthlead poisoningGeneral Medicinemedicine.diseaseCatalaseRatsOxidative StressLeadThioglycolates030217 neurology & neurosurgeryOxidative stressmedicine.drugDrug and chemical toxicology
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Chemoimmunotherapy with methotrexate, cytarabine, thiotepa, and rituximab (MATRix regimen) in patients with primary CNS lymphoma: results of the firs…

2016

BACKGROUND: Standard treatment for patients with primary CNS lymphoma remains to be defined. Active therapies are often associated with increased risk of haematological or neurological toxicity. In this trial, we addressed the tolerability and efficacy of adding rituximab with or without thiotepa to methotrexate-cytarabine combination therapy (the MATRix regimen), followed by a second randomisation comparing consolidation with whole-brain radiotherapy or autologous stem cell transplantation in patients with primary CNS lymphoma. We report the results of the first randomisation in this Article.METHODS: For the international randomised phase 2 International Extranodal Lymphoma Study Group-32 …

MaleComparative Effectiveness ResearchTransplantation ConditioningGastrointestinal DiseasesDenmarkMedizinKaplan-Meier EstimateDexamethasoneCentral Nervous System NeoplasmsDeath Sudden0302 clinical medicineIntraocular LymphomaGermanyAntineoplastic Combined Chemotherapy ProtocolsMedicineStandard treatmentOptic Nerve NeoplasmsPoisoningRemission InductionCytarabineHematopoietic Stem Cell TransplantationAnemiaHematologyInduction ChemotherapyAcute Kidney InjuryMiddle AgedCombined Modality TherapyMagnetic Resonance Imaging3. Good healthStrokeTreatment OutcomeTolerabilityItaly030220 oncology & carcinogenesischemoimmunotherapyRituximabFemaleNeurotoxicity SyndromesChemical and Drug Induced Liver InjuryRituximabSwitzerlandmedicine.drugMucositismedicine.medical_specialtyLymphoma B-CellNeutropeniaThioTEPAInfectionsTransplantation AutologousDisease-Free Survival03 medical and health sciencesprimary CNS lymphomaChemoimmunotherapyInternal medicineJournal Articleprimary CNS lymphoma chemoimmunotherapyHumansbusiness.industryThrombosismedicine.diseaseThrombocytopeniaUnited KingdomSurgeryTransplantationRegimenMethotrexateHeart InjuriesHyperglycemiaRadiotherapy Adjuvantbusiness030217 neurology & neurosurgeryFebrile neutropeniaThiotepaFollow-Up StudiesThe Lancet. Haematology
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Relationship between lead absorption and iron status and its association with oxidative stress markers in lead-exposed workers

2021

Background: The emission of lead (Pb) occurring during the extraction, processing and industrial applications of this element remains a significant environmental risk factor. The absorbability of lead in humans is strongly associated with the general health status of exposed individuals. Existing mineral deficiencies are considered being a predisposition to an increased Pb uptake. Both, iron deficiency and lead poisoning are the major caus-ative factors responsible for the prevalence of anemia within the vulnerable population, especially in children. Although some of the intervention programs of counteracting lead poisoning by iron supplementation proved to be effective in the Pb-exposed po…

MaleExposed PopulationAnemiaIronPopulationPhysiologymedicine.disease_causeBiochemistryAntioxidantsLead poisoningInorganic ChemistryLead exposurechemistry.chemical_compoundmedicineHumansChildeducationeducation.field_of_studymedicine.diagnostic_testbusiness.industryIron statusZinc protoporphyrinROSIron DeficienciesIron deficiencymedicine.diseaseLead PoisoningOxidative StressBlood leadLeadchemistrySerum ironMolecular MedicinebusinessBiomarkersOxidative stressJournal of Trace Elements in Medicine and Biology
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Pancuronium improves the neuromuscular transmission defect of human organophosphate intoxication.

1990

Two patients with acute severe organophosphate intoxication showed (1) single evoked compound muscle action potentials (CMAP) with repetitive discharges and (2) prominent decremental responses of CMAP with 20 and 50 Hz supramaximal nerve stimulation. Following the intravenous injection of single small doses of pancuronium, marked improvement in these abnormalities occurred and persisted for several hours. We postulate that the physiologic improvement following low-dose pancuronium results from blockade of acetylcholine receptors, especially those located on the terminal axon responsible for antidromic backfiring.

MaleInsecticidesNeuromuscular transmissionNeuromuscular JunctionAction PotentialsSuicide AttemptedElectromyographyNeurotransmissionIsoindolesOrganophosphate poisoningSynaptic TransmissionNeuromuscular junctionOrganophosphate PoisoningmedicineHumansPancuroniumAxonAcetylcholine receptormedicine.diagnostic_testParathionbusiness.industryMusclesOrganothiophosphatesOrganothiophosphorus Compoundsmedicine.diseaseAntidromicMedian Nervemedicine.anatomical_structureAnesthesiaNeurology (clinical)businessNeurology
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Near fatal percutaneous paraquat poisoning

1983

A fatal paraquat poisoning can occur when relatively large areas of skin are contaminated with a concentrated solution of paraquat (Gramoxone). A paraquat absorption takes place of the same magnitude as that with an equal dose per os. In the presence of mechanical or chemical lesion of the skin the percutaneous paraquat absorption is distinctly enhanced.

MaleParaquatinorganic chemicalsTime FactorsPercutaneousAccident preventionSkin AbsorptionPoison controlAbsorption (skin)LesionNecrosischemistry.chemical_compoundParaquatDrug DiscoveryAnimalsHumansMedicineheterocyclic compoundsGenetics (clinical)Skinintegumentary systembusiness.industryRats Inbred StrainsGeneral MedicinePARAQUAT POISONINGRatschemistryAccidents HomeChild PreschoolAnesthesiaMolecular MedicineDermal toxicityFemalemedicine.symptombusinessKlinische Wochenschrift
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A possible biomarker for methadone related deaths

2017

Abstract Methadone (MTH) concentrations in those dying of MTH toxicity totally overlap concentrations where the presence of MTH is only an incidental finding, making it very difficult to make distinctions in actual cases. A biomarker, be it anatomical or biochemical for MTH toxicity is badly needed, particularly if that markers were known to disrupt effective ventilation. Because the brainstem houses the regulatory centers for cardiorespiratory-control enters, it would seem to be the most likely anatomical site to seek abnormalities in cardiorespiratory control. Objective To locate and describe the cells of nucleus of the solitary tract (TS)(NTS) in human brainstem and determine if neuronal…

MalePathologyNecrosisApoptosisAutopsyCohort Studies0302 clinical medicineRetrospective StudieMedicineForensic PathologyNeuronsPoisoningSolitary tractGeneral MedicineRostral ventrolateral medullaNecrosiImmunohistochemistryCaspase 9Narcotic030220 oncology & carcinogenesisToxicityFemaleBrainstemmedicine.symptomBrainstemCaspase-9HumanNarcoticsAdultProgrammed cell deathmedicine.medical_specialty2734Pathology and Forensic MedicineNecrosisForensic ToxicologyYoung Adult03 medical and health sciencesSettore MED/43 - Medicina LegaleSolitary NucleuSolitary NucleusNeurotoxicityHumansRetrospective Studiesbusiness.industryfungiApoptosiBiomarkerNeuronApoptosisApoptosis; Biomarker; Brainstem; Caspase-9; Methadone; Neurotoxicity; Adult; Apoptosis; Brain Stem; Caspase 9; Cohort Studies; Female; Forensic Pathology; Forensic Toxicology; Humans; Immunohistochemistry; Male; Methadone; Narcotics; Necrosis; Neurons; Poisoning; Retrospective Studies; Solitary Nucleus; Young Adult; 2734; LawCohort StudiebusinessLawMethadone030217 neurology & neurosurgeryBrain Stem
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TIMP expression in toxic and cholestatic liver injury in rat.

1997

Abstract Background/Aims: Hepatic fibrosis is a dynamic pathological process with a net accumulation of extracellular matrix proteins. Recent evidence suggests that besides their increased synthesis, inhibition of matrix degradation plays a significant role. ECM degradation occurs via metalloproteinases which are inhibited in situ by specific tissue inhibitors of metalloproteinases (TIMPs). The aim of our studies was to determine the expression of TIMPs during toxic liver injury and cholestatic liver injury leading to fibrosis. Methods: We examined the expression of TIMP-1, -2 and -3 in two different rat models for liver injury (intraperitoneal CCl 4 injection and bile duct ligation) by Nor…

MalePathologymedicine.medical_specialtyIn situ hybridizationCholestasis IntrahepaticMatrix metalloproteinaseBiologyRats Sprague-DawleyCholestasisFibrosisInternal medicinemedicineAnimalsNorthern blotIn Situ HybridizationLiver injuryTissue Inhibitor of Metalloproteinase-3Tissue Inhibitor of Metalloproteinase-2Tissue Inhibitor of Metalloproteinase-1HepatologyCarbon Tetrachloride PoisoningAcute-phase proteinTissue Inhibitor of Metalloproteinasesmedicine.diseaseRatsEndocrinologyLiverChemical and Drug Induced Liver InjuryHepatic fibrosisJournal of hepatology
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Endothelial function and serum concentration of toxic metals in frequent consumers of fish.

2014

BACKGROUND:Endothelial dysfunction is involved in the pathogenesis of atherosclerosis. Consumption of fish is associated with reduced cardiovascular risk, but there is paucity of data concerning its effect on endothelial function. Furthermore, investigation of the effects of fish consumption on health must take into account the ingestion of contaminants, including transition metals and some metalloids, which may have unfavorable effects on health, including those on the cardiovascular system. We investigated the association between fish consumption, endothelial function (flow mediated dilation of the brachial artery), and serum concentration of some toxic metals in apparently healthy people…

MaleSettore MED/09 - Medicina Internalcsh:MedicineToxicologyHeavy MetalsCarotid Intima-Media ThicknessVascular MedicineRisk FactorsMedicine and Health SciencesIngestionToxinsSettore MED/49 - Scienze Tecniche Dietetiche ApplicateEndothelial dysfunctionlcsh:ScienceMultidisciplinaryPoisoningFishesMiddle Agedmedicine.anatomical_structureFemaleResearch ArticleAdultmedicine.medical_specialtyEndotheliumToxic Agentschemistry.chemical_elementFood ContaminationBiologyPeripheral Arterial DiseaseInsulin resistanceDiabetes mellitusInternal medicineMetals HeavymedicineAnimalsHumansArsenicNutritionlcsh:RBiology and Life SciencesFeeding Behaviormedicine.diseaseAtherosclerosisMercury (element)DietHeavy Metal PoisoningEndocrinologychemistrylcsh:Qendothelial function fish toxic metals cardiovascular riskEndothelium VascularInsulin ResistanceSeleniumPloS one
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