Search results for "polyneuropathy"

showing 10 items of 54 documents

F-Wave study in patients with chronic renal failure on regular haemodialysis

1986

Motor nerve conduction along the entire length of ulnar and tibialis posterior nerves was studied in 30 uraemic patients and in 20 control subjects. The M and F latencies, MNCV (between the stimulus sites), FWCV (between the spinal cord and the stimulus sites) and F-ratio (conduction time ratio of proximal to distal segment) were assessed to evaluate the conduction in the proximal versus the distal nerve segment. In the uraemic patients, the slowing of nerve conduction involved both segments of the tested nerves to the same extent. In fact, the F-ratio did not show any significant difference between the two groups; in only one patient was its value lower than the normal limit in the tibiali…

AdultMaleResuscitationAdolescentNeural ConductionMotor nerveStimulus (physiology)Nerve conduction velocityF waveRenal DialysisReaction TimeHumansMedicineAgedMotor NeuronsNeural Conductionbusiness.industryAnatomyMiddle AgedSpinal cordmedicine.diseasemedicine.anatomical_structureNeurologyKidney Failure ChronicFemaleNeurology (clinical)businessPolyneuropathyJournal of the Neurological Sciences
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Neuromuscular electrophysiological disorders and muscle atrophy in mechanically-ventilated traumatic brain injury patients: New insights from a prosp…

2018

International audience; Erratum inCorrigendum to "Neuromuscular electrophysiological disorders and muscle atrophy in mechanically-ventilated traumatic brain injury patients: New insights from a prospective observational study" [J Crit Care 44 (2018) 87-94]. [J Crit Care. 2019]AbstractPURPOSE:It is unclear whether the muscular changes in mechanically-ventilated traumatic brain injury patients (TBI) are only associated with disuse or additionally to neuromuscular electrophysiological disorders (NED). The correlation between muscle atrophy and NED may affect functional outcomes and rehabilitation programs significantly.MATERIAL AND METHODS:An observational study was performed to investigate th…

AdultMaleTraumatic brain injuryCritical Illnessmedicine.medical_treatmentNeuromuscular disorderCritical Care and Intensive Care MedicineBicepsNeuromuscular electrical stimulationlaw.invention03 medical and health sciences0302 clinical medicinelawChronaxieBrain Injuries TraumaticmedicineHumansProspective StudiesCritical illness polyneuropathyMuscle SkeletalProspective cohort studyMechanical ventilationAnalysis of VarianceRehabilitationIntensive care unit-acquired weaknessbusiness.industryRehabilitation030208 emergency & critical care medicineNeuromuscular DiseasesMiddle Agedmedicine.diseaseRespiration ArtificialIntensive care unitMuscle atrophy3. Good healthIntensive Care UnitsMuscular Atrophy[ SDV.NEU ] Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]AnesthesiaFemale[SDV.NEU]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]medicine.symptombusiness[SDV.MHEP]Life Sciences [q-bio]/Human health and pathology030217 neurology & neurosurgeryCritical illness polyneuropathy
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Sensory phenotype and risk factors for painful diabetic neuropathy: a cross-sectional observational study.

2017

Different sensory profiles in diabetic distal symmetrical sensory-motor polyneuropathy (DSPN) may be associated with pain and the responsiveness to analgesia. We aimed to characterize sensory phenotypes of patients with painful and painless diabetic neuropathy and to assess demographic, clinical, metabolic, and electrophysiological parameters related to the presence of neuropathic pain in a large cohort of well-defined DSPN subjects. This observational cross-sectional multi-center cohort study (performed as part of the ncRNAPain EU consortium) of 232 subjects with nonpainful (n = 74) and painful (n = 158) DSPN associated with diabetes mellitus of type 1 and 2 (median age 63 years, range 21-…

AdultMalemedicine.medical_specialtyDiabetic neuropathyAnalgesic030209 endocrinology & metabolismNeurological examinationCohort Studies03 medical and health sciencesPolyneuropathiesYoung Adult0302 clinical medicineDiabetic NeuropathiesRisk FactorsInternal medicineDiabetes mellitusmedicineHumansAgedAged 80 and overNeurologic Examinationmedicine.diagnostic_testbusiness.industryMiddle Agedmedicine.diseaseAnesthesiology and Pain MedicineCross-Sectional StudiesPhenotypeNeurologyNeuropathic painPhysical therapyNeuralgiaPain catastrophizingFemaleNeurology (clinical)businessPolyneuropathy030217 neurology & neurosurgeryCohort studyPain
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Heterogeneous presentation of caspr2 antibody‐associated peripheral neuropathy – A case series

2019

Contactin-associated protein 2-like (caspr2) antibodies have been discovered recently. Since then a multitude of patients with caspr2 antibodies presenting with different neurological symptoms have been reported. Here, we describe three patients with caspr2 antibodies with different types of pain/no pain in combination with peripheral neuropathy. The first patient, a 33-year-old woman, presented with erythromelalgia-like pain and autonomic symptoms; the second patient, a 58-year-old man, with paresthesia and pain while walking together with signs of peripheral motor neuron hyperexcitability in combination with optic neuritis, and the third patient, a 74-year-old man, without any pain but wi…

AdultMalemedicine.medical_specialtyEncephalopathyPain03 medical and health sciences0302 clinical medicineHumansMedicinePeripheral motor neuronOptic neuritis030212 general & internal medicineAgedAutoantibodiesbiologybusiness.industryAutoantibodyPeripheral Nervous System DiseasesMiddle Agedmedicine.diseaseDermatologyAnesthesiology and Pain MedicinePeripheral neuropathybiology.proteinFemalePresentation (obstetrics)AntibodybusinessPolyneuropathy030217 neurology & neurosurgeryEuropean Journal of Pain
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Myo-, neuro-, gastrointestinal encephalopathy (MNGIE syndrome) due to partial deficiency of cytochrome-c-oxidase

1987

A 42-year-old woman had a 10-year history of external ophthalmoplegia, malabsorption resulting in chronic malnutrition, muscle atrophy and polyneuropathy. Computer tomography revealed hypodensity of her cerebral white matter. A metabolic disturbance consisted of lactic acidosis after moderate glucose loads with increased excretion of hydroxybutyric and fumaric acids. Post-mortem studies revealed gastrointestinal scleroderma as the morphological manifestation of her malabsorption syndrome, ocular and skeletal myopathy with ragged red fibers, peripheral neuropathy, vascular abnormalities of meningeal and peripheral nerve vessels. Biochemical examination of the liver and muscle tissues reveale…

AdultPathologymedicine.medical_specialtyMalabsorptionGastrointestinal DiseasesEncephalopathyRespiratory chainCytochrome-c Oxidase DeficiencyEyePathology and Forensic Medicine03 medical and health sciencesCellular and Molecular Neuroscience0302 clinical medicineMuscular DiseasesMitochondrial myopathymedicineHumansMuscular dystrophy030304 developmental biology2. Zero hungerBrain Diseases0303 health sciencesbusiness.industryPeripheral Nervous System DiseasesSyndromemedicine.diseaseMitochondria MusclePeripheral neuropathyLactic acidosisFemaleNeurology (clinical)businessPolyneuropathy030217 neurology & neurosurgeryActa Neuropathologica
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Evaluation of carpal tunnel syndrome in patients with polyneuropathy

1997

The difference between the median nerve latency to the second lumbrical muscle and the ulnar nerve latency to the second interosseous muscle (L-I DIFF) was tested in a prospective study to discriminate whether prolonged distal motor latency of the median nerve in patients with polyneuropathy (PNP) reflects an additional carpal tunnel syndrome (CTS). We investigated 92 patients (107 hands) with CTS, 30 patients (34 hands) with PNP, 22 patients (27 hands) with CTS and coexisting PNP (PNP+CTS), and 77 controls (87 hands). L-I DIFF was significantly prolonged in both the CTS and PNP+CTS patients as compared to PNP patients and controls. It proved to be the most specific test to differentiate be…

Adultinorganic chemicalsmedicine.medical_specialtyPhysiologyNeural ConductionNerve conduction velocityCellular and Molecular NeurosciencePhysiology (medical)medicineHumansheterocyclic compoundsIn patientNeurons AfferentProspective cohort studyUlnar nerveCarpal tunnel syndromeUlnar NerveAgedMotor Neuronsmedicine.diagnostic_testbusiness.industryMiddle Agedmedicine.diseaseCarpal Tunnel SyndromeMedian nerveMedian Nervenervous system diseasesSurgeryenzymes and coenzymes (carbohydrates)Evaluation Studies as TopicNerve conduction studyNeurology (clinical)businessPolyneuropathyDemyelinating DiseasesMuscle & Nerve
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Familial Amyloid Polyneuropathy

2013

Familial amyloid polyneuropathy (FAP; also known as familiar amyloidosis and hereditary amyloidosis) is an autosomal dominant inherited disease due to mutations of the transthyretin (TTR) gene coding for the corresponding protein, consisting of 127 amino acids. The gene is located on chromosome 18q. More than 100 different mutations are known. Other mutant precursor proteins produced in the liver, such as apolipoprotein I and II, lysozyme and fibrinogen Aα, may be of etiological importance as well. Amyloidogenic mutations of the TTR gene lead to decreased stability of the corresponding protein and subsequently to extracellular deposition of amyloid in several tissues (peripheral and autonom…

Amyloid Neuropathies Familialmedicine.medical_specialtyAmyloidbiologyApolipoprotein Bbusiness.industrymedicine.medical_treatmentAmyloidosisGastroenterologyGeneral MedicineDiseaseLiver transplantationmedicine.diseaseGastroenterologyLiver TransplantationTransthyretinAmyloid NeuropathyInternal medicinePrevalencebiology.proteinmedicineHumansPrealbuminbusinessPolyneuropathyDigestive Diseases
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251 SENSORY PROFILES IN PAINFUL VS. NON-PAINFUL CHEMOTHERAPY-INDUCED POLYNEUROPATHY

2007

Anesthesiology and Pain MedicineChemotherapy inducedbusiness.industryAnesthesiaMedicineSensory systembusinessmedicine.diseasePolyneuropathyEuropean Journal of Pain
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Ozone Therapy as treatment for severe cases without good evolution in ophthalmology [abstract]

2019

PURPOSE: In ophthalmology many diseases lead to irreversible blindness, something that implies millions of dollars in care because people with these difficulties loose their autonomy, becoming dependent in all their everyday activities.
 This presentation will focus on clinical cases of the following pathologies treated with Ozone Therapy because all these were already being treated in a conventional way without signs of improvement. They are primary open-angle glaucoma, age-related macular degeneration, diabetic retinopathy, occlusion of the central retinal artery, facial paralysis and herpetic polyneuropathy associated with orbital apex syndrome.
 
 CASE PRESENTATION: As an…

Central retinal arterymedicine.medical_specialtyVisual acuitygenetic structuresbusiness.industryGlaucomaDiabetic retinopathyMacular degenerationmedicine.diseaseOzone therapyeye diseasesFacial paralysismedicine.arteryOphthalmologyMedicinemedicine.symptombusinessPolyneuropathyJournal of Ozone Therapy
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INDUCTION OF VEGF AND B-FGF RELEASE BY ELECTRO NEUROSTIMULATION (FREMS) IN DIABETIC POLYNEUROPATHY

2008

DIABETIC POLYNEUROPATHY aging frems
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